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1 n inflammation, consolidation, flooding, and atelectasis.
2 ral ventilation is present it prevents lobar atelectasis.
3 oss of sighs, which was associated with lung atelectasis.
4 tilation in order to induce lobar absorption atelectasis.
5 pressure in the recruitment of diaphragmatic atelectasis.
6 ors, particularly alveolar derecruitment and atelectasis.
7 e sickness to airway obstruction, apnea, and atelectasis.
8 nical ventilation often develop pneumonia or atelectasis.
9 ty-related changes consistent with dependent atelectasis.
10 ociated lung injury is cyclic recruitment of atelectasis.
11 , primarily manifested by significantly less atelectasis.
12 tem bronchus was needed to relieve left lung atelectasis.
13 ry function to those with DPT and no rounded atelectasis.
14  15 of 40), pulmonary edema (40%, 16 of 40), atelectasis (10%, 4 of 40), adult respiratory distress s
15 s included hypertrophic scar formation (35), atelectasis (12), pleural effusion (13), recurrent stern
16  two, by slight thickening or minimal linear atelectasis; 16, by thicker linear or subsegmental atele
17 ontinuation of narcotics), 3 (0.3%) cases of atelectasis, 2 (0.2%) corneal abrasions, 1 (0.1%) subacu
18 st common complications were ileus (27%) and atelectasis (26%).
19 r in group 1 compared with group 2 patients: atelectasis, 44% vs. 73% (p < .001); postextubation stri
20                     After the development of atelectasis, a decremental positive end-expiratory press
21 nt positive end-expiratory pressure to avoid atelectasis, a tidal volume that is limited to less than
22 ventilation) or injury from ventilation with atelectasis and alveolar flooding at end-expiration (ope
23  mice die within 3 days of birth due to lung atelectasis and breathing failure.
24 ng hallmarks of respiratory distress such as atelectasis and hyaline membranes.
25 e correlates with characteristic patterns of atelectasis and pleural fluid collection on conventional
26                        Our patients had less atelectasis and pneumonia than previously published seri
27 ictive pulmonary disease, ineffective cough, atelectasis and pneumonia, and chronic respiratory insuf
28                      Mild frequent PPCs (eg, atelectasis and prolonged oxygen therapy need) deserve i
29 tion, C/EBPgamma-deficient newborns die from atelectasis and respiratory failure, which can be mitiga
30                                              Atelectasis and surfactant depletion may contribute to g
31 e was placed on a lung area with significant atelectasis and the following parameters measured: (1) t
32                      However, the effects of atelectasis (and recruitment) on aerated airspaces remai
33 at they totally collapsed at end expiration (atelectasis) and reinflated during inspiration.
34 tusion, acute respiratory distress syndrome, atelectasis) and the nonspecific radiographic and clinic
35 tusion, acute respiratory distress syndrome, atelectasis) and the nonspecific radiographic and clinic
36  four conditions: pneumonia, fluid overload, atelectasis, and acute respiratory distress syndrome.
37 tal lethality, lung abnormalities resembling atelectasis, and dwarfism characterized by aberrant cart
38 CE mechanics demonstrated alveolar collapse, atelectasis, and leukocyte infiltration.
39 h monolayer collapse pressures, help prevent atelectasis, and minimize the flow of lung surfactant ou
40 (stroke), pulmonary complication (pneumonia, atelectasis, and prolonged ventilation), and renal compl
41 ults in prenatal lung malformation, neonatal atelectasis, and respiratory failure.
42 rmal lungs (baseline), unilateral whole-lung atelectasis, and unilateral and bilateral lung injuries
43 EEP are useful not only to treat hypoxia and atelectasis as the consequence of one-lung ventilation,
44 injured pig lung, in the absence of cyclical atelectasis (as determined with dynamic computed tomogra
45 ree consecutive patients had left lower lobe atelectasis believed to be related to endotracheal tube
46 sing the left atrium and causing medium lobe atelectasis; bilateral pleural effusion was also present
47 riable shunt fraction, such as with cyclical atelectasis, but it is generally presumed to remain cons
48  days, six of seven animals developed patchy atelectasis; by day 11, two of seven animals demonstrate
49 lations suggests that the static behavior of atelectasis cannot be accurately extrapolated to predict
50 issure correlated with a curvilinear band of atelectasis coursing inferomedially and obliquely from i
51                                              Atelectasis develops in critically ill obese patients wh
52 trauma (high-stretch), but not atelectrauma (atelectasis), directly activates monocytes within the pu
53 sure (3 cm H2O) and sustained inflation; and atelectasis group received the same tidal volume as cont
54 ecrosis factor, in the high-stretch, but not atelectasis group.
