ライフサイエンスコーパス: attachment level

1 ssue formation, probing depths, and clinical attachment levels).

2 H loss were noted at the 5.1-5.4 mm clinical attachment level.

3 probing, visible plaque, probing depth, and attachment level.

4 eth lost during follow-up had worse baseline attachment level.

5 ing depth, bleeding on probing, and clinical attachment level.

6 XCL8 were positively related to the clinical attachment level.

7 cells, but NanI-producing strains had higher attachment levels.

8 PD), and vertical (VAL) and horizontal (HAL) attachment levels.

9 lative vertical (RVAL) and horizontal (RHAL) attachment levels.

10 gatively correlated, with MARGI-reported RNA attachment levels.

11 mm and 1.2 +/- 1.0 mm), and gain of clinical attachment level (2.1 +/- 1.0 mm and 3.0 +/- 1.0 mm), wh

12 nths: 1) probing depth; 2) relative clinical attachment level; 3) GR; 4) thickness of KT (TKT); and 5

13 depth; (2) probing depth (PD); (3) clinical attachment level; (4) width of keratinized tissue (wKT);

14 his discovery GWAS of interproximal clinical attachment level-a measure of lifetime periodontal tissu

15 is was measured by the percent of sites with attachment level (AL) > or = 3 mm.

16 E treatments, respectively, were similar for attachment level (AL; 6.2 +/- 1.8 mm and 6.1 +/- 1.7 mm)

17 Baseline attachment level and behavioral factors were significant

18 1 and 2 had significantly different clinical attachment level and gingival recession changes by the e

19 gy (AAP) criteria using measures of clinical attachment level and probing depth (PD).

20 e secondary outcomes were change in clinical attachment level and proportion of sites with bleeding o

21 cate a possible correlation between clinical attachment level and salivary IgG (P = 0.07; r(2) = 0.08

22 use (versus none) was associated with higher attachment levels and more teeth only among participants

23 GI], bleeding on probing [BOP], and clinical attachment level) and photographs from 53 participants (

24 e outcome (clinically determined periodontal attachment level) and predictors (self-reported dental s

25 ions included: 1) probing depth, 2) clinical attachment level, and 3) oral radiographs for alveolar c

26 ncluding periodontal probing depth, clinical attachment level, and bleeding on probing, as well as cr

27 al examination using probing depth, clinical attachment level, and bleeding on probing.

28 arameters related to probing depth, clinical attachment level, and bone loss around teeth increased t

29 included changes in probing depth, clinical attachment level, and defect fill as revealed by reentry

30 ed by measurement of probing depth, clinical attachment level, and extent and severity of disease.

31 by criteria based on probing depth, clinical attachment level, and extent and severity of periodontal

32 by criteria based on probing depth, clinical attachment level, and extent and severity of periodontal

33 Probing depth, clinical attachment level, and gingival and plaque indices in eac

34 , glycated hemoglobin, sampled-site clinical attachment level, and gingival index (P <0.05).

35 Recession height, probing depth, clinical attachment level, and keratinized tissue width and thick

36 8-OHdG, MDA, probing depth, clinical attachment level, and percentage of sites bleeding on pr

37 width, width of keratinized tissue, clinical attachment level, and probing depth, measured to the nea

38 width, width of keratinized tissue, clinical attachment level, and probing depth.

39 on the percentage of root coverage, probing attachment level, and the amount of keratinized tissue i

40 s were measured, and probing depth, clinical attachment levels, and bleeding on probing were used to

41 vements in probing depth reduction, clinical attachment levels, and bone fill have been demonstrated.

42 in the percentage of root coverage, probing attachment levels, and increased keratinized tissue.

43 rrelated with probing pocket depth, clinical attachment levels, and RANKL concentrations in GCF.

44 Preoperative probing depths, attachment levels, and transoperative bone measurements

45 nges in probing depths and relative clinical attachment levels appeared to reflect changes in the und

46 ue index, gingival index, probing depth, and attachment level at six sites per tooth.

47 arameters, including probing depth, clinical attachment level, bleeding on probing, and gingival and

48 nation included probing depth (PD), clinical attachment level, bleeding on probing, and plaque index.

