ライフサイエンスコーパス: atrial arrhythmia

1 ce or absence of induced bradyarrhythmias or atrial arrhythmias).

2 on (NVAF) in older adults with no history of atrial arrhythmia.

3 ng new target for understanding and managing atrial arrhythmia.

4 tion, nor did it facilitate sustenance of an atrial arrhythmia.

5 e pericardial knock, larger atrial size, and atrial arrhythmia.

6 cation of patients at risk for stroke during atrial arrhythmia.

7 ut was associated with a shorter duration of atrial arrhythmia.

8 investigating the mechanisms responsible for atrial arrhythmia.

9 characteristics may serve as predictors for atrial arrhythmias.

10 atrium and pulmonary veins may contribute to atrial arrhythmias.

11 velopment but an increased susceptibility to atrial arrhythmias.

12 ng that air pollution may be associated with atrial arrhythmias.

13 BB fibers may play a role in development of atrial arrhythmias.

14 hophysiology and mechanisms of postoperative atrial arrhythmias.

15 circuits are common mechanisms of recurrent atrial arrhythmias.

16 and its regulation by beta-AR stimulation on atrial arrhythmias.

17 cation of AVN conduction in the treatment of atrial arrhythmias.

18 chanisms by which mutant IKur contributes to atrial arrhythmias.

19 associated with early or late postoperative atrial arrhythmias.

20 (WT) littermates that lack I Ks to prolonged atrial arrhythmias.

21 annel and prolongation of repolarization and atrial arrhythmias.

22 ponent in the development of Ca(2+)-mediated atrial arrhythmias.

23 luenced by the development of stress-induced atrial arrhythmias.

24 ycardia pacemaker therapy, and no persistent atrial arrhythmias.

25 priate sinus tachycardia, and 4 patients had atrial arrhythmias.

26 hannels has been proposed as a treatment for atrial arrhythmias.

27 sions for MV surgery provide a substrate for atrial arrhythmias.

28 esized that CRP is elevated in patients with atrial arrhythmias.

29 riod, it may contribute to the substrate for atrial arrhythmias.

30 mia-free periods in patients with paroxysmal atrial arrhythmias.

31 e most consistent predictor of postoperative atrial arrhythmias.

32 may contribute to the development of ectopic atrial arrhythmias.

33 red to be associated with the development of atrial arrhythmias.

34 urea nitrogen, congestive heart failure and atrial arrhythmias.

35 relationship between sinus node disease and atrial arrhythmias.

36 s that may modify atrial conduction or treat atrial arrhythmias.

37 susceptibility to re-entrant ventricular and atrial arrhythmias.

38 ented with sinus node dysfunction and 10 had atrial arrhythmias.

39 are their mapping resolution in scar-related atrial arrhythmias.

40 is known about the potential role of JPH2 in atrial arrhythmias.

41 h sick sinus syndrome/dilated cardiomyopathy/atrial arrhythmias.

42 the treatment of cardiac disease, including atrial arrhythmias.

43 -up but did not reduce the recurrence of all atrial arrhythmias.

44 s to dynamic instabilities that may underlie atrial arrhythmias.

45 extrasystolic activity capable of initiating atrial arrhythmias.

46 that Pitx2 haploinsufficiency predisposes to atrial arrhythmias.

47 ated LRA-signaling pathways in prevention of atrial arrhythmias.

48 s in the delay in cardiac repolarization and atrial arrhythmias.

49 r AF ablation would reduce the occurrence of atrial arrhythmias.

50 tients admitted for dofetilide reloading for atrial arrhythmias, 102 were reloaded at a previously to

51 ne the location of left atrial stasis during atrial arrhythmia; 2) define the degree of stasis associ

52 vs 9.7 days; P < 0.001); and lower rates of atrial arrhythmia (27% vs 19%; P = 0.013) and pulmonary

53 intervention (10.7% vs 3.2%; P<.001) and new atrial arrhythmias (4.6% vs 1.5%; P =.004) occurred more

54 t ventricular ejection fraction (<50%; 45%), atrial arrhythmias (58%), and malignant ventricular arrh

55 iomyopathy, atrioventricular block (AVB), or atrial arrhythmias (AAs) and ventricular arrhythmias (VA

56 Rapid atrial arrhythmias affect the elderly who undergo cardia

57 is was a retrospective review of adults with atrial arrhythmia after Fontan operation who were evalua

58 nd embolic complication (TEC) in adults with atrial arrhythmia after Fontan operation.

