ライフサイエンスコーパス: atopic march

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1 ergic disease, is a phenomenon known as the "atopic march".

2 nt of asthma in a disease course called the 'atopic march'.

3 ay account for one mechanism underlying the 'atopic march'.

4 opic dermatitis (AD), a phenomenon known as 'atopic march'.

5 hma development in a phenomenon known as the atopic march.

6 orbidity, ADAM33 may also be involved in the atopic march.

7 ntervention studies to potentially alter the atopic march.

8 nantly eczema loci increase the risk for the atopic march.

9 on and allergic sensitization initiating the atopic march.

10 tigation into environmental modifiers of the atopic march.

11 tical to establishing a causal nature of the atopic march.

12 econdary prevention strategies to arrest the atopic march.

13 s) follow trajectory profiles resembling the atopic march.

14 oietin (TSLP) is thought to drive AD and the atopic march.

15 , may play a critical role in triggering the atopic march.

16 maintenance of allergic inflammation and the atopic march.

17 AD with the goal to stop or even reverse the atopic march.

18 gnized as being a treatable component of the atopic march.

19 by eight latent classes: no disease (51.3%), atopic march (3.1%), persistent eczema and wheeze (2.7%)

20 data reveal critical roles for IL-33 in the "atopic march" and will offer a new therapeutic target in

21 features with these diseases as part of the "atopic march." Controversy exists as to whether immune a

22 other allergic diseases, a link termed 'the atopic march', debate continues as to whether this repre

23 The atopic march describes the progression from atopic derma

24 The atopic march describes this temporal relationship in the

25 bling studies to understand the aetiology of atopic march diseases.

26 s on plausible biological mechanisms for the atopic march focus on defective skin barrier function an

27 The frequently observed atopic march from early AD to asthma, allergic rhinitis,

28 demonstrate a role for TSLP and IL-25 in the atopic march from skin sensitization to food allergic re

29 Although the concept of the atopic march has been refined and strengthened by many c

30 The term "atopic march" has been used to imply a natural progressi

31 The atopic march hypothesis proposes that eczema precedes th

32 pulmonary function with age, reflecting the atopic march in patients with AD.

33 a, there is yet no definitive proof that the atopic march is the primary causal factor in childhood a

34 manifestations of these symptoms followed an atopic march pattern.

35 A popular hypothesis known as the atopic march proposes a set of sequential allergy and re

36 AD and asthma, the mechanisms underlying the atopic march remain poorly understood.

37 In this context, the atopic march remains a promising area of investigation.

38 While the underlying cause of the atopic march remains unknown, recent evidence suggests t

39 llergic rhinitis, in a process known as the "atopic march." Signaling between epithelial cells and in

40 Mouse models of atopic march suggest that systemic, skin-derived thymic

41 triggers and is often the first step in the atopic march that results in asthma and allergic rhiniti

42 xamine the allergic phenotypes that form the atopic march, to determine whether such analyses of data

43 understanding of the role of microbes in the atopic march towards asthma, and in causing exacerbation

44 also spreads beyond the skin to initiate the atopic march, which includes food allergy, asthma, and a

45 s new insights into our understanding of the atopic march, with innate immunity initiating dermatitis

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