49 inical examinations including probing depth, attachment level, bleeding on probing, and root caries r

50 obtained first, with probing depth, clinical attachment level, bleeding on probing, plaque index scor

51 er, there were no significant differences in attachment levels, bleeding upon probing, or extent of s

52 vely related with probing depth and clinical attachment level; blood glucose was related only to blee

53 ism and periodontal lesions (i.e., decreased attachment level, bone loss, tooth mobility/migration, a

54 ith probing depth (PD) >/= 5 mm and clinical attachment level (CAL) >/= 3 mm were randomly divided in

55 with probing depth (PD) >/=5 mm and clinical attachment level (CAL) >/=2 mm at the same site.

56 defined as percentage of cases with clinical attachment level (CAL) >/=2.7 mm and linear bone growth

57 0.05) of mean PD (1.4 +/- 0.7 mm), clinical attachment level (CAL) (1.3 +/- 0.8 mm), and BOP (33.4%

58 und between viral load and moderate clinical attachment level (CAL) (rho = 0.638, P <0.05), CD4+ nadi

59 ) of at least 5 mm and 2 mm loss of clinical attachment level (CAL) after initial non-surgical therap

60 ed change in probing depth (PD) and clinical attachment level (CAL) after the therapy.

61 this study is to report changes in clinical attachment level (CAL) and bone fill of periodontal IBDs

62 Clinical attachment level (CAL) and bone height (radiographic or

63 s to assess the association between clinical attachment level (CAL) and bone mineral density (BMD) at

64 changes in clinical factors such as clinical attachment level (CAL) and gingival recession (GR).

65 alivary cytokines was found between clinical attachment level (CAL) and IL-21 (P = 0.02).

66 ncluded per-subject mean changes in clinical attachment level (CAL) and probing depth (PD) from basel

67 rimary efficacy parameters included clinical attachment level (CAL) and probing depth (PD).

68 ater mean gain in relative vertical clinical attachment level (CAL) and relative horizontal CAL were

69 dontal destruction was assessed via clinical attachment level (CAL) and the number of missing teeth.

70 related with probing depth (PD) and clinical attachment level (CAL) as well as with GCF levels of TNF

71 CF was inversely associated with clinical attachment level (CAL) at baseline before therapy in all

72 periodontal probing depth (PD) and clinical attachment level (CAL) at six sites per tooth.

73 y significant gain (P <0.05) in the clinical attachment level (CAL) between baseline and 6 months in

74 Root coverage (RC) and clinical attachment level (CAL) did not differ significantly betw

75 cket Probing Depth reduction (PPD), Clinical Attachment Level (CAL) gain and radiographic bone gain w

76 , probing depth (PD) reduction, and clinical attachment level (CAL) gain in both statin and placebo/n

77 obing depth (PD) reduction and mean clinical attachment level (CAL) gain was greater in the ALN group

78 n terms of probing depth reduction, clinical attachment level (CAL) gain, and bone level (clinical an

79 st improvements of recession depth, clinical attachment level (CAL) gain, and keratinized tissue (KT)

80 t postoperative probing depth (PD), clinical attachment level (CAL) gain, or radiographic defect reso

81 cally significant PD reductions and clinical attachment level (CAL) gains > or =2 mm compared to 56%

82 The clinical attachment level (CAL) measurements were stable througho

83 bing (BOP), probing depth (PD), and clinical attachment level (CAL) of all teeth were examined.

84 al of GTR treated sites in terms of clinical attachment level (CAL) stability and tooth loss.

85 d probing depth (PD) with a gain in clinical attachment level (CAL) to treat advanced periodontal dis

86 on probing, probing depth (PD), and clinical attachment level (CAL) were carried out in four sites pe

87 ding on probing, probing depth, and clinical attachment level (CAL) were determined.

88 on probing, probing depth (PD), and clinical attachment level (CAL) were measured at baseline and 3 a

89 bing (BOP), probing depth (PD), and clinical attachment level (CAL) were performed.

90 Probing depth (PD) and clinical attachment level (CAL) were recorded at baseline and aft

91 rs including probing depth (PD) and clinical attachment level (CAL) were recorded.

92 severity of disease measured as the clinical attachment level (CAL) when healthy and diseased groups

93 dex (GI), 2) probing depth (PD), 3) clinical attachment level (CAL), 4) radiologic defect depth, and

94 acterized using probing depth (PD), clinical attachment level (CAL), alveolar crest height (ACH), and

95 and 12 months, probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP) we

96 improvements in probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP) we

97 val index (GI), probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP), w

98 ental calculus, probing depth (PD), clinical attachment level (CAL), and bleeding on probing (BOP).

