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1 n heart rate, mean aortic pressure, or right atrial pressure.
2 -LVDP) was used as a surrogate for mean left atrial pressure.
3 d elevated minimal LV pressure and mean left atrial pressure.
4 gh increases in cardiac output (.Q) and left atrial pressure.
5 using capacity of carbon monoxide, and right atrial pressure.
6 ion of right ventricular function, and right atrial pressure.
7 result of a lowering of early diastolic left atrial pressure.
8 on end-diastolic arterial pressure and right atrial pressure.
9  +/- 6 mm Hg, p < 0.002) and increased right atrial pressure (10 +/- 6 vs. 8 +/- 4 mm Hg, p < 0.05).
10 roximately 2-fold greater increases in right atrial pressure (10+/-4 versus 6+/-3 mm Hg; P=0.02), pul
11 us 5.1+/-1.9 L/min; P=0.01), increased right atrial pressure (12+/-5 versus 4+/-1 mm Hg; P=0.0002), a
12 and diuretics administered to decrease right atrial pressure (18+/-2 to 7+/-1 mm Hg), pulmonary wedge
13 0 mg groups, respectively; P<0.05) and right atrial pressure (-2.0+/-0.4, -3.7+/-0.4, and -3.5+/-0.4
14 44 mm Hg to 12 +/- 6 mm Hg; p = 0.007), left atrial pressure (29 +/- 11 mm Hg to 20 +/- 8 mm Hg; p =
15                 Tolvaptan also reduced right atrial pressure (-4.4 +/- 6.9 mm Hg [p < 0.05], -4.3 +/-
16 as tested prospectively in the estimation of atrial pressure 50 patients.
17    There were significant decreases in right atrial pressure (9+/-1 to 7+/-1 mm Hg, p < 0.01 by ANOVA
18 ular resistance, significantly higher common atrial pressure after Fontan and significantly lower pos
19 tic venous flow dynamics relate best to mean atrial pressure and can be used clinically to estimate m
20 as no significant relation between mean left atrial pressure and deceleration time.
21 creased after endotoxin infusion, while left atrial pressure and left ventricular end-diastolic diame
22                                         Left atrial pressure and left ventricular end-diastolic diame
23 mic hypotension occurred with a fall in left atrial pressure and little change in left ventricular vo
24 levated SCr was associated with higher right atrial pressure and lower cardiac index.
25 yncopal patients presented with higher right atrial pressure and lower cardiac outputs with lower sur
26 , mean pulmonary artery pressure, mean right atrial pressure and mean left atrial pressure) at baseli
27 nges in hemodynamic values (except for right atrial pressure and mean pulmonary artery pressure) were
28 te, mean arterial blood pressure, mean right atrial pressure and peak airway pressure.
29 ases in cardiac output and decreases in left atrial pressure and peripheral resistance but without el
30 elow Veq, we used a servomotor to clamp left atrial pressure and produce nonfilling diastoles, allowi
31 re, pulmonary vascular resistance, and right atrial pressure and provided incremental prognostic valu
32 ormed during exercise, included higher right atrial pressure and pulmonary capillary wedge pressure a
33 mic changes and additionally decreased right atrial pressure and pulmonary vascular resistance.
34 ed for the simultaneous measurement of right atrial pressure and renal sympathetic nerve activity.
35 excursion correlated with WHO-FC, mean right atrial pressure and survival (P<0.05).
36 ure (27% to 39% decrease by 6 h), mean right atrial pressure and systemic vascular resistance, along
37 PV showed positive correlation between intra-atrial pressure and the number of waves emanating from t
38  atrial diastole, resulting in a higher late atrial pressure and thus a normal mean pressure.
39                                              Atrial pressure and volume loops were prepared from inva
40 right atrial, pulmonary artery and mean left atrial pressures and cardiac output were obtained.
