ライフサイエンスコーパス: atonia

1 activation and loss of postural muscle tone (atonia).

2 tion similar to waking accompanied by muscle atonia.

3 d sufficient for generating REM sleep muscle atonia.

4 sleep), yet also in promoting PS with muscle atonia.

5 iting brainstem regions that suppress muscle atonia.

6 rough which positive emotions trigger muscle atonia.

7 nts of the medullary circuitry mediating REM atonia.

8 pocampal EEG, rapid eye movements and muscle atonia.

9 rticipates in rapid eye movement (REM) sleep atonia.

10 are regulated and in turn produce REM sleep atonia.

11 seen during mesopontine stimulation-induced atonia.

12 ccompanied by rapid eye movements and muscle atonia.

13 ese IPSPs appeared exclusively during muscle atonia.

14 ons before and after carbachol-induced motor atonia.

15 infancy occurs against a backdrop of muscle atonia, a result that is consistent with the view that A

16 he relationship between MT and nuchal muscle atonia, a widely recognized component of REM sleep.

17 f consciousness) and to the spinal cord (for atonia and antinociception).

18 produces an REM sleep-like state with muscle atonia and cortical activation, both of which are cardin

19 pathology that can progressively afflict REM atonia and midbrain dopaminergic centers.

20 Brief episodes of muscle atonia and postural collapse resembling cataplexy were a

21 and mechanisms underlying REM sleep without atonia and RBD based on data in cat and rat are presente

22 lesions eliminate REM sleep skeletal muscle atonia and release elaborate behavior.

23 demonstrating independent pathways mediating atonia and the EEG components of REM provide a basis for

24 ry sources to determine their effects on REM atonia and using transgenic mice to identify the neurotr

25 to be recorded during the carbachol-induced atonia, and eight of these also during the subsequent re

26 rks in rodents are responsible for REM motor atonia by retrogradely tracing inputs to the spinal vent

27 e many common features, including the muscle atonia, cataplexy is a distinct state in mice.

28 al, cellular and synaptic basis of REM sleep atonia control is a critical step for treating many slee

29 The absence of atonia during rapid eye movement (REM) sleep and dream-e

30 The descending signal for postural muscle atonia during REM sleep is thought to originate from glu

31 erized by the loss of normal skeletal muscle atonia during REM sleep with prominent motor activity ac

32 t, in turn, probably serve to promote muscle atonia during REM sleep.

33 a failure of the circuitry regulating motor atonia during REM sleep.

34 medial medulla and spinal cord and regulates atonia during REM sleep.

35 to the medulla and spinal cord and regulates atonia during REM sleep.

36 ude that the inhibitory system that mediates atonia during the state of active sleep can be activated

37 litatory mechanism contributes to the muscle atonia elicited in the decerebrate animal and in the int

38 All but one were suppressed during the atonia in parallel to the suppression of XII, phrenic an

39 ventral medulla do not result in loss of REM atonia in rats.

40 duction in REM sleep, and loss of normal REM atonia in some individuals may partially protect against

41 d the mechanisms of carbachol-induced muscle atonia in the alpha-chloralose-anesthetized animal.

42 e (indicative of wakefulness) and periods of atonia (indicative of sleep).

43 intrusion of rapid eye movement sleep muscle atonia into wakefulness.

44 gical features of REM are normal except that atonia is absent and elaborate behaviors may be exhibite

45 s state in fetal sheep is neck nuchal muscle atonia (NA).

46 btained in unanesthetized cats during muscle atonia occurring during natural active sleep.

47 ning the neural circuits responsible for the atonia of active sleep.

48 f spinal motoneurons necessary for the motor atonia of rapid-eye movement (REM) sleep in cats.

49 in association with loss of skeletal muscle atonia of REM sleep.

50 a motor suppression similar to the postural atonia of REM sleep.

51 stration of rapid eye movement sleep without atonia on polysomnography.

52 ctivity was correlated with bilateral muscle atonia or blockage of locomotion.

53 PS with cortical activation, PS with muscle atonia, or W with muscle tone.

54 At all ages, muscle atonia preceded MT and persisted until awake behaviors o

55 ucleus and assess their behaviour during the atonia produced by microinjections of a cholinergic agon

56 observed in rapid eye movement sleep without atonia (REM-A), created in cats by bilateral pontine les

57 y give rise to the phenomenon of REM without atonia (REM-A), in which the electrophysiological featur

58 vity that occur during the carbachol-induced atonia suggest that a similar withdrawal of serotonergic

59 M resulted in an intermittent loss of muscle atonia, taking the form of exaggerated phasic muscle twi

60 Following the recovery from the atonia, the firing rates of the eight cells increased to

61 Following the induction of atonia, the membrane potential activity was dominated by

62 n membrane properties in adult cats in which atonia was produced by the injection of carbachol into t

63 the above drugs produced cataplexy or muscle atonia when perfused into either the ventral tegmental a

64 urred predominantly during periods of muscle atonia (with or without concurrent myoclonic twitching),