ライフサイエンスコーパス: at age

1 The number of infant deaths at age 0-6 months was similar in each group (50 in the p

2 ns and the risk for concurrent sensitization at age 0.5, 2, and 3 years, and mixed-effects regression

3 03 (95% CI: 1.02, 1.05) for cancer diagnosed at ages 0-14 years.

4 03 (95% CI: 1.02, 1.05) for cancer diagnosed at ages 0-19 years and 1.03 (95% CI: 1.02, 1.05) for can

5 patients with lymphoid neoplasms, diagnosed at ages 0-79 years during the period 1987-2011, and 160,

6 evalence of challenge-confirmed food allergy at age 1 and 4 years was 11.0% and 3.8%, respectively.

7 ed prospectively, and lung function measured at age 1 month and 7 years.

8 g exotropia was induced in two male macaques at age 1 month by sectioning the tendons of the medial r

9 from the time of unilateral cataract surgery at age 1 to 7 months to age 5 years, and to compare AL g

10 compared with 40% of children not sensitized at age 1 year but sensitized by age 5 years, and 17% of

11 eport the updated prevalence of food allergy at age 1 year from the whole cohort, and to report the p

12 ho previously attended the HealthNuts clinic at age 1 year or reported symptoms of a new food allergy

13 to death (or end of eligibility for outcome, at age 1 year) and pregnancy (gestation and each trimest

14 Follow-up started at age 1 year, and the children were followed up for as

15 immune profiles associated with egg allergy at age 1 year, determine the phenotypic changes that occ

16 h and cat and dog allergen levels in bedding at age 1 year.

17 es at birth were poorly predictive for those at ages 1 and 3 years.

18 World Health Organization normative z scores at ages 1 to 6 and 8, 10, and 12 months (defined a prior

19 technology, respectively, in sera collected at ages 1, 2, 3, 5, 6, 7, 10, 13, and 20 years.

20 food allergens was determined longitudinally at ages (1/2), 1(1/2), 4 and 6 years by specific IgE ass

21 and cooked egg was introduced to both groups at age 10 months.

22 t age 4 y predicted more restrictive feeding at age 10 y (B = 0.15; 95% CI: 0.11, 0.18).

23 restrictive feeding at age 4 y to child zBMI at age 10 y after adjustment for baseline zBMI.The conti

24 ntinued use of restrictive feeding practices at age 10 y appeared to be primarily a response of mothe

25 At age 10 years, inhalant allergic sensitization and foo

26 sensitization or physician-diagnosed allergy at age 10 years.

27 me was associated with low cognitive ability at age 11 (FWERacross P=0.0043).

28 dently associated with having a thinner RNFL at age 11 or 12 years.

29 ears, of whom 565 (56%) had been lead tested at age 11 years (54% male; 93% white).

30 Mean (SD) blood lead level at age 11 years was 10.99 (4.63) microg/dL.

31 re ascertained as blood lead levels measured at age 11 years.

32 The protocol was repeated at age 11-12 and 19 years, and 1516 participated in all

33 the Children's Eating Attitudes Test (ChEAT) at age 11.5 y and in whom we measured adiposity and bloo

34 associations of problematic eating attitudes at age 11.5 y with new-onset obesity (OR: 2.18; 95% CI:

35 lectrical impedance measurement of body fat) at age 11.5 years using the same data set in a cohort of

36 with minimal differences versus UK controls at ages 11 years (p=0.0449) and 15 years (p=0.17), and t

37 an increased risk of infantile atopic eczema at age 12 months, but no significant association between

38 ized raw egg challenge and egg sensitization at age 12 months.

39 me was an EW SPT response of 3 mm or greater at age 12 months.

40 ss of 89.1% (85.1-92.3) for those vaccinated at age 12-13 years.

41 sed at age 9-11 years, while IQ was assessed at age 12.

42 milk with allergic disease and lung function at ages 12 and 18 years.

43 frequency questionnaires that assessed diet at ages 12-13 years and 10 years previously.

44 vels of binge eating and overeating in males at age 13 y likely cause higher BMI at age 17 y.

45 ffect of the same disordered eating patterns at age 13 y on BMI at age 17 y via a split-sample approa

46 BMI at age 7 y on disordered eating patterns at age 13 y with the use of data from the Avon Longitudi

47 es and females and food restriction in males at age 13 y.

48 hood and increased risk of disordered eating at age 13 y.

49 risk birth cohort, the persistence of asthma at age 13 years was most strongly associated with outpat

50 allergen sensitization, and asthma diagnosis at age 13 years were evaluated.

