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1 Twenty-four cases were identified (14% attack rate).
2 orted by 79 of 702 household contacts (11.3% attack rate).
3 pirical estimates of H3N2's population level attack rate.
4 on coronal caries incidence, increment, and attack rate.
5 ith a significant reduction in the secondary attack rate.
6 g of future hospitalizations and the overall attack rate.
7 accinated population contributed to the high attack rate.
8 s develop clinical prion disease with a 100% attack rate.
9 has an advantage that increases with the T6S attack rate.
10 tion, which is consistent with 2009 pandemic attack rates.
11 significant public health impact given high attack rates.
12 ce age-specific and population-level illness attack rates.
13 tivated vaccine in a year with low influenza attack rates.
14 imilarity of mimics can thus lead to greater attack rates.
15 re it was associated with high infection and attack rates.
16 ke location-specific projections of epidemic attack rates.
17 ation of antigenic variants, and high annual attack rates.
18 age and effectiveness) to estimate influenza attack rates.
19 We calculated attack rates.
20 ts in the lower Danube valley and Bucharest (attack rate 12.4/100000 people) with a seroprevalence of
24 e through contacts at 2 day care facilities (attack rate, 88.1 per 1000); and (3) 15 (18%) were young
25 ed Fisher's exact test to compare unadjusted attack rates according to dose status and years since re
27 ng abnormal prion protein resulted in a 100% attack rate after its inoculation in transgenic mice ove
29 he primary agent of infection included a low attack rate among incompletely vaccinated children and a
31 e only significant predictor of illness; the attack rate among people who consumed oysters was 29 per
35 Based on return on investment and higher attack rates among children, we recommend prioritizing c
37 During the measles epidemic of 1989-1991, attack rates among nonwhite children <5 years of age wer
41 nd indirect VE was assessed by comparing the attack rates among unvaccinated members between high and
42 e facilities, including higher-than-expected attack rates among vaccinated staff, were reported in so
45 ately strong, the parasitoid with the higher attack rate and conversion efficiency (Ooencyrtus) achie
46 In contrast, the parasitoid with the lower attack rate and conversion efficiency but the shorter ha
49 was to identify a dose at which the cholera attack rate and the geometric mean purge were sufficient
50 c influenza viruses have consistently higher attack rates and are typically associated with greater m
51 rolonged infectiousness led to extraordinary attack rates and case-fatality rates among HIV-infected
52 impact of early vaccination on age-specific attack rates and evaluate the outcomes of different vacc
55 to simultaneously estimate the prey-specific attack rates and predator-specific interference (facilit
57 omatic infections, without which the overall attack rates and the level of herd immunity cannot be ac
58 studies on root caries incidence, increment, attack rate, and annual total (root + coronal) caries in
59 sceptibility, the basic reproductive number, attack rate, and infectious period, for 115 cities durin
60 of a pandemic, reducing the overall and peak attack rate, and reducing the number of cumulative death
61 red deer-adapted BSE resulted in 90% to 100% attack rates, and BSE from cattle failed to transmit, in
63 uals, defence is less often required because attack rates are lower and the costs of defence may be h
64 for outbreak peak timing, peak intensity and attack rate, are substantially improved for predicted le
66 age, sex, vaccination status, arrival date, attack rates (ARs), and case fatality ratios (CFRs) for
68 asures could substantially lower the illness attack rate before a highly efficacious vaccine could be
69 ates also differed significantly (P=.045) in attack rate between TAMU (86%) and Iowa (52%) or UCP (59
70 Overall VE was assessed by comparing the attack rates between high and low vaccine coverage strat
76 uring the peak of a pandemic can reduce peak attack rates by up to 40%, but has little impact on over
79 f either 20% or 30% gross influenza clinical attack rate (CAR), with a "low severity" scenario with c
80 ition is in turn influenced by daily illness attack rate, climate, and other environment factors.
