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1 n heart rate, mean aortic pressure, or right atrial pressure.
2 -LVDP) was used as a surrogate for mean left atrial pressure.
3 d elevated minimal LV pressure and mean left atrial pressure.
4 gh increases in cardiac output (.Q) and left atrial pressure.
5 using capacity of carbon monoxide, and right atrial pressure.
6 ion of right ventricular function, and right atrial pressure.
7 result of a lowering of early diastolic left atrial pressure.
8 on end-diastolic arterial pressure and right atrial pressure.
9 +/- 6 mm Hg, p < 0.002) and increased right atrial pressure (10 +/- 6 vs. 8 +/- 4 mm Hg, p < 0.05).
10 roximately 2-fold greater increases in right atrial pressure (10+/-4 versus 6+/-3 mm Hg; P=0.02), pul
11 us 5.1+/-1.9 L/min; P=0.01), increased right atrial pressure (12+/-5 versus 4+/-1 mm Hg; P=0.0002), a
12 and diuretics administered to decrease right atrial pressure (18+/-2 to 7+/-1 mm Hg), pulmonary wedge
13 0 mg groups, respectively; P<0.05) and right atrial pressure (-2.0+/-0.4, -3.7+/-0.4, and -3.5+/-0.4
14 44 mm Hg to 12 +/- 6 mm Hg; p = 0.007), left atrial pressure (29 +/- 11 mm Hg to 20 +/- 8 mm Hg; p =
17 There were significant decreases in right atrial pressure (9+/-1 to 7+/-1 mm Hg, p < 0.01 by ANOVA
18 ular resistance, significantly higher common atrial pressure after Fontan and significantly lower pos
19 tic venous flow dynamics relate best to mean atrial pressure and can be used clinically to estimate m
21 creased after endotoxin infusion, while left atrial pressure and left ventricular end-diastolic diame
23 mic hypotension occurred with a fall in left atrial pressure and little change in left ventricular vo
25 yncopal patients presented with higher right atrial pressure and lower cardiac outputs with lower sur
26 , mean pulmonary artery pressure, mean right atrial pressure and mean left atrial pressure) at baseli
27 nges in hemodynamic values (except for right atrial pressure and mean pulmonary artery pressure) were
29 ases in cardiac output and decreases in left atrial pressure and peripheral resistance but without el
30 elow Veq, we used a servomotor to clamp left atrial pressure and produce nonfilling diastoles, allowi
31 re, pulmonary vascular resistance, and right atrial pressure and provided incremental prognostic valu
32 ormed during exercise, included higher right atrial pressure and pulmonary capillary wedge pressure a
34 ed for the simultaneous measurement of right atrial pressure and renal sympathetic nerve activity.
36 ure (27% to 39% decrease by 6 h), mean right atrial pressure and systemic vascular resistance, along
37 PV showed positive correlation between intra-atrial pressure and the number of waves emanating from t
44 sion pressure (diastolic; aortic minus right atrial pressure) and cerebral perfusion pressure (mean a
46 aortic pressure, in the left atrium to left atrial pressure, and in all heart chambers to a decrease
47 rized by a decrease in cardiac output, right atrial pressure, and left ventricular (LV) end-diastolic
48 ikely because of marked increase in the left atrial pressure, and preload reduction may unmask the re
49 exercise tolerance, functional class, right atrial pressure, and vasodilator response to adenosine.
