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1 ic* and pulmonary* vascular resistances, and atrial natriuretic peptide*.
2 tion of the cardiac-derived peptide hormone, atrial natriuretic peptide.
3 lar results were obtained for N-terminal pro-atrial natriuretic peptide.
4 sue weight assessment and gene expression of atrial natriuretic peptide.
5 ibute to the breakdown of bradykinin and the atrial natriuretic peptide.
6 mall peptides such as insulin, glucagon, and atrial natriuretic peptide.
7 Mz-ChA-1 responded only to atrial natriuretic peptide.
8 ypoxia can induce synthesis and secretion of atrial natriuretic peptide.
9 aritide is a 25-amino-acid synthetic form of atrial natriuretic peptide.
10 ing in the presence of chronically increased atrial natriuretic peptide.
11 -type natriuretic peptide in comparison with atrial natriuretic peptide.
12 e of chronically increased concentrations of atrial natriuretic peptide.
13 All patients showed low serum levels of atrial natriuretic peptide.
14 re healthy and demonstrated normal levels of atrial natriuretic peptide.
15 r function, and severely decreased levels of atrial natriuretic peptide.
16 cant even after adjusting for N-terminal pro-atrial natriuretic peptide.
17 ous frameshift mutation in the gene encoding atrial natriuretic peptide.
18 c protease responsible for the activation of atrial natriuretic peptide.
19 described stable fragment of N-terminal pro-atrial natriuretic peptide.
20 eptide was significantly more effective than atrial natriuretic peptide (16-fold increase in cyclic G
21 ose of SDZ-WAG 994, significant increases in atrial natriuretic peptide (216 +/- 137 to 407 +/- 146 p
22 intact internal mammary artery, 1 microM of atrial natriuretic peptide (26-fold increase in cyclic G
24 C resulted in marked increases of myocardial atrial natriuretic peptide 4-hydroxy-2-nonenal (a marker
25 r greater, P=0.001), had increased levels of atrial natriuretic peptide (7.3 versus 4.9 pmol/L, P=0.0
26 t converts pro-atrial natriuretic peptide to atrial natriuretic peptide, a cardiac hormone that regul
28 mRNA from LV myocytes showed upregulation of atrial natriuretic peptide, a molecular marker of hypert
29 ms of natriuretic peptide receptors but that atrial natriuretic peptide acts primarily on the artery
30 n plasma adrenomedullin, with higher urinary atrial natriuretic peptide, adrenomedullin, and cGMP exc
31 lier than the reduction of plasma volume and atrial natriuretic peptide after HeartMate implantation,
32 measured plasma volume and plasma levels of atrial natriuretic peptide, aldosterone, renin, and argi
33 , B-type natriuretic peptide, N-terminal pro-atrial natriuretic peptide, aldosterone, renin, fibrinog
34 ohumoral factors, cytokines, endothelin, and atrial natriuretic peptide all may participate in HPA ax
35 n-1, CT-pro-arginine vasopressin, and MR-pro-atrial natriuretic peptide), alone or as a panel, could
36 ) of cardiac-specific genes (Nkx2.5, GATA-4, atrial natriuretic peptide, alpha- and beta-myosin heavy
39 leation, molecular up-regulation of released atrial natriuretic peptide, altered expression of classi
40 ion of the fetal gene program (ie, increased atrial natriuretic peptide and alpha skeletal actin gene
41 n human ventricles (n=13), message levels of atrial natriuretic peptide and AT(1) receptor were inver
42 for age and clinical covariates, N-terminal atrial natriuretic peptide and B-type natriuretic peptid
44 rdiovascular regulator that is stimulated by atrial natriuretic peptide and B-type natriuretic peptid
46 ions of natriuretic peptides (N-terminal pro-atrial natriuretic peptide and B-type natriuretic peptid
