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1 Ao junction can be a frequent source of left atrial tachycardia.
2 as used to simulate the site of origin of an atrial tachycardia.
3 of this study was freedom from recurrent AF/atrial tachycardia.
4 t ablation was performed for recurrent AF or atrial tachycardia.
5 adaptive molecular and cellular response to atrial tachycardia.
6 ), 49% of the patients remained free from AF/atrial tachycardia.
7 to induce paroxysmal atrial fibrillation or atrial tachycardia.
8 rial fibrillation, atrial flutter, and focal atrial tachycardia.
9 reentry, Wolff-Parkinson-White syndrome, and atrial tachycardia.
10 roxysmal atrial fibrillation, and paroxysmal atrial tachycardia.
11 dent right atrial reentry (n=7), and 1 focal atrial tachycardia.
12 nvolving the left atrium compared with right atrial tachycardias.
13 successful in eliminating left versus right atrial tachycardias.
14 These foci usually induce irregular atrial tachycardias.
15 aches for atrial flutters and macroreentrant atrial tachycardias.
16 in 26 (96%) of 27 (95% CI 81% to 100%) right atrial tachycardias.
17 ugh low-voltage regions and aids ablation of atrial tachycardias.
18 nd this drove 30% (7/23) of our postablation atrial tachycardias.
19 sence of LVA (<0.5 mV) and inducible regular atrial tachycardias.
20 lter recordings; and (4) higher incidence of atrial tachycardia (15% versus 41%, P=0.02) and atrial f
22 ilure (3%), liver abscesses (3%), paroxysmal atrial tachycardia (3%), thoracic pain (3%), upper gastr
24 s (age 49+/-16 years, 9 females) with either atrial tachycardia (6 patients) or atrial fibrillation t
25 tory of successfully ablated incessant focal atrial tachycardia 64+/-36 months prior, and 20 healthy
28 imilar to tachycardia cycle length; 2) focal atrial tachycardia: a P-A interval < or =-20 ms; and 3)
31 chronic tachycardias have been reported with atrial tachycardias, accessory pathway reciprocating tac
32 he AF substrates tested, including sustained atrial tachycardia/AF itself, enhanced post-RFA atrial t
35 with AF and 16 patients with atrial flutter/atrial tachycardia) after the initial procedure during a
42 A-A-V response was observed in all cases of atrial tachycardia and simulated atrial tachycardia, eve
43 % and 5%, respectively, also had episodes of atrial tachycardia and supraventricular tachyarrhythmia.
45 istry designed to document the prevalence of atrial tachycardia and/or fibrillation (AT/AF) of any du
46 identify the anatomic origin of focal right atrial tachycardias and to define their relation with th
47 prising atrial fibrillation, atrial flutter, atrial tachycardia, and supraventricular tachyarrhythmia
48 ntricular overdrive pacing was diagnostic of atrial tachycardia, and this maneuver could be applied t
49 nd long-term recurrence of AF/atrial flutter/atrial tachycardia are significantly lower using general
51 T to adenosine can immediately differentiate atrial tachycardia arising from a focal source from that
52 lly abnormal atrial tissue and mechanisms of atrial tachycardia (AT) after mitral valve (MV) surgery.
53 es recurrence of atrial fibrillation (AF) or atrial tachycardia (AT) after radiofrequency catheter ab
55 r radiofrequency catheter ablation (RFCA) of atrial tachycardia (AT) arising from the coronary sinus
56 g criteria unequivocally differentiate focal atrial tachycardia (AT) caused by microreentry, triggere
60 re TAVR: paroxysmal atrial fibrillation (AF)/atrial tachycardia (AT) in 28, advanced atrioventricular
61 eparate focal from atypical macro-re-entrant atrial tachycardia (AT) on the electrocardiogram (ECG).
62 lar fibrillation (VF) preceded by paroxysmal atrial tachycardia (AT) or atrial fibrillation (AF)-in p
63 Here, we assess the efficacy of optogenetic atrial tachycardia (AT) termination in human hearts usin
64 this study was to determine the mechanism of atrial tachycardia (AT) that occurs after ablation of at
66 he feasibility of optogenetic termination of atrial tachycardia (AT), comparing two different illumin
67 adenosine has mechanism-specific effects on atrial tachycardia (AT), such that adenosine terminates
68 atrial fibrillation (AF) or other sustained atrial tachycardia (AT), verified by monthly visits, amb
72 onal reciprocating tachycardia (PJRT; n=12), atrial tachycardia (AT; n=53), and inappropriate sinus t
73 wavefronts, but this is often difficult for atrial tachycardias (AT) after ablation of atrial fibril
74 activation mapping of reentrant scar-related atrial tachycardias (AT) allows efficient radiofrequency
76 onal linear ablation lines in the setting of atrial tachycardias (AT) in patients with persistent AF
80 y of CRT loss increased, the contribution of atrial tachycardia/atrial fibrillation and SAV/PAV to th
82 ostics explained 55.8% of pacing loss: 30.6% atrial tachycardia/atrial fibrillation; 16.6% premature
83 ary outcome was symptomatic recurrence of AF/atrial tachycardia/atrial flutter lasting >30 seconds, d
86 jective of this study is to describe complex atrial tachycardias (ATs) that occur after this approach
87 iled noninvasive localization of right-sided atrial tachycardia before radiofrequency catheter ablati
88 change in right atrial pressure, persistent atrial tachycardia caused ARP and ERP to fall by > 10%.
