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1 e also been associated with disorders on the autism spectrum.
3 er risk for intellectual disability (ID) and autism spectrum disorder (ASD) and affects an estimated
4 t is common among individuals diagnosed with autism spectrum disorder (ASD) and attention-deficit hyp
6 LTD dysregulation in several mouse models of autism spectrum disorder (ASD) and discuss whether LTD d
8 mmon monogenic and highly penetrant cause of autism spectrum disorder (ASD) and intellectual disabili
9 communication in children with non-syndromic autism spectrum disorder (ASD) and language impairment i
12 s neurodevelopmental disorders, particularly autism spectrum disorder (ASD) and schizophrenia (SCZ).
13 orts have demonstrated a higher incidence of autism spectrum disorder (ASD) and substantially elevate
14 psychiatric disorders with high comorbidity: autism spectrum disorder (ASD) and Tourette syndrome (TS
15 the largest collection of trios (5,947) with autism spectrum disorder (ASD) available, including 282
17 oximately one quarter of individuals with an autism spectrum disorder (ASD) display self-injurious be
19 tion of emerging epigenetic information with autism spectrum disorder (ASD) genetic results may eluci
20 eviously reported that infants who developed autism spectrum disorder (ASD) had increased cerebrospin
24 the association between maternal smoking and autism spectrum disorder (ASD) in offspring have produce
51 ividuals with a clinical diagnosis of either autism spectrum disorder (ASD) or intellectual disabilit
52 d flame retardants (BFRs) is associated with autism spectrum disorder (ASD) or intellectual disabilit
53 nctional impact of novel mutations linked to autism spectrum disorder (ASD) provides a deeper mechani
54 dysfunction and the high male prevalence in autism spectrum disorder (ASD) remain poorly understood.
61 n patients with developmental delay (DD) and autism spectrum disorder (ASD) suggests a different path
62 ing whole-genome sequencing of families with autism spectrum disorder (ASD) to build a resource (MSSN
63 rrently, there is no effective treatment for autism spectrum disorder (ASD), a developmental disabili
64 a (SCZ), major depressive disorder (MDD) and autism spectrum disorder (ASD), and CACNA1D for BD and A
65 tor MEF2C regulates multiple genes linked to autism spectrum disorder (ASD), and human MEF2C haploins
66 human MET gene imparts a replicated risk for autism spectrum disorder (ASD), and is implicated in the
67 bility (ID) and are strongly associated with autism spectrum disorder (ASD), attention deficit hypera
68 ntal factors contribute to the etiologies of autism spectrum disorder (ASD), but evidence of specific
69 change are among the diagnostic criteria for autism spectrum disorder (ASD), but little research has
70 ationship between peripheral BDNF levels and autism spectrum disorder (ASD), but the findings are inc
71 largement has been observed in children with autism spectrum disorder (ASD), but the timing of this p
72 isorders such as intellectual disability and autism spectrum disorder (ASD), but their dysfunction is
73 hannel NaV1.2 are important risk factors for autism spectrum disorder (ASD), developmental delay, and
74 FMRP loss causing the most common heritable autism spectrum disorder (ASD), fragile X syndrome (FXS)
75 rks in neuropsychiatric disorders, including Autism Spectrum Disorder (ASD), has been well establishe
76 n in at least a subgroup of individuals with autism spectrum disorder (ASD), including in those with
78 and environmental factors in the etiology of autism spectrum disorder (ASD), our limited understandin
79 syndrome (FXS), a heritable intellectual and autism spectrum disorder (ASD), results from the loss of
80 pmental process are seen in individuals with autism spectrum disorder (ASD), schizophrenia and intell
81 mber variant most frequently associated with autism spectrum disorder (ASD), schizophrenia, and comor
82 Fine motor skill impairments are common in autism spectrum disorder (ASD), significantly affecting
85 rce Exchange (AGRE) cohorts of children with autism spectrum disorder (ASD), we find strong statistic
86 nal disturbances are common in children with autism spectrum disorder (ASD), we sought to define the
87 nnectivity have been extensively reported in autism spectrum disorder (ASD), while their effects on t
88 ioral phenotypes.SIGNIFICANCE STATEMENT Many autism spectrum disorder (ASD)-linked mutations disrupt
108 PTEN is mutated in a subset of children with autism spectrum disorder (ASD); however, the mechanism b
109 h improved outcomes for children with severe autism spectrum disorder (ASD); however, there are long
110 tational age (OR, 1.01 [95% CI, 0.81-1.25]), autism spectrum disorder (HR, 0.83 [95% CI, 0.62-1.13]),
111 was somewhat elevated for ADHD with comorbid autism spectrum disorder (OR, 1.76; 95% CI, 1.37-2.26).
