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1 including dysthymia, chronic depression, and atypical depression.
2 fficacy data exist for patients with chronic atypical depression.
3 's relative ineffectiveness in patients with atypical depression.
4 ity in humans experiencing chronic stress or atypical depression.
6 o better than imipramine in the treatment of atypical depression, although fluoxetine was better tole
8 ween epidemiologically derived typologies of atypical depression and DSM-IV major depression with aty
9 inflammatory and metabolic dysregulation in atypical depression and HPA-axis hyperactivity in melanc
11 syndromes: (1) mild typical depression, (2) atypical depression, and (3) severe typical depression.
14 r study sought to reevaluate the validity of atypical depression by examining the demographic and cli
15 leptin resistance may benefit patients with atypical depression characterized by obesity-related met
17 y was conducted to increase understanding of atypical depression diagnosed using only the reversed ve
21 because its known efficacy for treatment of atypical depression helped to calibrate the appropriaten
23 pothesized that to normalize HPA function in atypical depression, MAOI would differ from TCA in decre
24 ) axis is frequently impaired in depression, atypical depression may exhibit increased feedback sensi
25 ials, to our knowledge, of psychotherapy for atypical depression or of cognitive therapy compared wit
26 tence of the normal stress response, whereas atypical depression resembles a stress response that has
28 and oversleeping can be used to identify an atypical depression subgroup that is distinct from other
29 Guide for the Hamilton Depression Scale With Atypical Depression Supplement (SIGH-ADS), the Mania Rat
30 oxidase inhibitors are effective in treating atypical depression, their side effects and prescription
33 ive than tricyclic antidepressants (TCA) for atypical depression, we hypothesized that to normalize H
34 depression who met the Columbia criteria for atypical depression were randomly assigned to receive fl
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