47 s less hypotensive than the cardiac peptides atrial natriuretic peptide and B-type natriuretic peptid
48 eased expression of cardiac embryonic genes (atrial natriuretic peptide and beta-myosin heavy chain)
49 logical properties with the cardiac hormones atrial natriuretic peptide and brain natriuretic peptide
50 NPR)-A is the primary signaling receptor for atrial natriuretic peptide and brain natriuretic peptide
52 thod to quantitate mRNA expression levels of atrial natriuretic peptide and c-fos in an in vitro mode
55 llows a stepwise pattern, with resistance to atrial natriuretic peptide and marked sympathetic activa
56 s opposite effects, as do excess circulating atrial natriuretic peptide and overactivity of various i
57 ated the TAC-induced increases of myocardial atrial natriuretic peptide and the oxidative stress mark
58 he expression of known hypertrophic markers (atrial natriuretic peptide) and transcription factor act
59 etic peptides (C-type natriuretic peptide >> atrial natriuretic peptide) and, to a much smaller exten
61 n, we found that norepinephrine, endothelin, atrial natriuretic peptide, and bradykinin had no signif
62 ion, LVEF, serum ACE, plasma angiotensin II, atrial natriuretic peptide, and brain natriuretic peptid
63 protein synthesis, cellular protein, prepro-atrial natriuretic peptide, and c-fos mRNAs and decrease
64 m salt (DEA/NO) or the natriuretic peptides, atrial natriuretic peptide, and C-type natriuretic pepti
65 itivities of their cGMP responses to DEA/NO, atrial natriuretic peptide, and C-type natriuretic pepti
66 rations for histology, middle neurofilament, atrial natriuretic peptide, and connexin (Cx) 43 reveale
67 agents at this time appear to be adenosine, atrial natriuretic peptide, and cyclosporine, with other
68 tches, the response of BKCa to nitric oxide, atrial natriuretic peptide, and dibutyryl cGMP (Bt2cGMP)
69 only marginally elevated by the addition of atrial natriuretic peptide, and in broken-cell preparati
70 ulate whereas somatostatin, cholecystokinin, atrial natriuretic peptide, and nitric oxide inhibit aci
71 ene-related peptide, adrenomedullin, amylin, atrial natriuretic peptide, and pituitary adenylate cycl
72 or norepinephrine, epinephrine, aldosterone, atrial natriuretic peptide, and plasma renin activity we
73 mic arteries with substance P, epoprostenol, atrial natriuretic peptide, and relaxin (concentration r
74 for reduced fat oxidation to affect cardiac atrial natriuretic peptide, and thus, induce adipose lip
75 lective PDE2 inhibitor BAY 60-7550 augmented atrial natriuretic peptide- and treprostinil-evoked pulm
77 e-A (GC-A) receptor is known to require both atrial natriuretic peptide (ANP) and an adenine nucleoti
78 used to map the antagonistic effects between atrial natriuretic peptide (ANP) and angiotensin II (Ang
79 the EGR1-regulated cardioprotective peptides atrial natriuretic peptide (ANP) and B-type natriuretic
82 of circulating cardiac natriuretic peptides, atrial natriuretic peptide (ANP) and B-type or brain nat
84 ctivation of a fetal gene program, including atrial natriuretic peptide (ANP) and brain natriuretic p
85 at baseline, messenger ribonucleic acid for atrial natriuretic peptide (ANP) and brain natriuretic p
86 , the expression of iNOS mRNA was induced by atrial natriuretic peptide (ANP) and C-type natriuretic
90 ct (IMCD) cells contributes to resistance to atrial natriuretic peptide (ANP) and the excessive sodiu
91 ntrations of plasma norepinephrine (PNE) and atrial natriuretic peptide (ANP) and the renin activity
93 he natriuretic signaling pathway in RPE with atrial natriuretic peptide (ANP) and with membrane-perme
