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1 trogram characteristics were analyzed during atrial pacing.
2 died and subjected to 5 hours of rapid right atrial pacing.
3 of sustained AF produced by prolonged right atrial pacing.
4 was created by six weeks of continuous rapid atrial pacing.
5 racellular Na+ concentration following rapid atrial pacing.
6 costeroids, mucolytics, bronchodilators, and atrial pacing.
7 0.003) in patients assigned to postoperative atrial pacing.
8 l activation may be demonstrated during left atrial pacing.
9 illation was induced and maintained by rapid atrial pacing.
10 AF was then induced by rapid right atrial pacing.
11 during multiple loading conditions and right atrial pacing.
12 tiarrhythmic drug therapies during dual-site atrial pacing.
13 ardial gene painting; the AF group had burst atrial pacing.
14 vention of AF is enhanced by dual-site right atrial pacing.
15 chieved in 86% of patients during dual right atrial pacing.
16 e (TA-ER isthmus) and one by rapid overdrive atrial pacing.
17 rval with administration of adenosine during atrial pacing.
18 flutter and could be tested after dual-site atrial pacing.
19 given incremental doses of adenosine during atrial pacing.
20 pacing compared with single-site high right atrial pacing.
21 laxation (Tau), respectively, to incremental atrial pacing.
22 me points during and after incremental rapid atrial pacing.
23 onsumption increased (P = 0.04) from rest to atrial pacing.
24 yocardial uptake also increased from rest to atrial pacing.
25 trial fibrillation (AF) was induced by rapid atrial pacing.
26 entricular pacing but synchronous with right atrial pacing.
27 Sustained AF was induced by rapid atrial pacing.
28 itions may be achieved by either exercise or atrial pacing.
29 which are usually achieved with exercise or atrial pacing.
31 entage of whole body lactate disposal during atrial pacing (15.0 +/- 4.4%) compared to rest (4.9 +/-
32 left bundle-branch ablation and either right atrial pacing (190 to 200 bpm) for 6 weeks (DHF) or 3 we
34 erwent left-bundle branch ablation and right atrial pacing (200 beats/min) for 6 weeks (DHF) or 3 wee
35 ed by left atrial pacing compared with right atrial pacing (21.6 +/- 9.1 versus 32.4 +/- 15.1 ms, res
36 omly assigned to each of 5 groups: (1) rapid atrial pacing (240 bpm) for 3 weeks (n=9), (2) ACEI (ben
37 omly assigned to each of 5 groups: (1) rapid atrial pacing (240 bpm) for 3 weeks (n=9), (2) concomita
38 ly assigned to one of five groups: (1) rapid atrial pacing (240 bpm) for 3 weeks (n=9), (2) concomita
39 ifferent during CS pacing compared with left atrial pacing (41+/-16 versus 33+/-17 ms), the total rig
42 ng reduced LVdP/dtmax compared with baseline atrial pacing (-7.1 +/- 4.1% and -6.9 +/- 4.3%, respecti
46 /-110 days (p < 0.0001) in single-site right atrial pacing and 195+/-96 days in dual-site right atria
48 AF) or atrial flutter induced by single-site atrial pacing and correlated its efficacy with clinical
52 asing lactate at rest which increases during atrial pacing, and (2) when arterial substrate delivery
53 ecorded during spontaneous rhythm, overdrive atrial pacing, and AF/AFL induced by acetylcholine (ACh;
54 ransfer and pacemaker implantation for burst atrial pacing, animals were evaluated daily for cardiac
55 dy was to assess simultaneous right and left atrial pacing as prophylaxis for postoperative atrial fi
56 l patients had TWA measured at baseline with atrial pacing at 100 bpm (600 ms), 109 bpm (550 ms), and
59 Chronotropic stimulation was induced with atrial pacing at 130 minutes(-1) (n=5) whereas inotropic
61 ns during normal sinus rhythm and fixed-rate atrial pacing at 17.2+/-1.8 (SEM) beats per minute great
62 five additional normal anesthetized canines, atrial pacing at 180 beats/min did not increase T-wave a
63 ry drug infusion in sinus rhythm followed by atrial pacing at 20, 40, and 60 beats per minute above t
65 2 +/- 13 versus 76.8 +/- 8 ms, P < 0.01) and atrial pacing at 400 ms (102 +/- 13 versus 91 +/- 9 ms,
66 nt open heart surgery were assigned to right atrial pacing at 45 bpm (RA-AAI; n=39), right atrial tri
72 general anesthesia, AF was induced by burst atrial pacing at baseline and after 1 h of LLTS or sham
74 tients with dual AV node pathway physiology, atrial pacing at three chosen drive cycle lengths (DCL1,
75 cation for pacing, were randomly assigned to atrial pacing (at 70 beats/min) versus minimal ventricul
76 1.02+/-0.07 mL/min per g; P<0.05) and during atrial pacing (baseline, 0.82+/-0.11; enalaprilat, 0.98+
77 l mapping during sinus rhythm, exercise, and atrial pacing, before and after serum potassium increase
78 ion of atrial tachyarrhythmia with dual-site atrial pacing compared with patients without suppression
79 atrial repolarization was minimized by left atrial pacing compared with right atrial pacing (21.6 +/
80 ght atrium remained unchanged with dual-site atrial pacing compared with single-site high right atria
85 0 bpm) for 6 weeks (DHF) or 3 weeks of right atrial pacing followed by 3 weeks of resynchronization b
86 up 2 dogs (N=6) underwent intermittent rapid atrial pacing followed by active or sham LL-VNS on alter
89 n was tested in distal segments: incremental atrial pacing for endothelium-dependent cases; and intra
91 lity, safety and efficacy of multisite right atrial pacing for prevention of atrial fibrillation (AF)
93 ll established, the efficacy of device-based atrial pacing for treating spontaneous atrial tachyarrhy
99 recurrent AF with long-term dual-site right atrial pacing has been demonstrated in our previous stud
