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1 s of high-sensitivity C-reactive protein and asymmetric dimethylarginine).
2                                              Asymmetric dimethylarginine (10 microM) or native low-de
3                                              Asymmetric dimethylarginine (ADMA) and homocysteine are
4                                              Asymmetric dimethylarginine (ADMA) and monomethyl argini
5              The endogenous methylarginines, asymmetric dimethylarginine (ADMA) and N (G)-monomethyl-
6 uanidino-methylated arginines (MA) including asymmetric dimethylarginine (ADMA) and N(G)-methyl-l-arg
7               The endogenous methylarginines asymmetric dimethylarginine (ADMA) and N(G)-monomethyl-L
8                           The NOS inhibitors asymmetric dimethylarginine (ADMA) and N-monomethylargin
9 anidinomethylated arginines (MAs), including asymmetric dimethylarginine (ADMA) and NG-methyl-L-argin
10 r (15)N-arginine intravenous infusion and on asymmetric dimethylarginine (ADMA) and symmetric dimethy
11 n mammalian cells: monomethylarginine (MMA), asymmetric dimethylarginine (ADMA) and symmetric dimethy
12                          Increased levels of asymmetric dimethylarginine (ADMA) are associated with e
13 oth fibroblast growth factor 23 (FGF-23) and asymmetric dimethylarginine (ADMA) are associated with p
14         The endogenous NO synthase inhibitor asymmetric dimethylarginine (ADMA) circulates in plasma,
15                           Elevated levels of asymmetric dimethylarginine (ADMA) correlate with risk f
16     We sought to determine if a reduction in asymmetric dimethylarginine (ADMA) enhances endothelial
17 ncentrations of the endogenous NOS inhibitor asymmetric dimethylarginine (ADMA) in the hypoxia-induce
18           We have recently demonstrated that asymmetric dimethylarginine (ADMA) induces the transloca
19               We sought to determine whether asymmetric dimethylarginine (ADMA) inhibits nitric oxide
20                                              Asymmetric dimethylarginine (ADMA) is an endogenous comp
21                                              Asymmetric dimethylarginine (ADMA) is an endogenous inhi
22                                              Asymmetric dimethylarginine (ADMA) is an endogenous inhi
23                                              Asymmetric dimethylarginine (ADMA) is an endogenous meta
24 decreased, and the inhibitor of NO synthesis asymmetric dimethylarginine (ADMA) is increased.
25       The impact of statin therapy on plasma asymmetric dimethylarginine (ADMA) levels has not been c
26 ne, and the endogenous NO synthase inhibitor asymmetric dimethylarginine (ADMA) were measured with li
27 ork implicating the endogenous NOS inhibitor asymmetric dimethylarginine (ADMA), a cardiotoxic hormon
28           DDAH1 is a key catabolic enzyme of asymmetric dimethylarginine (ADMA), a major endogenous n
29                                         Both asymmetric dimethylarginine (ADMA), an endogenous inhibi
30                                              Asymmetric dimethylarginine (ADMA), an endogenous inhibi
31             Elevated blood concentrations of asymmetric dimethylarginine (ADMA), an endogenous inhibi
32 rogens regulate the metabolism or release of asymmetric dimethylarginine (ADMA), an endogenous inhibi
33 ffects in part by elevating plasma levels of asymmetric dimethylarginine (ADMA), an endogenous inhibi
34 dy prospectively determined plasma levels of asymmetric dimethylarginine (ADMA), an endogenous nitric
35 l contributor to endothelial pathobiology is asymmetric dimethylarginine (ADMA), an endogenous nitric
36                                              Asymmetric dimethylarginine (ADMA), an endogenous nitric
37                          Increased levels of asymmetric dimethylarginine (ADMA), an endogenous nitric
38 l contributor to endothelial pathobiology is asymmetric dimethylarginine (ADMA), an endogenous NO syn
