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2 19%), secreted more apoB, and expressed less atrial natriuretic factor (ANF) and brain natriuretic pe
3 5 haploinsufficiency also markedly decreased atrial natriuretic factor (ANF) and connexin 40 (cx40) t
4 function, its synergism with partners at the atrial natriuretic factor (ANF) and connexin-40 (Cx40) p
5 are markers of cardiac hypertrophy including atrial natriuretic factor (ANF) and myosin light chain-2
6 (GSK3beta) is critical for transcription of atrial natriuretic factor (ANF) by beta-adrenergic recep
7 induced increases in NADPH oxidase activity, atrial natriuretic factor (ANF) expression, and cardiac
10 otic expression screen for activators of the atrial natriuretic factor (ANF) gene, a cardiac-specific
11 its DNA binding sites in the promoter of the atrial natriuretic factor (ANF) gene, an in vivo target
13 In contrast, while MnTMPyP inhibited basal atrial natriuretic factor (ANF) mRNA expression, the str
15 containing c-Jun and with the cardiomyocyte atrial natriuretic factor (anf) promoter and the cardiac
19 bx20, activates a cardiac-specific promoter (atrial natriuretic factor (ANF)) alone and synergistical
20 embryonic hearts expressed similar levels of atrial natriuretic factor (ANF), brain natriuretic pepti
22 perated with c-Raf to increase expression of atrial natriuretic factor (ANF), whereas N17Rac1 inhibit
25 We investigated the potential of a C-type atrial natriuretic factor (C-ANF) to image developing pl
26 ently, we reported the (64)Cu-labeled C-type atrial natriuretic factor (CANF) fragment for detecting
27 mg/mm; p<0.001) and reduced LV expression of atrial natriuretic factor (p<0.05), alpha-skeletal muscl
28 nduced expression of two hypertrophic genes (atrial natriuretic factor [ANF] and c-myc) and also enha
29 spholamban restored the expression levels of atrial natriuretic factor and alpha-skeletal actin mRNA
30 n neonatal hearts, ventricular expression of atrial natriuretic factor and alpha-skeletal actin was m
32 ificantly increased heart/body weight ratio, atrial natriuretic factor and beta-myosin heavy chain mR
33 e cross-sectional areas, hypertrophy markers atrial natriuretic factor and beta-myosin heavy chain).
34 ition, two cardiac hypertrophy marker genes, atrial natriuretic factor and beta-myosin heavy chain, w
35 en synthesis and hypertrophy markers such as atrial natriuretic factor and beta-myosin heavy chain.
36 enuated expression of the trabecular markers atrial natriuretic factor and bone morphogenic protein 1
37 el pathways associated with up-regulation of atrial natriuretic factor and brain natriuretic peptide
38 ist-induced expression of the embryonic gene atrial natriuretic factor and enlargement of cardiomyocy
39 f the expression of various genes, including atrial natriuretic factor and Ifi204 (encoding p204).