99 d examiners measured probing depth, clinical attachment level (CAL), and bleeding on probing on all t

100 Probing depth (PD), clinical attachment level (CAL), and bone PD were recorded.

101 Clinical parameters, including PD, clinical attachment level (CAL), and BOP, and GCF IL-1beta levels

102 ), probing depth (PD), relative GR, clinical attachment level (CAL), and cervical lesion height cover

103 bleeding index, probing depth (PD), clinical attachment level (CAL), and gingival marginal level, inc

104 val index (GI), probing depth (PD), clinical attachment level (CAL), and HbA1c level, of all particip

105 g index (mSBI), probing depth (PD), clinical attachment level (CAL), and IBD depth, were recorded at

106 Probing depth, clinical attachment level (CAL), and keratinized gingival width (

107 were plaque index, gingival index, clinical attachment level (CAL), and PD.

108 evaluated were probing depth (PD), clinical attachment level (CAL), and percentage of sites with PD

109 Measurements of probing depth (PD), clinical attachment level (CAL), and radiographic BDA were done a

110 ents, including probing depth (PD), clinical attachment level (CAL), and radiographs, were used to cl

111 ne examination, probing depth (PD), clinical attachment level (CAL), and recession (REC) were measure

112 ements included probing depth (PD), clinical attachment level (CAL), and recession.

113 duced probing depth (PD), increased clinical attachment level (CAL), and reduced bleeding on probing

114 gingival bleeding on probing (BOP), clinical attachment level (CAL), and surfaces with plaque were re

115 REC, probing depth, clinical attachment level (CAL), and width of keratinized tissue

116 mean changes in probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and g

117 eters including probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), and p

118 Probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

119 Probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

120 Full-mouth probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

121 asurements were probing depth (PD), clinical attachment level (CAL), bleeding on probing (BOP), gingi

122 analyzed, including probing depth, clinical attachment level (CAL), bleeding on probing, and percent

123 Probing depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque

124 At baseline, probing depth (PD), clinical attachment level (CAL), bleeding on probing, and plaque

125 Vertical recession depth (VRD), clinical attachment level (CAL), clinical probing depth (PD), and

126 d tissue (KTw), probing depth (PD), clinical attachment level (CAL), clinician rating of color and te

127 gingival index, probing depth (PD), clinical attachment level (CAL), defect base level (DBL), and cre

128 intrabony defects was the change in clinical attachment level (CAL), for furcations the change in hor

129 nd post-therapy probing depth (PD), clinical attachment level (CAL), gingival recession (GR), and rad

130 cal parameters (probing depth (PD), clinical attachment level (CAL), gingival recession (GR), gingiva

131 l parameters, with the exception of clinical attachment level (CAL), had significantly (P <0.05) impr

132 ere recession depth, probing depth, clinical attachment level (CAL), height of keratinized tissue (wK

133 ameters such as probing depth (PD), clinical attachment level (CAL), IBD depth, and percentage defect

134 ters, including probing depth (PD), clinical attachment level (CAL), IBD depth, and percentage defect

135 meters, such as probing depth (PD), clinical attachment level (CAL), intrabony defect depth, and perc

136 ical recession (VR), probing depth, clinical attachment level (CAL), keratinized tissue width (KTW),

137 que index (PI), probing depth (PD), clinical attachment level (CAL), modified gingival index (GI), an

138 ters, including probing depth (PD), clinical attachment level (CAL), plaque index, and gingival index

139 ed by measuring probing depth (PD), clinical attachment level (CAL), plaque, bleeding on probing, vis

140 seline and 6 and 12 months included clinical attachment level (CAL), probing depth (PD), and bleeding

141 The clinical attachment level (CAL), probing depth (PD), and gingival

142 using alveolar crest height (ACH), clinical attachment level (CAL), probing depth (PD), and percenta

143 cal gingival recession depth (VRD), clinical attachment level (CAL), probing depth (PD), and width of

144 ts meta-analyses were conducted for clinical attachment level (CAL), probing depth (PD), bleeding on

145 res of alveolar crest height (ACH), clinical attachment level (CAL), probing depth (PD), gingival ble

146 width of keratinized gingiva (KW), clinical attachment level (CAL), probing depth (PD), gingival ind

147 width of keratinized tissue (WKT), clinical attachment level (CAL), probing depth (PD), plaque index

148 width of keratinized tissue (WKT), clinical attachment level (CAL), probing depth (PD), plaque index

149 Probing depth (PD), clinical attachment level (CAL), recession (REC), and tooth mobil

150 d 6 months were probing depth (PD), clinical attachment level (CAL), recession height (RH), width of

151 Gain in clinical attachment level (CAL), reduction in probing depth (PD),

152 (GTR) support substantial gains in clinical attachment level (CAL), reductions in probing depth (PD)

153 on measures of probing depth (PD), clinical attachment level (CAL), the radiographic pattern and ext

154 ng on probing (BOP), probing depth, clinical attachment level (CAL), waist circumference (WC), hsCRP,

155 dex (mSBI), probing depth (PD), and clinical attachment level (CAL), were recorded at baseline (befor

156 aque index, probing depth (PD), and clinical attachment level (CAL), were recorded at baseline.