112 4), anencephaly (AOR 2.9, 95% CI: 1.0, 8.2), autism spectrum disorder [AOR 1.3, 95% credible interval
113 .2 (one of the most common genetic causes of autism spectrum disorder [ASD]) and assess how these fea
114 npatient or outpatient clinical diagnosis of autism spectrum disorder and attention-deficit/hyperacti
115 howed a positive genetic correlation between autism spectrum disorder and cognitive function in child
116 ain comorbid developmental conditions (i.e., autism spectrum disorder and congenital malformations),
117 in 24 age- and gender-matched patients with autism spectrum disorder and control subjects ranging in
119 id-pregnancy serum samples: association with autism spectrum disorder and intellectual disability.
120 and provide insight into the pathogenesis of autism spectrum disorder and intellectual disability.
121 h interactive domain 1B (ARID1B) gene causes autism spectrum disorder and intellectual disability; ho
122 robiota to neurobehavioral diseases, such as autism spectrum disorder and major depression, drawing u
128 genes and pathways that are dysregulated in autism spectrum disorder as well as fragile X syndrome.
130 ed vs 2.14% of unexposed were diagnosed with autism spectrum disorder by age 15 years, and 12.63% of
132 d a weak but significant association between autism spectrum disorder diagnosis and increased cerebel
133 ought to determine a combined effect size of autism spectrum disorder diagnosis on different measures
134 patients display intellectual disability and autism spectrum disorder due to silencing of the X-linke
137 re our findings showed that male adults with autism spectrum disorder have regional differences in br
138 estational age OR, 1.15 [95% CI, 1.06-1.25]; autism spectrum disorder hazard ratio [HR], 2.02 [95% CI
140 lity of the parent-mediated intervention for autism spectrum disorder in south Asia (PASS) in India a
149 een described in individuals presenting with autism spectrum disorder or mild intellectual disability
152 nsight into the neural substrates underlying autism spectrum disorder social symptom severity, and fu
153 mpared with unexposed siblings (incidence of autism spectrum disorder was 3.40 per 1000 person-years
155 Tract-based spatial statistics revealed that autism spectrum disorder was associated with significant
156 vious observations of a higher risk of child autism spectrum disorder with serotonergic antidepressan
157 ed together in typical and atypical (notably autism spectrum disorder) groups: imitation, biological
159 social interaction is a defining feature of autism spectrum disorder, a neurodevelopmental disorder
160 on deficit hyperactivity disorder (ADHD) and autism spectrum disorder, although most commonly conside
161 or treating social deficits in patients with autism spectrum disorder, and common genetic variants in
162 f syndromic intellectual disability (ID) and autism spectrum disorder, and somatic mutations are link
163 populations - specifically, individuals with autism spectrum disorder, ASD - may further illuminate s
164 (2-AG) have been reported in mouse models of autism spectrum disorder, but a causal role for striatal
165 tric and neurodegenerative diseases, such as autism spectrum disorder, depression and Alzheimer's dis
167 higher rates than UK controls of symptoms of autism spectrum disorder, disinhibited social engagement
168 rios across four neuropsychiatric disorders: autism spectrum disorder, epileptic encephalopathy, inte
169 ificantly enriched for genes associated with autism spectrum disorder, giving support to the idea tha
170 or speech and language and are implicated in autism spectrum disorder, in which language function can
171 direct measures of IQ to measure symptoms of autism spectrum disorder, inattention and overactivity,
172 omatin-remodeling subunit, in short stature, autism spectrum disorder, intellectual disability, and c
173 al disorders including primary microcephaly, autism spectrum disorder, intellectual disability, and s
174 ing aggregated data from 9,246 families with autism spectrum disorder, intellectual disability, or de
175 ne of the most prevalent monogenic causes of autism spectrum disorder, making it imperative to unders
176 increased risk of small for gestational age, autism spectrum disorder, or attention-deficit/hyperacti
177 ity in the etiology across schizophrenia and autism spectrum disorder, PVI circuits are altered in th
178 oter, enriched in subgroups of children with autism spectrum disorder, reduces transcription and disr
179 es of many neurological disorders, including autism spectrum disorder, schizophrenia, and Alzheimer's