40 trophy, increased expression of fetal genes (atrial natriuretic factor and skeletal alpha-actin), dec
42 , overexpression of NF-kappaB itself induced atrial natriuretic factor expression and cardiomyocyte e
43 ment, sarcomeric organization, and increased atrial natriuretic factor expression in association with
44 P79 attenuated cardiomyocyte hypertrophy and atrial natriuretic factor expression in response to angi
47 ha(1B)AR-induced hypertrophy and ventricular atrial natriuretic factor expression were significantly
48 ction, which induced myocyte hypertrophy and atrial natriuretic factor expression, protected against
52 ocardin A transactivated the promoter of the atrial natriuretic factor gene through the serum respons
53 ssion of cardiac-specific promoters from the atrial natriuretic factor gene, the b-type natriuretic p
56 ganization, increased cell size, and induced atrial natriuretic factor in cardiomyocytes plated on th
57 els of cardiac hypertrophy and expression of atrial natriuretic factor in Mekk1(-/-) animals, which s
58 s-sectional area, and hypertrophy-associated atrial natriuretic factor induction following pressure o
59 days) did not affect the trophic response or atrial natriuretic factor induction to pressure overload
60 c mass, myocyte size, hypertrophy-associated atrial natriuretic factor induction, and c-Jun N-termina
61 O mice shows a dramatic up-regulation of the atrial natriuretic factor message, a well-established bi
62 here were no significant changes in IGF-1 or atrial natriuretic factor mRNA levels in response to GH
63 in cell size, sarcomeric reorganization, and atrial natriuretic factor production were remarkably att
66 as co-transfected with SRF, but it repressed atrial natriuretic factor promoter activity, which was s
67 P-TF2dependent synergistic activation of the atrial natriuretic factor promoter by both GATA4 and the
68 educed the capability of SRF to activate the atrial natriuretic factor promoter that contains the ser
72 that TBX5 activates the transcription of an atrial natriuretic factor reporter construct and this ef
73 ing adult heart, steady-state mRNA levels of atrial natriuretic factor were increased in both the fet
74 ed in the morphants including an increase in atrial natriuretic factor, a hallmark for cardiac hypert
75 we find that fluorescence from Topaz-tagged atrial natriuretic factor, a peptidergic vesicle pH indi
76 LV fibrosis, and expression of fetal genes (atrial natriuretic factor, alpha-skeletal muscle actin,
77 cardiac expression of HF marker genes (GRK2, atrial natriuretic factor, and beta-myosin heavy chain).
78 yosin heavy chain, cardiac troponin I and T, atrial natriuretic factor, and cardiac transcription fac
79 RNA expression of brain natriuretic peptide, atrial natriuretic factor, and renin were significantly
80 pregulation of hypertrophy markers including atrial natriuretic factor, beta-MHC, and GATA4; and acti
81 onic and contractile protein genes including atrial natriuretic factor, beta-myosin heavy chain, and
82 expression of the hypertrophic marker genes, atrial natriuretic factor, brain natriuretic peptide, an
83 pressed genes including cardiac alpha actin, atrial natriuretic factor, cardiac myosin heavy chain al
84 o well-known markers of cardiac hypertrophy (atrial natriuretic factor, CARP, and beta-myosin heavy c
85 n reaction, we measured transcript levels of atrial natriuretic factor, myosin heavy chain-alpha and
86 hy like: alpha- and beta-myosin heavy chain, atrial natriuretic factor, phospholamban, and sarcoplasm
87 However, in vivo imaging of a neuropeptide, atrial natriuretic factor, tagged with green fluorescent
90 ytosis were developed by expressing a prepro-atrial natriuretic factor-green fluorescent protein fusi
91 iferase gene expression but no activation of atrial natriuretic factor-luciferase gene expression.
92 accumulation, coincident with activation of atrial natriuretic factor-luciferase gene expression.