41                           Arterial and right atrial pressures and end-tidal CO2 were measured.
42                                         Left atrial pressures and LV volumes and pressures were measu
43 alculated from Doppler indices reflective of atrial pressures and the diastolic filling volume.
44 sion pressure (diastolic; aortic minus right atrial pressure) and cerebral perfusion pressure (mean a
45 (MAP), pulmonary artery pressure (PAP), left atrial pressure, and cardiac output (CO).
46  aortic pressure, in the left atrium to left atrial pressure, and in all heart chambers to a decrease
47 rized by a decrease in cardiac output, right atrial pressure, and left ventricular (LV) end-diastolic
48 ikely because of marked increase in the left atrial pressure, and preload reduction may unmask the re
49  exercise tolerance, functional class, right atrial pressure, and vasodilator response to adenosine.
50 rdiac output; mean aortic, pulmonary or left atrial pressures; and peak positive and negative first d
51 edural monitoring included vital signs, left atrial pressure, arterial blood pressure, cerebral perfu
52 urements of left ventricular inflow and left atrial pressures, ascending aortic pressure, thermodilut
53  the modified Bernoulli equation, with right atrial pressure assumed to be 10 mm Hg.
54 ass at diagnosis (P < 0.001), and high right atrial pressure at diagnosis (P = 0.002).
55 re, mean right atrial pressure and mean left atrial pressure) at baseline, during 60 min of atrial fi
56 reload was abruptly reduced by clamping left atrial pressure between 0 and -2 mm Hg in seven open-che
57 y arterial pressure of < 25 mm Hg and a left atrial pressure between 2 and 5 mm Hg.
58 hypotension or perioperative changes in left atrial pressure, brain natriuretic peptide levels, lacti
59        Breathing NO decreased the mean right atrial pressure by 12 +/- 3%, mean pulmonary arterial pr
60  congestion were produced by increasing left atrial pressure by 2 mmHg.
61  balloon in the left atrium to increase left atrial pressure by 5 mmHg.
62 High pulmonary vascular resistance and right atrial pressure by invasive hemodynamic measurements wer
63 scular resistance (by 29%; P=0.03) and right atrial pressure (by 40%; P=0.007), but with only modest
64  measured lung lymph flow after raising left atrial pressure (by inflating a balloon) in sheep that w
65 ion, left ventricular end-diastolic and left atrial pressure can rise to extremely high levels.
66  invasively derived measurements, mean right atrial pressure cardiac index, and mixed venous oxygen s
67  of 6MWD, pulmonary arterial pressure, right atrial pressure, cardiac index and pulmonary vascular re
68 th Organization functional class, mean right atrial pressure, cardiac index, and mixed venous oxygen
69  and mean systemic arterial pressures (MAP), atrial pressures, cardiac output, and arterial blood gas
70                                    With left atrial pressure clamping, maximal LV pressure decreased
71 domized to the PAC arm (n = 194), only right atrial pressure correlated weakly with baseline SCr (r =
72  aging has been proposed in which early left atrial pressure could be low in the aged heart but rise
73                                        Right atrial pressure decreased 52% (P=0.012), pulmonary arter
74 .1 +/- 1.1 to 13.2 +/- 1.3 mm Hg; mean right atrial pressure decreased from 10.9 +/- 1 to 4.8 +/- 1.0
75 whereas diastolic left ventricular and right atrial pressures decreased significantly and proportiona
76 e data indicate that PAOP overestimates left atrial pressure during endotoxin shock, making it an ina
77  is feasible, seems to be safe, reduces left atrial pressure during exercise, and could be a new stra
78 (PPH), right atrial pressure may exceed left atrial pressure during exercise, resulting in a right-to
79 ogy was defined as inspiratory rise in right atrial pressure during right heart catheterization.