94 pertensive rat, implicated the gene encoding atrial natriuretic peptide (ANP) as a possible candidate
101 GMP, 1-methyl-3-isobutylxanthine (IBMX), and atrial natriuretic peptide (ANP) but not 8-Br-cAMP mimic
102 ed the hypothesis that nitric oxide (NO) and atrial natriuretic peptide (ANP) can attenuate the effec
104 ion (VE) natriuresis and renal resistance to atrial natriuretic peptide (ANP) characterize states of
107 Circulating natriuretic peptides such as atrial natriuretic peptide (ANP) counterbalance the effe
109 ibited hypertrophy concurrent with increased atrial natriuretic peptide (ANP) expression that depende
110 mice, TAC resulted in a 15-fold increase in atrial natriuretic peptide (ANP) expression, a 55% incre
112 with Src, modulated basal and ET-stimulated atrial natriuretic peptide (ANP) gene promoter activity,
113 tudy was to investigate the impact of rs5065 atrial natriuretic peptide (ANP) gene variant on coronar
119 osure to 3-morpholino-sydnonimine (SIN-1) or atrial natriuretic peptide (ANP) in the absence or in th
120 e main receptor that mediates the effects of atrial natriuretic peptide (ANP) in the regulation of pl
121 assess the dynamics of nitric oxide (NO) and atrial natriuretic peptide (ANP) induced synthesis of in
136 ug 44b administered at 10 mg/kg po on plasma atrial natriuretic peptide (ANP) levels in conscious rat
137 failure is characterized by elevated plasma atrial natriuretic peptide (ANP) levels, but little is k
138 nd norepinephrine (NE) increased both OP and atrial natriuretic peptide (ANP) mRNA levels twofold to
139 of this study was to examine the effects of atrial natriuretic peptide (ANP) on cytosolic Ca2+ oscil
140 the impact of arginine vasopressin (AVP) and atrial natriuretic peptide (ANP) on sodium homeostasis i
141 intravital microscopy to study the effect of atrial natriuretic peptide (ANP) on the extrasplenic mic
142 the major and possibly the only receptor for atrial natriuretic peptide (ANP) or B-type natriuretic p
145 orskolin), we found that low doses of either atrial natriuretic peptide (ANP) or NO donors potentiate
147 extracellular hormone binding domain of the atrial natriuretic peptide (ANP) receptor contains two p
150 y and guanylyl cyclase catalytic) domains of atrial natriuretic peptide (ANP) receptor-A (Npra) in po
153 ism for antiapoptotic signaling initiated by atrial natriuretic peptide (ANP) stimulation leading to
154 ier would attenuate the action of endogenous atrial natriuretic peptide (ANP) to increase vascular pe
157 ive partition analysis indicated that (125)I-atrial natriuretic peptide (ANP) was rapidly released in
158 12-amino acid extension to the C terminus of atrial natriuretic peptide (ANP) was recently geneticall
159 ition, plasma arginine vasopressin (AVP) and atrial natriuretic peptide (ANP) were determined at the
160 enzyme) is a cardiac protease that activates atrial natriuretic peptide (ANP), a cardiac hormone that
161 nd treat systemic hypertension by expressing atrial natriuretic peptide (ANP), a hormone able to decr
163 RP17 in cardiomyocytes enhances secretion of atrial natriuretic peptide (ANP), a regulator of blood p
164 al structures of the complexes of NPR-C with atrial natriuretic peptide (ANP), and with brain natriur
167 translated region of NPPA, the gene encoding atrial natriuretic peptide (ANP), is associated with blo
168 blood pressure and fluid homeostasis by the atrial natriuretic peptide (ANP), making PDE2-type enzym
169 nous neuropeptides, endothelin-3 (ET-3), and atrial natriuretic peptide (ANP), modulate the prolifera
