ライフサイエンスコーパス: assays were carried out

1 best MIP was morphologically characterized and equilibrium assays were carried out.

2 g the forward and the gamma-glutamyl transferase (gamma-GT) assays were carried out.

3 appropriate protein-binding, enzymatic, and nuclear import assays were carried out.

4 450R transcription initiation, nuclear run-on transcription assays were carried out.

5 ngs were harvested after 7 weeks and TDC and STR1 enzymatic assays were carried out.

6 Then, migration assays were carried out and an evaluation of the potential ri

7 closure in cax1, cax3, and cax1/cax3 when stomatal movement assays were carried out at a lower extracellular pH.

8 S.c. xenograft assays were carried out in a severe combined immunodeficient

9 Proliferation, tube formation, and migration assays were carried out in RVECs isolated from wild-type and

10 Parallel assays were carried out in the absence of virus in order to q

11 r cell adhesion molecule was affected when cell aggregation assays were carried out in the presence of >/=38 mM ethanol.

12 to develop a multiplex assay, in which the IL-1R and IL-5R assays were carried out in the same well with each receptor i

13 color through reactions of copigmentation or condensation, assays were carried out in wine model systems with different

14 Furthermore, a series of ligand competition assays were carried out on a single lectin super-microarray t

15 e disequilibrium with these were identified, and functional assays were carried out on all 23 SNPs.

16 Radioligand binding assays were carried out on all synthesized peptides, includin

17 Both the PTH assay and IL-5 assays were carried out on microliter volumes and demonstrate

18 ce, circular dichroism, and electrophoretic gel retardation assays were carried out on recombinant 80-aa residue proteins

19 The behavioral assays were carried out on tethered cells lacking CheA and Ch

20 erially expressed and refolded, and then RIA and GH-binding assays were carried out on the purified recombinant proteins.

21 fected by subtype C HIV-1 were constructed, and competition assays were carried out to compare the fitness between the tr

22 Electrophoretic mobility shift assays were carried out to determine the binding affinity to

23 nal killing between the two E. coli strains, transformation assays were carried out to determine the relative sensitiviti

24 In-depth functional assays were carried out to document the impact of lncRNA on t

25 Fluorescence polarization assays were carried out to evaluate the ability of terphenyl

26 ed mutagenesis, enzyme kinetics as well as UDP-bead binding assays were carried out to investigate the role of each DXD m

27 Single-molecule microscopy and stopped-flow kinetics assays were carried out to understand the microtubule polymer

28 these nanoassemblies is easily controlled, and proteolytic assays were carried out under both excess enzyme and excess s

29 For such a broad range of activities, reaction assays were carried out under many different conditions, prev

30 Trypsin inhibition assays were carried out using a modified rat trypsin, C191A/C

31 n vitro enzyme-linked immunosorbent assays and bead-binding assays were carried out using BSA conjugated with high and lo

32 To probe for interactions with GHR, in vitro binding assays were carried out using glutathione S-transferase-GHR f

33 TR4 activity and phosphorylation assays were carried out using hepatocytes and various TR4 wil

34 Analytical performance and specificity assays were carried out using human serum and different prote

35 targets of MisR regulation, electrophoretic mobility shift assays were carried out using purified MisR-His(6) protein.

36 5 antifungal mechanism in vivo, both binding and fungicidal assays were carried out using S. cerevisiae isogenic SSA1/SSA

37 Chloramphenicol acetyltransferase assays were carried out with constructs containing the kpsF 5

38 Binding assays were carried out with HEK-293 cells expressing either

39 Binding assays were carried out with HEK-293 cells expressing either

40 Binding assays were carried out with HEK-293 cells expressing either

41 Binding assays were carried out with HEK-293 cells expressing either

42 ed increase in fitness was more pronounced when competition assays were carried out with IFN-alpha-treated cells, suggest

43 Competitive microtubule binding assays were carried out with monomeric motor domains of the m

44 function encoded by the intact 3.2-kb par region, stability assays were carried out with ParD provided in trans by a comp

45 ro EC branch point formation, EC invasion, and EC migration assays were carried out with preop, POD 7 to 13 and 14 to 20

46 tified as VPg, therefore yeast two-hybrid and GST pull-down assays were carried out with proteins encoded by the pvr1 all

47 Similar assays were carried out with whole mouse eyes in situ.