50 rdiac output; mean aortic, pulmonary or left atrial pressures; and peak positive and negative first d
51 edural monitoring included vital signs, left atrial pressure, arterial blood pressure, cerebral perfu
52 urements of left ventricular inflow and left atrial pressures, ascending aortic pressure, thermodilut
55 re, mean right atrial pressure and mean left atrial pressure) at baseline, during 60 min of atrial fi
56 reload was abruptly reduced by clamping left atrial pressure between 0 and -2 mm Hg in seven open-che
58 hypotension or perioperative changes in left atrial pressure, brain natriuretic peptide levels, lacti
62 High pulmonary vascular resistance and right atrial pressure by invasive hemodynamic measurements wer
63 scular resistance (by 29%; P=0.03) and right atrial pressure (by 40%; P=0.007), but with only modest
64 measured lung lymph flow after raising left atrial pressure (by inflating a balloon) in sheep that w
66 invasively derived measurements, mean right atrial pressure cardiac index, and mixed venous oxygen s
67 of 6MWD, pulmonary arterial pressure, right atrial pressure, cardiac index and pulmonary vascular re
68 th Organization functional class, mean right atrial pressure, cardiac index, and mixed venous oxygen
69 and mean systemic arterial pressures (MAP), atrial pressures, cardiac output, and arterial blood gas
71 domized to the PAC arm (n = 194), only right atrial pressure correlated weakly with baseline SCr (r =
72 aging has been proposed in which early left atrial pressure could be low in the aged heart but rise
74 .1 +/- 1.1 to 13.2 +/- 1.3 mm Hg; mean right atrial pressure decreased from 10.9 +/- 1 to 4.8 +/- 1.0
75 whereas diastolic left ventricular and right atrial pressures decreased significantly and proportiona
76 e data indicate that PAOP overestimates left atrial pressure during endotoxin shock, making it an ina
77 is feasible, seems to be safe, reduces left atrial pressure during exercise, and could be a new stra
78 (PPH), right atrial pressure may exceed left atrial pressure during exercise, resulting in a right-to
80 rial contraction (a wave); point 2, the left atrial pressure during the start of ventricular systole;
81 ial filling (v wave); point 4, earliest left atrial pressure during ventricular filling; and the line
82 mean difference between the aortic and right atrial pressures during the release phase of chest compr
83 terogeneous clinical syndrome, elevated left atrial pressure-either at rest or with exertion-is a com
89 complex but characterised by increased left atrial pressure, especially during exertion, which might
90 dge pressure fell from 31 to 18 mm Hg, right atrial pressure from 15 to 8 mm Hg, and SVR from 1,780 t
91 ters also improved with a reduction in right atrial pressure from 22 mm Hg at baseline, to 9 mm Hg an
92 Hg to 11.5+/-3.7 mm Hg; p < .001), and right atrial pressures (from 7.3+/-2.5 mm Hg to 9.8+/-2.5 mm H
96 ic variables such as cardiac index and right atrial pressure have consistently been associated with s
97 leaflets, which equals left ventricular-left atrial pressure, have been proposed to explain this dyna
100 % CI, 1.76-12.88; P=0.0021), increased right atrial pressure (HR, 1.34; 95% CI, 0.95-1.90; P=0.0992 a
102 ilure, interventions to reduce elevated left atrial pressure improve symptoms and reduce the risk of
105 describe the design of REDUCE Elevated Left Atrial Pressure in Heart Failure (REDUCE LAP-HF I), the
109 A paradoxical inspiratory rise in right atrial pressure (in contrast to the normal fall during i
112 for the first time that an increase in intra-atrial pressure increases the rate and organization of w
113 ignificant interaction between SCr and right atrial pressures (interaction P<0.0001); increased SCr b
114 pulmonary arterial pressure, left and right atrial pressures, intracranial pressure, body temperatur
117 of the mitral valve increased the mean left atrial pressure (LAP) by approximately 2.6 and 3.8 mmHg,
119 s have limitations in the prediction of left atrial pressure (LAP) in patients with mitral valve dise
120 ing pressures to direct measurements of left atrial pressure (LAP) via catheterization in 100 patient
122 Hemodynamic parameters, including mean left atrial pressure (LAP, in mm Hg), mean pulmonary artery p
123 ith higher pulmonary arterial and mean right atrial pressures, lower cardiac index, and impaired exer
125 c peptide (BNP), and hemodynamics with right atrial pressure <8 mm Hg and cardiac index >2.5 mg/kg/mi
126 ion of right ventricular function with right atrial pressure <8 mm Hg and cardiac index >2.5 to 3.0 l
127 ascular resistance, cardiac index, and right atrial pressure may be used to stratify risk of death.
128 primary pulmonary hypertension (PPH), right atrial pressure may exceed left atrial pressure during e
129 ators of RV-PV function (i.e., resting right atrial pressure, mean PA pressure, pulmonary vascular re
130 Active treatment significantly lowered right atrial pressure, mean pulmonary artery pressure, and pul
133 vival on univariate analysis were mean right atrial pressure (mRAP) (p < 0.0001), mPAP (p = 0.034), R
134 a Cox proportional hazards model: mean right atrial pressure (mRAP) more than or equal to 14 mm Hg an
135 renal vascular conductance (RVC), mean right atrial pressure (mRAtP), urine flow, glomerular filtrati
139 relaxation, LV end-diastolic pressure, right atrial pressure, or pulmonary pressure in either patient
140 dynamic values, including pulmonary and left atrial pressures, or intrathoracic impedance, which is r
142 e dP/dt (p < 0.05), an increase in mean left atrial pressure (p < 0.05) and a prolongation of tau, th
143 s of DHEA-S were associated with lower right atrial pressure (P = 0.02) and pulmonary vascular resist
144 e, mean pulmonary artery pressure, and right atrial pressure (P</=0.001, 0.003, 0.017, and 0.031, res
145 0.001) in association with decreases in left atrial pressure (P<0.001), peripheral resistance (P=0.01
146 his study compared a prediction of mean left atrial pressure (P(LA)) ascertained by Doppler echocardi
147 ssociation functional class (P=0.009), right atrial pressure (P=0.037), and stroke volume (P=0.043).