59 Survival free of any atrial arrhythmia after one procedure was not significan

60 2 months, the drug-free rate of freedom from atrial arrhythmias after 1 or 2 procedures was 60.2% (95

61 uperior to CA in achieving freedom from left atrial arrhythmias after 12 months of follow-up, althoug

62 Effectiveness outcomes included freedom from atrial arrhythmias after ablation and proportion of part

63 significantly associated with recurrence of atrial arrhythmias after catheter ablation for AF.

64 2 patients with congenital heart disease and atrial arrhythmias, age 32.0 +/- 18.0 years, 45.2% femal

65 significant interaction between a history of atrial arrhythmia and percent pacing.

66 ter, in patients undergoing cardioversion of atrial arrhythmias and in patients with mitral valve dis

67 conduction disturbance and the occurrence of atrial arrhythmias and low left ventricular ejection fra

68 Increased expression of Rho GDIalpha led to atrial arrhythmias and mild ventricular hypertrophy in a

69 uces the incidence of clinically significant atrial arrhythmias and need for cardioversion/hospitaliz

70 Drug-induced BS is associated with atrial arrhythmias and SND.

71 cular symptoms, including a low incidence of atrial arrhythmias and syncope.

72 ons in patients starting sotalol therapy for atrial arrhythmias and to identify factors that might pr

73 Whether beta-blockers reduce atrial arrhythmias and, when added to an angiotensin-con

74 ardia, atrioventricular conduction block, or atrial arrhythmias) and dilated cardiomyopathy.

75 on class 1 or 3 antiarrhythmic drugs (11 for atrial arrhythmias), and 2 were on amiodarone as a bridg

76 ablation reinterventions in 13 patients for atrial arrhythmia, and cardioversions in 15 patients.

77 ed by the implanted valve, the occurrence of atrial arrhythmias, and associated comorbidities.

78 ornia, CHD was associated with incident CHF, atrial arrhythmias, and fetal growth restriction and com

79 associated with myocardial ischemic damage, atrial arrhythmias, and intra-atrial conduction delay.

80 icated in the development and maintenance of atrial arrhythmias, and is characterized by expansion of

81 Patients likely to have severe stasis during atrial arrhythmia are those with left ventricular dilati

82 Ventricular and atrial arrhythmias are common after acute myocardial inf

83 Atrial arrhythmias are common both before and after surg

84 Although stress-induced atrial arrhythmias are common during exercise testing, t

85 Atrial arrhythmias are common early after atrial fibrill

86 directed at prophylaxis and acute therapy of atrial arrhythmias are discussed as are recommendations

87 Atrial arrhythmias are frequent, and atrial flutter may

88 -term ventricular dysfunction, although late atrial arrhythmias are more likely to be encountered.

89 Atrial arrhythmias are the most common complication enco

90 l use for the treatment of heart failure and atrial arrhythmia, are potent inhibitors of DNA double-s

91 for IART now includes algorithms to prevent atrial arrhythmias, as well as antitachycardia pacing, w

92 arrhythmias lasting more than 24 hours; (2) atrial arrhythmias associated with severe symptoms requi

93 y sought to assess the types and patterns of atrial arrhythmias, associated factors, and age-related

94 Freedom from atrial arrhythmias at 12 months postprocedure was simila

95 our understanding of the pathophysiology of atrial arrhythmias, but also to the development of AF ma

96 I drug that is used for the cardioversion of atrial arrhythmias, but it can cause torsade de pointes.

97 of Scn2b in mice results in ventricular and atrial arrhythmias, consistent with reported SCN2B mutat

98 bidities, including coronary artery disease, atrial arrhythmias, diabetes mellitus, and chronic kidne

99 Conversion was defined as termination of the atrial arrhythmia during or within 60 min after infusion

100 dy was to determine if atrial ectopy (AE) or atrial arrhythmias during exercise are predictive of an

101 , 123 patients (31.9%) experienced recurrent atrial arrhythmias during the 1-year follow-up.

