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1 iple sclerosis, on MMP-9 expression in human astroglioma and fibrosarcoma cell lines and primary astr
2  II MHC and CIITA mRNA accumulation in human astroglioma and fibrosarcoma cell lines, but TGF-beta do
3 e substantial productive HIV infection of an astroglioma cell line (U87MG) and human fetal astrocytes
4 strocytes, primary human astrocytes, a human astroglioma cell line CRT, and primary rat microglia.
5 ssive, on MMP-2 gene expression in two human astroglioma cell lines (U251-MG and CRT).
6                                   Stable U87 astroglioma cell lines expressing CD4 and individual chi
7  Here, we report that OSM treatment of human astroglioma cell lines increases VEGF levels by approxim
8 hown that primary human astrocytes and human astroglioma cell lines produce the CXC chemokines IFN-ga
9   Similar results were observed in two human astroglioma cell lines, CRT and STT; TGF-beta inhibited
10 nstitutive activity of this gene in invasive astroglioma cell lines.
11 is inhibitory effect of IL-1beta using human astroglioma cell lines.
12 its HIV-1 infection mediated by CCR5 in U-87 astroglioma cells but has no effect on infection of CXCR
13 Stat1 alpha mRNA were introduced in CH235-MG astroglioma cells by transient transfection; such treatm
14 olic pulse-chase labeling studies of U373-MG astroglioma cells indicated that turnover of the carbohy
15   This novel mechanism of VEGF regulation in astroglioma cells may be active in pathophysiological st
16 st that CXCR4-mediated signaling pathways in astroglioma cells may be another mechanism for these cel
17 sults in decreased invasiveness of the human astroglioma cells through an extracellular matrix.
18           MMP-2 is highly expressed in human astroglioma cells, and contributes to the invasiveness o
19  activation of the type IV CIITA promoter in astroglioma cells, indicating that the inhibitory influe
20 newborn and adult infected rats and cultured astroglioma cells, shunting tryptophan degradation towar
21 MO, but not IDO, in vitro in BDV-infected C6 astroglioma cells.
22 ne receptor expression and function on human astroglioma cells.
23 haB isolated from rat heart tissue and human astroglioma cells.
24 ssion by siRNA inhibited proliferation of C6 astroglioma cells.
25  in HCMV- or adenovirus vector-infected U373 astroglioma cells.
26 othelial cells are cultured alone or with C6 astrogliomas grown on a separate culture surface.
27 o constitute an important component of human astroglioma invasion.
28 a may have beneficial effects in attenuating astroglioma invasive properties.
29      We demonstrate that HTLV-1 infection in astrogliomas results in a robust induction of interleuki
30 tional relevance of CXCR4 expression, stable astroglioma transfectants expressing high levels of CXCR
31 have distinct quantities of CD4 and in human astroglioma U87MG-CD4 cells that express a large quantit

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