157 bing (BOP), probing depth (PD), and clinical attachment level (CAL), were recorded before the treatme

158 x, plaque index, probing depth, and clinical attachment level (CAL), were recorded.

159 bing (BOP), probing depth (PD), and clinical attachment level (CAL), which were recorded at baseline

160 index (PI), probing depth (PD), and clinical attachment level (CAL).

161 dex (mSBI), probing depth (PD), and clinical attachment level (CAL).

162 que score, bleeding on probing, and clinical attachment level (CAL).

163 The primary outcome variable was clinical attachment level (CAL).

164 bing (BOP), probing depth (PD), and clinical attachment level (CAL).

165 , mean probing depth (PD), and mean clinical attachment level (CAL).

166 bing depth, gingival recession, and clinical attachment level (CAL).

167 (GI); 4) probing depth (PD); and 5) clinical attachment level (CAL).

168 dex (mSBI), probing depth (PD), and clinical attachment level (CAL).

169 (FMBS), gingival recession, PD, and clinical attachment level (CAL).

170 margin position (GMP), and relative clinical attachment level (CAL).

171 th including probing depth (PD) and clinical attachment level (CAL).

172 zed tissue, probing depth (PD), and clinical attachment level (CAL).

173 index (GI), probing depth (PD), and clinical attachment level (CAL).

174 depth (PD), gingival recession, and clinical attachment level (CAL).

175 in probing depth (PD) and gains in clinical attachment level (CAL).

176 fferences in probing depth (PD) and clinical attachment level (CAL).

177 dex (mSBI), probing depth (PD), and clinical attachment level (CAL).

178 BOP); 3) probing depth (PD); and 4) clinical attachment level (CAL).

179 (BS); 3) probing depth (PD); and 4) clinical attachment level (CAL).

180 e primary outcome was the change in clinical attachment level (CAL).

181 n in probing depth (PD) and gain in clinical attachment level (CAL).

182 f the 12-month follow-up period: 1) clinical attachment level (CAL); 2) presence or absence of mobili

183 periodontal probing depth (PD); 2) clinical attachment level (CAL); 3) gingival recession; and 4) pe

184 t (NSPT): 1) probing depth (PD); 2) clinical attachment level (CAL); 3) plaque index (PI); 4) gingiva

185 x (mSBI); 3) probing depth (PD); 4) clinical attachment level (CAL); and 5) IBD depth.

186 Effectiveness measurements included clinical attachment levels (CAL) and gingival recession (GR) meas

187 Clinical attachment levels (CAL) and N-benzoyl-dl-arginine-2-naph

188 Probing depths (PD) and clinical attachment levels (CAL) were measured before treatment a

189 measures of probing depth (PD) and clinical attachment levels (CAL) with 95% confidence intervals (C

190 d upon probing pocket depths (PPD), clinical attachment levels (CAL), and whole-mouth gingival bleedi

191 Conventional probing depths, clinical attachment levels (CAL), gingival index (GI), and plaque

192 (probing depths [PDs] and vertical clinical attachment level [CAL-V]) and standardized radiographs w

193 index, probing depth [PD], vertical clinical attachment level [CAL-V]) were available for patients at

194 aque index, probing depth [PD], and clinical attachment level [CAL]) were recorded at baseline and 2

195 aque index, probing depth [PD], and clinical attachment level [CAL]) were recorded at baseline, 2 mon

196 aque index, probing depth [PD], and clinical attachment level [CAL]) were recorded at baseline, 3 mon

197 index [GI], probing depth [PD], and clinical attachment level [CAL]).