93 onomous accumulation of the endocytic tracer atrial natriuretic factor-red fluorescent protein at ear
102 r greater, P=0.001), had increased levels of atrial natriuretic peptide (7.3 versus 4.9 pmol/L, P=0.0
105 the EGR1-regulated cardioprotective peptides atrial natriuretic peptide (ANP) and B-type natriuretic
107 of circulating cardiac natriuretic peptides, atrial natriuretic peptide (ANP) and B-type or brain nat
109 ctivation of a fetal gene program, including atrial natriuretic peptide (ANP) and brain natriuretic p
110 at baseline, messenger ribonucleic acid for atrial natriuretic peptide (ANP) and brain natriuretic p
111 ct (IMCD) cells contributes to resistance to atrial natriuretic peptide (ANP) and the excessive sodiu
113 he natriuretic signaling pathway in RPE with atrial natriuretic peptide (ANP) and with membrane-perme
120 Circulating natriuretic peptides such as atrial natriuretic peptide (ANP) counterbalance the effe
121 ibited hypertrophy concurrent with increased atrial natriuretic peptide (ANP) expression that depende
122 mice, TAC resulted in a 15-fold increase in atrial natriuretic peptide (ANP) expression, a 55% incre
123 with Src, modulated basal and ET-stimulated atrial natriuretic peptide (ANP) gene promoter activity,
124 tudy was to investigate the impact of rs5065 atrial natriuretic peptide (ANP) gene variant on coronar
130 e main receptor that mediates the effects of atrial natriuretic peptide (ANP) in the regulation of pl
131 assess the dynamics of nitric oxide (NO) and atrial natriuretic peptide (ANP) induced synthesis of in
142 of this study was to examine the effects of atrial natriuretic peptide (ANP) on cytosolic Ca2+ oscil
143 intravital microscopy to study the effect of atrial natriuretic peptide (ANP) on the extrasplenic mic
144 the major and possibly the only receptor for atrial natriuretic peptide (ANP) or B-type natriuretic p
146 orskolin), we found that low doses of either atrial natriuretic peptide (ANP) or NO donors potentiate
148 extracellular hormone binding domain of the atrial natriuretic peptide (ANP) receptor contains two p
150 y and guanylyl cyclase catalytic) domains of atrial natriuretic peptide (ANP) receptor-A (Npra) in po
152 ism for antiapoptotic signaling initiated by atrial natriuretic peptide (ANP) stimulation leading to
153 ier would attenuate the action of endogenous atrial natriuretic peptide (ANP) to increase vascular pe
156 ive partition analysis indicated that (125)I-atrial natriuretic peptide (ANP) was rapidly released in
157 12-amino acid extension to the C terminus of atrial natriuretic peptide (ANP) was recently geneticall
158 enzyme) is a cardiac protease that activates atrial natriuretic peptide (ANP), a cardiac hormone that
159 nd treat systemic hypertension by expressing atrial natriuretic peptide (ANP), a hormone able to decr
160 RP17 in cardiomyocytes enhances secretion of atrial natriuretic peptide (ANP), a regulator of blood p
161 al structures of the complexes of NPR-C with atrial natriuretic peptide (ANP), and with brain natriur
163 translated region of NPPA, the gene encoding atrial natriuretic peptide (ANP), is associated with blo
164 blood pressure and fluid homeostasis by the atrial natriuretic peptide (ANP), making PDE2-type enzym
167 pro-atrial natriuretic peptide (pro-ANP) to atrial natriuretic peptide (ANP), suggesting that corin
168 ases in vascular permeability in response to atrial natriuretic peptide (ANP), which acts with the ki
169 GC) activity of the receptor protein in both atrial natriuretic peptide (ANP)-dependent and -independ
171 exin A6 (Anxa6) to be a crucial regulator of atrial natriuretic peptide (ANP)-mediated counterhypertr
172 component of the intracellular modulation of atrial natriuretic peptide (ANP)-stimulated activation o
178 udy tested the hypothesis that activation of atrial natriuretic peptide (ANP)/cGMP/protein kinase G s
181 s the diagnostic utility of mid-regional pro-atrial natriuretic peptide (MR-proANP) for the diagnosis