102 Electrocardiograms during atrial arrhythmia episodes were reviewed by an electroph

103 also increases the susceptibility to develop atrial arrhythmias facilitated by spontaneous Ca(2+) rel

104 Each species displayed an irregular atrial arrhythmia following oil exposure, indicating a h

105 Although sinus node dysfunction (SND) and atrial arrhythmias frequently coexist and interact, the

106 The primary end point, freedom from left atrial arrhythmia >30 seconds without antiarrhythmic dru

107 Patients with a history of sustained atrial arrhythmia had a stronger baroreflex than those w

108 its effect on the electrical remodeling and atrial arrhythmia have never been explored.

109 oximately one-third of patients experiencing atrial arrhythmias have AF secondary to pulmonary vein-l

110 The incidence and mechanisms of these late atrial arrhythmias have not been thoroughly described.

111 iabetes, coronary artery disease, history of atrial arrhythmias, history of ventricular arrhythmias,

112 n any particular venous drainage pattern and atrial arrhythmia; however, patients with a separate ost

113 IART is the most common presenting atrial arrhythmia in patients with congenital heart dise

114 included dyspnea or heart failure in 67% and atrial arrhythmias in 62%.

115 observed to immediately precede the onset of atrial arrhythmias in 73% of episodes.

116 1 mumol/l was more effective in suppressing atrial arrhythmias in atria with reduced Pitx2c mRNA lev

117 m generator that is involved in Ca-dependent atrial arrhythmias in CPVT.

118 We compared the incidence of atrial arrhythmias in double-lung transplant patients ve

119 c electrophysiology (EP) and inducibility of atrial arrhythmias in MHC-TGFcys33ser transgenic mice (T

120 ein demonstrates increased susceptibility to atrial arrhythmias in mice where Notch has been transien

121 Similar odds were found for atrial arrhythmias in noncomplex (OR, 8.2; 95% CI, 3.0-2

122 ess effective when used for the treatment of atrial arrhythmias in pediatric patients compared with i

123 intervals and the appearance of progressive atrial arrhythmias in RTEF-1 mice.

124 lay an important role in the pathogenesis of atrial arrhythmias in scenarios where VIP release is inc

125 ac melanocyte-like cells and did not develop atrial arrhythmias in the absence of DCT.

126 Atrial arrhythmias in this population can lead to decomp

127 d stimulation, Pitx2(null+/-) adult mice had atrial arrhythmias, including AFL and atrial tachycardia

128 ortant role in triggering and/or maintaining atrial arrhythmias, including atrial fibrillation (AF).

129 he sinoatrial node (SAN) as a participant in atrial arrhythmias, including atrial flutter (AFL) and a

130 tions of n-3 PUFA effects on ventricular and atrial arrhythmias, including studies in patients with i

131 Permanent atrial arrhythmias increased with age from 3.1% to 22.6%

132 evices used as prophylaxis for postoperative atrial arrhythmias; intravenous amiodarone for destabili

133 Prior studies suggest that stroke during atrial arrhythmia is related to stasis in either the bod

134 branch block (kappa 0.78), and very good for atrial arrhythmias (kappa 0.83).

135 kappa 0.56 to 0.70); and good/very good for atrial arrhythmias (kappa 0.84 and 0.79) and bundle bran

136 d of 3 months, AF recurrence (defined as any atrial arrhythmia lasting >/=30 s) was detected using se

137 nd point of the study was a composite of (1) atrial arrhythmias lasting more than 24 hours; (2) atria

138 Atrial arrhythmias may reflect underlying left atrial en

139 ence of advanced age at operation, symptoms, atrial arrhythmias, mitral regurgitation or moderately i

140 albumin levels increased, with tinnitus and atrial arrhythmias more common, in the salsalate group c

141 ies toward developing arrhythmias, with left atrial arrhythmias more commonly observed.