198 erapy clinical (probing depth [PD], clinical attachment level [CAL], and gingival recession [GR]) and

199 que index [PI], probing depth [PD], clinical attachment level [CAL], and percentage of sites with ble

200 l measurements (probing depth [PD], clinical attachment level [CAL], recession, mobility, plaque inde

201 Clinical attachment levels (CALs; primary outcome), probing depth

202 points examined included changes in clinical attachment level, changes in bone level/fill, and probin

203 mple size >500, half-mouth minimum, clinical attachment level) containing prevalence data on destruct

204 and Drug Administration (FDA), for clinical attachment level (DeltaCAL), mean change in radiographic

205 ive trait of CP (mean interproximal clinical attachment level determined by full-mouth periodontal ex

206 Probing depth (PD), clinical attachment level, dichotomous presence or absence of sup

207 h improvement in probing depths and clinical attachment levels following non-surgical and various sur

208 ding on probing, probing depth, and relative attachment level from a customized probing stent.

209 bsolute change in probing depth and clinical attachment level from baseline to 1-year follow-up.

210 uating plaque index, probing depth, clinical attachment level, furcation involvement, bleeding on pro

211 p had statistically significant open probing attachment level gain (95% confidence level, 3.18 to 4.3

212 Outcomes were clinical attachment level gain (CALg) and probing depth reduction

213 measure was absolute change in open probing attachment level gain and percentage defect resolution f

214 , gingival index, plaque index, and clinical attachment level gain at 90 days, demonstrating effectiv

215 eduction in deep probing depths and clinical attachment level gain in three of four specimens, in add

216 pared to 13.8% by SRP alone), and a clinical attachment level gain of 1.16 mm (compared to 0.80 mm by

217 actor-BB (rhPDGF-BB) led to greater clinical attachment level gain of approximately 1 mm compared to

218 greater probing depth reduction and clinical attachment level gain than the control group after 3 and

219 The mechanism for this attachment level gain to the non-root planed tooth is no

220 was 2.97 mm (95% CI: 2.38-3.56 mm), clinical attachment level gain was 1.65 mm (95% CI: 1.17-2.13 mm)

221 Probing depth reduction and clinical attachment level gain were obtained in three-fourths of

222 ical (RVCAL) and horizontal (RHCAL) clinical attachment level gain were shown to be greater in the AL

223 However, HBF, recession, clinical attachment level gain, and probing depth reduction at fu

224 luded probing depth (PD) reduction, clinical attachment level gain, bleeding on probing (BOP) reducti

225 arameters: probing depth reduction, clinical attachment level gain, bleeding on probing reduction, an

226 A, the probing depth reductions and clinical attachment level gains found in each group were not stat

227 Attachment level gains were significantly improved from

228 us was determined by probing depth, clinical attachment level, gingival bleeding index, and the prese

229 tal parameters (probing depth [PD], clinical attachment level, gingival index, bleeding on probing, a

230 ecession width, probing depth (PD), clinical attachment level, gingival index, plaque index, patient

231 imary outcome, bleeding on probing, clinical attachment level, gingival recession, interleukin-1beta,

232 s, and 6 months were probing depth, clinical attachment level, GR height, width of keratinized gingiv

233 mm, bleeding on probing [BOP], and clinical attachment level >/= 2 mm) and one healthy site (PD </=

234 rproximal probing depth >/=6 mm and clinical attachment level >/=4 mm were randomized into two groups

235 exhibiting probing depth >/=4 mm or clinical attachment level >/=4 mm.

236 rs of periodontal probing depth and clinical attachment level, have been proven in multiple clinical

237 ent in the following: 1) horizontal clinical attachment level (HCAL); 2) vertical clinical attachment

238 ve correlations with probing depth, clinical attachment level, IL-1beta, and IL-6.

239 Mean PD reduction (P = 0.002) and attachment level improvements (P = 0.012) were significa

240 rapy in reducing probing depth and improving attachment levels in smokers with moderate to advanced a

241 subjects were stratified based on gender and attachment level into two groups.

242 However, if gain in clinical attachment level is considered the gold standard for non

243 , and 12 months included recession, clinical attachment level, keratinized tissue height, and plaque

244 bing depths (mean: 3.18 mm; SD: 0.59 mm) and attachment levels (mean: 3.93 mm; SD: 0.19) at 6 years w

245 s that are heavily influenced by probing and attachment level measurements alone.

246 g index, probing depths, recession, clinical attachment level, mobility, furcation involvement, numbe

247 g index, probing depths, recession, clinical attachment level, mobility, furcation involvement, numbe

248 dental cleaning; and baseline factors (worst attachment level of > or =7 mm, not flossing, a molar to

249 (2.42 mm vs. 1.32 mm) and a gain in clinical attachment level of 22% versus 7% (1.58 mm vs. 0.42 mm)

250 mm with mean post-surgical PD of 3.17 mm and attachment levels of 4.05 mm, based on subject means.