171 f the endogenous anti-proliferative peptide, atrial natriuretic peptide (ANP), on the ability of MAPK
174 pro-atrial natriuretic peptide (pro-ANP) to atrial natriuretic peptide (ANP), suggesting that corin
175 ases in vascular permeability in response to atrial natriuretic peptide (ANP), which acts with the ki
176 GC) activity of the receptor protein in both atrial natriuretic peptide (ANP)-dependent and -independ
178 exin A6 (Anxa6) to be a crucial regulator of atrial natriuretic peptide (ANP)-mediated counterhypertr
179 component of the intracellular modulation of atrial natriuretic peptide (ANP)-stimulated activation o
193 udy tested the hypothesis that activation of atrial natriuretic peptide (ANP)/cGMP/protein kinase G s
194 lamines, renin, aldosterone, angiotensin and atrial natriuretic peptides (ANP, N-ANP and BNP) were me
196 contribute) is the key process through which atrial natriuretic peptide attenuates elevations in cyto
197 ales, cardiac expression of the precursor of atrial natriuretic peptide B and of adrenomedullin also
199 assessed the relations of plasma N-terminal atrial natriuretic peptide, B-type natriuretic peptide,
200 l, carbonyls), hypertrophic gene expression (atrial natriuretic peptide, B-type natriuretic peptide,
201 tricular expressions of the genes coding for atrial natriuretic peptide, beta myosin heavy chain, med
202 levels by more than 50% within 3 h and 125I-atrial natriuretic peptide binding by approximately 50%
203 a lesser extent, VD3 effected a reduction in atrial natriuretic peptide, brain natriuretic peptide, a
204 g expression of the hypertrophy gene markers atrial natriuretic peptide, brain natriuretic peptide, b
207 or-stimulated cardiomyocyte contractility by atrial natriuretic peptide/cGMP signaling in early cardi
210 This report implicates perturbation of the atrial natriuretic peptide-cyclic guanosine monophosphat
211 g both the renin-angiotensin-aldosterone and atrial natriuretic peptide-degrading systems simultaneou
212 f rats with RO-25-6760 for 7 d increased the atrial natriuretic peptide-dependent excretion of sodium
216 ressed in mice revealed that this unexpected atrial natriuretic peptide effect is brought about by sp
217 the addition of cGMP intracellularly, or of atrial natriuretic peptide extracellularly, stimulated i
218 guanylyl cyclase-A (GC-A), the receptor for atrial natriuretic peptide, five are conserved within GC
219 Phase 2 began with de Bold's discovery of atrial natriuretic peptide, followed by isolation of the
220 iogenesis, increased Akt activity, decreased atrial natriuretic peptide gene expression, and maintena
221 ed with the negative regulation of the human atrial natriuretic peptide gene promoter by these mutant
222 ntricular myocytes, with increased secretory atrial natriuretic peptide granules and degenerative mye
223 ed the effect of Ras overexpression on human atrial natriuretic peptide (hANP) gene expression, a mar
224 ns, the most intriguing have been the use of atrial natriuretic peptide in patients with nonoliguric
225 angiotensin II, aldosterone, vasopressin and atrial natriuretic peptide in the exercise and control g
226 angiotensin II, aldosterone, vasopressin and atrial natriuretic peptide in the training and control g
227 mm(2), P < 0.01) and marker gene expression (atrial natriuretic peptide increased by 409 +/- 32%, and
229 rprisingly, L-CPT1 hearts contained elevated atrial natriuretic peptide, indicating induction of hype
230 lls, both the NO donor S-nitrosocysteine and atrial natriuretic peptide induced SSG1 phosphorylation,
234 rdiomyocytes expression of the stress-marker atrial natriuretic peptide is suppressed by EMD 57033.