80 rial contraction (a wave); point 2, the left atrial pressure during the start of ventricular systole;
81 ial filling (v wave); point 4, earliest left atrial pressure during ventricular filling; and the line
82 mean difference between the aortic and right atrial pressures during the release phase of chest compr
83 terogeneous clinical syndrome, elevated left atrial pressure-either at rest or with exertion-is a com
84                                         Left atrial pressure elevation during dextran infusion increa
85                                   Thus, left atrial pressure elevation increased lymph flow less in d
86                              Absence of left atrial pressure elevation was based on combined hemodyna
87          For comparison, we also raised left atrial pressure elevation, plasma oncotic pressures in d
88      We hypothesized that elevation of intra-atrial pressure elicits high-frequency and spatio-tempor
89  complex but characterised by increased left atrial pressure, especially during exertion, which might
90 dge pressure fell from 31 to 18 mm Hg, right atrial pressure from 15 to 8 mm Hg, and SVR from 1,780 t
91 ters also improved with a reduction in right atrial pressure from 22 mm Hg at baseline, to 9 mm Hg an
92 Hg to 11.5+/-3.7 mm Hg; p < .001), and right atrial pressures (from 7.3+/-2.5 mm Hg to 9.8+/-2.5 mm H
93 %, exercise PCWP >/=25 mm Hg, and PCWP-right atrial pressure gradient >/=5 mm Hg.
94 time, < 180 m/s, which indicated a mean left atrial pressure &gt; or = 20 mm Hg, were both 100%.
95                                     At intra-atrial pressures &gt;10 cm H2O, the maximum dominant freque
96 ic variables such as cardiac index and right atrial pressure have consistently been associated with s
97 leaflets, which equals left ventricular-left atrial pressure, have been proposed to explain this dyna
98 on pulmonary-capillary wedge pressure, right atrial pressure, heart rate, or cardiac output.
99 ricle (HR, 10.5; P=0.0429), and higher right atrial pressure (HR, 1.3 per 1 mm Hg; P=0.0016).
100 % CI, 1.76-12.88; P=0.0021), increased right atrial pressure (HR, 1.34; 95% CI, 0.95-1.90; P=0.0992 a
101                                        Intra-atrial pressure (IAP) was increased in steps of 2 to 3 c
102 ilure, interventions to reduce elevated left atrial pressure improve symptoms and reduce the risk of
103 more accurate than P(PAO) in estimating left atrial pressure in cardiac surgical patients.
104 er echocardiographic variables and mean left atrial pressure in group A patients.
105  describe the design of REDUCE Elevated Left Atrial Pressure in Heart Failure (REDUCE LAP-HF I), the
106 ial of a device-based therapy to reduce left atrial pressure in HFpEF.
107                     The REDUCe Elevated Left Atrial Pressure in Patients with Heart Failure (REDUCE L
108        REDUCE LAP-HF I (Reduce Elevated Left Atrial Pressure in Patients With Heart Failure) was a ph
109      A paradoxical inspiratory rise in right atrial pressure (in contrast to the normal fall during i
110                                        Right atrial pressure increased by 2.5 +/- 1.8 mm Hg (P=0.002)
111                                         Left atrial pressure increases at exercise with an average up
112 for the first time that an increase in intra-atrial pressure increases the rate and organization of w
113 ignificant interaction between SCr and right atrial pressures (interaction P<0.0001); increased SCr b
114  pulmonary arterial pressure, left and right atrial pressures, intracranial pressure, body temperatur
115 xhibiting directionally opposite firing when atrial pressure is perturbed.
116               The effect of raising the left atrial pressure (LAP) acutely above 25 mmHg (to cause pu
117  of the mitral valve increased the mean left atrial pressure (LAP) by approximately 2.6 and 3.8 mmHg,
118                                The mean left atrial pressure (LAP) correlated well with the septal si
119 s have limitations in the prediction of left atrial pressure (LAP) in patients with mitral valve dise
120 ing pressures to direct measurements of left atrial pressure (LAP) via catheterization in 100 patient
121 ally clinically significant increase in left atrial pressure (LAP).
