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1 alvular aortic stenosis, and 2.8% for primum atrial septal defect.
2 One infant had an atrial septal defect.
3 undergo surgical repair of an ostium primum atrial septal defect.
4 22 patients and 10 control subjects with an atrial septal defect.
5 er responding to surgical closure of a large atrial septal defect.
6 gical approaches for simple lesions, such as atrial septal defect.
7 nt with tricuspid atresia and large secundum atrial septal defect.
8 in a large family with dominantly inherited atrial septal defect.
9 n of the great arteries, and ventricular and atrial septal defects.
10 ccluder for percutaneous closure of secundum atrial septal defects.
11 heart syndrome but inversely associated with atrial septal defects.
12 fety and efficacy of device closure of large atrial septal defects.
13 interaction affect muscular ventricular and atrial septal defects.
14 rmed on 9 kindreds with familial CHD, 4 with atrial septal defects, 2 with patent ductus arteriosus,
15 ich ventricular septal defects (22/47, 47%), atrial septal defects (20/47, 43%), patent ductus arteri
16 Principal features of IART circuits were atrial septal defect (4 patients), atriotomy (3 patients
18 5+/-14 years) with PHT and associated CHD (9 atrial septal defect, 7 ventricular septal defect, 4 tra
19 tion of the great arteries (8%), ventricular/atrial septal defects (8%), left ventricular outflow obs
22 including aortic coarctation, ventricular or atrial septal defect, abnormal mitral valve, aortic root
23 ere at highest risk of IE in comparison with atrial septal defects (adjusted rate ratio, 95% confiden
24 phisms that confer greater susceptibility to atrial septal defect and atrioventricular septal defects
25 epilepticus, congestive heart failure due to atrial septal defect and hypernatremic dehydration due t
26 monary stenosis in 2 and subaortic membrane, atrial septal defect and mitral regurgitation in 1 patie
27 re moderate to large VSD in two patients and atrial septal defect and unlimited differential diagnosi
29 iac malformations, including ventricular and atrial septal defects and a thin ventricular myocardium.
30 iations between carbon monoxide and isolated atrial septal defects and between ozone and isolated ven
32 onsistent in magnitude were detected between atrial septal defects and bromoform (aOR = 1.56; 95% CI:
35 domain, p.G115W, was identified in familial atrial septal defects and demonstrated decreased transac
36 tion factor, were reported to cause secundum atrial septal defects and result in atrioventricular (AV
37 al defect device for use in multifenestrated atrial septal defects and the fenestrated atrial septal
39 n autosomal dominant skeletal dysplasia with atrial septal defects, and familial total anomalous pulm
40 D (ie, excluding ventricular septal defects, atrial septal defects, and pulmonary valve stenosis) occ
42 were positively associated with the risks of atrial septal defect (aORs ranging from 1.29 to 2.17), p
43 these pollutants and daily risks of secundum atrial septal defect, aortic coarctation, hypoplastic le
46 defects (aRR, 0.85; 95% CI, 0.75-0.96), and atrial septal defects (aRR, 0.82; 95% CI, 0.69-0.95) but
47 370), left-sided lesions (n = 160), secundum atrial septal defect (ASD) (n = 71), and Ebstein's malfo
48 ht atrium to the right ventricle and both an atrial septal defect (ASD) and a ventricular septal defe
49 r (RV) volume after transcatheter closure of atrial septal defect (ASD) and to investigate factors th
53 x 10(-)(7)) with the risk of ostium secundum atrial septal defect (ASD) in the discovery cohort (N =
58 MYH6 can cause an autosomal dominant form of atrial septal defect (ASD), whereas others have identifi
60 owing isolated procedures: device closure of atrial septal defect (ASD); device closure of patent duc
61 use of permanent synthetic implants to close atrial septal defects (ASD) and patent foramen ovale (PF
62 form transcatheter closures of secundum type atrial septal defects (ASD) and to assess ASD size and c
63 m in the embryo and persistent ostium primum atrial septal defects (ASD) in approximately 20% of adul
67 fourth generation buttoned device in dosing atrial septal defects (ASDs) and to test the hypothesis
69 generations of closure devices for secundum atrial septal defects (ASDs) at a single institution.
