1 enting the main source for adaptation in the auditory nerve.
2 ere it is localized to ganglion cells in the auditory nerve.
3 vities <1.2 kHz was enhanced relative to the auditory nerve.
4 bnormal synaptic structure in endings of the auditory nerve.
5 lely on the relative timing of spikes in the auditory nerve.
6 e development of spontaneous activity in the auditory nerve.
7 y transmit timing information encoded by the auditory nerve.
8 ial during high-frequency stimulation of the auditory nerve.
9 not block EPSPs produced by stimulating the auditory nerve.
10 -deaf people to electrically stimulate their auditory nerve.
11 amate, the neurotransmitter release from the auditory nerve.
12 l signals to produce maximum activity in the auditory nerve.
13 eus receive the bulk of their input from the auditory nerve.
14 n, a procedure that requires survival of the auditory nerve.
15 diameter and neurofilament regulation in the auditory nerve.
16 precise stimulus-driven spike timing in the auditory nerve.
17 cess most likely underlies adaptation in the auditory nerve.
18 ng and chronic electrical stimulation of the auditory nerve.
19 annel's ability to effectively stimulate the auditory nerve.
20 unction of terminal unmyelinated portions of auditory nerve.
21 ost always higher than those observed in the auditory nerve.
22 and processes first-order afferents from the auditory nerve.
23 e and sets the stage for cell replacement of auditory nerve.
24 lus (CS) applied to the posterior eighth, or auditory, nerve, a positive slope of CR acquisition was
25 e abnormalities persist after restoration of auditory nerve activity by a cochlear implant, the proce
26 This had the benefit that BCs relayed auditory nerve activity even when synapses showed signif
27 ent of a neuronal correlate of tinnitus when auditory nerve activity is reduced due to the earplug.
28 Elimination of auditory nerve activity results in atrophy and death of
29 Elimination of auditory nerve activity results in death and atrophy of
30 empts to relate organ of Corti structure and auditory nerve activity to the morphology of primary syn
31 is significantly influenced by sound-evoked auditory nerve activity.
32 Examination of the sural nerve and the auditory nerve adjacent to the brainstem showed marked l
33 cochlear nucleus (VCN) is from glutamatergic auditory nerve afferents, but the VCN is also innervated
34 tory synapses formed by two distinct inputs, auditory nerve (AN) and parallel fibers (PF), on differe
35 ed this in the mouse cochlear nucleus, where auditory nerve (AN) fibers contact bushy cells (BCs) at
36 onstructed in 3D the trajectories of labeled auditory nerve (AN) fibers following multiunit recording
37 e of sound levels over which firing rates of auditory nerve (AN) fibers grows rapidly with level shif
38 convergent inputs from high-spontaneous rate auditory nerve (AN) fibers, with no special currents and
39 fect neurotransmitter release by presynaptic auditory nerve (AN) fibers.
40 otemporal pattern of discharges across their auditory nerve (AN) inputs ().
41 nological model of the inner hair cell (IHC)-auditory nerve (AN) synapse successfully explained neura
42 auditory brainstem implants that bypass the auditory nerve and directly stimulate auditory processin
43 Shocks to the auditory nerve and granule cell domains evoked glutamate
44 putatively reflecting decreased inputs from auditory nerve and increased inputs from nonauditory str
45 ributed to a return of spike activity in the auditory nerve and may help explain cochlear implant ben
46 Brain slices containing the auditory nerve and NM on both sides were obtained from h
47 nses to sound can first be recorded from the auditory nerve and observed behaviourally from 10-12 day
48 ells receive excitatory inputs from both the auditory nerve and parallel fibers; cartwheel cells rece
49 ribbons, followed by reduced activity of the auditory nerve and reduced centrally generated wave II a
50 of anemia disrupts normal development of the auditory nerve and results in altered conduction velocit
51 d VGLUT2, are differentially associated with auditory nerve and somatosensory inputs to the CN, respe
52 t occurs in the cochlear nucleus (CN), where auditory nerve and somatosensory pathways converge.