180 t (attention-deficit/hyperactivity disorder, autism spectrum disorder, schizophrenia, bipolar disorde
181 it/hyperactivity disorder, eating disorders, autism spectrum disorder, substance use disorders, anxie
182 typically developing individuals but not in autism spectrum disorder, the magnitude of social predic
183 mutations have recently been associated with autism spectrum disorder, which parallels previous evide
184 fficulties that enhance the understanding of autism spectrum disorder- and schizophrenia-related dime
209 many neurodevelopmental disorders including autism spectrum disorder; however, little is known about
220 ess in understanding the genetic etiology of autism spectrum disorders (ASD) has fueled remarkable ad
224 nly implicated factors in neurodevelopmental autism spectrum disorders (ASD), characterized, in part,
225 f heritable intellectual disability (ID) and autism spectrum disorders (ASD), Fragile X syndrome (FXS
226 tor 5 (mGluR5) have therapeutic potential in autism spectrum disorders (ASD), including tuberous scle
234 pain sensitivity is commonly associated with autism spectrum disorders (ASDs) and affects the life qu
235 stnatal onset neurological disorders such as autism spectrum disorders (ASDs) and intellectual disabi
237 dent that there is a genetic contribution to autism spectrum disorders (ASDs) and other neural disord
238 e de novo synonymous mutations identified in autism spectrum disorders (ASDs) and schizophrenia (SCZ)
239 With an onset under the age of 3 years, autism spectrum disorders (ASDs) are now understood as d
240 have been described in some individuals with autism spectrum disorders (ASDs) as well as their family
241 (LGD) mutations contribute significantly to autism spectrum disorders (ASDs) but have been primarily
243 een prenatal exposure to antidepressants and autism spectrum disorders (ASDs) in children, with incon
246 hanisms driving brain development at risk in autism spectrum disorders (ASDs) remain mostly unknown.
247 coding polymorphism has been associated with autism spectrum disorders (ASDs) within a subgroup of pa
248 sory hypersensitivity is a common symptom in autism spectrum disorders (ASDs), including fragile X sy
255 ffecting gaze behavior toward faces, such as autism spectrum disorders and congenital prosopagnosia.
256 ining 1 (PTCHD1) is mutated in patients with autism spectrum disorders and intellectual disabilities
257 hat these rearrangements are associated with autism spectrum disorders and mirror phenotypes of obesi
258 BP5 CNV found in 0.5%-1% of individuals with autism spectrum disorders and schizophrenia and whose re
266 ysiology of macrocephaly/autism syndrome and autism spectrum disorders featuring dysregulated mTOR si
268 ltural bridges shaped public discourse about autism spectrum disorders on Facebook over the course of
269 icated in neurodevelopmental disorders, like autism spectrum disorders or attention-deficit hyper-act
270 a profile of traits that have been linked to autism spectrum disorders, and are thought to encompass
271 Neurodevelopmental disorders, including autism spectrum disorders, are highly male biased, but t
272 mood affective disorders, anxiety disorders, autism spectrum disorders, attention deficit/hyperactivi
273 ntribute to many clinical disorders, such as autism spectrum disorders, corpus callosum agenesis, Jou
274 ed clusters for enrichment of schizophrenia, autism spectrum disorders, developmental delay and intel
275 able research into the genetic background of autism spectrum disorders, environmental factors are lik
276 tive immune system function is implicated in autism spectrum disorders, including Fragile X syndrome.
277 y of neurodevelopmental disorders, including autism spectrum disorders, intellectual disability and s
278 though human RIM-BPs have been implicated in autism spectrum disorders, little is known about the rol
280 understanding of the complex neurobiology of autism spectrum disorders, valid disease models are pivo
281 translation of dyrk1a, a Down syndrome- and autism spectrum disorders-related gene, is dependent on
296 ore, our finding of enrichment was unique to autism spectrum disorders; brain gene expression signatu
297 logy of neurodevelopmental disorders such as autism spectrum disorders; neurodegenerative disorders s
298 eneration of epilepsy, neuropathic pain, and autism spectrum disorders; thus, it is important to char
300 neuropsychiatric disorders like bipolar and autism spectrum where 40 Hz ASSR deficits have been docu
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