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1 ect which was prevented by pretreatment with atipamezole.
2 ter than those following dexmedetomidine and atipamezole.
3 gonists, such as yohimbine, rauwolscine, and atipamezole.
5 of the specific alpha2-adrenergic antagonist atipamezole (1.5 mg/kg i.p.) after chronic treatment wit
7 rmined for the selective alphaAR antagonists atipamezole (8.79), rauwolscine (7.75), 2-(2,6-dimethoxy
9 increasing central noradrenergic tone using atipamezole, an alpha-2 adrenoceptor antagonist, could i
13 followed by atipamezole injection (clonidine-atipamezole) demonstrated dramatic behavioral effects in
14 of the thoracic spinal cord in the clonidine-atipamezole group compared to a sham-operated atipamezol
18 NE or of the alpha2-adrenoceptor antagonist, atipamezole, in the mouse amygdala produces localized ex
21 eated chronically with clonidine followed by atipamezole injection (clonidine-atipamezole) demonstrat
23 nists of alpha(1)- (terazosin) or alpha(2)- (atipamezole) receptors or of either the partial alpha(1)
24 a 2-AR-selective antagonists, rauwolscine or atipamezole, reversed the functional effects of dexmedet
25 (saline-saline, clonidine-saline and saline-atipamezole) showed no overt unusual behavioral effects
26 istration of the selective alpha2-antagonist atipamezole to rats chronically treated with the alpha2-
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