148 ostoperative central venous (P<0.001) common atrial pressure (P=0.042), inotropic score (P<0.001), an
149 tio, 5.3; P<0.001), whereas changes in right atrial pressure (P=0.36) and pulmonary capillary wedge p
151 blockade and the absence of change in right atrial pressure, persistent atrial tachycardia caused AR
152 sion pressure is not thought to reflect left atrial pressure (Pla) when alveolar pressure (PA) exceed
153 peak systolic blood pressure (Ps), mean left atrial pressure (PLA), and Doppler-derived IVRT (IVRTDop
154 te wedge pressure (Pcw) from transmural left atrial pressure (Platm) by elevating pleural pressure an
155 nd EDPR gradients, in conjunction with right atrial pressure, provide Doppler estimates of pulmonary
156 vice that allows shunting to reduce the left atrial pressure provides clinical and hemodynamic benefi
158 3 months (p=0.035), with no changes in right atrial pressure, pulmonary arterial pressure, or pulmona
160 Mean pulmonary arterial pressure, mean left atrial pressure, pulmonary vascular resistance, and stat
161 t (=pulmonary capillary wedge pressure-right atrial pressure; r=0.67; P=0.003), suggesting relief of
163 Diastolic dysfunction was defined as right atrial pressure (RAP) >/=15 mm Hg (right ventricular [RV
164 nal pediatric hemodynamic cutpoints of right atrial pressure (RAP) >12 mm Hg or pulmonary capillary w
165 ographic and invasive measures of mean right atrial pressure (RAP) (r = 0.863; p < 0.0001), systolic
168 dictive value of coronary fistulae and right atrial pressure (RAP) score (comprising the tricuspid va
169 data exists as to the relation of mean right atrial pressure (RAP) to Doppler parameters of right atr
171 dynamic determinants included elevated right atrial pressure, reduced pulmonary artery pulse pressure
172 ue disease, functional class III, mean right atrial pressure, resting systolic blood pressure and hea
173 stolic filling was determined by a simulated atrial pressure source that was either constant or varie
174 terial blood pressure (to 30-35 mm Hg), both atrial pressures, systemic oxygen consumption (by 35%),
176 ling wave indexes had the best relation with atrial pressure, the highest being for systolic filling
177 filling have been applied to determine left atrial pressure, their accuracy has been limited by the
178 ventricular end-diastolic diameter, and left atrial pressure vs. left ventricular end-diastolic diame
179 <0.01) per 10-mL/m(2) decrease and for right atrial pressure was 1.05 (95% confidence interval, 1.02-
181 gnificant increase in activity when the left atrial pressure was acutely elevated in both intact and
186 ivity (% change in RSNA/mm Hg change in left atrial pressure) was markedly attenuated after PL (pre,
187 l sympathetic nerve activity/mmHg mean right atrial pressure) was measured during isotonic saline vol
188 itation, low cardiac index, and raised right atrial pressure were associated with poor survival for b
189 e renal failure or elevated postbypass right atrial pressure were at increased risk for early mortali
190 fusion pressure, systemic pressure, and left atrial pressure were continuously monitored, electronica
191 fusion pressure, systemic pressure, and left atrial pressure were continuously monitored, electronica
192 stance, stroke volume index (SVI), and right atrial pressure were independently associated with death
193 l pressure, arterial blood pressure and left atrial pressure were measured in paralysed, anaesthetize
197 re (pulmonary capillary wedge pressure-right atrial pressure), which reflects LV preload independent
200 ary perfusion pressure (CPP) (aortic - right atrial pressure) without epinephrine (A-CPR 21 +/- 8 mm
201 p = 0.577; Q = 14.64, I(2) = 79.51%), right atrial pressure (WMD: 1.01 mmHg, 95%CI: -0.93, 2.96, p =
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