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1 with the body, suggesting a more generalized attention deficit.
2 attery assessed both spatial and non-spatial attention deficits.
3 d provided the anatomical basis for kinds of attention deficits.
4 e) produced a systematic exaggeration of the attention deficits.
5 riance for motor and language than memory or attention deficits.
6 ameters are neural markers of musicality and attention deficits.
7 diminished thalamic gain control can lead to attention deficits.
8 isability (ID), and frequently presents with attention deficits.
10 creased in prefrontal cortex of rats showing attention deficits after developmental malnutrition.
12 gical disorders, including schizophrenia and attention deficit and hyperactivity disorder (ADHD).
14 , the overall group demonstrated significant attention deficits and a significantly greater frequency
17 TRN-restricted deletion of Ptchd1 leads to attention deficits and hyperactivity, both of which are
18 1DS have high prevalence of autism spectrum, attention deficit, and anxiety disorders, as well as psy
22 ies suggested that Chrna7 deficient mice had attention deficits, but were normal in baseline behavior
24 teria were systematic childhood diagnosis of attention-deficit disorder, ADHD, or a research diagnost
26 disorders, like autism spectrum disorders or attention-deficit hyper-activity disorders, which are kn
27 irst-line treatment of choice in adults with attention-deficit hyperactive disorder (ADHD) and some s
29 g been recognized that many individuals with attention deficit hyperactivity disorder (ADHD) also hav
31 son's disease (PD) as well as treatments for attention deficit hyperactivity disorder (ADHD) and depr
32 ociated with autism spectrum disorder (ASD), attention deficit hyperactivity disorder (ADHD) and schi
33 and to neuropsychiatric disorders including attention deficit hyperactivity disorder (ADHD) and schi
34 or (GRM) GFIN, previously observed to impact attention deficit hyperactivity disorder (ADHD) and schi
35 number of psychiatric conditions, including attention deficit hyperactivity disorder (ADHD) and subs
37 esses tied to the adult outcome of childhood attention deficit hyperactivity disorder (ADHD) could gu
44 d treatment of children and adolescents with attention deficit hyperactivity disorder (ADHD) is uncle
46 olescents and young adults without childhood attention deficit hyperactivity disorder (ADHD) often pr
47 This study examined the effects of childhood attention deficit hyperactivity disorder (ADHD) symptoms
48 tism, bipolar disorder, major depression and attention deficit hyperactivity disorder (ADHD) using ge
50 speech impairment, variable expressivity of attention deficit hyperactivity disorder (ADHD), and mot
51 ed executive inhibition is a core deficit of attention deficit hyperactivity disorder (ADHD), which i
52 moderate the effect of stress on severity of attention deficit hyperactivity disorder (ADHD), with st
53 s to excess mortality among individuals with attention deficit hyperactivity disorder (ADHD), yet ass
54 4, and illustrate a use case of HaploReg for attention deficit hyperactivity disorder (ADHD)-associat
66 (mood and anxiety), behavioral disturbance (attention deficit hyperactivity disorder and conduct dis
67 ase guanfacine is approved for children with attention deficit hyperactivity disorder but not well st
68 he study of monozygotic twins discordant for attention deficit hyperactivity disorder can elucidate m
69 ited understanding of why many children with attention deficit hyperactivity disorder do not outgrow
70 on between the persistence and remittance of attention deficit hyperactivity disorder may provide a f
71 ed with neurotransmitter dysregulation, e.g. attention deficit hyperactivity disorder or Alzheimer's
73 0.05 and P </= 0.01) with schizophrenia and attention deficit hyperactivity disorder were also nomin
74 ers, including excessive daytime sleepiness, attention deficit hyperactivity disorder, Alzheimer dise
75 bsessive-compulsive disorder and behaviours, attention deficit hyperactivity disorder, and autistic s
76 adolescent psychopathology, such as anxiety, attention deficit hyperactivity disorder, and substance
77 sease, schizophrenia, autism, depression and attention deficit hyperactivity disorder, as well as age
78 ur candidates under clinical development for attention deficit hyperactivity disorder, binge eating d
79 sensory gating, and social behavior and that attention deficit hyperactivity disorder, bipolar disord
80 lycerides, type 2 diabetes, waist-hip ratio, attention deficit hyperactivity disorder, bipolar disord
81 but high variability in patients with autism/attention deficit hyperactivity disorder, compared with
83 eneral anesthesia exposure early in life and attention deficit hyperactivity disorder, language proce
84 nge of neuropsychiatric illnesses, including attention deficit hyperactivity disorder, mood disorders