251 rease, but no significant effects on probing attachment level or percentage of root surface coverage.

252 efect coverage, keratinized tissue, clinical attachment level, or clinical healing for treatment of r

253 ontal probing depth (P <0.05), mean clinical attachment level (P <0.05), and sites with bleeding on p

254 l groups both for PD (P = 0.03) and clinical attachment level (P = 0.11).

255 rs recorded included probing depth, clinical attachment level, plaque index, and gingival index.

256 Probing depth, clinical attachment level, plaque index, and papilla bleeding ind

257 ates to determine the level of importance of attachment level, probing depth, furcation involvement,

258 elists determined the level of importance of attachment level, probing depth, mobility, plaque, infla

259 significant improvements in clinical status (attachment level, probing depth, plaque, gingivitis, and

260 Attachment levels, probing depths, plaque levels, degree

261 Attachment levels, probing depths, plaque levels, degree

262 to teeth with an initial reduced periodontal attachment level, provided adequate periodontal treatmen

263 ding index, probing depth (PD), and relative attachment level (RAL) were recorded at baseline and 3,

264 g index (mSBI), probing depth (PD), relative attachment level (RAL), and gingival marginal level (GML

265 ight measured apico-coronally (KG), relative attachment level (RAL), probing depths (PD), bleeding on

266 Relative clinical attachment level (RCAL) and probing depth (PD) measures

267 uch as probing depth (PD), relative clinical attachment level (rCAL), and gingival margin level (GML)

268 3) probing depth (PD); 4) relative clinical attachment level (rCAL); and 5) gingival marginal level

269 (PI), gingival index (GI), relative clinical attachment levels (RCAL) to the stent, recession depth (

270 ngival index (GI), PD, and relative clinical attachment levels (rCALs) was done at baseline and at 4

271 ional periodontal measures, such as clinical attachment level, recession, and bleeding on probing.

272 eeding index, mobility, furcations, clinical attachment level, recession, and periodontal status did

273 Relative clinical attachment level (relative CAL) and probing depth (PD) m

274 achment level (RVAL) and relative horizontal attachment level (RHAL), and intrabony defect depth were

275 index, probing depth (PD), relative vertical attachment level (RVAL) and relative horizontal attachme

276 ve vertical and relative horizontal clinical attachment level [rvCAL and rhCAL], intrabony defect dep

277 n specific combinations of probing depth and attachment level values.

278 tical probing depth (VPD), vertical clinical attachment level (VCAL), gingival recession, and horizon

279 ttachment level (HCAL); 2) vertical clinical attachment level (VCAL); 3) probing depth (PD); and 4) l

280 Gain in clinical attachment level was 2.8 +/- 0.6 mm in OF and 2.3 +/- 0.

281 bing (BOP), probing depth (PD), and clinical attachment level, was performed at all PMT visits during

282 on probing, probing depth (PD), and clinical attachment level were carried out in four sites per toot

283 amount of keratinized gingiva, and clinical attachment level were evaluated.

284 ng, suppuration, probing depth, and clinical attachment level were measured at all teeth present.

285 Probing depth and clinical attachment level were measured at baseline and 1 year.

286 ameters including probing depth and clinical attachment level were measured, and a gingival tissue sa

287 ingival indices, probing depth, and clinical attachment level were measured.

288 probing depth, keratinized tissue (KT), and attachment level were recorded at baseline and 8 months

289 robing depth (PD), recession depth (RD), and attachment level were recorded.

290 ion, keratinized tissue, probing depths, and attachment levels were made initially, at 3 months, and

291 bleeding scores, probing depths and clinical attachment levels were observed for both test and contro

292 Probing depth and clinical attachment levels were obtained.

293 ing on probing, probing depths, and clinical attachment levels were performed at baseline, after 6 we

294 Gingival and periodontal pocket depth and attachment levels were recorded.

295 gingival index, probing depth, and clinical attachment level) were recorded.

296 x, plaque index, probing depth, and clinical attachment level, were recorded before GCF collection.

297 x, plaque index, probing depth, and clinical attachment level, were recorded.

298 ctions in probing depths and improvements in attachment levels while producing little or no increase

299 ctions in probing depths and improvements in attachment levels while producing no detectable recessio

300 ded probing depth, recession depth, clinical attachment level, width of keratinized tissue, mobility,