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1 s of high-sensitivity C-reactive protein and asymmetric dimethylarginine).
6 uanidino-methylated arginines (MA) including asymmetric dimethylarginine (ADMA) and N(G)-methyl-l-arg
9 anidinomethylated arginines (MAs), including asymmetric dimethylarginine (ADMA) and NG-methyl-L-argin
10 r (15)N-arginine intravenous infusion and on asymmetric dimethylarginine (ADMA) and symmetric dimethy
11 n mammalian cells: monomethylarginine (MMA), asymmetric dimethylarginine (ADMA) and symmetric dimethy
13 oth fibroblast growth factor 23 (FGF-23) and asymmetric dimethylarginine (ADMA) are associated with p
17 ncentrations of the endogenous NOS inhibitor asymmetric dimethylarginine (ADMA) in the hypoxia-induce
26 ne, and the endogenous NO synthase inhibitor asymmetric dimethylarginine (ADMA) were measured with li
27 ork implicating the endogenous NOS inhibitor asymmetric dimethylarginine (ADMA), a cardiotoxic hormon
32 rogens regulate the metabolism or release of asymmetric dimethylarginine (ADMA), an endogenous inhibi
33 ffects in part by elevating plasma levels of asymmetric dimethylarginine (ADMA), an endogenous inhibi
34 dy prospectively determined plasma levels of asymmetric dimethylarginine (ADMA), an endogenous nitric
35 l contributor to endothelial pathobiology is asymmetric dimethylarginine (ADMA), an endogenous nitric
38 l contributor to endothelial pathobiology is asymmetric dimethylarginine (ADMA), an endogenous NO syn
40 aw the next morning for soluble CD40 ligand, asymmetric dimethylarginine (ADMA), and nitrotyrosine le
41 1) strictly generates monomethylarginine and asymmetric dimethylarginine (ADMA), but not symmetric di
42 risk factors, total homocysteine (tHcy) and asymmetric dimethylarginine (ADMA), correlate with decre
43 Plasma was assayed for endothelin 1 (ET-1), asymmetric dimethylarginine (ADMA), intercellular adhesi
44 dogenous inhibitor of nitric oxide synthase, asymmetric dimethylarginine (ADMA), is elevated in patie
45 a levels of arginine, citrulline, ornithine, asymmetric dimethylarginine (ADMA), symmetric dimethylar
46 function and raised plasma concentrations of asymmetric dimethylarginine (ADMA), the endogenous inhib
56 e endogenous nitric oxide synthase inhibitor asymmetric dimethylarginine [ADMA, via inhibition of dim
57 bin [TAT] complex), endothelial dysfunction (asymmetric dimethylarginine [ADMA]), and platelet-derive
59 vels of apolipoprotein AI and high levels of asymmetric dimethylarginine, an endogenous inhibitor of
60 lammation (C-reactive protein), or levels of asymmetric dimethylarginine, an endogenous inhibitor of
62 that 3.7% of the molecules naturally contain asymmetric dimethylarginine and/or monomethylarginine.
63 Decompartmentalized hemoglobin, arginase 1, asymmetric dimethylarginine, and adenine nucleotides are
64 1(-/-) mice showed a significant increase of asymmetric dimethylarginine concentration in plasma (1.4
72 eduction in the level of p-cresol sulfate or asymmetric dimethylarginine to significant reductions in
74 uremic milieu contains increased amounts of asymmetric dimethylarginine, we speculate that such accu
75 stable nitric oxide oxidation products, and asymmetric dimethylarginine were abnormal in patients wi
76 plasma levels of l-arginine, arginase-1, and asymmetric dimethylarginine were measured at serial time
78 il, betaine, symmetric dimethylarginine, and asymmetric-dimethylarginine), were also increased in uri
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