236 he placebo group, P=.001) and reduced plasma atrial natriuretic peptide levels (-2.9 versus 26.9 pg/m
240 odilatin, the renally synthesized isoform of atrial natriuretic peptide, may improve pulmonary conges
241 ulate the reduced fat oxidation and elevated atrial natriuretic peptide message of cardiac hypertroph
243 s the diagnostic utility of mid-regional pro-atrial natriuretic peptide (MR-proANP) for the diagnosis
244 enomedullin (MR-proADM), and midregional pro-atrial natriuretic peptide (MR-proANP) in a large prospe
245 sess the prognostic value of midregional pro-atrial natriuretic peptide (MR-proANP) in patients after
246 4 cardiovascular biomarkers, midregional pro-atrial natriuretic peptide (MR-proANP), midregional pro-
247 1-receptor mRNA and protein, upregulation of atrial natriuretic peptide mRNA expression, and increase
248 of diastolic function, nor the induction in atrial natriuretic peptide mRNA in the hypertrophic vent
250 gical domains: neurohormonal (N-terminal pro-atrial natriuretic peptide [N-ANP], B-type natriuretic p
251 action of numerous other regulators such as atrial natriuretic peptide, neurotensin, and orexin B ar
253 n AMI is the increased cardiac expression of atrial natriuretic peptide (NP) and B-type NP, with thei
254 gated the effectiveness of plasma N-terminal atrial natriuretic peptide (NT-ANP) and brain natriureti
255 of this study was to examine the effects of atrial natriuretic peptide on potentially harmful elevat
256 th increased plasma levels of N-terminal pro-atrial natriuretic peptide (p = 0.002), after adjustment
257 in men, P<0.001 in women) and N-terminal pro-atrial natriuretic peptide (P<0.001 in men, P=0.001 in w
259 one (NT-pro-BNP), and N-terminal fragment of atrial natriuretic peptide pro-hormone (NT-pro-ANP) leve
260 93 cells, we showed that corin converted pro-atrial natriuretic peptide (pro-ANP) to atrial natriuret
261 In cell-based studies, corin converted pro-atrial natriuretic peptide (pro-ANP) to mature ANP, sugg
262 e hypertension, and corin activation and pro-atrial natriuretic peptide processing activity were unde
264 hese pathways, related to the stimulation of atrial natriuretic peptide production and secretion.
265 iously undiscovered, mechanism through which atrial natriuretic peptide protects rat hepatocytes, and
266 two categorically different models (lack of atrial natriuretic peptide receptor A; overexpression of
269 sues and [3H]phenylalanine incorporation and atrial natriuretic peptide secretion as markers of hyper
272 gregate (amylin, ACTH, LHRH, angiotensin II, atrial natriuretic peptide, somatostatin, oxytocin, neur
274 3T3 cells was increased by prior exposure to atrial natriuretic peptide, suggesting that hormone bind
277 -type natriuretic peptide and N-terminal pro-atrial natriuretic peptide to the risk of death from any
278 e was 24% lower (P<0.001) and N-terminal pro-atrial natriuretic peptide was 16% lower (P<0.001); in w
279 e was 29% lower (P<0.001) and N-terminal pro-atrial natriuretic peptide was 18% lower (P<0.001).
282 ncluded vasoactive peptides; the vasodilator atrial natriuretic peptide was induced by CSD, while the
283 - or 12-week-old animals, and the PCH marker atrial natriuretic peptide was not different in young ve
284 f response to C-type natriuretic peptide and atrial natriuretic peptide was the same as in the intact
285 gic hypertrophy (beta-myosin heavy chain and atrial natriuretic peptide) was measured with a quantita
287 the approximately 180-kDa fragment activated atrial natriuretic peptide, whereas the approximately 16
288 NO donors on COX-2-mRNA expression; and (c) atrial natriuretic peptide, which increases cellular cGM
290 mplants to produce sufficient amounts of the atrial natriuretic peptide, which reduced the blood pres
291 -type natriuretic peptide and N-terminal pro-atrial natriuretic peptide with metabolic risk factors,
292 estry, and identified associations of plasma atrial natriuretic peptide with rs5068 (P = 8 x 10(-70))
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