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1 ts were change in flow-mediated dilation and augmentation index.
2 ere seen in either flow-mediated dilation or augmentation index.
3 n independent predictor of higher hs-CRP and augmentation index.
4 o influence on augmentation index (change in augmentation index, -0.4%; 95% CI, -1.7 to 0.8; P=0.5) o
5                        NO3(-) reduced aortic augmentation index (132.2+/-16.7% versus 141.4+/-21.9%;
6 ry flow reserve, (3) pulse wave velocity and augmentation index, (4) circulating NT-proBNP (N-termina
7 +/- 1.0 m/s vs. -0.1 +/- 0.9 m/s, p < 0.01), augmentation index (-5.2 +/- 6.1% vs. -1.4 +/- 5.9%, p <
8 orearm mitochondrial oxidative function, and augmentation index (a marker of arterial wave reflection
9 sterone, and endothelin (ET)-1 together with augmentation index, a measure of arterial stiffness, wer
10            The peak reductions in peripheral augmentation index after the WA, AO, and CO meals (-9.5%
11 01), AI d (r = -0.17, P = 0.06), the central augmentation index (AI c ) (r = 0.61, P < 0.001) or AI c
12  AI rd , a combination of AI r and diastolic augmentation index (AI d ) with a weight alpha, to achie
13                                       Radial augmentation index (AI r ) can be more conveniently meas
14                                              Augmentation index (AI(a)) and timing of the reflected p
15 tid-femoral pulse wave velocity (cf-PWV) and augmentation index (AI) at a steady state.
16 emoral pulse wave velocity (PWV) and carotid augmentation index (AI)].
17 t the hypothesis that age-related changes in augmentation index (AIx) are more prominent in younger i
18                                              Augmentation index (AIx) is a measure of systemic arteri
19                                              Augmentation index (AIx) is widely used as a measure of
20 pressure (BP), pulse wave velocity (PWV) and augmentation index (AIx) were assessed in 130 subjects (
21            Pulse wave velocity (PWV) and the augmentation index (AIx) were assessed noninvasively and
22 and dose on the change in blood pressure and augmentation index (AIx) were determined.
23 s, LV wall thickness and dimensions, central augmentation index (AIx), aortic pulse wave velocity (aP
24    Pulse wave analysis was used to determine augmentation index (AIx), which provides a measure of sy
25  using pulse wave velocity (PWV) and central augmentation index (AIx).
26 emoral pulse wave velocity (PWV), and aortic augmentation index (AIx).
27 % vs. placebo -0.69 +/- 2.8%; p = 0.017) and augmentation index (allopurinol -2.8 +/- 5.1% vs. placeb
28 hanges in air pollution were associated with augmentation index and augmentation pressure at several
29 t-term exposure to air pollution and central augmentation index and augmentation pressure, correlates
30 ate capabilities for measuring radial artery augmentation index and pulse pressure velocity.
31 elocity and radial tonometry-derived central augmentation index and subendocardial viability ratio we
32  arterial stiffness (pulse wave velocity and augmentation index) and blood pressure were also not sig
33 ntral pulse pressure, augmentation pressure, augmentation index, and mean arterial pressure.
34 tic modulus, impedance, pulse wave velocity, augmentation index, and pulse pressure amplification) ar
35 6 mm Hg (95% confidence interval, 2.4-20.7), augmentation index, and pulse wave velocity without chan
36                          Brachial stiffness, augmentation index, and systemic arterial compliance wer
37 -femoral pulse wave velocity (cfPWV), aortic augmentation index, and systemic arterial compliance.
38                                              Augmentation index (as %) and augmentation pressure (in
39 n of either meal significantly decreased the augmentation index at 2 and 4 h (P < 0.002) and signific
40 a and augments pressure in late systole [ie, augmentation index = (augmented pressure/pulse pressure)
41 mpliance estimates but may underestimate the augmentation index because the latter requires greater f
42 ulse wave velocity (beta = -0.09, p = 0.04), augmentation index (beta = -0.11, p = 0.03), and subendo
43 artery (CCA-IMT), pulse wave velocity (PWV), augmentation index, blood pressure (BP), and vascular bi
44 arkedly change wave reflection amplitude and augmentation index by altering stiffness of the muscular
45 tic impedance [Zc]) and late-systolic loads (augmentation index [cAI]; late pressure-time integral [P
46 y arterial pulse-wave velocity (Doppler) and augmentation index (carotid tonometry) declined with ver
47            Calculations included the carotid augmentation index, carotid artery compliance and the di
48 pulse wave velocity [PWV]), wave reflection (augmentation index, carotid-brachial pressure amplificat
49 tion, carotid artery intima-media thickness, augmentation index, central blood pressure, subendocardi
50                                 In contrast, augmentation index, central pulse pressure, and pulse pr
51 -1.9 to 1.0], P=0.6) and had no influence on augmentation index (change in augmentation index, -0.4%;
52                            HLS increased the augmentation index compared with the other test meals (P
53 ial stiffness (pulse wave velocity [PWV] and augmentation index corrected for heart rate [AI@75]) wer
54  The secondary outcomes were CFPWV, FWA, and augmentation index during examination cycle 8.
55 ex, height, weight, end-diastolic LV volume, augmentation index, end-systolic pressure, and cardiovas
56 oCor Mx system was used to derive the aortic augmentation index from radial artery pulse pressure wav
57                                      Central augmentation index improved significantly with allopurin
58 sodilatation was calculated as the change in augmentation index in response to an endothelium-depende
59 essure augmentation was determined using the augmentation index in the ascending aorta (AIaa) and dis
60  arterial stiffness (pulse wave velocity and augmentation index) in 20 adult patients with hypertensi
61                                              Augmentation index increased after transplant and was gr
62 a strong inverse relationship between HR and augmentation index, indicative of increased wave reflect
63  levels and 3 measures of vascular function (augmentation index, mean arterial pressure, and pulse pr
64 , 1.6 [95% CI, 1.3-2.0] per 1 SD; P < .001), augmentation index (OR, 1.7 [95% CI, 1.4-2.0] per 1 SD;
65 ention did not significantly change CCA-IMT, augmentation index, or BP, but pulse pressure variabilit
66 eactive protein (P(interaction) < 0.001) and augmentation index (P = 0.06) values at or above the 75t
67  had higher hs-CRP (P=0.014), higher central augmentation index (P=0.015), and lower glutathione leve
68 function (P=0.009), and improved the central augmentation index (P=0.015).
69 lood pressure, mean arterial pressure (MAP), augmentation index, pulse wave velocity (PWV), and intim
70 = [Reflected/Forward wave amplitude] x 100), augmentation index ([Second/First systolic peak] x 100)
71 rterial compliance to 6 +/- 7% accuracy, and augmentation index to within -7% points (30 +/- 45% accu
72  a decrease in pulse wave velocity (PWV) and augmentation index up to 26 h after the walk.
73 nts/cm(3) IQR increase) were associated with augmentation index values that were 0.8% (95% confidence
74               In healthy volunteers, PWV and augmentation index were associated both with black carbo
75 is cohort, higher aortic stiffness, FWA, and augmentation index were associated with higher risk of i
76                      Pulse wave velocity and augmentation index were improved only after anti-IL-12/2
77 Aortic pulse wave velocity (PWV) and carotid augmentation index were reduced only with SR (p < 0.05).
78  Six months after RD aortic augmentation and augmentation index were significantly reduced by -11 mm
79 y [CFPWV], forward wave amplitude [FWA], and augmentation index) were examined over a 7-year period i
80 ticles and PM2.5, and an increase in PWV and augmentation index with NO2 and ultrafine particles.

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