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1 esis and possible therapy for infant biliary atresia.
2 6 years (IQR 0.58-3.0) and 64.3% had biliary atresia.
3 amprey thrives despite developmental biliary atresia.
4 role in pathogenesis in experimental biliary atresia.
5 thout tracheo-esophageal fistula or tracheal atresia.
6  and embryos predominantly develop pulmonary atresia.
7 drives the phenotype of experimental biliary atresia.
8 ion to primary biliary cirrhosis and biliary atresia.
9 mice a partial rupture, resulting in choanal atresia.
10 ith isolated SMCP and/or unexplained choanal atresia.
11 mune response in the pathogenesis of biliary atresia.
12 e surrounding follicular cells did not begin atresia.
13 ion of apoptosis in a mouse model of biliary atresia.
14  have furthered our understanding of biliary atresia.
15 the early pathogenesis of congenital biliary atresia.
16  in the pathogenesis of experimental biliary atresia.
17 ant clinical problem in infants with biliary atresia.
18 tion, or to die by programmed cell death, or atresia.
19  mice using an experimental model of biliary atresia.
20 omes, screening, and pathogenesis of biliary atresia.
21 ntributing factor in the etiology of biliary atresia.
22 bility to induce the murine model of biliary atresia.
23 es, and structural proteins at all phases of atresia.
24 jury and obstruction in experimental biliary atresia.
25 ogy of Fallot with complete pulmonary artery atresia.
26 ng laryngeal atresia and the other, tracheal atresia.
27 disease progression in patients with biliary atresia.
28  reserve by early adulthood without inducing atresia.
29 e balance between follicular development and atresia.
30 g a mouse model of rotavirus-induced biliary atresia.
31 athogenesis of the embryonic form of biliary atresia.
32 utor to the pathogenesis of neonatal biliary atresia.
33 s after liver transplantation due to biliary atresia.
34  esophagus did not correlate with esophageal atresia.
35 as not sufficient for diagnosis of pulmonary atresia.
36 ngiography in depicting extrahepatic biliary atresia.
37 es within the livers of infants with biliary atresia.
38 cular hypoplasiawith aortic and mitral valve atresia.
39 , resulting in an attenuated form of biliary atresia.
40  mild pulmonary stenosis to severe pulmonary atresia.
41 bile ducts of mice with experimental biliary atresia.
42 ng sphincter-of-Oddi dysfunction and biliary atresia.
43 ion, showed a limited effect on ovulation or atresia.
44 rapid increase of macrophages and a surge of atresia.
45 ious adverse events in children with biliary atresia.
46 before the surge of macrophage migration and atresia.
47 oses were Ebstein's anomaly (40%), pulmonary atresia (11%), and tetralogy of Fallot (8%).
48 logy of Fallot (15 patients, 25%), tricuspid atresia (12 patients, 20%), Ebstein's anomaly (4 patient
49 omy (5.8% [$21227436]), and small-intestinal atresia (5.1% [$18840546]).
50 dence interval, 45.8 to 64.4); and tricuspid atresia, 74.6% (95% confidence interval, 62.4 to 83.4).
51 rvival was obtained in children with biliary atresia (82%, 79%, and 78% at 1, 5, and 10 years, respec
52             Among TOF, 35 (9%) had pulmonary atresia, 98 (26%) had a palliative procedure before repa
53 sus rotavirus (RRV) can also lead to biliary atresia (a neonatal human disease) in mice.
54  patients with complex pulmonary stenosis or atresia, a detailed delineation of all sources of pulmon
55 est in whether rotavirus could cause biliary atresia, an idiopathic, obliterative infantile disease o
56  congenital malformations comprising choanal atresia, anal abnormalities, post-axial polydactyly, hea
57 e for neonatal disorders such as oesophageal atresia and biliary atresia through clinical trials beca
58 patients with complex pulmonary stenosis and atresia and can be considered a noninvasive alternative
59 rtility associated with increased follicular atresia and decreased rates of ovulation.
60 f variceal bleeding in children with biliary atresia and high-risk gastroesophageal varices.
61 n causes of cholestatic jaundice are biliary atresia and idiopathic neonatal hepatitis (INH).
62 rs in response to Shh knockdown is pulmonary atresia and is directly related to the abnormal developm
63 ously identified in a patient with tricuspid atresia and large secundum atrial septal defect.