55                                 In contrast, atelectasis had no effect on perfusate cytokines compare
56             Individuals with DPT and rounded atelectasis had similar pulmonary function to those with
57                        Patients with rounded atelectasis have a history of asbestos exposure or pleur
58 pared with sham, LI animals had irreversible atelectasis, higher lung infection rates (P<0.0001) and
59 tress syndrome in preterm infants, including atelectasis, hyaline membranes, and the lack of pulmonar
60  preceded by a recruitment maneuver reverses atelectasis, improves lung mechanics, distribution of ve
61 ring cyclic recruitment and derecruitment of atelectasis in acute respiratory failure and might harm
62 itive end-expiratory pressure for recruiting atelectasis in dependent and diaphragmatic regions.
63                                         Lung atelectasis in mutants correlates with reduced levels of
64 ges in 21 (33%), perigraft leak in 13 (21%), atelectasis in six (10%), mural thrombus within the sten
65 7, the chest radiographs showed patchy lobar atelectasis in six animals, which cleared by day 11 in a
66 ce regarding the prevention of postoperative atelectasis in sub-Saharan Africa.
67                      Saline lavage increased atelectasis (increase in nonaerated tissue from 1.2% to
68 ventilation in the control group resulted in atelectasis, increased concentrations of bronchoalveolar
69 pithelium and endothelium that causes edema, atelectasis, inflammation, and fibrosis.
70                                      Rounded atelectasis is an atypical form of lung collapse that us
71                        In acute lung injury, atelectasis is common and frequently develops in the dep
72 Overdistension of aerated lung occurs during atelectasis is detectable using clinically relevant magn
73 pirometry in the prevention of postoperative atelectasis is inconclusive.
74 the lung contusions and none of the cases of atelectasis, laceration, or pneumonia (P = .0001).
75 w, deep ventilation was associated with less atelectasis, less alveolar formation, and more elastin w
76 mal values, and that in lobar pneumonia with atelectasis, lung HA would be further decreased because
77 d by pneumonia (n = 2), pleural effusion and atelectasis (n = 1), or liver abscess (n = 1).
78 herapy by nasal cannula (n = 235; 19.6%) and atelectasis (n = 206; 17.1%).
79                  After 14 days of persistent atelectasis of the left lung despite thorascopic decorti
80                                      Rounded atelectasis of the lung is well described in medical lit
81 ung conditions" (p < 0.0001) and "unilateral atelectasis" (p = 0.0026).
82                                              Atelectasis plus lobar pneumonia further decreased lung
83  perioperative pulmonary risk and focuses on atelectasis, pneumonia, and respiratory failure.
84 eep vein thrombosis, respiratory depression, atelectasis, pneumonia, ileus, and postoperative nausea
85 r complications were anastomotic leak (13%), atelectasis/pneumonia (2%), intrathoracic hemorrhage, re
86  infrequent: wound infection/dehiscence, 3%, atelectasis/pneumonia, 2%, intrathoracic hemorrhage, rec
87   However, differential diagnosis of rounded atelectasis poses a challenge to pulmonary specialists a
88 ng all offspring within 24 h of birth due to atelectasis-producing pulmonary hypoxia, which recapitul
89 %) had other noninfectious etiologies (e.g., atelectasis, pulmonary edema, and shock).
90 ations include pleural effusion, hemothorax, atelectasis, pulmonary edema, diaphragmatic dysfunction,
91 eural sparing was also evaluated in cases of atelectasis, pulmonary laceration, and a control group o
92 rio theorizes that the initiating problem is atelectasis that develops as a result of a surfactant ab
93                                              Atelectasis, the most common finding, was present in 20
94         Pulmonary complications ranging from atelectasis to acute respiratory failure are common caus
95 tasis; 16, by thicker linear or subsegmental atelectasis; two, by contiguous tumor infiltration; one,
96 I, 1.7-2.2; p < 10), including pneumothorax, atelectasis, ventilator-associated pneumonia, hypoglycem
97                                              Atelectasis was demonstrated using computerized tomograp
98   An animal model of obesity with reversible atelectasis was developed by placing fluid filled bags o
99 antly different for pneumonia, but dependent atelectasis was more common in patients with early ARDS
100 etained if the five patients with radiologic atelectasis were excluded from analysis.
101 atively combinations of fibrosis, edema, and atelectasis, were present in all premature infants.
102  lowering distal airway pressure may lead to atelectasis, whereas raising distal airway pressure may
103 s breathing caused a significant increase in atelectasis with cyclic collapse.

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