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1 produced no reduction in the severity of the audiogenic seizure.
2 rotein synthesis, exaggerated mGluR-LTD, and audiogenic seizures.
3 areas for the initiation and propagation of audiogenic seizures.
4 relate with susceptibility and resistance to audiogenic seizures.
5 der, including sudden unexplained deaths and audiogenic seizures.
6 that is critical in the initiation of reflex audiogenic seizures.
7 behaviour, defective social interaction and audiogenic seizures.
8 fore unrecognized specific susceptibility to audiogenic seizures.
10 initiation site in the neuronal network for audiogenic seizure (AGS) in rats undergoing ethanol with
11 (IC) central nucleus (ICc), is critical for audiogenic seizure (AGS) initiation in the genetically e
13 implicated in the neuronal network for these audiogenic seizures (AGS) in animals undergoing ETX.
14 pathway is also implicated in the network of audiogenic seizures (AGS) in genetically epilepsy-prone
16 iculus (DLSC) play a role in the network for audiogenic seizures (AGS) in genetically epilepsy-prone
17 us studies indicate that daily repetition of audiogenic seizures (AGS) leads to audiogenic 'kindling'
18 In rodents, ETX results in susceptibility to audiogenic seizures (AGS), and the DLSC are implicated a
19 t-ictal analgesia occurs in GEPRs, following audiogenic seizures (AGS), and whether this analgesia in
20 (GEPR-3s) exhibits generalized onset clonic audiogenic seizures (AGS), but following repetitive AGS
21 ucture in the requisite neuronal network for audiogenic seizures (AGS), which is confined to the brai
26 controls to flurothyl, PTZ, kainic acid, and audiogenic seizures and enhanced sensitivity (i.e., seiz
27 ependent LTD, neocortical hyperexcitability, audiogenic seizures, and altered behaviors, including an
28 mice exhibited impaired motor coordination, audiogenic seizures, and brainstem neurodegeneration.
29 tigation studied the mode of inheritance for audiogenic seizures by crossing the Frings mouse with th
30 3ahl variant may account for the hearing and audiogenic seizure differences observed between Frings a
35 ice all exhibit an enhanced tendency to have audiogenic seizures in response to white noise stimuli a
37 scribe a mutation in a novel gene underlying audiogenic seizures in the Frings mouse, providing a val
41 loss (age-related hearing loss 5, ahl5) and audiogenic seizures (juvenile audiogenic monogenic seizu
45 mg/kg had no effect in 4/4 rats made acutely audiogenic seizure-prone by infusion of bicuculline into
46 entricular (i.c.v.) administration of GRP to audiogenic seizure-prone DBA/2 mice prior to exposure to
49 acoustic hypersensitivity and propensity for audiogenic seizures, suggesting altered auditory respons
50 trols and a group of animals displaying high audiogenic seizure susceptibility (100% AGS) (HAGS), and
51 e human ortholog of the gene responsible for audiogenic seizure susceptibility in Frings and BUB/BnJ
52 ve GSK3 promotes locomotor hyperactivity and audiogenic seizure susceptibility in FX mice, raising th
55 receptor 1), also known as MASS1 (monogenic audiogenic seizure susceptible 1), is an orphan G protei
56 rings strain, 391 of the 836 N2 progeny were audiogenic seizure susceptible, a finding consistent wit
57 the seizure gene, named mass1 for monogenic audiogenic seizure susceptible, to an approximately 3.6
59 ological dysfunction, neurodegeneration, and audiogenic seizures that manifest beginning in early adu
60 eurological applications, such as preventing audiogenic seizures that originate in the auditory midbr
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