85 lphenidate (MPH), a first-line treatment for attention deficit hyperactivity disorder, on D2/3 recept
86 a clinical measure of attention--symptoms of attention deficit hyperactivity disorder--from resting-s
98 he proportion who were clinically at risk of attention deficit hyperactivity disorders after Bonferro
99 ndings are mixed on the relationship between attention-deficit hyperactivity disorder (ADHD) and youn
101 ral studies have questioned the stability of attention-deficit hyperactivity disorder (ADHD) from chi
111 tine, drugs widely used for the treatment of attention-deficit hyperactivity disorder (ADHD), suppres
112 n two male siblings who share a diagnosis of attention-deficit hyperactivity disorder (ADHD), with ot
116 Brain Imaging Data Exchange (ABIDE) and the Attention-Deficit Hyperactivity Disorder (ADHD-200) data
118 ole in neuropsychiatric disorders, including attention-deficit hyperactivity disorder and drug addict
119 orders, including schizophrenia, depression, attention-deficit hyperactivity disorder and substance a
120 dividuals with major depressive disorder and attention-deficit hyperactivity disorder from their cont
121 associations of smoking with drug and AUDs, attention-deficit hyperactivity disorder, bipolar disord
122 evelopmental disabilities, including autism, attention-deficit hyperactivity disorder, dyslexia, and
125 al-reported symptoms of depression, anxiety, attention/deficit hyperactivity disorder (ADHD), opposit
127 elations were found between extraversion and attention-deficit-hyperactivity disorder (ADHD) and betw
129 gnaling in humans associated with addiction, attention-deficit/ hyperactivity disorder, schizophrenia
130 mental exposure to PBDEs and intelligence or Attention Deficit/Hyperactivity Disorder (ADHD) and atte
131 gression is widely observed in children with attention deficit/hyperactivity disorder (ADHD) and has
132 ental report of learning disability (LD) and attention deficit/hyperactivity disorder (ADHD) in child
133 renatal exposure to PFASs is associated with attention deficit/hyperactivity disorder (ADHD) or child
134 features, including intellectual disability, attention deficit/hyperactivity disorder (ADHD), and aut
135 um PCB levels in infancy are associated with attention deficit/hyperactivity disorder (ADHD)-related
138 d 3.3-fold (95% CI: 1.4, 7.7) higher odds of attention deficit/hyperactivity disorder problems compar
139 opmental disorders, including schizophrenia, attention deficit/hyperactivity disorder, and autism spe
140 neurologic disorders in offspring, including attention deficit/hyperactivity disorder, autism, and sc
141 nxiety disorders, autism spectrum disorders, attention deficit/hyperactivity disorder, severe learnin
144 [0.2%]; adjusted RR, 4.3; 95% CI, 2.3-8.2), attention-deficit/hyperactivity disorder (189 cases [5.3
145 ar bipolar disorder (94.2 [1.69]; P = .004), attention-deficit/hyperactivity disorder (96.3 [0.91]; P
146 ofiles in autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) - preval
150 compared and contrasted with trajectories in attention-deficit/hyperactivity disorder (ADHD) and chil
151 dication has long been effective in treating attention-deficit/hyperactivity disorder (ADHD) and is c
153 lphenidate (MPH) is used clinically to treat attention-deficit/hyperactivity disorder (ADHD) and off-
157 earch has demonstrated that individuals with attention-deficit/hyperactivity disorder (ADHD) are more
158 sses underlying persistence and remission of attention-deficit/hyperactivity disorder (ADHD) are poor
162 gical and hemodynamic activity is altered in attention-deficit/hyperactivity disorder (ADHD) during t
170 ic resonance imaging (fMRI) studies of adult attention-deficit/hyperactivity disorder (ADHD) have rev
171 Previous neuroimaging investigations in attention-deficit/hyperactivity disorder (ADHD) have sep
172 volumetric differences were associated with attention-deficit/hyperactivity disorder (ADHD) in a rec
173 ys been a key criterion for the diagnosis of attention-deficit/hyperactivity disorder (ADHD) in adult
192 p between autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD) is incre
198 A neurobiological-based classification of attention-deficit/hyperactivity disorder (ADHD) subtypes
199 based on network strength at rest predicted attention-deficit/hyperactivity disorder (ADHD) symptoms
201 ssociated with a categorical ASD diagnosis); attention-deficit/hyperactivity disorder (ADHD) traits (
202 when obsessive-compulsive disorder (OCD) and attention-deficit/hyperactivity disorder (ADHD) were exc
203 elating maternal smoking during pregnancy to attention-deficit/hyperactivity disorder (ADHD) without
204 modeling complex brain disorders, including attention-deficit/hyperactivity disorder (ADHD), aggress
205 to investigate the genetic susceptibility of Attention-Deficit/Hyperactivity Disorder (ADHD), but wit
