1             PIN II has two or more layers of atypical cells.

2      PIN I lesions have one or two layers of atypical cells.

3 iewed by the consensus panel for potentially atypical cells.

4 e aspiration biopsy was done and revealed no atypical cells.

5                                    The first atypical cells are detected in the pituitary intermediat

6                    The presence of 3 or more atypical cells at the dermal-epidermal junction (DEJ) by

7    Of these, the presence of pagetoid cells, atypical cells at the dermal-epidermal junction, and irr

8                                              Atypical cells contain no wild-type copy of the Rb gene

9                                         Many atypical cells contained large hypertrophic cytoplasm an

10 d to the periphery, demonstrating that these atypical cells contributed to the peripheral T cell pool

11                                        These atypical cells could be associated with an underlying ep

12     Human embryonic stem (hES) cells show an atypical cell-cycle regulation characterized by a high p

13                      When, as a result of an atypical cell division, a cell is displaced from a hair

14  grade I-II, indicated by multiple layers of atypical cells, is seen consistently by 12 weeks of chem

15 agetoid cells and follicular localization of atypical cells, respectively.

16 as exfoliative erythroderma with circulating atypical cells, Sezary syndrome (SS).

17                       PIN IV lesions contain atypical cells that fill the lumen and bulge focally int

18                    The earliest Rb-deficient atypical cells were identified in the intermediate and a

19                                              Atypical cells were seen with cytologic examination of f