51 At age 13 years, a BMI z score of 1 was associated with

52 children sensitized by age 1 year had asthma at age 13 years, compared with 40% of children not sensi

53 Compared with women with menarche at age 13 years, women who had their first menstruation

54 95% CI, 0.4-2.3), was associated with asthma at age 13 years.

55 born into the cohort, 772 attended follow-up at age 13-16 years between July 22, 2011, and Nov 11, 20

56 We used follow-up data at age 13-16 years from the Manchester Asthma and Allerg

57 as a result of cancer 6 to 9 years earlier, at ages 13 to 16 years.

58 ss and steeper discounting of future rewards at age 14 also predicts problematic drug use at age 16,

59 ign to track 144 novelty-seeking adolescents at age 14 and 16 to determine whether neural activity in

60 l cortex) regions during reward anticipation at age 14 predicts problematic drug use at age 16.

61 An SD increase in UVB exposure at age 14 to 19 years (OR, 0.81; 95% CI, 0.71-0.92) and

62 e of IPV at age 14 weeks or two doses of IPV at age 14 weeks and 18 weeks.

63 eks, and 14 weeks and either one dose of IPV at age 14 weeks or two doses of IPV at age 14 weeks and

64 how that contact with mental health services at age 14 years by adolescents with a mental disorder re

65 ay, and faces tasks-were collected in youths at age 14, as part of the IMAGEN study.

66 members with complete data on visual acuity at age 15 or 16 years, measured in 1961, 1974, and 1986,

67 gains in the population-wide life expectancy at age 15 years since the introduction of ART, and the s

68 charged from inpatient psychiatric treatment at ages 15 to 44 years.

69 Human Sertoli cells obtained from men at ages 15, 23, 36 and 40 were cultured in vitro.

70 offending, respectively, as adverse outcomes at ages 15-35 years.

71 young adult cancer survivors (AYA [diagnosed at ages 15-39 years]) with those of women without a canc

72 lood pressure (OR: 1.25; 95% CI: 0.99, 1.58) at age 16 y.

73 gnitive performance indexed by school grades at age 16 years and IQ test scores at military conscript

74 The mean school grades at age 16 years and IQ test scores at military conscript

75 ma, rhinitis, and aeroallergen sensitization at age 16 years and with incidence of asthma between 8 a

76 educational achievement over time, up to 9% at age 16, accounting for 15% of the heritable variance.

77 at age 14 also predicts problematic drug use at age 16, but the neural responses independently predic

78 At age 16, only 53% of patients were in high school.

79 PS septiles differed by a whole school grade at age 16.

80 tion at age 14 predicts problematic drug use at age 16.

81 isordered eating patterns at age 13 y on BMI at age 17 y via a split-sample approach in the ALSPAC.

82 in males at age 13 y likely cause higher BMI at age 17 y.

83 NAFLD was diagnosed in 15.2% of adolescents at age 17 years.

84 Peak mortality hazard at ages 17 to 24 years was confirmed in the subgroup of

85 f 32.0 degrees C reduces death or disability at age 18 months in infants with hypoxic-ischemic enceph

86 nd a clinical examination (47% participated) at age 18 to 19 years.

87 utcomes on discharge to home, at 1 year, and at age 18 to 24 months' PMA and neurodevelopmental asses

88 d with a faster decline in SI and a lower SI at age 18 years, independent of adiposity.

89 ine in disposition index (DI) and a lower DI at age 18 years.

90 ow-up were also associated with a higher BMI at age 18 years.

91 and IQ test scores at military conscription at age 18 years.

92 hormone replacement therapy use, somatotype at age 18, benign breast disease, mammographic density,

93 dy mass index (BMI; weight (kg)/height (m)2) at age 18-21 years, BMI at baseline, and change in BMI d

94 Multisensitization at age 19 years was significantly associated with early

95 ional adjustment for ointment use for eczema at age 2 months, and cross-lagged modeling showed no con

96 ng MRI in neonates and behavioral inhibition at age 2 using the Infant-Toddler Social and Emotional A

97 on was assessed by dual X-ray absorptiometry at age 2 weeks.

98 rculating IGF-I, and total and abdominal fat at age 2 weeks.