64             It was associated with pulmonary atresia and moderate or severe aortic regurgitation in u
65 ring from tetralogy of Fallot with pulmonary atresia and multiple aortopulmonary collaterals and fami
66           Advances in treatments for biliary atresia and necrotising enterocolitis have been achieved
67 erferon gamma in 65% of infants with biliary atresia and no diseased control.
68 le duct remnant in all patients with biliary atresia and only 1 control.
69 ct remnants, and peripheral blood of biliary atresia and other cholestatic disease controls were char
70 ar and humoral autoimmunity in human biliary atresia and possible interventional strategies therefore
71 formation for assessing prognosis in biliary atresia and primary biliary cirrhosis; and important cli
72 cedures such as portoenterostomy for biliary atresia and repair of esophageal atresia and tracheoesop
73  affect infants and children-such as biliary atresia and Reye's syndrome; and (3) redefinition of the
74  on concepts from the mouse model of biliary atresia and rotavirus vaccination programs.
75 wo cases of CHAOS with one showing laryngeal atresia and the other, tracheal atresia.
76            A primary diagnosis of intestinal atresia and the presence of the ileocecal valve may conf
77 for biliary atresia and repair of esophageal atresia and tracheoesophageal fistula.
78 virus (RRV)- induced murine model of biliary atresia and whether the T cells are sufficient to result
79 t survival was best in children with biliary atresia and worst in adults with malignancy.
80 that, based on rates of oocyte degeneration (atresia) and clearance, are needed to continuously reple
81 4 with cryptogenic cirrhosis, 4 with biliary atresia, and 10 normal subjects.
82 (48%) with tetralogy of Fallot and pulmonary atresia, and 4 of 54 (8%) with prosthetic valve endocard
83 ere confirmed (cleft palate, cleft lip, anal atresia, and hypospadias).
84 ngiomas, pyriform aperture stenosis, choanal atresia, and laryngeal webs, may also have adverse effec
85 rythroblastosis fetalis, septicemia, biliary atresia, and other causes of hyperbilirubinemia.
86                          Male sex, pulmonary atresia, and previous palliations emerged as predictors
87 , familial intrahepatic cholestasis, biliary atresia, and primary sclerosing cholangitis, and clinica
88 sis, primary sclerosing cholangitis, biliary atresia, and progressive familial intrahepatic cholestas
89 congenital diaphragmatic hernia, oesophageal atresia, and ruptured omphalocele or gastroschisis.
90 ents of autoimmunity exist in murine biliary atresia, and the progressive bile duct injury is due in
91                  The pathogenesis of biliary atresia appears to involve immune-mediated fibro-obliter
92 curring events such as ovulation and ovarian atresia are accompanied with tissue destruction and repa
93 on, embryonic and perinatal forms of biliary atresia are distinguished by gene expression profiling.
94 le duct obstruction in children with biliary atresia are largely unknown.
95 , primary sclerosing cholangitis and biliary atresia are thought to be immune-mediated cholangiopathi
96 etically programmed animal model for biliary atresia, as it loses its bile ducts and gallbladder duri
97 ent hepatoportoenterostomy (HPE) for biliary atresia at 9 U.S. pediatric centers between 1997 and 200
98  = 5) and perinatal (n = 6) forms of biliary atresia at the time of diagnosis and hybridized them aga
99 omplications of portal hypoplasia in biliary atresia (BA) and acute rejection (AR) are still major co
100 ing Kasai portoenterostomy (KPE) for biliary atresia (BA) and to examine associations between these p
101 ent in infants with cirrhosis due to biliary atresia (BA) as early as the time of evaluation for live
102                        Children with biliary atresia (BA) have increased maternal cells in their live
103                                      Biliary atresia (BA) is a destructive cholangiopathy of childhoo
104                                      Biliary atresia (BA) is a devastating disease of childhood for w
105                                      Biliary atresia (BA) is a fibroinflammatory obstruction of the e
106                                      Biliary atresia (BA) is a neonatal cholangiopathy of unknown eti
107                                      Biliary atresia (BA) is a neonatal obstructive cholangiopathy th
108                                      Biliary atresia (BA) is a progressive fibroinflammatory disorder
109                                      Biliary atresia (BA) is a progressive fibroinflammatory obstruct
110                                      Biliary atresia (BA) is a progressive, inflammatory cholangiopat
111                                      Biliary atresia (BA) is a rare disease in infants, with unknown
112                                      Biliary atresia (BA) is notable for marked ductular reaction and
113 theory regarding the pathogenesis of biliary atresia (BA) is that bile duct injury is initiated by a
114                      The etiology of biliary atresia (BA) is unknown.
115 e, rhesus rotavirus (RRV) can induce biliary atresia (BA), a disease resulting in inflammatory obstru
116                                      Biliary atresia (BA), a progressive fibroinflammatory disorder o
117 th rhesus rotavirus (RRV) results in biliary atresia (BA), and this condition is influenced by the ho
118                                      Biliary atresia (BA), the most common cause of end-stage liver d
119 asonography (US) in the diagnosis of biliary atresia (BA), with surgery as the reference standard.