122  Hemodynamic parameters, including mean left atrial pressure (LAP, in mm Hg), mean pulmonary artery p
123 ith higher pulmonary arterial and mean right atrial pressures, lower cardiac index, and impaired exer
124  best predicted death in patients with right atrial pressure &lt;10 mm Hg.
125 c peptide (BNP), and hemodynamics with right atrial pressure &lt;8 mm Hg and cardiac index >2.5 mg/kg/mi
126 ion of right ventricular function with right atrial pressure &lt;8 mm Hg and cardiac index >2.5 to 3.0 l
127 ascular resistance, cardiac index, and right atrial pressure may be used to stratify risk of death.
128  primary pulmonary hypertension (PPH), right atrial pressure may exceed left atrial pressure during e
129 ators of RV-PV function (i.e., resting right atrial pressure, mean PA pressure, pulmonary vascular re
130 Active treatment significantly lowered right atrial pressure, mean pulmonary artery pressure, and pul
131 ft atrium was directly catheterized for left atrial pressure measurements.
132  that a mechanical approach to reducing left atrial pressure might be effective in HFPEF.
133 vival on univariate analysis were mean right atrial pressure (mRAP) (p < 0.0001), mPAP (p = 0.034), R
134 a Cox proportional hazards model: mean right atrial pressure (mRAP) more than or equal to 14 mm Hg an
135 renal vascular conductance (RVC), mean right atrial pressure (mRAtP), urine flow, glomerular filtrati
136                            Increases in left atrial pressure of 5 mmHg increased RAR activity from 3.
137  approach to AF under conditions of elevated atrial pressure or volume.
138 ing mean arterial pressure, heart rate, left atrial pressure, or left ventricular dP/dt.
139 relaxation, LV end-diastolic pressure, right atrial pressure, or pulmonary pressure in either patient
140 dynamic values, including pulmonary and left atrial pressures, or intrathoracic impedance, which is r
141 rotein C pathway, in rats subjected to acute atrial pressure overload caused by aortic banding.
142 e dP/dt (p < 0.05), an increase in mean left atrial pressure (p < 0.05) and a prolongation of tau, th
143 s of DHEA-S were associated with lower right atrial pressure (P = 0.02) and pulmonary vascular resist
144 e, mean pulmonary artery pressure, and right atrial pressure (P</=0.001, 0.003, 0.017, and 0.031, res
145 0.001) in association with decreases in left atrial pressure (P<0.001), peripheral resistance (P=0.01
146 his study compared a prediction of mean left atrial pressure (P(LA)) ascertained by Doppler echocardi
147 ssociation functional class (P=0.009), right atrial pressure (P=0.037), and stroke volume (P=0.043).
148 ostoperative central venous (P<0.001) common atrial pressure (P=0.042), inotropic score (P<0.001), an
149 tio, 5.3; P<0.001), whereas changes in right atrial pressure (P=0.36) and pulmonary capillary wedge p
150                                Elevated left atrial pressure, particularly during exercise, is a key
151  blockade and the absence of change in right atrial pressure, persistent atrial tachycardia caused AR
152 sion pressure is not thought to reflect left atrial pressure (Pla) when alveolar pressure (PA) exceed
153 peak systolic blood pressure (Ps), mean left atrial pressure (PLA), and Doppler-derived IVRT (IVRTDop
154 te wedge pressure (Pcw) from transmural left atrial pressure (Platm) by elevating pleural pressure an
155 nd EDPR gradients, in conjunction with right atrial pressure, provide Doppler estimates of pulmonary
156 vice that allows shunting to reduce the left atrial pressure provides clinical and hemodynamic benefi
157                  In controls subjects, right atrial pressure, pulmonary arterial pressure, and pulmon
158 3 months (p=0.035), with no changes in right atrial pressure, pulmonary arterial pressure, or pulmona
159                 LV pressure and volume, left atrial pressure, pulmonary artery pressure and flow, and
160  Mean pulmonary arterial pressure, mean left atrial pressure, pulmonary vascular resistance, and stat
161 t (=pulmonary capillary wedge pressure-right atrial pressure; r=0.67; P=0.003), suggesting relief of
162  pressure (PCWP) (32 to 14 mm Hg), and right atrial pressure (RA) (19 to 9 mm Hg).