53 lcium-binding proteins were expressed in the auditory nerve, and CR immunoreactivity labeled the firs
54 cochlear turns and a reduced activity of the auditory nerve (auditory brainstem response wave I).
55 ade probably was not caused by damage to the auditory nerve, because the lesioned animals showed inta
56 gan of Corti initiate electrical activity in auditory nerves before hearing, pointing to an essential
57 ntitative differences between trigeminal and auditory nerve boutons in terms of their localization on
58 constrained by temporal phase locking in the auditory nerve but may instead stem from higher-level co
59 ar implants deliver electrical pulses to the auditory nerve by relying on sophisticated signal proces
60 The myelinated fibers of the auditory nerve can be divided into two separate populati
61 ending mammalian auditory pathway beyond the auditory nerve can be predicted based on coding principl
62 Sound-evoked spikes in the auditory nerve can phase-lock with submillisecond precis
63 her with the role of neurons embedded in the auditory nerve [cochlear root neurons (CRNs)], recently
64 ed low amplitude negative potentials without auditory nerve compound action potentials.
65 Thus AMPA receptors postsynaptic to the auditory nerve differ from those postsynaptic to paralle
66 n is a standard clinical tool for diagnosing auditory nerve disorders due to acoustic neuromas.
67 Subjects with auditory nerve disorders had normal loudness adaptation
68 greater magnitude in ribbon synapse than in auditory nerve disorders.
69 ode of auditory prosthesis using penetrating auditory-nerve electrodes that permit frequency-specific
70 em, from pulse trains presented with various auditory-nerve electrodes to phase-locked activity of ne
71 The large endings of the auditory nerve, endbulbs of Held, were studied because t
72 lobular bushy cells of the AVCN receive huge auditory nerve endings specialized for high fidelity neu
73 eafness results in synaptic abnormalities in auditory nerve endings.
74 s intervene between mechanical vibration and auditory nerve excitation.
75 We review the mechanical origin of auditory-nerve excitation, focusing on comparisons of th
76 input, because shocks to the cut end of the auditory nerve excited Golgi cells with excitatory posts
77 antly inherited neuropathy of both optic and auditory nerves expressed by impaired visual acuity, mod
78 The auditory nerve feeds information through several paralle
79 ximately 1 year after deefferentation, acute auditory nerve fiber (ANF) recordings were made from les
80 nce the encoding of the onset of synchronous auditory nerve fiber activity.
81 bulbs arise from the ascending branch of the auditory nerve fiber and contact the cell body of spheri
82 detectors of the coincident firing of their auditory nerve fiber inputs.
83 ells, which cannot be restored by increasing auditory nerve fiber recruitment.
84 ity) of the auditory periphery by modulating auditory nerve fiber response properties.
85 s from mammalian inner hair cells (IHCs) and auditory nerve fiber terminals that typically receive in
86 Here we report that several basic auditory nerve fiber tuning properties can be accounted
87 ases of basilar-membrane (BM) vibrations and auditory-nerve fiber responses to tones at a basal site
88 ule cell domains (GCD) of CN, whereas type I auditory nerve fibers (ANFs) project to the magnocellula
89 Physiological thresholds for auditory nerve fibers and cochlear nucleus neurons are t
90 range of intensities than those reported for auditory nerve fibers and cochlear nucleus neurons.
91 NA and NM receive input from bifurcating auditory nerve fibers and initiate processing pathways s
92 Retrogradely-labeled auditory nerve fibers and the majority of labeled multip
93 term depression (LTD), whereas those between auditory nerve fibers and their targets do not.
94 to detect the coincident firing of groups of auditory nerve fibers and to convey the precise timing o
95 ls in brainstem slices during stimulation of auditory nerve fibers at 35 degrees C.
96 or initiating bursts of action potentials in auditory nerve fibers before the onset of hearing.
