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1 tery and abdominal aorta of 7 rabbits fed an atherogenic diet.
2 Ac) or (Sc) peptide before initiation of the atherogenic diet.
3 een the 3 groups on either a chow diet or an atherogenic diet.
4 s on a chow diet, and sixfold in mice fed an atherogenic diet.
5 ty lipoprotein receptor Ldlr(-/-) mice on an atherogenic diet.
6 esterase-null mice fed either control or an atherogenic diet.
7 ment in T-cell-deficient mice fed a high fat atherogenic diet.
8 ercholesterolemia induced by feeding them an atherogenic diet.
9 wine by 6 months' feeding of a hypercaloric, atherogenic diet.
10 etes, obesity, a sedentary lifestyle, and an atherogenic diet.
11 culating proatherogenic cells in mice fed an atherogenic diet.
12 mice fed either a standard rodent chow or an atherogenic diet.
13 cteristics of autoimmunity in response to an atherogenic diet.
14 G-LDL or in isolated aortae from mice fed an atherogenic diet.
15 h muscle cell content after 8 or 16 weeks of atherogenic diet.
16 e non-Tg littermates on B6 background fed an atherogenic diet.
17 n in EC-specific STAT3 knock-out mice on the atherogenic diet.
18 loss, increased physical activity, and anti-atherogenic diet.
19 plaque collagenases) after 5 and 10 weeks of atherogenic diet.
20 ontransgenic littermate controls were fed an atherogenic diet.
21 half of them deficient in TIMP-1, was fed an atherogenic diet.
22 tic lesions in LDL receptor-null mice fed an atherogenic diet.
23 tery and abdominal aorta of 7 rabbits fed an atherogenic diet.
24 001) in angiotensin II-treated animals on an atherogenic diet.
25 animals receiving either standard chow or an atherogenic diet.
26 ls were placed on either standard chow or an atherogenic diet.
27 rmation in LDL receptor-deficient mice on an atherogenic diet.
28 by C57BL/6 mice fed the bile acid-containing atherogenic diet.
29 e for 24 to 36 hours before the onset of the atherogenic diet.
30 ipids and lipid deposition in animals fed on atherogenic diets.
32 eroma in 33 rabbits by balloon injury and an atherogenic diet (0.3% cholesterol and 4.7% coconut oil)
33 male heterozygous apoE-deficient mice fed an atherogenic diet (2 x 10(3) +/- 2.5 x 10(3) microm2 in h
34 0.5% kcal from fat) (fructose group, n = 9), atherogenic diet (20% kcal from fructose and 46% kcal fr
35 rger than C57BL/6 wild type mice when fed an atherogenic diet (37,123 +/- 3485 microm2 versus 16, 688
38 atherosclerosis was induced in 18 rabbits by atherogenic diet and double balloon endothelial denudati
40 ts and the animals randomized to continue an atherogenic diet (atherosclerosis progression) or resume
42 decreased in B6 mice upon challenge with the atherogenic diet but increased in C3H, indicating that p
43 -cholesterol levels on both the chow and the atherogenic diets, but this locus did not contribute to
44 ression of vascular damage in response to an atherogenic diet by quantifying the in vivo accumulation
45 nd tempers chronic inflammatory responses to atherogenic diet by restraining proinflammatory signalin
50 n rats as well as rabbits, fed on normal and atherogenic diet containing 10% of SL1 and SL2 (experime
54 component of aortic aneurysms induced by an atherogenic diet containing cholate in mice deficient in
57 ild-type and heterozygous counterparts on an atherogenic diet despite having lower levels of apolipop
60 therogenic diet group, n = 13), and modified atherogenic diet (different source of fat and higher pro
63 ces or prevents vascular disease, we treated atherogenic diet-fed ldlr(-/-) mice with recombinant ost
65 e New Zealand White rabbits that were fed an atherogenic diet for 1 week before injury and until euth
68 ide transfer protein gene, were placed on an atherogenic diet for 16 weeks, then either made diabetic
73 Thirty-two cynomolgus monkeys were fed an atherogenic diet for 2 years (progression phase) and the
75 variectomized cynomolgus monkeys were fed an atherogenic diet for 30 months; 25 received 175 microg/d
78 Six cynomolgus monkeys were fed a high-fat atherogenic diet for 44 months and then a low-fat regres
84 2% cholesterol and 0.7% cholate by weight) (atherogenic diet group, n = 13), and modified atherogeni
86 ments demonstrated that B6Tg2576 mice fed an atherogenic diet had more spatial learning impairment th
87 mice were fed with either a high-cholesterol atherogenic diet (HCD) or matching normal diet (ND), res
88 in rabbits, or the rabbits were placed on an atherogenic diet, hepatic mRNA for apoJ was increased by
92 oprotein E-deficient (ApoE(-/-)) mice fed an atherogenic diet highlighted DC efficacy in effector T-c
93 ed lesion development in apoE+/- mice fed an atherogenic diet, indicating that the adverse effect is
96 ssess an antioxidant protective role against atherogenic diet induced oxidative stress in cardiac, he
99 12 weeks of being fed a cholesterol-enriched atherogenic diet, mice injected with GdCl(3) exhibited 5
101 ificantly inhibited the alteration effect of atherogenic diet on the lipid profile and antioxidant en
103 micromol/L in monkeys fed an unsupplemented atherogenic diet (P<0.01) but did not increase in monkey
105 After 2 months under a 1.25% cholesterol atherogenic diet, Pfn(+/-)Ldlr(-/-) (PfnHet) exhibited a
107 oteins isolated from transgenic mice fed the atherogenic diet promoted cholesterol efflux from choles
108 eficiency in LDLr-/- and apoE-/- mice fed an atherogenic diet resulted in increased aortic cholestero
110 ant administration of allosamidin and fed an atherogenic diet showed aggravated atherosclerotic lesio
113 0.01) but did not increase in monkeys fed an atherogenic diet supplemented with B vitamins (3.8+/-0.3
114 ntrol and six other experimental groups (fed atherogenic diet supplemented with different doses of P.
115 netic behavior in cynomolgus macaques fed an atherogenic diet supplemented with either fish oil (1.6
116 were fed an atherogenic diet (control) or an atherogenic diet supplemented with vitamin E, vitamins E
122 L/6J backcross progeny fed either chow or an atherogenic diet to detect QTLs that regulate high-densi
129 n observed in the arteries of rabbits fed on atherogenic diets was significantly reduced when structu
131 lammatory when the mice are maintained on an atherogenic diet, when they are injected with LDL-derive
132 streptozotocin-induced diabetes consuming an atherogenic diet, without changes in gross glomerular mo
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