142 Atrial arrhythmias occur commonly after cardiac surgery

143 Atrial arrhythmias occur frequently, and their occurrenc

144 Transient atrial arrhythmia occurred in 5 patients after implantat

145 AF recurrences were defined as any atrial arrhythmia occurring within the first week (early

146 endage thrombus formation, but stroke during atrial arrhythmia occurs frequently in the absence of ap

147 confidence interval, 1.01-11.6, P<0.05) and atrial arrhythmias (odds ratio, 5.1; 95% confidence inte

148 patients (86%) and 5 patients were free from atrial arrhythmia off AADs and on AADs, respectively.

149 veins and freedom from recurrent symptomatic atrial arrhythmia off all antiarrhythmic drugs at 12 mon

150 The primary study end point was freedom from atrial arrhythmias off antiarrhythmic drugs at 1 year af

151 dure (p < 0.001) and higher freedom from all atrial arrhythmias (p = 0.003).

152 ial fibrillation increases in prevalence and atrial arrhythmias progressively become permanent as the

153 d mechanical ventilation, pneumonia, sepsis, atrial arrhythmias, pulmonary embolism, need for early r

154 on of aggressive BP treatment did not reduce atrial arrhythmia recurrence after catheter ablation for

155 rial surface and 18 months of follow-up, the atrial arrhythmia recurrence rate was 15% after 1.4 +/-

156 rders, but its applicability for terminating atrial arrhythmias remains largely unexplored.

157 2/RyR2 ratios can promote SR Ca(2+) leak and atrial arrhythmias, representing a potential novel thera

158 lmonary connection are at risk for recurrent atrial arrhythmia requiring catheter ablation.

159 In 20 additional patients with scar-related atrial arrhythmias, similar sequential mapping with both

160 Exclusion criteria were history of atrial arrhythmia, stroke, valvular or congenital heart

161 Rapid atrial arrhythmias such as atrial fibrillation (AF) pred

162 duce the incidence of spontaneous or induced atrial arrhythmias, suggesting that neuromodulation may

163 vein(s) tended to have a higher frequency of atrial arrhythmia than those with other patterns (P =.05

164 The CS may also generate focal atrial arrhythmias that may play a role in triggering an

165 seful and safe treatment alternative for the atrial arrhythmias that occur after cardiac surgery.

166 In patients with scar-related atrial arrhythmias, the total area of bipolar voltage <0

167 PMCA1(cko) hearts became more susceptible to atrial arrhythmias under rapid programmed electrical sti

168 Incident CHF, atrial arrhythmias, ventricular arrhythmias, and materna

169 Early recurrence of atrial arrhythmia was an independent predictor of late r

170 pattern was determined, and association with atrial arrhythmia was assessed with the chi(2) and Fishe

171 d 82% (LPeAF) at 1 year and freedom from all atrial arrhythmias was 77% (PAF), 75% (PeAF), and 57% (L

172 ntrol study design, CRP in 131 patients with atrial arrhythmias was compared with CRP in 71 control p

173 Freedom from atrial arrhythmias was demonstrated in 12 of 20 (60%) an

174 Recurrence of atrial arrhythmias was the primary endpoint.

175 Subjects with a history of atrial arrhythmia were more likely to be paced < or =92%

176 -proven ATTRwt, poor functional capacity and atrial arrhythmias were common clinical features.

177 cardiomyopathy was identified in 6 patients, atrial arrhythmias were detected in 9 patients, and sudd

178 thy-linked JPH2 mutation not associated with atrial arrhythmias were not significantly different from

179 tients admitted for dofetilide reloading for atrial arrhythmias were retrospectively reviewed.

180 ardia, sinus pauses, and a susceptibility to atrial arrhythmias, which contribute to a phenotype rese

181 red eighty-seven patients with no history of atrial arrhythmia who had a preoperative BNP level and h

182 The primary end point was freedom from atrial arrhythmia with absence of persistent complicatio

183 e Cav1.3-null mutant mice showed evidence of atrial arrhythmias, with inducible atrial flutter and fi

184 n of the LAA improved long-term freedom from atrial arrhythmias without increasing complications.

185 of recurrence was defined as the absence of atrial arrhythmias without using any antiarrhythmic agen