97 old stimulus levels, the frequency tuning of auditory nerve fibers closely paralleled that of basilar
98 These results suggest that auditory nerve fibers encode a broad set of natural soun
99 Auditory nerve fibers exhibited a recovery of normal syn
100 Auditory nerve fibers from mutants had normal threshold,
101 Cells excited predominantly by auditory nerve fibers had AMPA receptors with exceptiona
102 racterized the response properties of single auditory nerve fibers in mice lacking Bassoon, a scaffol
103 Terminals of auditory nerve fibers in the multipolar cell area includ
104 ically by recording spike times from chicken auditory nerve fibers in vivo while stimulating with rep
105 the amount of sound-evoked spike activity in auditory nerve fibers influences terminal morphology and
106 Auditory nerve fibers innervate narrow, topographically
107 s to tones of a basilar membrane site and of auditory nerve fibers innervating neighboring inner hair
108 ndrites of octopus cells cross the bundle of auditory nerve fibers just proximal to where the fibers
109 A reduction in the number of myelinated auditory nerve fibers leads to a reduced maximal firing
110 ontaneous activity, we hypothesized that the auditory nerve fibers might initially form topographical
111 endbulb of Held synapse, which is formed by auditory nerve fibers onto bushy cells (BCs) in the ante
112 ndbulb of Held synapses, which are formed by auditory nerve fibers onto bushy cells in the cochlear n
113 m its high-dimensional sensory inputs-30,000 auditory nerve fibers or 10(6) optic nerve fibers-a mana
114 spiral ganglion, to study the projections of auditory nerve fibers representing a narrow band of freq
115 Repetitive stimulation of presynaptic auditory nerve fibers resulted in acute depression of EP
116 In the CN, espin-positive auditory nerve fibers showed a projection pattern typica
117 llel fibers synapse on apical dendrites, and auditory nerve fibers synapse on basal dendrites.
118 This wave evokes firing in auditory nerve fibers that are tuned to high frequencies
119 rites of fusiform cells in the deep layer by auditory nerve fibers through synapses that do not show
120 Octopus cells are excited by auditory nerve fibers through the activation of rapid, c
121 te-level functions, and adaptation of single auditory nerve fibers were measured in chinchillas with
122 white cats had partial hearing and possessed auditory nerve fibers with a wide range of spontaneous a
123 at encodes the timing of firing of groups of auditory nerve fibers with exceptional precision.
124 can cause a selective loss of high-threshold auditory nerve fibers without affecting absolute sensiti
125 f the wave I potential (generated by primary auditory nerve fibers) but normal amplitudes of the more
126 ith excitation mediated by granule cells and auditory nerve fibers, and shapes the output of the DCN
127 In whole-cell recordings from targets of auditory nerve fibers, octopus and T stellate cells, min
128 neurons receive excitatory input from either auditory nerve fibers, parallel fibers, or both fiber sy
129 ular to the tonotopically organized array of auditory nerve fibers, placing the earliest arriving inp
130 ing of midbrain neurons approximated that of auditory nerve fibers.
131 wn as an obligatory relay center for primary auditory nerve fibers.
132 pattern of firing in the tonotopic array of auditory nerve fibers.
133 ous activity and no sound-evoked activity in auditory nerve fibers.
134 e input from slowly conducting, unmyelinated auditory nerve fibers.
135 t noise exposure, is associated with loss of auditory nerve fibers.
136 e and timing of the postsynaptic response in auditory nerve fibers.
137 a their ribbon synapses into firing rates in auditory nerve fibers.
138 may underlie fundamental characteristics of auditory nerve fibers.
139 cells is converted into a firing pattern in auditory nerve fibers.
140 Auditory-nerve fibers (ANFs) in the cat have been subdiv
141 illa cochlea were inferred from responses of auditory-nerve fibers (ANFs) to threshold- and moderate-
142 shape changes of threshold tuning curves of auditory-nerve fibers along the cochlea.