120 o naturally occurring outbreaks of a biliary atresia (BA)-like disease in livestock.
121 er fibrosis, a grave complication of biliary atresia (BA).
122 rotavirus (RRV)-induced experimental biliary atresia (BA).
123 e three main human cholangiopathies (biliary atresia [BA], primary biliary cholangitis [PBC], and pri
124 tional epidermolysis associated with pyloric atresia/beta4 keratinocytes.
125 ular septum, single ventricle, and tricuspid atresia born in 1996 to 2003 were identified from the Te
126  populate the livers of infants with biliary atresia, but it is unknown whether neonatal lymphocytes
127 duces a disease similar to infantile biliary atresia, but previous attempts to correlate reovirus inf
128                         For example, choanal atresia (CA) is a congenital craniofacial anomaly in whi
129                     In hearts with pulmonary atresia, cardiac neural crest-derived cells, which form
130 s with features of vertebral anomalies, anal atresia, cardiovascular anomalies, trachea-oesophageal f
131 ed immunodeficiency with multiple intestinal atresias (CID-MIA) is a rare hereditary disease characte
132                                      Biliary atresia continues to represent a major challenge with ma
133 d anomalies include ocular coloboma, choanal atresia, cranial nerve defects, distinctive external and
134  defects, tetralogy of Fallot, and tricuspid atresia, defects that resemble those associated with mut
135 st 60 years, successful repair of esophageal atresia (EA) and distal tracheoesophageal fistula (TEF)
136 IM 600992), a condition including esophageal atresia (EA) and tracheoesophageal fistula (TEF), in whi
137 imary repair (aged <3 months) with pulmonary atresia (early-PA group; n=49), and primary repair betwe
138           Epidermolysis bullosa with pyloric atresia (EB-PA), manifesting with neonatal blistering an
139       During and after developmental biliary atresia, expression of cyp7a1 in intestine increased by
140 sis, including organ and digit duplications, atresia, fistulas, hypertelorism, cleft palate and hamar
141  and haematocervix with cervical and vaginal atresia found on the left side (classification 1.2) with
142 s of ocular coloboma, heart defects, choanal atresia, growth retardation, genital abnormalities, and
143 ere bilateral microphthalmia and oesophageal atresia has a de novo missense mutation, R74P, that alte
144 very of the pathogenic mechanisms of biliary atresia has been limited by the inability to study extra
145               Livers of infants with biliary atresia have a coordinated activation of genes involved
146 k formation, and that embryos with pulmonary atresia have an outflow tract septum.
147  and neonatal mice with experimental biliary atresia have shown increased expression of proapoptosis
148 , reintervention was associated with virtual atresia (hazard ratio [HR], 0.51; 95% confidence interva
149 dition is characterized by bilateral choanal atresia, hearing loss, cleft lip and/or palate, and othe
150 ound in 11 independent patients with biliary atresia, hepatitis BC, alcohol, primary biliary cirrhosi
151 Relatively common conditions such as biliary atresia, however, remain largely unexplained and the vir
152  mimic of Tetralogy of Fallot with pulmonary atresia; however, subsequent reports describe only a sin
153 uctal stent were less likely to have virtual atresia (HR, 0.36; 95% CI, 0.15-0.85; P=0.02) and more l
154                        Patients with biliary atresia (i.e., obliteration of the biliary tree) suffer
155 neonatal conditions reported were intestinal atresia in 28 (54.9%) studies, abdominal wall defects in
156 23 subjects and was accompanied by pulmonary atresia in 8.
157 e searched for genomic signatures of biliary atresia in affected infants.
158  re-examine whether rotavirus causes biliary atresia in children are discussed based on concepts from
159 f four patients, unilateral transverse sinus atresia in eight, and a narrowed superior sagittal sinus
160 nce 1999, all infants with suspected biliary atresia in England and Wales, UK, have been referred to
161 pathology analogous to that found in biliary atresia in humans, and high levels of T3SA+ antigen in b
162 ship between rotavirus infection and biliary atresia in humans.