182 enomedullin (MR-proADM), and midregional pro-atrial natriuretic peptide (MR-proANP) in a large prospe
183 sess the prognostic value of midregional pro-atrial natriuretic peptide (MR-proANP) in patients after
184 4 cardiovascular biomarkers, midregional pro-atrial natriuretic peptide (MR-proANP), midregional pro-
185 n AMI is the increased cardiac expression of atrial natriuretic peptide (NP) and B-type NP, with thei
186 th increased plasma levels of N-terminal pro-atrial natriuretic peptide (p = 0.002), after adjustment
187 in men, P<0.001 in women) and N-terminal pro-atrial natriuretic peptide (P<0.001 in men, P=0.001 in w
188 93 cells, we showed that corin converted pro-atrial natriuretic peptide (pro-ANP) to atrial natriuret
189 In cell-based studies, corin converted pro-atrial natriuretic peptide (pro-ANP) to mature ANP, sugg
190 C resulted in marked increases of myocardial atrial natriuretic peptide 4-hydroxy-2-nonenal (a marker
191 gical domains: neurohormonal (N-terminal pro-atrial natriuretic peptide [N-ANP], B-type natriuretic p
192 ohumoral factors, cytokines, endothelin, and atrial natriuretic peptide all may participate in HPA ax
195 ion of the fetal gene program (ie, increased atrial natriuretic peptide and alpha skeletal actin gene
196 for age and clinical covariates, N-terminal atrial natriuretic peptide and B-type natriuretic peptid
198 rdiovascular regulator that is stimulated by atrial natriuretic peptide and B-type natriuretic peptid
199 ions of natriuretic peptides (N-terminal pro-atrial natriuretic peptide and B-type natriuretic peptid
200 s less hypotensive than the cardiac peptides atrial natriuretic peptide and B-type natriuretic peptid
201 eased expression of cardiac embryonic genes (atrial natriuretic peptide and beta-myosin heavy chain)
202 logical properties with the cardiac hormones atrial natriuretic peptide and brain natriuretic peptide
203 NPR)-A is the primary signaling receptor for atrial natriuretic peptide and brain natriuretic peptide
207 ated the TAC-induced increases of myocardial atrial natriuretic peptide and the oxidative stress mark
209 contribute) is the key process through which atrial natriuretic peptide attenuates elevations in cyto
210 ales, cardiac expression of the precursor of atrial natriuretic peptide B and of adrenomedullin also
214 ressed in mice revealed that this unexpected atrial natriuretic peptide effect is brought about by sp
215 iogenesis, increased Akt activity, decreased atrial natriuretic peptide gene expression, and maintena
216 ntricular myocytes, with increased secretory atrial natriuretic peptide granules and degenerative mye
217 ns, the most intriguing have been the use of atrial natriuretic peptide in patients with nonoliguric
218 mm(2), P < 0.01) and marker gene expression (atrial natriuretic peptide increased by 409 +/- 32%, and
220 lls, both the NO donor S-nitrosocysteine and atrial natriuretic peptide induced SSG1 phosphorylation,
224 rdiomyocytes expression of the stress-marker atrial natriuretic peptide is suppressed by EMD 57033.
227 ulate the reduced fat oxidation and elevated atrial natriuretic peptide message of cardiac hypertroph
228 of this study was to examine the effects of atrial natriuretic peptide on potentially harmful elevat
229 one (NT-pro-BNP), and N-terminal fragment of atrial natriuretic peptide pro-hormone (NT-pro-ANP) leve
230 e hypertension, and corin activation and pro-atrial natriuretic peptide processing activity were unde
232 hese pathways, related to the stimulation of atrial natriuretic peptide production and secretion.
233 iously undiscovered, mechanism through which atrial natriuretic peptide protects rat hepatocytes, and
234 two categorically different models (lack of atrial natriuretic peptide receptor A; overexpression of
237 -type natriuretic peptide and N-terminal pro-atrial natriuretic peptide to the risk of death from any
238 e was 24% lower (P<0.001) and N-terminal pro-atrial natriuretic peptide was 16% lower (P<0.001); in w
239 e was 29% lower (P<0.001) and N-terminal pro-atrial natriuretic peptide was 18% lower (P<0.001).