143 The responses to sound of auditory-nerve fibers are well known in many animals but
144 ear implants cannot activate selectively the auditory-nerve fibers having low characteristic frequenc
145 otential tuning curves against the tuning of auditory-nerve fibers in experimental animals and use co
146 Some investigators have claimed that the auditory-nerve fibers of humans are more sharply tuned t
147 etect the coincident activation of groups of auditory nerve fibres by broadband transient sounds, com
148 ry ganglion cells and central and peripheral auditory nerve fibres within the cochlea.
149 ency-bandwidth dependence similar to that of auditory nerve fibres, and yield significantly greater c
150 a gives rise to V-shaped tuning functions in auditory nerve fibres, but by the level of the inferior
151 bly increased spontaneous activity of single auditory nerve fibres, while concanavalin A had no effec
152 emperature-sensitive auditory neuropathy) or auditory nerve fibres.
153 kely to induce neurotransmitter release onto auditory nerve fibres.
154 taneous and the sound-evoked activity of the auditory nerve fibres.
155 r, the mechanisms responsible for initiating auditory nerve firing in the absence of sound have not b
156 r similar to those observed by others in the auditory nerve following acoustic trauma, and suggest th
157 d in our previous study and by others in the auditory nerve following less severe acoustic trauma, an
158 eive most of their excitatory input from the auditory nerve; fusiform cells receive excitatory inputs
159 ts in the ongoing timing of responses in the auditory nerve, generally but not always resulting in sh
160 electrode array that directly stimulates the auditory nerve, has greatly benefited many individuals w
161 ansmit phase-locked action potentials of the auditory nerve in a pathway that contributes to sound lo
162 Synapses of the contralateral auditory nerve in early implanted cats also exhibited sy
163 as evaluated by unilaterally stimulating the auditory nerve in media containing the metabotropic glut
164 receptors while unilaterally stimulating the auditory nerve in vitro.
165 pathy were probably related to a decrease of auditory nerve input accompanying axonal disease.
166 mEPSCs in cells with auditory nerve input alone were inhibited by philanthoto
167 ns in the central auditory system to reduced auditory nerve input in the absence of elevated hearing
168 Deprivation of auditory nerve input in young mice results in dramatic n
169 Auditory nerve input to the principal neurons of the AVC
170 CN) where principal cells integrate primary, auditory nerve input with modulatory, parallel fiber inp
171 Golgi cells probably receive auditory nerve input, because shocks to the cut end of t
172 in the parallel fiber inputs, but not in the auditory nerve inputs innervating the same DCN principal
173 us cells prevent firing if the activation of auditory nerve inputs is not sufficiently synchronous an
174 n of intensity selectivity from nonselective auditory nerve inputs remain largely unclear.
175 r ultrastructural characteristics of primary auditory nerve inputs were similar across the rostral an
176 gnocellular ventral CN (VCN), which receives auditory nerve inputs.
177 al and thus lowers the response threshold to auditory nerve inputs.
178 ange adaptation observed at the level of the auditory nerve is located peripheral to the spike genera
179 Damage to the auditory nerve is postulated to trigger compensatory lon
180 of the current investigation was to generate auditory nerve-like glutamatergic neurons from ES cells.
181 (pancreatic beta-cell, hematopoietic cells, auditory nerve) maintained normal function but pachytene
182 Deaf patients without an intact auditory nerve may be helped by the next generation of a
183 eural coding, predicted from a computational auditory nerve model, with perception of vocoded speech
184 itory system is very immature at birth, with auditory nerve neurons initially exhibiting very low or
185 and after deafferentation of the excitatory auditory nerve (nVIII) inputs.