90 urity of the extrastimulus and time to first atrial tachycardia complex were directly correlated (R=0
92 matic or asymptomatic AF, atrial flutter, or atrial tachycardia), detected by either scheduled or uns
94 ll cases of atrial tachycardia and simulated atrial tachycardia, even in the presence of dual atriove
98 CFAE arm had a higher incidence of organized atrial tachycardia/flutter (24/33 [73%] versus 11/31 [35
100 euvers, it can be difficult to differentiate atrial tachycardia from other forms of paroxysmal suprav
101 ecurrence of symptomatic AF, atrial flutter, atrial tachycardia (HR, 0.56 [95% CI, 0.33-0.95]; P = .0
104 FAE ablation did not improve freedom from AF/atrial tachycardia in patients with paroxysmal or persis
105 prove the overall rate of freedom from AF or atrial tachycardia in patients with persistent AF (OR, 1
106 itive and specific for the identification of atrial tachycardia in the electrophysiology laboratory.
107 The most common finding was rapid, irregular atrial tachycardias in the region of the pulmonary veins
110 ce had atrial arrhythmias, including AFL and atrial tachycardia, indicating that Pitx2 haploinsuffici
111 change included atrial premature complexes, atrial tachycardia, interpolated ventricular premature c
115 ined as an episode of AF, atrial flutter, or atrial tachycardia lasting >30 seconds after a 3-month b
116 acterize the circuit of macroreentrant right atrial tachycardia (MacroAT) in patients after surgical
118 Recent studies have suggested that right atrial tachycardias may also have a characteristic anato
119 romic reciprocating tachycardia (n = 43), 3) atrial tachycardia (n = 19) and 4) atrial tachycardia si
121 n 47 cases: macroreentrant (n = 25) or focal atrial tachycardia (n = 8), atrioventricular nodal reent
122 l fibrillation (n=13), atrial flutter (n=4), atrial tachycardia (n=3), idiopathic ventricular tachyca
123 A trend toward a higher incidence of left atrial tachycardia occurrence in the wide antral circumf
124 that approximately two thirds of focal right atrial tachycardias occurring in the absence of structur
125 erm procedural outcome or freedom from AF or atrial tachycardia (odds ratio [OR], 0.80; 95% confidenc
127 77 reablation group patients were free of AF/atrial tachycardia on no AADs; in contrast, in the AAD g
128 thermore, 2 of 7 ablation dogs had sustained atrial tachycardias, one of which was successfully ablat
133 ree of target arrhythmia was achieved in all atrial tachycardia patients; however, a nontargeted vent
137 ar follow-up, freedom from AF/atrial flutter/atrial tachycardia recurrence was significantly higher i
138 er rate of consecutive procedures because of atrial tachycardia recurrences (P = 0.003; HR, 1.71; 95%
139 ivation pattern of localized reentry (LR) in atrial tachycardia remains incompletely understood.
140 (control, n=16) and 3 canine AF-models: (1) atrial tachycardia remodeling (ATR; n=16) induced by atr
141 0.10.1+/-0.00.5+/-0.40.3+/-0.1 ATR indicates atrial tachycardia remodeling; CAF, chronic atrial fibri
143 schemes of atrial flutter and macroreentrant atrial tachycardias, reviews the technique of radiofrequ
144 = 43), 3) atrial tachycardia (n = 19) and 4) atrial tachycardia simulated by demand atrial pacing in
145 0 (28%) of 35 consecutive patients with left atrial tachycardia, the arrhythmia originated from the M
146 ntribute to the positive inotropy and sinus (atrial) tachycardia traditionally attributed to chronic,
147 es including hypertrophic cardiomyopathy and atrial tachycardia, tumor predisposition, and cognitive
149 ibrillation, occurrence of atrial flutter or atrial tachycardia, use of antiarrhythmic drugs, or repe
154 verall, the rate of recurrent arrhythmia (AF/atrial tachycardia) was 21% at 1 year, 11% between 1 and
161 aroxysmal atrial fibrillation and paroxysmal atrial tachycardia, which suggests that simultaneous sym
162 Five patients were documented to have focal atrial tachycardia, which was mapped to the coronary sin
163 from atrial fibrillation, atrial flutter, or atrial tachycardia while not receiving antiarrhythmic me
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