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1 e atrioventricular block at the level of the atrioventricular node).
2 the remaining 7 for pathway proximity to the atrioventricular node.
3 observed in the conduction properties of the atrioventricular node.
4  with antibody-associated disease beyond the atrioventricular node.
5  tract, atrial septum, and in sinoatrial and atrioventricular node.
6 embryos appear to lack the primordium of the atrioventricular node.
7 n, we focused on genetic modification of the atrioventricular node.
8 t half maximum of 10 mm was delivered to the atrioventricular node.
9   Bradycardia was induced by ablation of the atrioventricular node.
10  is virtually absent from the sinoatrial and atrioventricular nodes.
11 raphy or the discovery of the sinoatrial and atrioventricular nodes.
12 a position midway between the sinoatrial and atrioventricular nodes.
13    Fourteen piglets 8 weeks of age underwent atrioventricular node ablation and were paced from eithe
14     Patients with cAVB were identified by an atrioventricular node ablation or diagnosis of third-deg
15 G telemetry devices underwent radiofrequency atrioventricular node ablation to produce AVB.
16 s, beam positions, and particle numbers) for atrioventricular node ablation was conducted.
17                               Radiofrequency atrioventricular node ablation was performed in patients
18                                              Atrioventricular node ablation with pacemaker insertion
19 gation, we tested the feasibility of in vivo atrioventricular node ablation, in Langendorff-perfused
20                                        After atrioventricular node ablation, the right ventricular ap
21 ial fibrillation should be considered before atrioventricular node ablation.
22 o undergo either pulmonary-vein isolation or atrioventricular-node ablation with biventricular pacing
23     Pulmonary-vein isolation was superior to atrioventricular-node ablation with biventricular pacing
24 dema in another; in the group that underwent atrioventricular-node ablation with biventricular pacing
25 nths (60, vs. 82 in the group that underwent atrioventricular-node ablation with biventricular pacing
26 t pulmonary-vein isolation, and 40 underwent atrioventricular-node ablation with biventricular pacing
27 vagal denervation of the atria and sinus and atrioventricular nodes also denervated the ventricles.
28 ck-down of Prox1 restored the anatomy of the atrioventricular node and His-Purkinje network both of w
29 rug therapy, radio-frequency ablation of the atrioventricular node and implantation of a permanent pa
30 d all patients who underwent ablation of the atrioventricular node and implantation of a permanent pa
31               Radiofrequency ablation of the atrioventricular node and permanent pacing are used for
32 l of the ventricular rate by ablation of the atrioventricular node and permanent pacing does not adve
33 uired for optimal impulse propagation in the atrioventricular node and stabilizes the level of the co
34 f the heart that become the sinoatrial node, atrioventricular node, and bundle of His.
35                                          The atrioventricular node artery passed close to the coronar
36                           Distances from the atrioventricular node artery to the coronary sinus, from
37 coordinate the spread of excitation from the atrioventricular node (AV node) to ventricular myocardiu
38 lt mice display conduction defects below the atrioventricular node (AVN) as determined by invasive el
39                Posteroseptal ablation of the atrioventricular node (AVN) has been proposed as a means
40                                          The atrioventricular node (AVN) is the only normal electrica
41 ng of electrical activity in rabbit isolated atrioventricular node (AVN) preparations that, in the ma
42                          PGVS applied to the atrioventricular node (AVN) slows nodal conduction.
43  supply to the sinuatrial node (SAN) and the atrioventricular node (AVN) with 64-section computed tom
44 lude injury of the sinoatrial node (SAN) and atrioventricular node (AVN), requiring cardiac rhythm ma
45  of interconnected structures, including the atrioventricular node (AVN), the central connection poin
46 ince the first description of the anatomical atrioventricular nodes (AVNs), a large number of studies
47 mitral) AT that can be overcome by injecting atrioventricular node blockers and signal averaging, res
48 lopment and postnatal function of the murine atrioventricular node by promoting cell-cycle exit of sp
49 pagation of excitation in the sinoatrial and atrioventricular nodes by shortening the space constant
50            cGi overexpression in the porcine atrioventricular node causes physiologically relevant he
51 ntaining nuclear integrity in heart disease, atrioventricular node cells lacking lamin A exhibit incr
52 tly higher expressed in the right atrium and atrioventricular node compared with left ventricle (P=5.
53            Ablation did not adversely affect atrioventricular node conduction, nor did it facilitate
54                          Diagnosing atypical atrioventricular node-dependent long RP supraventricular
55 nduction system disease, including sinus and atrioventricular node dysfunction.
56            Ibutilide prolonged the antegrade atrioventricular node effective refractory period (ERP)
57 enesis and for maturation and maintenance of atrioventricular node function throughout life.
58    Ibutilide prolonged refractoriness of the atrioventricular node, His-Purkinje system, and AP.
59 The magnetic catheter was used to ablate the atrioventricular node in 4 animals and to perform linear
60 th atrial fibrillation after ablation of the atrioventricular node is similar to expected survival in
61 s complication of NLS is inflammation of the atrioventricular node leading to congenital heart block
62 ing of Tbx5(del/+) mice suggested a specific atrioventricular node maturation failure.
63  This case indicates that although the adult atrioventricular node may be relatively resistant to the
64 via an accessory pathway (n = 4) or via twin atrioventricular nodes (n = 4), ventricular tachycardia
65 n40(-)/connexin45(+) cells is missing in the atrioventricular node of Nkx2-5 heterozygous KO mice.
66 result from abnormalities of the sinus node, atrioventricular node, or the His-Purkinje system.
67 ardia is useful for differentiating atypical atrioventricular node re-entrant tachycardia (AVNRT) fro
68 rant junctional tachycardia (JT) and typical atrioventricular node re-entry tachycardia (AVNRT).
69     The study was performed to document that atrioventricular node reciprocating tachycardia (AVNRT)
70 ntricular (AV) reentry (AVRT; n=59), typical atrioventricular node reentry (AVNRT; n=82), atypical AV
71 ac conduction-system defects that affect the atrioventricular node, resulting in bradycardia.
72 atients with double firing properties of the atrioventricular node, separating these into discrete ty
73 atrioventricular bundle during the period of atrioventricular node specification, which results in fe
74            The function of the sinus and the atrioventricular node was preserved in most patients, bu
75 ate electrical conduction through the distal atrioventricular node, where conducting cells densely in
76 n based on this mechanism could occur in the atrioventricular node, where multiple branching is struc
77 d channel, subtype 4 staining in the compact atrioventricular node with some retention of hyperpolari

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