241 ncluded vasoactive peptides; the vasodilator atrial natriuretic peptide was induced by CSD, while the
242 - or 12-week-old animals, and the PCH marker atrial natriuretic peptide was not different in young ve
244 -type natriuretic peptide and N-terminal pro-atrial natriuretic peptide with metabolic risk factors,
245 estry, and identified associations of plasma atrial natriuretic peptide with rs5068 (P = 8 x 10(-70))
246 he expression of known hypertrophic markers (atrial natriuretic peptide) and transcription factor act
247 etic peptides (C-type natriuretic peptide >> atrial natriuretic peptide) and, to a much smaller exten
249 gic hypertrophy (beta-myosin heavy chain and atrial natriuretic peptide) was measured with a quantita
250 n-1, CT-pro-arginine vasopressin, and MR-pro-atrial natriuretic peptide), alone or as a panel, could
252 t converts pro-atrial natriuretic peptide to atrial natriuretic peptide, a cardiac hormone that regul
253 n plasma adrenomedullin, with higher urinary atrial natriuretic peptide, adrenomedullin, and cGMP exc
254 , B-type natriuretic peptide, N-terminal pro-atrial natriuretic peptide, aldosterone, renin, fibrinog
255 ) of cardiac-specific genes (Nkx2.5, GATA-4, atrial natriuretic peptide, alpha- and beta-myosin heavy
256 leation, molecular up-regulation of released atrial natriuretic peptide, altered expression of classi
258 ion, LVEF, serum ACE, plasma angiotensin II, atrial natriuretic peptide, and brain natriuretic peptid
259 m salt (DEA/NO) or the natriuretic peptides, atrial natriuretic peptide, and C-type natriuretic pepti
260 itivities of their cGMP responses to DEA/NO, atrial natriuretic peptide, and C-type natriuretic pepti
261 rations for histology, middle neurofilament, atrial natriuretic peptide, and connexin (Cx) 43 reveale
262 agents at this time appear to be adenosine, atrial natriuretic peptide, and cyclosporine, with other
263 ulate whereas somatostatin, cholecystokinin, atrial natriuretic peptide, and nitric oxide inhibit aci
264 ene-related peptide, adrenomedullin, amylin, atrial natriuretic peptide, and pituitary adenylate cycl
265 mic arteries with substance P, epoprostenol, atrial natriuretic peptide, and relaxin (concentration r
266 for reduced fat oxidation to affect cardiac atrial natriuretic peptide, and thus, induce adipose lip
267 assessed the relations of plasma N-terminal atrial natriuretic peptide, B-type natriuretic peptide,
268 l, carbonyls), hypertrophic gene expression (atrial natriuretic peptide, B-type natriuretic peptide,
269 tricular expressions of the genes coding for atrial natriuretic peptide, beta myosin heavy chain, med
270 g expression of the hypertrophy gene markers atrial natriuretic peptide, brain natriuretic peptide, b
271 Phase 2 began with de Bold's discovery of atrial natriuretic peptide, followed by isolation of the
272 rprisingly, L-CPT1 hearts contained elevated atrial natriuretic peptide, indicating induction of hype
273 odilatin, the renally synthesized isoform of atrial natriuretic peptide, may improve pulmonary conges
274 action of numerous other regulators such as atrial natriuretic peptide, neurotensin, and orexin B ar
276 gregate (amylin, ACTH, LHRH, angiotensin II, atrial natriuretic peptide, somatostatin, oxytocin, neur
277 3T3 cells was increased by prior exposure to atrial natriuretic peptide, suggesting that hormone bind
279 the approximately 180-kDa fragment activated atrial natriuretic peptide, whereas the approximately 16
281 mplants to produce sufficient amounts of the atrial natriuretic peptide, which reduced the blood pres
282 lective PDE2 inhibitor BAY 60-7550 augmented atrial natriuretic peptide- and treprostinil-evoked pulm
284 This report implicates perturbation of the atrial natriuretic peptide-cyclic guanosine monophosphat
285 g both the renin-angiotensin-aldosterone and atrial natriuretic peptide-degrading systems simultaneou
286 f rats with RO-25-6760 for 7 d increased the atrial natriuretic peptide-dependent excretion of sodium
298 or-stimulated cardiomyocyte contractility by atrial natriuretic peptide/cGMP signaling in early cardi
299 lamines, renin, aldosterone, angiotensin and atrial natriuretic peptides (ANP, N-ANP and BNP) were me
300 The NPPA gene codes for the precursor of atrial natriuretic polypeptide, suggesting that NPPA may
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