163   Diastolic dysfunction was defined as right atrial pressure (RAP) >/=15 mm Hg (right ventricular [RV
164 nal pediatric hemodynamic cutpoints of right atrial pressure (RAP) >12 mm Hg or pulmonary capillary w
165 ographic and invasive measures of mean right atrial pressure (RAP) (r = 0.863; p < 0.0001), systolic
166                               Although right atrial pressure (RAP) and pulmonary capillary wedge pres
167  AF was associated with an increase of right atrial pressure (RAP) and right atrial dilatation.
168 dictive value of coronary fistulae and right atrial pressure (RAP) score (comprising the tricuspid va
169 data exists as to the relation of mean right atrial pressure (RAP) to Doppler parameters of right atr
170             Measurements included mean right atrial pressure (RAP), mean pulmonary arterial pressure
171 dynamic determinants included elevated right atrial pressure, reduced pulmonary artery pulse pressure
172 ue disease, functional class III, mean right atrial pressure, resting systolic blood pressure and hea
173 stolic filling was determined by a simulated atrial pressure source that was either constant or varie
174 terial blood pressure (to 30-35 mm Hg), both atrial pressures, systemic oxygen consumption (by 35%),
175       Cyanotic patients had lower mean right atrial pressures than the control subjects (4+/-3 versus
176 ling wave indexes had the best relation with atrial pressure, the highest being for systolic filling
177  filling have been applied to determine left atrial pressure, their accuracy has been limited by the
178 ventricular end-diastolic diameter, and left atrial pressure vs. left ventricular end-diastolic diame
179 <0.01) per 10-mL/m(2) decrease and for right atrial pressure was 1.05 (95% confidence interval, 1.02-
180                         Average initial left atrial pressure was 31 mm Hg.
181 gnificant increase in activity when the left atrial pressure was acutely elevated in both intact and
182                                     The left atrial pressure was directly related to the E/A ratio (r
183               In the 123 patients, the right atrial pressure was less than 10 mm Hg in 49 patients, t
184                                         Left atrial pressure was measured with a micromanometer cathe
185                               Exercise right atrial pressure was the highest in MI+DD followed by MI-
186 ivity (% change in RSNA/mm Hg change in left atrial pressure) was markedly attenuated after PL (pre,
187 l sympathetic nerve activity/mmHg mean right atrial pressure) was measured during isotonic saline vol
188 itation, low cardiac index, and raised right atrial pressure were associated with poor survival for b
189 e renal failure or elevated postbypass right atrial pressure were at increased risk for early mortali
190 fusion pressure, systemic pressure, and left atrial pressure were continuously monitored, electronica
191 fusion pressure, systemic pressure, and left atrial pressure were continuously monitored, electronica
192 stance, stroke volume index (SVI), and right atrial pressure were independently associated with death
193 l pressure, arterial blood pressure and left atrial pressure were measured in paralysed, anaesthetize
194                                SVI and right atrial pressure were the hemodynamic variables that were
195                             Aortic and right atrial pressures were measured with micromanometers.
196                             Aortic and right atrial pressures were measured with micromanometers.
197 re (pulmonary capillary wedge pressure-right atrial pressure), which reflects LV preload independent
198       The dissociation between PAOP and left atrial pressure, while left ventricular and -diastolic d
199                        Nitrite reduced right atrial pressures, with no effect on cardiac output or st
200 ary perfusion pressure (CPP) (aortic - right atrial pressure) without epinephrine (A-CPR 21 +/- 8 mm
201  p = 0.577; Q = 14.64, I(2) = 79.51%), right atrial pressure (WMD: 1.01 mmHg, 95%CI: -0.93, 2.96, p =

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