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1 al recognition memory and auditory function (auditory brainstem response).
2 accompanying hearing loss (estimated by the auditory brainstem response).
3 neuropathy, as measured using Wave I of the auditory brainstem response.
4 w greater between-trial phase-locking in the auditory brainstem response.
5 orresponding decline in the amplitude of the auditory brainstem response.
6 d intact cochlear amplification but impaired auditory brainstem responses.
7 hs of age, after which they display abnormal auditory brainstem responses.
8 al day 7 (P7) to P96 using voltage-clamp and auditory brainstem responses.
9 ional knock-out (cKO) mice exhibit decreased auditory brainstem responses.
10 with reduced acoustic startle and distorted auditory brainstem responses.
11 h DT at P2, had normal hair cells and normal auditory brainstem responses.
12 c emissions and 70-80 dB threshold shifts in auditory brainstem responses.
13 have normal auditory thresholds but abnormal auditory brainstem responses.
14 nd WT mice in spontaneous eyeblink activity, auditory brainstem response (ABR) amplitudes, and tail-f
15 and functional hearing measures, such as the auditory brainstem response (ABR) and distortion product
17 the CN participates in the generation of the auditory brainstem response (ABR) and receives direct in
18 system function is being measured using the auditory brainstem response (ABR) or distortion product
19 ion product otoacoustic emission (DPOAE) and auditory brainstem response (ABR) thresholds during and
22 the results of our previous study that used auditory brainstem response (ABR) thresholds to identify
25 In the current study, a QTL analysis for auditory brainstem response (ABR) thresholds, which indi
26 chlear responses, both in the neural output [auditory brainstem response (ABR) wave 1] and in outer h
27 sensory hair cells, 5) significantly delayed auditory brainstem response (ABR) wave I latencies at lo
32 either Coch 131-150 or beta-tectorin 71-90, auditory brainstem responses (ABR) showed significant he
34 w2J/dfw2J homozygotes exhibit no discernible auditory brainstem responses (ABR) to sound pressure lev
35 ng testing as a function of sound frequency (auditory brainstem response -- ABR thresholds, and disto
36 ndent auditory threshold shifts (measured by auditory brainstem response, ABR) of up to 73 dB (16 kHz
37 ng loss and cochlear pathology, we collected auditory brainstem responses (ABRs) and determined cochl
40 mine this relationship further, we collected auditory brainstem responses (ABRs) from rhesus monkeys
43 C), which is obtained typically by recording auditory brainstem responses (ABRs)-the BIC reflects the
50 ells, in adult mice virtually eliminated the auditory brainstem response and acoustic startle reflex,
51 s seen by morphology and cochlear functions (auditory brainstem response and otoacoustic emissions).
53 d cochlear microphonics, as well as abnormal auditory brainstem responses and cortical auditory-evoke
54 mal cochlear function as indicated by normal auditory brainstem responses and distortion product otoa
56 hlear nerve fibers, and using measurement of auditory brainstem responses and otoacoustic emissions t
57 isplatin-induced ototoxicity, as measured by auditory brainstem responses and scanning electron micro
58 d noise, a normal sense of balance, a normal auditory brainstem response, and normal transduction cur
59 latency and distortion in the wave I of the auditory brainstem responses, and elevated sensitivity t
60 ion in cochlear neural responses, as seen in auditory brainstem responses, and increased the loss of
61 estly increased interpeak intervals in their auditory brainstem responses, and substantially longer l
62 are approximately 10 dB more sensitive than auditory brainstem responses, and they are very sharply
65 of both strains were selected with matching auditory brainstem response audiograms and gap detection
66 e resulted in a temporary threshold shift in auditory brainstem responses but a persistent increase i
67 mouse, the ducky mouse (du), showed elevated auditory brainstem response click and frequency-dependen
68 Homozygous mutant mice had no detectable auditory brainstem response, displayed highly disorganiz
69 congenital profound deafness, as assessed by auditory brainstem response, distortion product otoacous
70 ng distortion product otoacoustic emissions, auditory brainstem responses, envelope following respons
73 ials) and auditory nerve/brainstem activity (auditory brainstem responses) have made it possible to d
79 ll transduction and decreased suprathreshold auditory brainstem response input/output gain in WT mice
80 nacin treatment of engrafted animals reduced auditory brainstem response interpeak latency, indicativ
82 distortion product otoacoustic emission and auditory brainstem response measurements in Col11a2 -/-
84 vidence from human temporal bone studies and auditory brainstem response measures suggests that this
87 When tested for endocochlear potential and auditory brainstem response, PVM/M-depleted animals show
88 temporary threshold shift (TTS), evident in auditory brainstem response recordings as sound levels r
89 loss at 3 weeks after infection, measured by auditory brainstem response recordings, correlated to th
94 bjects with tinnitus and a normal audiogram, auditory brainstem responses show a significantly reduce
95 less variability when tapping to a beat have auditory brainstem responses that are less variable as w
96 erely hearing-impaired, as shown by elevated auditory brainstem response thresholds and absent endoco
97 osure, nor were click- or noise-burst-evoked auditory brainstem response thresholds different from co
98 outer HCs is disrupted in Prox1DTA mice and auditory brainstem response thresholds in adults are 40-
99 ed higher hair cell survival rates and lower auditory brainstem response thresholds in injected ears
105 ividual differences observed in behavior and auditory brainstem response timing to cochlear synaptopa
107 develop a mathematical method to measure the auditory brainstem response to running speech, an acoust
109 without developmental dyslexia by measuring auditory brainstem responses to a speech syllable presen
110 tones; and (ii) physiological adaptation of auditory brainstem responses to clicks as a function of
111 processing of complex sounds such as speech (auditory brainstem responses to speech and other complex
112 oor readers have significantly more variable auditory brainstem responses to speech than do good read
113 e duration of the wave V-V(n) complex of the auditory brainstem response was studied, as was the effe
117 topathy in humans, namely, modestly abnormal auditory brainstem response Wave I/Wave V ratios in the
118 on were unaffected in these mutant mice, but auditory brainstem response wave-I amplitude was reduced
119 mal data, we demonstrate that the latency of auditory brainstem response wave-V in noise reflects aud
124 exhibit normal hearing as measured by evoked auditory brainstem responses, which suggests that the ne
125 ermore, 3-week-old double mutant mice lacked auditory brainstem responses, which were present in thei
126 fness characterized by an absent or abnormal auditory brainstem response with preservation of outer h
127 encies and interpeak latencies, evaluated by auditory brainstem response within 48 h after birth, wer
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