99 Human cognitive function was assessed at age 2 y with the Bayley Scales of Infant Development

100 network is related to behavioral inhibition at age 2 years beyond sociodemographic and familial fact

101 as double-blinded; some unmasking took place at age 2 years for an interim analysis, but participants

102 = 67; 6.5%]), the late phenotype with onset at age 2 years or older (n = 50; 4.8%), and the never/in

103 irth cohort study had a clinical examination at age 2 years to assess eczema and allergen-specific Ig

104 s/muL or more and aeroallergen sensitization at age 2 years were each associated with increased risk

105 e cognitive, language, and motor development at age 2 years, with adjusted composite score mean diffe

106 n MLPT infants and term-born control infants at age 2 years.

107 related these maps to behavioral inhibition at age 2, covarying for sex, social risk, and motion dur

108 the Environment (CHARGE) case-control study at age 2-5 y, were clinically confirmed to have ASD (n=2

109 rks is associated with behavioral inhibition at age 2.

110 oencephalopathy in seven patients presenting at ages 2 to 4 months with progressive microcephaly, spa

111 k characteristics (n = 910) and BMI z scores at ages 2, 3, and 4 y were examined with the use of mult

112 Blood samples were collected at ages 2, 4, 6, and 11 years, and serum-specific IgE le

113 sociated with a higher offspring BMI z score at ages 2-4 y.

114 as 0.7%-11.5% at ages <2 years and 0.8%-3.3% at ages 2-4 years.

115 rglycemia, oxidative stress, and nephropathy at age 20 weeks compared with their db/m littermates.

116 and bone mineral density (BMD) were measured at age 20 y through the use of dual-energy X-ray absorpt

117 At age 20 years, their IgE recognized most frequently De

118 At age 20, the patient developed spots of healthy-lookin

119 who attended for routine cervical screening at age 20-21 years.

120 At age 22 years, 262 members of a Faroese birth cohort,

121 tal condition that can be reliably diagnosed at age 24 months.

122 Calculus, pocket, or bleeding presence at age 24 years separately presented fair accuracy.

123 Oral examinations were performed at ages 24 and 31 years in the Pelotas 1982 birth cohort

124 ce age-standardised rates of cervical cancer at ages 25-64 years by 19%, from 15.1 in 2016 to 12.2 pe

125 d trimesters and urine samples from children at age 3 or 4 years.

126 hnically diverse study population of infants at age 3 to 6 months, who were enrolled in Vitamin D Ant

127 n longitudinal analyses, H. pylori infection at age 3 was inversely associated with incidence of atop

128 oth child asthma and vitamin D concentration at age 3 y did not modify the association between matern

129 associated with an increased risk of asthma at age 3 years after adjusting for common confounders (r

130 antigens based on serum specific IgE levels at age 3 years in high-risk children.

131 At age 3 years, children were assessed for asthma, aller

132 associated with enhanced cytokine responses at age 3 years, including IFN-alpha and IL-10 responses

133 al blood mononuclear cell cytokine responses at age 3 years.

134 heeze or food or environmental sensitization at age 3 years.

135 sensitization and recurrent wheeze assessed at age 3 years.

136 quent development of these allergic diseases at age 3 years.

137 timulated peripheral blood mononuclear cells at age 3 years.

138 all, 166 (36%) children had recurrent wheeze at age 3 years.

139 At age 3, sensitization to foods (milk, egg, peanut, soy

140 Intestinal microbiome samples were collected at age 3-6 months in children participating in the follo

141 ed 1871 participants who reported family SES at ages 3 to 18 years and were evaluated for LV structur

142 in the United States with diabetes diagnosed at age 30 years or older.

143 pients with CF should initiate CRC screening at age 30 years within 2 years of the transplantation be

144 th a 0.1% reduction in adult annual earnings at age 30.

145 Periodontitis at age 31 years according to six classifications was use

146 The outcome measures at age 35 years were social disadvantage (divorced or se

147 s, with the exception of weekly cannabis use at age 35 years, which remained independently associated

148 program implementation, 2-dose MCV coverage at age 36 months exceeded that obtained at age 60 months

149 cognitive function and socioeconomic status at age 38 years and with declines in IQ and with downwar

150 onomic status (primary outcome) was assessed at age 38 years using the New Zealand Socioeconomic Inde

151 ing Speed (secondary outcomes) were assessed at age 38 years using the Wechsler Adult Intelligence Sc

152 Among blood-tested participants included at age 38 years, mean WAIS-IV score was 101.16 (14.82) a

153 1037 original participants, 1007 were alive at age 38 years, of whom 565 (56%) had been lead tested

154 tests of cognitive function in the offspring at age 4 and 6-12 y.