186 ificity of the primary afferent axons of the auditory nerve occurs in late gestation or early postnat
187 of the spiral ganglion neurons (SGNs) of the auditory nerve occurs with age and in response to acoust
188 wave, we recorded from single fibers in the auditory nerve of anesthetized cat in response to harmon
189 The auditory nerve on one side of a chick brainstem slice wa
190 The auditory nerve on one side of the slice was stimulated f
191 ochlea suppress sound-evoked activity of the auditory nerve on two time scales via one nicotinic rece
192 c stimulation or electric stimulation of the auditory nerve or the brainstem.
193 ic startle circuit in rats consisting of the auditory nerve, posteroventral cochlear nucleus, an area
194 NF application induced significant growth of auditory nerve presynaptic boutons that convey the condi
195 In the month posthatch, the auditory nerve projection to the NM matures, and appears
196 postnatal maturation of the primary afferent auditory nerve projections from the cat cochlear spiral
197 Toward the eventual goal of auditory nerve replacement, the aim of the current inves
198 typical neurons in primary visual cortex or auditory nerve, respectively.
199 nd 85% from injections of the trigeminal and auditory nerves, respectively) were apposed to somata or
200 term effects of cochlear de-efferentation on auditory nerve response properties in adult chinchillas.
201 s to pairs of stimuli, based on single-fiber auditory nerve responses at 70 and 50 dB sound pressure
202 e examined the trial-to-trial variability of auditory nerve responses during short-term rate-adaptati
203 shift in BF that characterizes maturation of auditory nerve responses during the same period.
204 milliseconds) is responsible for modulating auditory nerve responses to acoustic stimulation.
205 easurements of basilar membrane velocity and auditory nerve responses to sound, has demonstrated sign
206 inor signal transformations, the polarity of auditory-nerve responses does not conform with tradition
207 Near-threshold, auditory-nerve responses to low-frequency tones are sync
208 normal media, unilateral stimulation of the auditory nerve resulted in darker Y10B immunolabeling of
209 on from the turtle by pairing trigeminal and auditory nerve stimulation.
210 ochlea removal and in vitro after unilateral auditory nerve stimulation.
211 macaques using a stimulus design employed in auditory nerve studies of pitch encoding.
212 he influence of neurotrophin gene therapy on auditory nerve survival and peripheral sprouting in Pou4
213 ceptor, mGluR1 alpha, were found only at the auditory nerve synapse.
214 port describes the morphologic plasticity of auditory nerve synapses in response to ototoxic deafenin
215 AMPA receptors expressed at auditory nerve synapses in the mammalian and avian cochl
216 lopment of synaptic function was examined at auditory nerve synapses in the rostromedial region of th
217 In accordance, auditory nerve synapses in the VCN are functional at E15
218 However, receptors at auditory nerve synapses on cells that also receive paral
219 MPA receptor by increasing GluR4 subunits in auditory nerve synapses.
220 postsynaptic membranes of parallel fiber and auditory nerve synapses.
221 The results show that trigeminal and auditory nerve terminal fields occupy primarily the soma
222 eristics of the endbulb of Held, a prominent auditory nerve terminal in the cochlear nucleus.
223 Thus, elimination of auditory nerve terminals and pruning of axonal branches
224 c auditory nerve terminals, although rostral auditory nerve terminals contained a greater concentrati
225 ansmission at the end-bulb synapse formed by auditory nerve terminals onto the soma of neurons in the
226 The percentage of primary auditory nerve terminals was larger in caudal AVCN, wher
227 volume fraction were similar for axosomatic auditory nerve terminals, although rostral auditory nerv
228 eak conditioned stimulus (CS) applied to the auditory nerve that immediately precedes an unconditione
229 In auditory nerve, this form of adaptation is likely mediat
230 amine the timing of action potentials in cat auditory nerve to broadband noise presented at different
231 ea activated proximal myelinated portions of auditory nerve to restore hearing.
232 tion potential that is transmitted along the auditory nerve to the cochlear nucleus in the brainstem
233 Axons are extended from the bundle of auditory nerve toward some of the new hair cells, sugges
234 (tINs) in the hindbrain, at the level of the auditory nerve, with long, ipsilateral, descending axons
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