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1 ransferase activity, interacts with the ATM (ataxia-telangiectasia-mutated) protein.
2 cardiomyocyte nuclear foci of phosphorylated ataxia telangiectasia mutated protein, an upstream regul
4 ency virus type 1) require the activation of ataxia telangiectasia mutated protein (ATM) and/or Mre11
6 , the protein defective in NBS, functions in ataxia telangiectasia mutated protein (ATM)-dependent si
9 ng pathway, specific protein events, such as ataxia-telangiectasia mutated protein (ATM) activation a
10 eine, an inhibitor of the checkpoint kinases ataxia-telangiectasia mutated protein (ATM) and ataxia-t
11 age-sensing molecules involved were probably ataxia-telangiectasia mutated protein (ATM) and DNA-depe
12 ced gastric cancer, especially in those with ataxia-telangiectasia mutated protein (ATM)-negative tum
14 of the surrogate markers of DNA damage H2AX, ataxia telangiectasia mutated protein, DNA-dependent pro
15 osphorylation at threonine 188 (T188) by the ataxia telangiectasia mutated protein kinase (ATM) and t
20 hat RAP80 can function as a substrate of the ataxia-telangiectasia mutated protein kinase in vitro, w
22 s to decrease the cellular level of the ATM (ataxia telangiectasia mutated) protein, known to repress
23 anism by which SUMOylation and activation of ataxia-telangiectasia-mutated protein, PKCdelta, caspase
25 es of the mammalian cell cycle, and the ATM (ataxia telangiectasia mutated) protein plays a role in i
28 induced phosphorylation of DNA damage marker ataxia-telangiectasia-mutated protein S1981 preceded act
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