ライフサイエンスコーパス: atheromatous plaque

1 ole for secretory SMCs in the development of atheromatous plaque.

2 m (Lu-Tex) is a photosensitizer that targets atheromatous plaque.

3 ET-1 and its precursor, big ET-1, within the atheromatous plaque.

4 racterized by the presence of lesions called atheromatous plaques.

5 as reference segments and 1318 segments with atheromatous plaques.

6 , a pathogen also linked to endocarditis and atheromatous plaques.

7 mutans has been found with high frequency in atheromatous plaques.

8 abetes, aortic dimension, the presence of an atheromatous plaque and smoke in the left atrium.

9 overexpression of collagenolytic enzymes in atheromatous plaques and implicated MMPs in the destabil

10 e-related increase in the incidence of aorta atheromatous plaques and periaortic vascular channels in

11 ynthesis may influence both the stability of atheromatous plaques and the development of restenotic l

12 ent reductions in the inflammatory status of atheromatous plaque, and suggest that this effect may ha

13 ies have been detected in circulation and in atheromatous plaques, and immune complexes (ICs) formed

14 PA and vWF may correlate with instability of atheromatous plaques, and that their decrease after coro

15 s plaques < or = abdominal aortic aneurysm < atheromatous plaques; and correlate with macrophage cont

16 ity lipoprotein (OxHDL) within the intima of atheromatous plaques as well as in plasma; however, its

17 In our study, atheromatous plaques (ATH) and macroscopically intact ti

18 Pathological changes consisting of atheromatous plaques, atherosclerotic microaneurysms ext

19 We found that both the coronary atheromatous plaque burden and smoking habit are associa

20 que in the coronary arteries is a marker for atheromatous-plaque burden and is predictive of future r

21 Fibrous and atheromatous plaques but not normal arteries contained N

22 nical events but may evolve into complicated atheromatous plaques characterized by an accumulation of

23 Finally, atheromatous plaques contained higher levels of proinfla

24 The core of an atheromatous plaque contains lipids, macrophages, and ce

25 hought to play an important role in coronary atheromatous plaque destabilization.

26 Atheromatous plaque disruption often presents as complex

27 s have been developed that ablate or section atheromatous plaque during percutaneous coronary interve

28 h, composition, and rupture of intracoronary atheromatous plaque-factors that define the natural hist

29 Metformin attenuated Ang-II-induced atheromatous plaque formation and aortic aneurysm in Apo

30 regressing angiotensin II (Ang-II)-mediated atheromatous plaque formation in ApoE(-/-) mice.

31 have demonstrated a high incidence of CMV in atheromatous plaques from the coronary circulation.

32 sease, but the paucity of neutrophils in the atheromatous plaque has led to neglect of its potential

33 Improved understanding of the biology of atheromatous plaques has led to the concept of plaque vu

34 ynamic therapy (PDT) agent that localizes in atheromatous plaque in which it can be activated by far-

35 associated with irregularity and rupture of atheromatous plaques in both the carotid and coronary ar

36 al microbiota to the bloodstream and then to atheromatous plaques in carotid or other peripheral arte

37 t C pneumoniae can frequently be detected in atheromatous plaques in coronary arteries.

38 tic and asymptomatic patients would identify atheromatous plaques independently of stenosis.

39 showed that the enhanced glycolytic flux in atheromatous plaques of ApoE(-/-) mice was associated wi

40 lut1 connects the enhanced glucose uptake in atheromatous plaques of ApoE(-/-) mice with their myelop

41 o cause clinical manifestations (vulnerable, atheromatous plaques) or those less frequently associate

42 therosclerotic lesions (P < 0.004) and fewer atheromatous plaques (P < 0.008) when compared with ApoE

43 Histological analysis of the atheromatous plaques showed no difference between lesion

44 al for atraumatic and effective debulking of atheromatous plaque through a biological mechanism, the

45 play a significant role in atherogenesis and atheromatous plaque vulnerability and may determine rapi

46 abounded in the macrophage-rich shoulders of atheromatous plaques with histological features of vulne