155 white (EW) of less than 2 mm were randomized at age 4 months to receive whole-egg powder or placebo (

156 y and safety of early hen's egg introduction at age 4 to 6 months to prevent hen's egg allergy in the

157 d sex- and age-adjusted BMI SD scores (zBMI) at age 4 y predicted more restrictive feeding at age 10

158 emporal association from restrictive feeding at age 4 y to child zBMI at age 10 y after adjustment fo

159 associated with IgE sensitization to peanut at age 4 years (adjusted odds ratio, 1.88; 95% CI, 1.03-

160 To compare structural outcome at age 4 years of eyes treated with intravitreal injecti

161 Late-onset peanut allergy at age 4 years was rare (0.2%).

162 At age 4 years, parents completed a questionnaire (81.3%

163 lergy, asthma, eczema, and allergic rhinitis at age 4 years.

164 ne-homocysteine methyltransferase)-null mice at age 4, 12, 24, and 52 wk (N = 8) and observed elevati

165 At age 4, we calculated a CM-risk score (n=386) as the s

166 core indicates a higher cardiometabolic risk at age 4.

167 At ages 4 and 10 y, restrictive feeding was assessed wit

168 ncy and child neuropsychological development at ages 4-5 y.The multicenter prospective mother-child c

169 Plan between July 1, 1991 and June 30, 2008 at age 40 years or older, and matched five controls by y

170 ed screening method, initiation of screening at age 40 years, 5-year re-screening and 3-year surveill

171 Colonoscopy every 5 years, starting at age 40 years, was the optimal colonoscopy strategy fo

172 red average-risk screening options beginning at age 40 years.

173 and 100 generally healthy patients recruited at ages 40 to 65 years.

174 associated with reduced risk of hypertension at ages 40-49 years (odds ratio = 0.92, 95% confidence i

175 In addition, risk of hypertension at ages 40-49 years decreased with increasing duration o

176 ween breastfeeding and incident hypertension at ages 40-65 years using data collected from 1995 to 20

177 s to 6% (range: 6-7%) among women vaccinated at age 45 years.

178 To report the prevalence of anisometropia at age 5 years after unilateral intraocular lens (IOL) i

179 IgE to Der p 1 or Der p 23 at age 5 years or less predicted asthma at school age.

180 If the goal is emmetropia at age 5 years, then the immediate postoperative hyperme

181 5 years, and 17% of children not sensitized at age 5 years.

182 udophakic eyes had significant anisometropia at age 5 years.

183 Anisometropia was calculated at age 5 years.

184 omen had no effect on child neurodevelopment at age 5-6 years.

185 s (Strengths and Difficulties Questionnaire) at ages 5 years, 7 years, and 11 years and maternal psyc

186 ic crude and adjusted risk ratios for asthma at ages 5-9 years were calculated using Poisson regressi

187 The cumulative incidences at age 50 years among CCSS low-risk groups were < 5%, co

188 Screening should begin at age 50 years in average-risk persons, except in Afric

189 The cumulative burden of grade 1-5 CHCs at age 50 years was highest in survivors of CNS malignan

190 d DXA and quantitative CT screening starting at age 55 with quantitative CT screening every 5 years (

191 Exposure to mites at age 6 and 18 months was assessed by measuring Der p 1

192 Samples were collected at age 6 and 8 weeks of life.

193 if) ligand 8 (CXCL8) were measured in plasma at age 6 months (N = 214) and 7 years (N = 277) in child

194 Sensitization was evaluated at age 6 months, 18 months, 4 years, and 6 years by skin

195 Elevated LPS-stimulated-Treg percentage at age 6 was associated with increased risk of asthma (a

196 C)-Pediatric model, we simulated EID testing at age 6 weeks for HIV-exposed infants without and with

197 age 6 weeks, 10 weeks, and 14 weeks; or bOPV at age 6 weeks, 10 weeks, and 14 weeks and either one do

198 llocated villages (clusters) to either: tOPV at age 6 weeks, 10 weeks, and 14 weeks; or bOPV at age 6

199 At age 6 y, fat mass index (FMI) and fat-free mass index

200 D and residential location both at birth and at age 6 years (i.e., closer to average age at diagnosis

201 level of 150 ng/mL or more predicted asthma at age 6 years (odds ratio [OR], 2.3; 95% CI, 1.3-4.4).

202 ach associated with increased risk of asthma at age 6 years (OR, 3.1; 95% CI, 1.7-6.0 and OR, 3.3; 95

203 Using the residential address at age 6 years produced similar results; however, areas

204 ers do not predict doctors' diagnosed asthma at age 6 years.