39 AT) and assessed associations with arginine, asymmetric dimethylarginine (ADMA), and hemolysis.
40 aw the next morning for soluble CD40 ligand, asymmetric dimethylarginine (ADMA), and nitrotyrosine le
41 1) strictly generates monomethylarginine and asymmetric dimethylarginine (ADMA), but not symmetric di
42  risk factors, total homocysteine (tHcy) and asymmetric dimethylarginine (ADMA), correlate with decre
43  Plasma was assayed for endothelin 1 (ET-1), asymmetric dimethylarginine (ADMA), intercellular adhesi
44 dogenous inhibitor of nitric oxide synthase, asymmetric dimethylarginine (ADMA), is elevated in patie
45 a levels of arginine, citrulline, ornithine, asymmetric dimethylarginine (ADMA), symmetric dimethylar
46 function and raised plasma concentrations of asymmetric dimethylarginine (ADMA), the endogenous inhib
47                                              Asymmetric dimethylarginine (ADMA), which inhibits NO sy
48 e endogenous nitric-oxide synthase inhibitor asymmetric dimethylarginine (ADMA).
49  systemic levels of l-arginine and increased asymmetric dimethylarginine (ADMA).
50 nverts TbPRMT7 into a type I PRMT, producing asymmetric dimethylarginine (ADMA).
51 genous nitric oxide synthase (NOS) inhibitor asymmetric dimethylarginine (ADMA).
52 onist of NO synthase has been characterized: asymmetric dimethylarginine (ADMA).
53                                We focused on asymmetric dimethylarginine (ADMA, the endogenous inhibi
54             CS patients had higher levels of asymmetric dimethylarginine (ADMA; P<0.0001), symmetric
55                                              Asymmetric-dimethylarginine (ADMA) limits NO production
56 e endogenous nitric oxide synthase inhibitor asymmetric dimethylarginine [ADMA, via inhibition of dim
57 bin [TAT] complex), endothelial dysfunction (asymmetric dimethylarginine [ADMA]), and platelet-derive
58                                              Asymmetric dimethylarginine also stimulated MCP-1 format
59 vels of apolipoprotein AI and high levels of asymmetric dimethylarginine, an endogenous inhibitor of
60 lammation (C-reactive protein), or levels of asymmetric dimethylarginine, an endogenous inhibitor of
61 would be expected to lead to accumulation of asymmetric dimethylarginine and inhibition of NOS.
62 that 3.7% of the molecules naturally contain asymmetric dimethylarginine and/or monomethylarginine.
63  Decompartmentalized hemoglobin, arginase 1, asymmetric dimethylarginine, and adenine nucleotides are
64 1(-/-) mice showed a significant increase of asymmetric dimethylarginine concentration in plasma (1.4
65                                              Asymmetric dimethylarginine increases endothelial oxidat
66                                              Asymmetric dimethylarginine-induced monocyte binding was
67                                              Asymmetric dimethylarginine is an endogenous inhibitor o
68                                              Asymmetric dimethylarginine is eliminated largely by the
69  vascular cell adhesion molecule (VCAM), and asymmetric dimethylarginine levels were measured.
70                                              Asymmetric dimethylarginine may be an endogenous proathe
71                                              Asymmetric dimethylarginine plays a role in endothelial
72 eduction in the level of p-cresol sulfate or asymmetric dimethylarginine to significant reductions in
73 ype II activity) as well as small amounts of asymmetric dimethylarginine (type I activity).
74  uremic milieu contains increased amounts of asymmetric dimethylarginine, we speculate that such accu
75  stable nitric oxide oxidation products, and asymmetric dimethylarginine were abnormal in patients wi
76 plasma levels of l-arginine, arginase-1, and asymmetric dimethylarginine were measured at serial time
77 vely small amounts of monomethylarginine and asymmetric dimethylarginine were produced.
78 il, betaine, symmetric dimethylarginine, and asymmetric-dimethylarginine), were also increased in uri

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