163  important for biliary infection and biliary atresia in mice.
164 erum of mice and children and causes biliary atresia in neonatal mice.
165   Using a model of rotavirus-induced biliary atresia in newborn mice, we found that activated NK cell
166 falcine sinus associated with straight sinus atresia in one patient and suggested transverse sinus th
167 indications for transplantation were biliary atresia in seven, fulminant hepatic failure in six, chro
168 ing and a significant increase in follicular atresia in the 30 ppb group were observed.
169 lts in intestine after developmental biliary atresia, in addition to known mechanisms, such as the re
170 dies in the rotavirus mouse model of biliary atresia indicate that infection of biliary epithelium is
171 onfidence interval, 49.9 to 61.7); pulmonary atresia intact ventricular septum, 55.7% (95% confidence
172 h hypoplastic left heart syndrome, pulmonary atresia intact ventricular septum, single ventricle, and
173 ial intrahepatic cholestasis type 1, biliary atresia, intrahepatic cholestasis of pregnancy, and prim
174                                      Biliary atresia is a devastating disorder of the newborn in whic
175                                      Biliary atresia is a fibro-inflammatory cholangiopathy that obst
176                                      Biliary atresia is a neonatal obstructive cholangiopathy that pr
177                                      Biliary atresia is a rare disease of infancy, which has changed
178                                      Biliary atresia is an inflammatory fibrosclerosing lesion of the
179                                      Biliary atresia is an inflammatory, fibrosclerosing neonatal cho
180                                      Biliary atresia is associated with oligoclonal expansions of CD4
181 ween ovarian folliculogenesis and follicular atresia is critical for female fertility and is strictly
182 onic and perinatal clinical forms of biliary atresia is largely undefined.
183                  The pathogenesis of biliary atresia is not known; one theory is that of a virus-indu
184                         Preantral follicular atresia is observed within the few follicles that develo
185                                      Biliary atresia is the leading cause of cholestasis in infants y
186                                      Biliary atresia is the most common cause of end-stage liver dise
187                                      Biliary atresia is the most common cause of end-stage liver dise
188                                      Biliary atresia is the most common cholangiopathy of childhood.
189                                   A follicle atresia kinetic constant (k(A)) was modeled for the two
190 and are associated with extrahepatic biliary atresia, lead to a loss of membrane recognition, but do
191      Sox17 haploinsufficiency causes biliary atresia-like phenotypes and hepatitis in late organogene
192 iary disease, including extrahepatic biliary atresia, liver disease and transplantation in cystic fib
193                Short stature, auditory canal atresia, mandibular hypoplasia, and skeletal abnormaliti
194 gh much is known about management of biliary atresia, many aspects are poorly understood, including i
195 opment of 46XX females can result in vaginal atresia, masculinization of the urethra, a single urogen
196           Gastroschisis and small-intestinal atresia may be particularly high-yield targets for multi
197 irty-two patients with pulmonary stenosis or atresia (median age: 4.7 years, range: 1 day to 46.9 yea
198                          Multiple intestinal atresia (MIA) is a rare cause of bowel obstruction that
199 ar and cellular basis of multiple intestinal atresia (MIA).
200 astic left heart syndrome (n=346), tricuspid atresia (n=103), tetralogy of Fallot (n=127), pulmonary
201  septal defects (n=22), tricuspid hypoplasia/atresia (n=13), and coronary artery fistulas (n=16).
202 ded tetralogy of Fallot (TOF) with pulmonary atresia (n=13), TOF with pulmonary stenosis (n=4), post-
203 103), tetralogy of Fallot (n=127), pulmonary atresia (n=177), heterotaxy syndrome (n=38), and other (
204  followed by gastroschisis (n=5), intestinal atresia (n=5), and necrotizing enterocolitis (n=4).
205 and had the underlying diagnoses: intestinal atresia, necrotizing enterocolitis, gastroschisis, and m
206 tions in CHD7 cause Coloboma, Heart Disease, Atresia of Choanae, Retardation of Growth and/or Develop
207 our with learning deficits had hypoplasia or atresia of cingulum.