205 50 at young adulthood) and UK controls (n=52 at age 6 years; n=39 at young adulthood) had similarly l

206 heir gender are "really, really smart." Also at age 6, girls begin to avoid activities said to be for

207 areas, or early vaccination prior to travel at age 6-11 months.

208 rm, wrist, scaphoid bone, clavicle, or ankle at age 6-13 y.

209 the Bayley Scales of Infant Development and at age 6.5 y with a hippocampus-dependent delayed-recall

210 fter further controlling for body mass index at age 6.5 y, problematic eating attitudes remained posi

211 tional analysis, current H. pylori infection at age 6.5 years was inversely, though not significantly

212 alence of atopy and "any allergic condition" at age 6.5 years.

213 Infants were followed up at ages 6 (n = 2956) and 12 (n = 2872) months and atopic

214 d less than 6 months in an institution (n=67 at ages 6 years; n=50 at young adulthood) and UK control

215 o more than 6 months in an institution (n=98 at ages 6 years; n=72 at young adulthood) had persistent

216 Serum 25(OH)D was assessed at ages 6, 14, 17, and 20 y, and whole-body bone mineral

217 ld increase in the risk of offspring obesity at ages 6-11 y (adjusted RR: 2.39; 95% CI: 1.97, 2.89) a

218 hom we measured adiposity and blood pressure at ages 6.5, 11.5, and 16 y.

219 rage at age 36 months exceeded that obtained at age 60 months in historical cohorts recommended to re

220 gham and Rotterdam cohorts was 34.69%-38.45% at age 60-64 y, 30.76%-40.26% at 65-69 y, and 33.3%-35.1

221 was associated with muscle related outcomes at age 60-64, (2) these associations were modified by 25

222 ges in uninsured rates among trauma patients at age 64 versus 65 years and whether there are associat

223 percentage-point increase in rehabilitation at age 64 versus 65 years, enabling an additional 1-in-1

224 ciated with mean-level cognitive performance at age 65 y, but not with rate of cognitive change.

225 r's disease (A+T+N-, A+T-N+, and A+T+N+; 86% at age 65 years and 51% at age 80 years) or with suspect

226 ophysiology (A-T+N-, A-T-N+, and A-T+N+; 92% at age 65 years and 78% at age 80 years).

227 e years lived in different dependency states at age 65 years in 1991 and 2011, and new projections of

228 Cumulative 20-year dementia risks at age 65 years were 30.13% (95% CI, 26.87%-32.93%) and

229 ssociated with changes in insurance coverage at age 65 years.

230 ing in the lowest quartile for lung function at age 7 may have long-term consequences for the develop

231 ater ADHD trajectories; and then followed up at age 7 to investigate the stability of associations ac

232 Current asthma at age 7 was defined by asthma medications dispensed at

233 .02, 0.15) and risk of overweight or obesity at age 7 y [adjusted RR (aRR) comparing the highest with

234 Z and a higher risk of overweight or obesity at age 7 y among children born after pregnancies complic

235 dy data.MR results indicated that higher BMI at age 7 y likely causes higher levels of binge eating a

236 e for BMI to assess the causal effect of BMI at age 7 y on disordered eating patterns at age 13 y wit

237 sk of treatment with inhaled corticosteroids at age 7 years (adjusted odds ratio, 4.01 [95% confidenc

238 and supplements during pregnancy and asthma at age 7 years when the diagnosis is more reliable than

239 ong children with below-average BMI z scores at age 7 years, a score increase of 0.5 from ages 7 to 1

240 ong children with above-average BMI z scores at age 7 years, a score increase of 0.5 from ages 7 to 1

241 At age 7 years, children had acquired 69.5% to 74.5% of

242 eanut introduction, and peanut sensitization at age 7 years.

243 (95% CI 0.21-0.29) increase in offspring BMI at age 7, with similar results at later ages and when FM

244 irect effects mediated through later poverty at age 7-14 years (beta = -0.01, 95% confidence interval

245 unction z score) on child cognitive function at age 7-14 years (i.e., joint mediators beta = -0.07, 9

246 ge <7 years) and poverty in later childhood (at age 7-14 years) with cognitive function at age 7-14 y

247 (at age 7-14 years) with cognitive function at age 7-14 years.

248 0.01) and school attendance/home environment at age 7-14 years.

