ライフサイエンスコーパス: athletic

1 risk of joint damage during participation in athletics.

2 ts adolescents involved in overhead throwing athletics.

3 n and adolescents participating in organized athletics.

4 nistered before participation in high school athletics.

5 n information source, improved perception of athletic abilities and strength-training self-efficacy,

6 al talent selection techniques in predicting athletic ability, and careful consideration should be gi

7 uscle dysfunction, fatigue, and weakening of athletic ability.

8 ed as a natural and safe method of improving athletic ability.

9 Sudden cardiac death during athletic activities is a highly visible event, and contr

10 d participation by the general population in athletic activities leads to increased trauma to bones,

11 dings in obese children, children engaged in athletic activities, children with renal disease, childr

12 s and positional changes that may occur with athletic activities.

13 Nonparticipation in competitive athletic activity and disease-specific treatment for ide

14 recognized as a cause of sudden death during athletic activity because of its association with ventri

15 In athletic adolescents, spondylolysis is the most common o

16 that inspires Texas A&M Aggies to victory in athletic and academic competitions.

17 ores for young athletes with a concussion in athletic and clinical settings.

18 Dust samples from a mixed-use athletic and educational facility were subjected to micr

19 Online questionnaires ascertained athletic and military history.

20 Specific athletic and occupational activities result in predictab

21 s continued all normal activities, including athletics and driving.

22 Athletics and vanity aside, there are many reasons for w

23 erence for disqualification from competitive athletics are now a standard for management decisions wh

24 eath in Athletes and the National Collegiate Athletic Association (2002 to 2011).

25 death were identified in National Collegiate Athletic Association (NCAA) athletes from 2004 to 2008 b

26 the incidence of SCD in National Collegiate Athletic Association (NCAA) student-athletes and assess

27 in studies of Division 1 National Collegiate Athletic Association and Italian athletes (ages 18 to 25

28 rom data provided by the National Collegiate Athletic Association and the National Federation of Stat

29 The rate of SCD in National Collegiate Athletic Association athletes is high, with males, black

30 The National Collegiate Athletic Association convened a multidisciplinary task f

31 A database of all National Collegiate Athletic Association deaths (2003-2013) was developed.

32 bservational study of 29 National Collegiate Athletic Association Division I football players include

33 n varsity athletics at a National Collegiate Athletic Association Division I university between 1999

34 The National Collegiate Athletic Association Injury Surveillance Program include

35 The National Collegiate Athletic Association Injury Surveillance Program include

36 nd Outcomes Network; and National Collegiate Athletic Association Injury Surveillance Program, respec

37 A total of 1110 National Collegiate Athletic Association member colleges and universities we

38 ll male athletes who participated in varsity athletics at a National Collegiate Athletic Association

39 hallenging because little is known regarding athletic cardiac remodeling in these athletes or athlete

40 athletes of all ages, and serve the greater athletic community and our nation as a whole, by allowin

41 ac emergencies in basketball players and the athletic community at large.

42 e stimulant drugs whose use is prohibited in athletic competition by the World Anti-Doping Agency (WA

43 reatments, and to educate patients in proper athletic conditioning.

44 er a training session (78 cases) or a formal athletic contest (43 cases), with 80 deaths (63%) occurr

45 cess was obtained from the team physician or athletic director; preparticipation screening forms were

46 egetarian diet is compatible with successful athletic endeavor.

47 Collected were daily injury and athletic exposure (AE) reports, a baseline questionnaire

48 son, and the incidence rate was 1.2 per 1000 athletic exposure hours (95% CI, 0.9-1.6).

49 ayers, there were 59 concussions with 43 742 athletic exposure hours.

50 every 10 000 athletic exposures, in which an athletic exposure is defined as one athlete participatin

51 Data on athletic exposure were available for 34 American footbal

52 ately 2.5 concussions occur for every 10 000 athletic exposures, in which an athletic exposure is def

53 , the recognized cardiovascular risks of the athletic field are now extended to include cardiac arres

54 Furthermore, participation in competitive athletics for athletes with life-threatening arrhythmias

55 mportant to develop tools that differentiate athletic from cardiomyopathic change.

56 As our society's interest in competitive athletics has grown, so has the participation of our you

57 Female participation in athletics has increased significantly over the past seve

58 ere [stages III and IV]); informant-reported athletic history and, for players who died in 2014 or la

59 a surface lesion, especially in the young or athletic individual.

60 To properly diagnose and manage athletic injuries in adolescent girls, pediatric health

61 This review identifies athletic injuries that are unique to or especially commo

62 We hypothesized that athletic left ventricular hypertrophy is a consequence o

63 osis of hypertrophic cardiomyopathy in young athletic men should, therefore, not be made purely on th

64 quirements between middle-aged sedentary and athletic men, suggesting that energy requirements of hea

65 ow VO2max is set and concludes that the more athletic one is, the more VO2max is sensitive to O2 tran

66 ities were social rather than educational or athletic (P<.05).

67 Revised management recommendations regarding athletic participation after acute myocarditis have heig

68 In recent years, athletic participation has more than doubled in all majo

69 Athletic participation in high school football.

70 In the initial year of athletic participation, 19.2% of football athletes had h

71 final (19.2% versus 10.2%; P=0.001) years of athletic participation.

72 diatricians can appropriately care for their athletic patients.

73 olic androgenic steroids are used to enhance athletic performance and appearance.

74 (AAS) are illicitly administered to enhance athletic performance and body image.