208  knockouts), including abnormal stenosis and atresia of the aorta, defective arterial branching from
209  CHARGE (coloboma of the eye, heart defects, atresia of the choanae, retardation of growth and/or dev
210 yndrome (coloboma of the eye, heart anomaly, atresia of the choanae, retardation, and genital and ear
211 acterized by ocular Coloboma, Heart defects, Atresia of the choanae, Retarded growth and development,
212 acterized by ocular coloboma, heart defects, atresia of the choanae, retarded growth and development,
213 defects (Coloboma of the eye, Heart defects, Atresia of the choanae, severe Retardation of growth and
214 velopment of the mesonephros in males causes atresia of the homolateral ejaculatory duct that results
215 ictly needed, explaining the random culling (atresia) of precursor cells in bilaterians.
216 was increased in patients with porta hepatis atresia (Ohi type II and III vs type I; HR: 2.03, P = 0.
217  were abnormal but not suggestive of biliary atresia (one false-negative finding); 12 were consistent
218 nd 123 (39.9%) an operation to treat biliary atresia or choledochal cyst in the preceding year.
219                                              Atresia or hypoplasia of crura and body of fornices was
220 n bile duct (CBD) disorders, such as biliary atresia or ischemic strictures, is restricted by the lac
221 owever, in contrast to patients with biliary atresia or other forms of cholestasis who develop progre
222 patients (one lost to follow-up) had biliary atresia or other surgical lesions; two were abnormal but
223 stein's anomaly (4 patients, 6.5%), pulmonic atresia or stenosis (7 patients, 11.5%), truncus arterio
224 isk factors for mortality, whereas pulmonary atresia or stenosis and pulmonary artery banding were as
225 s with primary biliary cirrhosis and biliary atresia or with Alagille syndrome, two major pediatric c
226  developed ovaries but had excess follicular atresia, ovulation of defective oocytes, and severely re
227 icantly higher occurrence of pulmonary valve atresia (P = 0.001) compared with cases without a detect
228 cases for which 39 (93%) were due to biliary atresia (P<0.001).
229                            In TOF, pulmonary atresia (P=0.003), male sex (P=0.01) and previous pallia
230 ts with pulmonary stenosis (PS) or pulmonary atresia (PA).
231 vating cytokine IL-33 is elevated in biliary atresia patient serum and in the livers and bile ducts o
232 onfiguration in a paediatric tricuspid valve atresia patient; (3) establish whether the widely used c
233               In the murine model of biliary atresia, perinatal exposure to rhesus rotavirus (RRV) re
234 d compared these phenotypes to the pulmonary atresia phenotype observed following the systemic loss o
235        Senescence may play a role in biliary atresia, primary sclerosing cholangitis, cellular reject
236 tic left heart syndrome and gastrointestinal atresias, probably due to haploinsufficiency for the nei
237 treated with cyclopamine exhibited pulmonary atresia, pulmonary stenosis, and persistent truncus arte
238 ulticenter, double-blind Steroids in Biliary Atresia Randomized Trial (START) was conducted in 140 in
239 r, increased apoptosis of GCs and follicular atresia, reduced ovulation rate, and a dramatic decline
240  as an early outcome in all cases of biliary atresia referred from one of the three centres.
241 3 low), aortopexy (7), laparoscopic duodenal atresia repair (4), and various major cardiac operations
242  for anastomotic strictures after esophageal atresia repair is feasible and acceptably safe and provi
243  tonsillectomy and/or adenoidectomy, choanal atresia repair, and/or treatment of gastroesophageal ref
244 n, neonatal hemochromatosis, and the Biliary Atresia Research Consortium have been summarized.
245 ding ocular coloboma, heart defects, choanal atresia, retarded growth and development, genitourinary
246 E syndrome (coloboma, heart defects, choanal atresia, retarded growth, genital anomalies, and ear ano
247 ed penetrance, such as cleft palate, choanal atresia, septal defects of the heart, haemorrhages, pren
248 were found for spina bifida, cleft lip, anal atresia, severe congenital heart defects in general, or
249 al injury and obstruction within 7 days, and atresia shortly thereafter.
250           Gastroschisis and small intestinal atresia (SIA) are birth defects that are thought to aris
251 with cleft palate (aOR = 1.23) and anorectal atresia/stenosis (aOR = 1.40).