72 rnelia de Lange syndrome-associated secundum atrial septal defects (ASDs) caused by NIPBL mutations,
74 was to quantitate shunt flow volumes through atrial septal defects (ASDs) in a chronic animal model w
75 spected anomalous pulmonary veins (APVs) and atrial septal defects (ASDs) using fast cine magnetic re
78 went surgical correction of an ostium primum atrial septal defect at our institution at a mean age of
79 en <15 years) with large, isolated, secundum atrial septal defects (balloon-stretched diameter >/=34
80 septal defect (SVASD) differs from secundum atrial septal defect by its atrial septal location and i
81 ients undergoing the surgical creation of an atrial septal defect by the Blalock-Hanlon technique.
82 ven patients (6%) in the pitavastatin group (atrial septal defect, chronic obstructive pulmonary dise
83 alve procedure (9%), patent foramen ovale or atrial septal defect closure (23%), cardiac metastasecto
85 and frequent concomitant procedures such as atrial septal defect closure, arrhythmia surgery (the ma
86 enomic analyses of probands with heterotaxy, atrial septal defects, conotruncal, and left ventricular
89 ed Amplatzer Ductal Occluder, the cribriform atrial septal defect device for use in multifenestrated
91 ar that evaluated the safety and efficacy of atrial septal defect devices, patent ductus occluders, a
92 oninvasive procedure to create or enlarge an atrial septal defect for the treatment of neonates with
94 obese women had significantly higher ORs for atrial septal defects, hypoplastic left heart syndrome,
95 tachycardia (AT) after SR-CHD were studied (atrial septal defect in 6, tetralogy of Fallot in 4, and
97 l lymphopenia, intermittent neutropenia, and atrial septal defects in 3 members of a consanguineous k
98 pathophysiology, treatment, and outcomes of atrial septal defects in children and adult patients in
100 .I263V mutation in TLL1 was identified in an atrial septal defects kindred and is predicted to affect
101 ts with repaired "simple" lesions such as an atrial septal defect may not have normal survival if the
103 patients underwent repair of a secundum-type atrial septal defect (n=12) or patent foramen ovale (n=5
104 microm in aerodynamic diameter and isolated atrial septal defects (odds ratio = 2.27, 95% confidence
106 uals who had had transcatheter closure of an atrial septal defect or patent foramen ovale to assess h
107 eatinine > or =2 mg/dL (OR 4.7), restrictive atrial septal defect (OR 2.7) and, in staged surgery, at
108 e an embryologic basis for the prevalence of atrial septal defects (ostium primum and secundum), vent
110 uch as the transcatheter closure of secundum atrial septal defects, patent foramen ovale, patent duct
112 ting surgery for congenital heart disease (9 atrial septal defect repair, 4 Fontan, 2 Mustard, 2 Senn
113 eak (prior Mustard procedure), closure of an atrial septal defect, repair of partial anomalous pulmon
114 t of the TARP syndrome, talipes equinovarus, atrial septal defect, Robin sequence, and persistent lef
115 tations, but not a mutation causing secundum atrial septal defects (S52F), demonstrated impaired prot
118 g man with IAA associated with sinus venosus atrial septal defect (SVD) and partial anomalous pulmona
120 of great vessels, ventricular septal defect, atrial septal defect, tetralogy of Fallot, coarctation o
121 g these tools, specifically in patients with atrial septal defects, tetralogy of Fallot, single ventr
122 mid-to-long-term data for device closure of atrial septal defects, the incidence of late cardiac ero
123 ccluder for Percutaneous Closure of Secundum Atrial Septal Defects; this study was approved by the Fo
124 reports the pilot in vivo study to create an atrial septal defect through the use of extracardiac app
125 ting on children after closure of a secundum atrial septal defect through the use of surgery (n=26) o
126 y the type of defect, from a low of 4.7% for atrial septal defects to a high of 28% for hypoplastic l
127 2 patients without prior diagnosis of PFO or atrial septal defect undergoing surgery at the Cleveland
130 ry for 2 cohorts: (1) all patients with CHD (atrial septal defect, ventricular septal defect, tetralo
136 s decreased between 1990 and 2011 except for atrial septal defects, which increased significantly.
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