75 ephedra or ephedrine used for weight loss or athletic performance and case reports of adverse events

76 Concerned with the improvement of athletic performance and the prevention of injury, adole

77 ey achieved at younger ages, but declines in athletic performance are inevitable with ageing.

78 ls, iron deficiency often not only decreases athletic performance but also impairs immune function an

79 to increase limb range of movement prior to athletic performance but it is unclear which component o

80 Examination of athletic performance has been used to discern the functi

81 tal muscle and/or skin perfusion might limit athletic performance in hot environments.

82 afest and most effective agents in enhancing athletic performance in this group are creatine, branche

83 on the association of genetic variation with athletic performance in young athletes is available; how

84 , and evidence to support use of ephedra for athletic performance is insufficient.

85 pecific effects of circadian misalignment on athletic performance under natural conditions.

86 No trials of ephedra and athletic performance were found; 7 trials of ephedrine w

87 growth hormone is reportedly used to enhance athletic performance, although its safety and efficacy f

88 genes are consistently associated with elite athletic performance, and none are linked strongly enoug

89 vers a number of the agents known to enhance athletic performance, and their possible role in preserv

90 g work productivity, voluntary activity, and athletic performance, are addressed.

91 ld stimulates adaptations towards maximising athletic performance, but is unlikely to have further be

92 metimes are used for weight loss or enhanced athletic performance, but the efficacy and safety of the

93 s the decades-long, controlled diminution in athletic performance, should not be seen as a disease pr

94 with at least 8 weeks of follow-up; and for athletic performance, those having no minimum follow-up.

95 e the existing literature on the genetics of athletic performance, with particular consideration for

96 omote the desired adaptations for maximising athletic performance.

97 on and is purported to increase strength and athletic performance.

98 late key physiological processes involved in athletic performance.

99 isease recovery, while males are superior in athletic performance.

100 Leucine has been suggested to improve athletic performance.

101 y determine the effects of growth hormone on athletic performance.

102 that loss of myostatin function can enhance athletic performance.

103 y of ephedra and ephedrine on weight loss or athletic performance; disagreements were resolved by con

104 training environment, is important for elite athletic performance; however, few genes are consistentl

105 The sexual differences in longevity and athletic performances are attributed to the tradeoff bet

106 es are superior in longevity, while males in athletic performances.

107 Shoulder disease is common in the athletic population and may arise as a consequence of a

108 n suggested, although not studied in a large athletic population comparing black and white athletes.

109 ersy of excessive volumes of exercise in the athletic population is discussed.

110 Shoulder injuries are common in the athletic population.

111 early osteoarthritic changes in the injured athletic population.

112 deficiency anemia is likely to be higher in athletic populations and groups, especially in younger f

113 screening for cardiovascular disease in such athletic populations may not be justifiable.

114 Such studies could include clinical and athletic populations to integrate nutritional and exerci

115 This has implications for athletic populations, as well as conditions characterise

116 hysiologists wishing to provide a measure of athletic potential or to characterize subjects in exerci

117 ardiomyopathies, and defining guidelines for athletic preparticipation screening.

118 Athletic pubalgia (AP) is a leading cause of athlete los

119 epicts patterns of findings in patients with athletic pubalgia, including rectus abdominis insertiona

120 ility of the hip, myotendinous injuries, and athletic pubalgia.

121 Athletic remodeling, both on a macroscopic and cellular

122 nificantly lower percentage body fat, higher athletic scores, higher fruit intake, lower total serum

123 mass index, higher estimated VO2max, higher athletic scores, lower ratios of total serum cholesterol

124 Preparticipation athletic screening for cardiovascular disease with stand

125 exist to aid decision making in clinical and athletic settings.

126 However, athletic status had no effect on substrate oxidation.

127 , a third study examining the positive Black-athletic stereotypical association fails to demonstrate

128 le is associated with increased LM and elite athletic success.

129 ly enough to warrant their use in predicting athletic success.

130 ide range of patients: from the sedentary to athletic, the toddler to the pre-teen.The osteochondrose

131 wever, with growing female representation in athletics, the sport community has become increasingly a

132 are providers, and others involved in female athletics to allow early identification and intervention

133 Synthetic athletic tracks and turf areas for outdoor sporting grou

134 moved from participation and evaluated by an athletic trainer or physician prior to returning to part

135 At schools that employ at least one athletic trainer, most high school athletes who sustain

136 ort-related concussions will be cared for by athletic trainers and primary care physicians.

137 Data were collected by athletic trainers at youth, high school, and collegiate

138 Athletic trainers attended each practice and game during

139 lete exposure information were documented by athletic trainers during practices and games.

140 practitioners and 244 schools (34%) allowed athletic trainers to perform examinations.

141 tionally, multidisciplinary teams comprising athletic trainers, physical therapists, primary care spo

142 e geared toward athletes as well as coaches, athletic trainers, school nurses, primary care providers

143 ience sample of 12 NFL teams at professional athletic training facilities between April and July 2007

144 iac remodeling occurs in response to regular athletic training, and the degree of remodeling is assoc

145 Athletic training, such as that undertaken by ballet dan

146 ificantly affected by diet, but not by prior athletic training.

147 The National Athletic Treatment, Injury and Outcomes Network program

148 Youth Football Surveillance System; National Athletic Treatment, Injury and Outcomes Network; and Nat

149 The National Athletic Treatment, Injury, and Outcomes Network Program

150 esponding to unanticipated cardiac events in athletic venues.

151 by echocardiography in a large population of athletic versus sedentary healthy subjects.

152 iological mechanisms for plaque formation in athletic versus sedentary men.

153 Gaining the benefits of participation in athletics while minimizing the risk of osteoarthritis re