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1 rotein's function and has been implicated in atherothrombotic disease.
2 concluded that CRP is probably a mediator of atherothrombotic disease.
3 enetics of platelet reactivity pertaining to atherothrombotic disease.
4 n prothrombotic genes with atherogenesis and atherothrombotic disease.
5 ent vascular abnormality in the evolution of atherothrombotic disease.
6 ombin generation in normal hemostasis and in atherothrombotic disease.
7 rs of platelet and endothelial function, and atherothrombotic disease.
8 ed circulating pool of tissue factor (TF) in atherothrombotic diseases.
9 ry markers has been examined in a variety of atherothrombotic diseases.
10 omising approaches to the treatment of human atherothrombotic diseases.
11 gen are associated with an increased risk of atherothrombotic diseases although a causative correlati
13 ay a critical role in the pathophysiology of atherothrombotic disease, and aspirin is the most common
14 ronary syndromes and other manifestations of atherothrombotic disease are primarily caused by atheros
16 uantitative assessment of atherosclerotic or atherothrombotic disease during its natural history and
17 associated with the presence of subclinical atherothrombotic disease (e.g. carotid wall thickness) a
18 echanism for accelerated plaque necrosis and atherothrombotic disease in patients with sitosterolemia
19 AR1 signaling with MMP inhibitors, including atherothrombotic disease, in-stent restenosis, heart fai
20 pathological remodeling processes including atherothrombotic disease, inflammation, angiogenesis, an
21 ease, but the link between hyperglycemia and atherothrombotic disease is not completely understood.
22 Hypercholesterolemia is a risk factor for atherothrombotic disease, largely attributed to its impa
23 rkers that reflect the clinical potential of atherothrombotic disease may allow more precise risk str
24 the Executive Committee of the Prevention of Atherothrombotic Disease Network to issue a "call to act
25 atients >/= 45 years of age with established atherothrombotic disease or >/= 3 risk factors for ather
26 stry, which evaluates subjects with clinical atherothrombotic disease or risk factors for its develop
28 possible etiologic role for C. pneumoniae in atherothrombotic disease, raising questions about the co
30 d TF may contribute to the increased risk of atherothrombotic disease that accompanies these conditio
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