249 nce of any positive SPT increased from 20.6% at age 7-8 years to 30.6% at 11-12 years, and 42.1% at 1

250 Animals were the primary sensitizers at age 7-8 years, 16.3%, followed by pollen, 12.4%.

251 At age 7-9 years, schizophrenia polygenic risk scores sh

252 tained an association with ADHD trajectories at age 7.

253 troke cases) with measured weight and height at ages 7 to 13 years.

254 n 3 children (2 male and 1 female; diagnosed at ages 7, 20, and 28 months).

255 g)/height (m)2), and body surface area (BSA) at ages 7-13 years and birth weight are associated with

256 Height at ages 7-13 years was significantly associated with MM,

257 who were diagnosed with a primary malignancy at age 70 years or older and were treated with chemother

258 At age 75 years or older, major upper gastrointestinal b

259 n of being LGA with the prevalence of asthma at age 8 in atopic and non-atopic children and the role

260 led cluster-randomized trials were revisited at age 8 y between February 2013 and June 2014.

261 size, body composition, and metabolic health at age 8 y in preterm-born children who were randomly as

262 Nutritional habits at age 8 y were assessed by using a 3-d nutritional diar

263 re assessed by using a 3-d nutritional diary.At age 8 y, no differences were found in body size, body

264 controlled trial were enrolled for follow-up at age 8 y.

265 We assessed ADHD-related behaviors at age 8 years using Conners' Teacher Rating Scale-Revis

266 proportion of total VLC n-3 PUFAs in plasma at age 8 years was associated with a reduced risk of pre

267 Dietary TAC at age 8 years was estimated by combining information on

268 and arachidonic acid [AA]) in blood samples at age 8 years were measured for 940 children from the p

269 At age 8 years, disparities in the prevalence of ideal B

270 om 2003 through 2005, 516 boys were enrolled at age 8-9 years and followed for up to 10 years.

271 MRI data were collected at ages 8 and 10.

272 -N+, and A+T+N+; 86% at age 65 years and 51% at age 80 years) or with suspected non-Alzheimer's patho

273 -N+, and A-T+N+; 92% at age 65 years and 78% at age 80 years).

274 At age 9 months, birds looked more at a tool moving a pi

275 months (intervention group) or 1 dose of MV at age 9 months, in accordance with current practice (co

276 ied from 55% (53-56%) among women vaccinated at age 9 years to 6% (range: 6-7%) among women vaccinate

277 consumption and sleep quality were assessed at age 9-11 years, while IQ was assessed at age 12.

278 001-04, we re-enrolled 19 274 (70%) children at age 9-12 years, and randomly selected 2879 from the 1

279 in pregnancy with offspring metabolic health at age 9-16 y in a longitudinal cohort that oversampled

280 h adult-onset of asthma (defined as starting at age >/=18 years) as compared with childhood-onset sev

281 a group and those with no psychotic disorder at age >/=25 years.

282 208 patients with incident AF-related events at age >/=80 and known prior AF, only 19 (9.1%) were ant

283 tal incident ischaemic strokes, 369 occurred at age >/=80 years, of which 124 (33.6%) were in non-ant

284 ld (280 vs 19), rising to 50-fold (189 vs 4) at age >/=80 years.

285 d OR, 8.0; P = .001), first wheezing episode at age less than 12 months (adjusted OR, 7.3; P = .007),

286 ears, women who had their first menstruation at age </=11 years had a 51% higher risk of developing G

287 The study included 8,383 ALL cases diagnosed at age </=19 years in 2000-2010.

288 -onset inflammatory bowel disease (diagnosis at age <16 years) were excluded.

289 ated the associations of early-life poverty (at age <7 years) and poverty in later childhood (at age

290 nce in the general population was 0.7%-11.5% at ages <2 years and 0.8%-3.3% at ages 2-4 years.

291 ent effects of H. pylori infection (measured at age of 3, 5 and 6.5 years) on prevalence and incidenc

292 py and reported allergic disorders (measured at age of 6.5 years) were determined using multiple logi

293 was not significantly associated with asthma at age of 8 in atopic and non-atopic children.

294 lete data on asthma, anthropometry and atopy at age of 8 years, and potential confounders were availa

295 hibit a transient increase in vessel density at ages P10-P12 due to delayed vessel pruning.

296 ogous therapies, many of which will be aimed at aged patients.

297 monkeys looked preferentially at faces, even at ages prior to the emergence of face domains, but face

298 ulation-based birth cohort) were followed up at age six and half years.

299 ossibility for alternative therapies applied at ages thought to be recalcitrant to recovery.

300 tract length and volumes in subjects who are at ages when the growth is fastest.