101 e AVN, AF was simulated by random high right atrial pacing in 11 atrial-AVN rabbit heart preparations
107 applied a novel technique of dual-site right atrial pacing in an unselected group of consecutive pati
109 myocardial ischaemia induced by incremental atrial pacing in patients with coronary artery disease c
110 LV function and the response to incremental atrial pacing in patients with dilated cardiomyopathy (n
111 acy and safety evaluation of dual-site right atrial pacing in patients with drug-refractory atrial fi
112 nd 4) atrial tachycardia simulated by demand atrial pacing in patients with inducible atrioventricula
113 eterization was performed at rest and during atrial pacing in patients with mild IPAH (n = 10) compar
116 ing rest and increased heart work induced by atrial pacing in seven healthy men and women (age, 49.7
117 ial fibrillation (AF); and 2) the ability of atrial pacing in single- and dual-site modes to increase
118 re and after the lesions was assessed during atrial pacing in sinus rhythm from the medial and latera
119 oup 2 = radiofrequency energy applied during atrial pacing in sinus rhythm from the proximal coronary
120 fter which further energy was applied during atrial pacing in sinus rhythm until isthmus conduction b
122 this study was to determine the efficacy of atrial pacing in the prevention of atrial fibrillation f
123 f atrial repolarization is minimized by left atrial pacing in this preparation with no further advant
128 right ventricle during AF (RA-irregular) and atrial pacing inhibited by atrial sensing mode at a rate
129 rpose of this study was to determine whether atrial pacing is a safe alternative to minimal (backup-o
131 he purpose of this study was to determine if atrial pacing is effective in reducing postoperative atr
134 ax) and aortic pulse pressure) compared with atrial pacing-LBBB, and this improvement correlated with
136 The MCS included 2589 patients with Accufix atrial pacing leads that were implanted at or who were f
137 istration of small doses of adenosine during atrial pacing led consistently to transient block of imp
138 RR interval during atrial fibrillation/rapid atrial pacing</=250 ms (or antegrade effective refractor
144 le pericarditis model (n = 10) and the rapid atrial pacing model (400 beats/min for 6 weeks, n = 6).
146 ed was the efficacy of two single-site right atrial pacing modes (high right atrium and coronary sinu
149 ), either spontaneously (n = 36) or by rapid atrial pacing (n = 50), with AFL being typical in the ma
150 tion (n=7) and human studies involving fixed atrial pacing (n=10), passive head-up tilt testing (n=11
151 ned from dogs with AF induced by rapid right atrial pacing (n=6 for left atrial, n=4 for right atrial
152 ) were randomized to one of three groups: no atrial pacing (NAP), right atrial pacing (RAP) or biatri
155 red at a fixed rate after at least 1 week of atrial pacing only (baseline), during short-term (2 h) a
158 40; 95 percent CI, 1.04-1.89); postoperative atrial pacing (OR, 1.27; 95 percent CI, 1.00-1.62); and
159 15.6 pg/ml 60 min after termination of rapid atrial pacing (p < 0.0001), changes that were mirrored a
160 pacing and 195+/-96 days in dual-site right atrial pacing (p < 0.005 versus single-site pacing and p
162 sted for age, sex, lower rate limit, percent atrial pacing, percent biventricular pacing, and implant
163 (1) at baseline, (2) at 10 and 30 minutes of atrial pacing plus intravenous norepinephrine infusion,
164 differentially decreased to ventricular vs. atrial pacing post-MI (63% in control vs. 44% in MI to v
165 and it used 3 different methods of overdrive atrial pacing (Ramp, Burst+, and 50-Hz burst) to treat A
171 cular (AV) node dysfunction, rate-responsive atrial pacing should be used in the majority of cases.
175 We compared a new two-stage transesophageal atrial pacing stress echocardiography (TAPSE) protocol w
183 itions (intracoronary 5% dextrose in water), atrial-pacing tachycardia decreased Tau by 13% (P<0.05),
186 he subgroup of 41 patients treated only with atrial pacing therapies was analyzed, the reduction in b
187 ry active (functional) hyperaemia induced by atrial pacing to a high rate in the denervated heart of
189 We tested the ability of dual-site right atrial pacing to prevent atrial fibrillation (AF) or atr
190 f catecholamines; 2) correction of fever; 3) atrial pacing to restore synchrony; 4) digoxin; 5) pheny
191 tients and the Atrial Fibrillation Reduction Atrial Pacing Trial enrolled 2580 patients receiving a d
192 l (RA) pacing was performed for 3 minutes in atrial pacing triggered by ventricular sensing mode trig
193 id not exhibit spontaneous AF but that rapid atrial pacing unmasked an increased vulnerability to AF
195 nted dogs, we found that rapid (400 min(-1)) atrial pacing was associated with attenuation of the atr
200 study comparing dual- and single-site right atrial pacing was performed followed by a long-term effi
204 X2 transgenic mouse and a pig model of rapid atrial pacing, we found that oxidation products (such as
205 t baseline and after LV epicardial and right atrial pacing with high-resolution Doppler and contrast
206 ke also increased significantly from rest to atrial pacing with no significant differences in fractio
207 r or fibrillation was induced by rapid right atrial pacing with or without a right atrial free wall c
209 ose uptake increased (P = 0.04) from rest to atrial pacing with unchanged fractional extraction (rest
211 pose of this study was to determine if burst atrial pacing would have an effect on terminating atrial
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