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1 iple sclerosis, on MMP-9 expression in human astroglioma and fibrosarcoma cell lines and primary astr
2 II MHC and CIITA mRNA accumulation in human astroglioma and fibrosarcoma cell lines, but TGF-beta do
3 e substantial productive HIV infection of an astroglioma cell line (U87MG) and human fetal astrocytes
4 strocytes, primary human astrocytes, a human astroglioma cell line CRT, and primary rat microglia.
7 Here, we report that OSM treatment of human astroglioma cell lines increases VEGF levels by approxim
8 hown that primary human astrocytes and human astroglioma cell lines produce the CXC chemokines IFN-ga
9 Similar results were observed in two human astroglioma cell lines, CRT and STT; TGF-beta inhibited
12 its HIV-1 infection mediated by CCR5 in U-87 astroglioma cells but has no effect on infection of CXCR
13 Stat1 alpha mRNA were introduced in CH235-MG astroglioma cells by transient transfection; such treatm
14 olic pulse-chase labeling studies of U373-MG astroglioma cells indicated that turnover of the carbohy
15 This novel mechanism of VEGF regulation in astroglioma cells may be active in pathophysiological st
16 st that CXCR4-mediated signaling pathways in astroglioma cells may be another mechanism for these cel
19 activation of the type IV CIITA promoter in astroglioma cells, indicating that the inhibitory influe
20 newborn and adult infected rats and cultured astroglioma cells, shunting tryptophan degradation towar
30 tional relevance of CXCR4 expression, stable astroglioma transfectants expressing high levels of CXCR
31 have distinct quantities of CD4 and in human astroglioma U87MG-CD4 cells that express a large quantit
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