206 phenidate hydrochloride for the treatment of attention-deficit/hyperactivity disorder (ADHD), little
207 ivity, a multifaceted behavioral hallmark of attention-deficit/hyperactivity disorder (ADHD), strongl
208 er to better understand the underpinnings of attention-deficit/hyperactivity disorder (ADHD), we targ
224 riants with long repeats have been linked to attention-deficit/hyperactivity disorder (ADHD); however
227 disorder (HR, 0.83 [95% CI, 0.62-1.13]), or attention-deficit/hyperactivity disorder (HR, 0.99 [95%
228 h schizophrenia (N=3540), autism (N=16 146), attention-deficit/hyperactivity disorder (N=18 726) and
232 disorders, including infantile parkinsonism, attention-deficit/hyperactivity disorder and autism spec
233 s characterized by high impulsivity, such as attention-deficit/hyperactivity disorder and bipolar dis
235 opment of novel pharmacologic treatments for attention-deficit/hyperactivity disorder and other condi
236 r across sex, after excluding relatives with attention-deficit/hyperactivity disorder and probands wi
237 l relevance to the etiology and treatment of attention-deficit/hyperactivity disorder and related dis
238 hiatric disorders listed in DSM-5, including attention-deficit/hyperactivity disorder and substance u
239 e risk of suicidal behavior in patients with attention-deficit/hyperactivity disorder and suggests th
240 rgent results were obtained for the level of attention-deficit/hyperactivity disorder and the influen
241 disorder and suggests that individuals with attention-deficit/hyperactivity disorder and their famil
243 ed vs 5.46% of unexposed were diagnosed with attention-deficit/hyperactivity disorder by age 15 years
244 sits in 2007-2010 but represented 55.9% when attention-deficit/hyperactivity disorder codes were excl
245 e drugs, antihypertensive drugs, sleep aids, attention-deficit/hyperactivity disorder drugs, and anti
246 for the treatment of insomnia [sleep aids], attention-deficit/hyperactivity disorder drugs, antidepr
248 hazard ratio [HR], 2.02 [95% CI, 1.80-2.26]; attention-deficit/hyperactivity disorder HR, 2.21 [95% C
249 disorders such as schizophrenia, autism, and attention-deficit/hyperactivity disorder in humans.
250 al diagnosis of autism spectrum disorder and attention-deficit/hyperactivity disorder in offspring.
251 tipsychotics, the most common diagnoses were attention-deficit/hyperactivity disorder in younger chil
254 While there was no association with comorbid attention-deficit/hyperactivity disorder or IQ, age rang
256 tion (outlier magnitude) relates to specific attention-deficit/hyperactivity disorder symptoms (hyper
257 externalizing behavioral problems, including attention-deficit/hyperactivity disorder symptoms and co
258 ediate factors (child executive function and attention-deficit/hyperactivity disorder symptoms) and c
259 ants contributes to slower processing speed, attention-deficit/hyperactivity disorder symptoms, and e
261 s between ICN dysmaturation and diagnosis of attention-deficit/hyperactivity disorder were identified
263 pes of care and inpatient well-newborn care, attention-deficit/hyperactivity disorder, and asthma amo
264 ial behavior, posttraumatic stress disorder, attention-deficit/hyperactivity disorder, and conduct pr
265 ral psychiatric diseases, such as addiction, attention-deficit/hyperactivity disorder, and schizophre
266 al psychiatric disorders, such as addiction, attention-deficit/hyperactivity disorder, and schizophre
267 rodevelopmental disorders, including autism, attention-deficit/hyperactivity disorder, and schizophre
268 ts with a psychiatric diagnosis of interest (attention-deficit/hyperactivity disorder, autism spectru
270 between them and schizophrenia, depression, attention-deficit/hyperactivity disorder, eating disorde
271 nidate, a common and effective treatment for attention-deficit/hyperactivity disorder, in healthy adu
273 disorders (intellectual disability, autism, attention-deficit/hyperactivity disorder, schizophrenia,
274 the genetic signature of DD overlapped with attention-deficit/hyperactivity disorder, schizophrenia,
275 of suicidal behavior among individuals with attention-deficit/hyperactivity disorder, the developmen
286 xiety, disruptive mood dysregulation, and/or attention-deficit/hyperactivity disorders and 22 healthy
287 and posttraumatic stress) and externalizing (attention-deficit/hyperactivity, intermittent explosive,
288 d Interview for Prodromal Syndromes, and for attention-deficit/hyperactivity, substance-related, and
291 ecently reported that spatial and nonspatial attention deficits in stroke patients with hemispatial n
293 e effect of oxytocin on reversing non-social attention deficits is a particularly novel finding, and
296 f reality, disruption of working memory, and attention deficits that characterize these disorders.
300 network normalization." METHODS: We measured attention deficits with a neuropsychological battery and
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