81 ty data (eg, to estimate household secondary attack rate, contact patterns within hospitals, and effe
82 ipants diagnosed with typhoid infection (ie, attack rate), defined as persistent fever of 38 degrees
83 that the feedback between basal biomass and attack rates destabilizes the trophic interactions, lead
84 47% to 67%) in 2000-2001; however, influenza attack rates differed between these 2 periods (in the pl
85 orial capacity translated into a significant attack rate during the 2015 outbreak, with a subsequent
89 lance data can be used to estimate influenza attack rates during the 2012-2013 and 2013-2014 seasons
96 States, we estimated the household secondary attack rate for pandemic H1N1 to be 27.3% [95% confidenc
101 ack rate for severe rotavirus diarrhea, a 3% attack rate for severe nonrotavirus diarrhea in the popu
104 There were no significant differences in the attack rates for diarrhoea, dysentery, or respiratory in
108 ns of such measures for studies of secondary attack rates, for the persistence of infection in human
109 ive (12 months) virus-associated symptomatic attack rates from 12 countries and symptomatic case fata
110 ed hospitalization-related (in-hospital) VTE attack rates from 2005 to 2010 ranged from 251 to 306 (1
114 ts who exhibit symptoms [household secondary attack rate (hSAR)], this metric is difficult to interpr
116 /14) in household contacts, and the clinical attack rate (ie, the proportion of persons seropositive
117 provides the largest reduction in infection attack rate if the efficacy of 5-fold fractional-dose va
120 existing heterologous immunity which reduced attack rates in adults during 2009 had substantially dec
124 e of Bahia in Brazil which suggests that the attack rate is unidentifiable with monthly data in Bahia
125 cause a severe worldwide epidemic, with high attack rates, large numbers of deaths and hospitalizatio
126 orkers at the outbreak hospital, yielding an attack rate of 10% among potentially exposed outbreak ho
128 n U.S. laboratories yearly and calculated an attack rate of 13/100,000 microbiologists between 1996 a
129 the model predicts a reduction to an illness attack rate of 2% (95% Cl: 0.2, 16) and a death rate of
131 ion, the model predicts an influenza illness attack rate of 33% of the population (95% confidence int
133 malaria (9.1 person-years), resulting in an attack rate of 5.8 cases per person-year (95% CI, 4.3 to
135 hi (Quailes strain) inoculum required for an attack rate of 60%-75% in typhoid-naive volunteers when
144 to infection in both mice and ticks, a high attack rate of ticks on mice, a high density of larval t
147 incorporating epidemic curves with clinical attack rates of 20% or 30% in a single wave of illness,
148 erlands, we estimated combined influenza A/B attack rates of 29.2% (95% credible interval (CI): 21.6,
149 achieve the primary objective, resulting in attack rates of 55% (11/20) or 65% (13/20), respectively
150 as a function of varying values of true VE, attack rates of rotavirus and nonrotavirus diarrhea in t
151 oyed by extremely large dispersals and large attacking rates of insects, thus creating multiple attra
152 on the intermediate predator, as long as the attack rate on the intermediate predator is relatively l
153 e prey are vigilant, increasing top predator attack rates on the intermediate predator reduces compet
154 e numbers of undetected infections, and high attack rates on- and off-farm, even beyond a 5-km high-r
155 of this model should minimize differences in attack rates or severity when groups are challenged at d
160 nor respiratory diseases (1933 and 1935), an attack rate ratio analysis of the decline of diphtheria
162 ed heart problems 2 to 3 years following the attacks (rate ratios, 4.67 at 2 years and 3.22 at 3 year
163 rable outcomes in terms of optimal infection attack rate reduction, peak incidence reduction or peak
167 monstrate that estimates of ferret secondary attack rate (SAR) explain 66% of the variation in human
168 a meta-analysis of Ebola household secondary attack rate (SAR), disaggregating by type of exposure (d
170 at day 5 of introduction and a 30% clinical attack rate scenario, epidemic peak would be delayed by
173 uenza pandemic that has age-specific illness attack rates similar to the Asian pandemic in 1957-1958
175 estimated SIA coverage tended to have lower attack rates (Spearman Correlation Coefficient=-0.63), p
176 tive strategy for reduction of the infection attack rate that would be robust with a large margin for
177 ver transmission, we calculate the infection attack rate (the proportion of population infected over
179 e doses can be estimated from the cumulative attack rate up to the point antibiotic prophylaxis begin
180 nd adolescents aged 1 to 14 years, secondary attack rates varied according to age and by disease and
181 ed on estimates for Antioquia, Colombia, the attack rate varies between 4% and 26% for a low (below 1
182 te-to-severe rt-PCR-confirmed influenza, the attack rate was 0.62% (16 cases) in the QIV group and 2.
184 4 recipients, the RT-PCR-confirmed influenza attack rate was 2.2% (96 cases among 4303 participants)
189 he cholera epidemic in Haiti, the cumulative attack rate was 6.1%, with cases reported in all 10 depa
190 exposed to unvaccinated cases, the secondary attack rate was 71.5% if they were unvaccinated and 15.1
194 1-50.0%) in Colombia which suggests that the attack rate was most likely less than 50%; and 32.4% (95
196 45% and 3%, respectively, and the estimated attack rates were 29% and 0%, respectively, for an appar
197 Rakai District, the estimated infection and attack rates were 68% and 41%, respectively, and 55% of
198 Moreover, we estimated that the infection attack rates were 78.0% (95% confidence interval (CI): 6
200 children < or = 2 years old, and the highest attack rates were for infants 9-11 months (65/100,000) a
202 wever, for the remote population, the lowest attack rates were obtained for adults older than 50 year
205 ections produced more intuitively consistent attack rates when households were stratified by the spee
206 up to 40%, but has little impact on overall attack rates, whereas case isolation or household quaran
208 strategies substantially reduced the illness attack rate, with a maximal reduction in the attack rate
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