ライフサイエンスコーパス: atrioventricular conduction

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1 ncreased left ventricle diameter and delayed atrioventricular conduction.

2 (-15)) with PR interval, a marker of cardiac atrioventricular conduction.

3 ges and congenital lack of macrophages delay atrioventricular conduction.

4 ed interatrial conduction without disturbing atrioventricular conduction.

5 us associated with cardiac malformations and atrioventricular conduction abnormalities was mapped to

6 on of NKX2-5 is linked to septal defects and atrioventricular conduction abnormalities, early lethali

7 TAVI is associated with atrioventricular-conduction abnormalities requiring PPM

8 tion during arrhythmias, including accessory atrioventricular conduction activation.

9 onduction system abnormalities with aberrant atrioventricular conduction and an increased rate of arr

10 imaging, to assess Ca(2+) handling, revealed atrioventricular conduction and excitation-contraction w

11 ein kinase (AMPK), most notably, accelerated atrioventricular conduction and impaired AMPK activation

12 Galphai2 overexpression suppressed baseline atrioventricular conduction and slowed the heart rate du

13 065 patients with sinus-node disease, intact atrioventricular conduction, and a normal QRS interval t

14 ophysiology in humans, including heart rate, atrioventricular conduction, and ventricular repolarizat

15 We conclude that while CVMs controlling atrioventricular conduction are distributed with a peak

16 Rapid atrial rates and intrinsic atrioventricular conduction, as well as congenital mitra

17 ough the inhomogeneous and extremely complex atrioventricular conduction axis is much deeper than it

18 e of permanent pacemaker (PPM) placement for atrioventricular conduction block (AV block) after opera

19 Atrioventricular conduction block and arrhythmias caused

20 Atrioventricular conduction block is a complication of o

21 bserved change in rhythm including temporary atrioventricular conduction block or tachycardia termina

22 ay contribute to paravalvular regurgitation, atrioventricular conduction block, and mitral or coronar

23 onduction-system disease (sinus bradycardia, atrioventricular conduction block, or atrial arrhythmias

24 itation (Wolff-Parkinson-White syndrome) and atrioventricular conduction block.

25 ate heart rate variability, sinus pause, and atrioventricular conduction block.

26 e that most commonly involves some degree of atrioventricular conduction blockade.

27 dysfunction manifested by atropine-sensitive atrioventricular conduction blocks and bradycardia that

28 owing seizures, SENP2-deficient mice develop atrioventricular conduction blocks and cardiac asystole.

29 manifestations of neonatal lupus, comprising atrioventricular conduction defects and cardiomyopathy,

30 he cardiac transcription factor Nkx2-5 cause atrioventricular conduction defects in humans by unknown

31 entricular arrhythmias, atrial fibrillation, atrioventricular conduction defects, and death by 4 mont

32 ands and family members was characterized by atrioventricular conduction disturbances (61% and 44%, r

33 of the homeodomain, were not associated with atrioventricular conduction disturbances, and were not f

34 d for patients with underlying sinus node or atrioventricular conduction disturbances, for patients w

35 Patients with DM develop atrioventricular conduction disturbances, the principal

36 5 in the myocardium leads to prolongation of atrioventricular conduction, due in part to activation o

37 1) diverse atrial rhythms and mechanisms of atrioventricular conduction during 2 degrees AVB; 2) jun

38 ate, only a few genes for familial sinus and atrioventricular conduction dysfunction are known, and t

39 RATIONALE: Familial sinus node and atrioventricular conduction dysfunction is a rare disord

40 ular response since many drugs that decrease atrioventricular conduction have negative inotropic effe

41 praventricular tachycardia mechanism was 1:1 atrioventricular conduction in 22 patients and supravent

42 Transplantation of EECTs in vivo restored atrioventricular conduction in a rat model of complete h

43 of the carboxyl zinc-finger of Gata6 alters atrioventricular conduction in postnatal life as assesse

44 tionally selective effects on heart rate and atrioventricular conduction in the rat.

45 one made during pacing to measure changes in atrioventricular conduction (P-R interval) independent o

46 ysiological testing demonstrated alternative atrioventricular conduction pathways consistent with WPW

47 l [CI] = 1.3 to 5.1; p trend <0.001), slower atrioventricular conduction (PR interval +7.2 ms, 95% CI

48 electrocardiographic measures of heart rate, atrioventricular conduction (PR interval), ventricular r

49 entricular rate (p < 0.001) and reduced both atrioventricular conduction (PR segment-p = 0.02; PR int

50 f new pacemaker features designed to promote atrioventricular conduction, preserve ventricular conduc

51 2 +/- 1.7 RF applications) without affecting atrioventricular conduction properties.

52 Maximal changes in atrioventricular conduction resulted from more rostral s

53 d synergize to activate transcription in the atrioventricular conduction system.

54 he inner AV canal muscle layer at e.d. 13.5, atrioventricular conduction through the canal was abolis

55 motropic state of the heart (P-R interval or atrioventricular conduction time) in response to selecti

56 and aortic bodies caused an increase in the atrioventricular conduction time.

57 We demonstrate that a ring of atrioventricular conduction tissue develops at 40 hours

58 notch1b are necessary for the development of atrioventricular conduction tissue.

59 Overall, the ratio of the change in atrioventricular conduction to the change in heart rate

60 lrhodopsin-2-expressing macrophages improves atrioventricular conduction, whereas conditional deletio

61 He is remembered for his insight into atrioventricular conduction, which is as valid today as

62 Delivery of 130 Gy caused disturbance of atrioventricular conduction with transition into complet

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