252 sia/stenosis (aOR = 2.97) including duodenal atresia/stenosis (aOR = 2.48), and atrial septal defect
253 gmatic hernia (aOR = 2.58), small intestinal atresia/stenosis (aOR = 2.97) including duodenal atresia
254 he prevalence of rectal and large intestinal atresia/stenosis was higher for ART births compared with
255 he aorta, cleft lip, cleft palate, anorectal atresia/stenosis, and limb reduction (upper limit of 95%
256  networks previously unrecognized in biliary atresia, such as the complement components C3ar-1 and C1
257 nique autosomal-recessive lymphedema-choanal atresia syndrome.
258                            Tracheal agenesis/atresia (TA) is a rare but fatal congenital disease in w
259 subjects from families with congenital aural atresia that were heterozygous for TSHZ1 loss-of-functio
260                                      Biliary atresia, the most common cause of childhood cirrhosis, i
261 ; however, a subset of patients with biliary atresia, the most common childhood cholangiopathy, exhib
262 ve finding); 12 were consistent with biliary atresia (three false-positive findings); four demonstrat
263 vered that the sea lamprey adapts to biliary atresia through a unique mechanism of de novo synthesis
264 ders such as oesophageal atresia and biliary atresia through clinical trials because of the rarity of
265  we show that androgens attenuate follicular atresia through nuclear and extranuclear signaling pathw
266 d drive the phenotypic expression of biliary atresia, thus constituting a potential therapeutic targe
267  We hypothesized that the T cells in biliary atresia tissue expressed related TCRs, suggesting that t
268 s (7.5% [95% CI, 3.5%-13.8%]) with pulmonary atresia to 497 of 801 (62.0% [58.7%-65.4%]) with coarcta
269 we used a rotavirus-induced model of biliary atresia to investigate the entire biliary transcriptome
270 fants with tetralogy of Fallot and pulmonary atresia (TOF/PA) and to prospectively validate these pre
271  adults with surgically corrected esophageal atresia/tracheaesophageal fistula (EA/TEF).
272 ntact ventricular septum and 28 with virtual atresia) underwent RV decompression at median 3 (25th-75
273 ollicle reserve is reduced by maturation and atresia until menopause ensues.
274 ll intestine for diseases such as intestinal atresias, volvulus, and necrotizing enterocolitis.
275 plantation or death in children with biliary atresia was determined.
276                                   Intestinal atresia was the most common diagnosis in the deceased gr
277 long with numbers of ovarian macrophages and atresia wave.
278 nfants born in the UK with suspected biliary atresia were investigated in regional centres, and, if c
279                     148 infants with biliary atresia were treated between January, 1999, and June, 20
280 d chronic biliary disorders, such as biliary atresia, which remains the most common paediatric chroni
281 low-up was predicted by PN dependence and SB atresia, while maximal absolute SB width by birth weight
282 cts including overriding aorta and pulmonary atresia, while none of the sham-operated controls were a
283                   Among infants with biliary atresia who have undergone hepatoportoenterostomy, high-
284 3712]), tracheoesophageal fistula/esophageal atresia (WIQR, $39206; median, $105259 [IQR, $87335-$126
285 TC7A) for mutations associated with multiple atresia with combined immunodeficiency.
286 aling in the myocardium results in tricuspid atresia with hypoplastic right ventricle associated with
287          The syndrome of multiple intestinal atresia with immunodeficiency is a rare, invariably fata
288 ith higher risk of thrombosis were pulmonary atresia with intact ventricular septum (hazard ratio [HR
289 ing catheter valve perforation for pulmonary atresia with intact ventricular septum (PAIVS) 21 years
290 ongitudinal series of fetuses with pulmonary atresia with intact ventricular septum (PAIVS) and/or cr
291 ibe the morphologic variability in pulmonary atresia with intact ventricular septum (PAIVS) within a
292      Ninety-nine patients (71 with pulmonary atresia with intact ventricular septum and 28 with virtu
293                      Patients with pulmonary atresia with intact ventricular septum deemed suitable f
294                                    Pulmonary atresia with intact ventricular septum is rarely associa
295 (RV) decompression in infants with pulmonary atresia with intact ventricular septum vary widely.
296 es undergoing RV decompression for pulmonary atresia with intact ventricular septum were included fro
297 a relatively common birth defect, esophageal atresia with or without tracheoesophageal fistula (EA/TE
298 al diaphragmatic hernia (CDH) and esophageal atresia with tracheoesophageal fistula (EA/TEF) can be r
299 ble-inlet left ventricle (DILV) or tricuspid atresia with transposed great arteries (TA-TGA).
300 ly repaired tetralogy of Fallot or pulmonary atresia with ventricular septal defect.

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