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1 ally prevented by the endonuclease inhibitor aurintricarboxylic acid.
5 usly-unreported FEN1 inhibitors, among which aurintricarboxylic acid and NSC-13755 (an arylstibonic d
6 blockers, a NO generator, N-acetylcysteine, aurintricarboxylic acid, and actinomycin D occurred at a
7 and genistein) but was inhibited by Zn(2+), aurintricarboxylic acid, and various protease inhibitors
8 OPAC compounds inhibited the FP-based assay: aurintricarboxylic acid (ATA) (IC50=1.4 muM), suramin so
9 show that treatment with the PMCA4 inhibitor aurintricarboxylic acid (ATA) inhibits and reverses card
10 Janus tyrosine kinase (JAK)-STAT signaling [aurintricarboxylic acid (ATA), (E)-2-cyano-3-(3,4-dihydr
11 single Jak-Stat pathway can be activated by aurintricarboxylic acid (ATA), a negatively charged trip
13 goal of this study was to determine whether aurintricarboxylic acid (ATA), an endonuclease inhibitor
14 ); (3) DNA laddering which can be blocked by aurintricarboxylic acid (ATA), an inhibitor of apoptosis
16 e most potent and specific YopH inhibitor is aurintricarboxylic acid (ATA), which exhibits a Ki value
17 ases by intracerebroventricular injection of aurintricarboxylic acid (ATA, 20 micrograms/5 microliter
18 2+) ions, and was inhibited by Zn(2+) and by aurintricarboxylic acid; it was not inhibited by G-actin
20 Similarly, the survival-promoting agents aurintricarboxylic acid, N-acetylcysteine, the nitric ox
21 onucleolytic activity was inhibited by Zn2+, aurintricarboxylic acid, N-ethylmaleimide, spermine, and
22 3 autocatalytic activity, suggesting that an aurintricarboxylic acid-sensitive Ced-3/ICE-related prot
23 ow concentrations (< or = 0.1 micromol/L) of aurintricarboxylic acid were capable of suppressing TPP
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