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1 retardation, Down syndrome, cerebral palsy, autism spectrum disorder).
2 been linked to neurological diseases such as autism spectrum disorder.
3 me, epilepsy, major depressive disorder, and autism spectrum disorder.
4 ocial signal was altered in individuals with autism spectrum disorder.
5 cluding schizophrenia, bipolar disorder, and autism spectrum disorder.
6 attention-deficit/hyperactivity disorder and autism spectrum disorder.
7 ation increases the likelihood of developing autism spectrum disorder.
8 sability, and the most common known cause of autism spectrum disorder.
9 n deficit/hyperactivity disorder (ADHD), and autism spectrum disorder.
10 , 1.6%-2.6%) of children were diagnosed with autism spectrum disorder.
11 delay/intellectual disability, epilepsy, and autism spectrum disorder.
12 pressant exposure during pregnancy and child autism spectrum disorder.
13 plicated as a central node of dysfunction in autism spectrum disorder.
14 ically diagnosed with fragile X syndrome and autism spectrum disorder.
15 system is overactivated in individuals with autism spectrum disorder.
16 ly presents with intellectual disability and autism spectrum disorder.
17 contrasting language performance observed in autism spectrum disorder.
18 ultiple forms of intellectual disability and autism spectrum disorder.
19 nces in white matter networks in adults with autism spectrum disorder.
20 otion recognition, relationship quality, and autism spectrum disorder.
21 cally developing children, and children with Autism Spectrum Disorder.
22 ome is the most common monogenetic cause for autism spectrum disorder.
23 rapies to help ameliorate social deficits in autism spectrum disorder.
24 area affected in both fragile X syndrome and autism spectrum disorder.
25 es or contribute to the pathogenesis of some autism spectrum disorders.
26 ritable cause of intellectual disability and autism spectrum disorders.
27 s neuromotor deficits in startle disease and autism spectrum disorders.
28 isorders and are among the most prevalent in autism spectrum disorders.
29 cial interaction is a diagnostic hallmark of autism spectrum disorders.
30 vior toward faces, such as prosopagnosia and autism spectrum disorders.
31 ing protein, Rbfox1, as a candidate gene for autism spectrum disorders.
32 ng VIPUR to de novo variants associated with autism spectrum disorders.
33 ttention deficit/hyperactivity disorder, and autism spectrum disorders.
34 the possibility of personalized medicine for autism spectrum disorders.
35 ctions with central molecular hubs linked to autism spectrum disorders.
36 ar disease states such as CHARGE syndrome or autism spectrum disorders.
37 standing DA developmental conditions such as autism spectrum disorders.
38 d to human hyperekplexia/startle disease and autism spectrum disorders.
39 social interaction is a defining feature of autism spectrum disorder, a neurodevelopmental disorder
41 on deficit hyperactivity disorder (ADHD) and autism spectrum disorder, although most commonly conside
42 npatient or outpatient clinical diagnosis of autism spectrum disorder and attention-deficit/hyperacti
43 howed a positive genetic correlation between autism spectrum disorder and cognitive function in child
44 ain comorbid developmental conditions (i.e., autism spectrum disorder and congenital malformations),
45 in 24 age- and gender-matched patients with autism spectrum disorder and control subjects ranging in
47 id-pregnancy serum samples: association with autism spectrum disorder and intellectual disability.
48 and provide insight into the pathogenesis of autism spectrum disorder and intellectual disability.
49 h interactive domain 1B (ARID1B) gene causes autism spectrum disorder and intellectual disability; ho
50 robiota to neurobehavioral diseases, such as autism spectrum disorder and major depression, drawing u
56 ffecting gaze behavior toward faces, such as autism spectrum disorders and congenital prosopagnosia.
57 ining 1 (PTCHD1) is mutated in patients with autism spectrum disorders and intellectual disabilities
58 hat these rearrangements are associated with autism spectrum disorders and mirror phenotypes of obesi
59 BP5 CNV found in 0.5%-1% of individuals with autism spectrum disorders and schizophrenia and whose re
64 or treating social deficits in patients with autism spectrum disorder, and common genetic variants in
65 f syndromic intellectual disability (ID) and autism spectrum disorder, and somatic mutations are link
66 a profile of traits that have been linked to autism spectrum disorders, and are thought to encompass
67 fficulties that enhance the understanding of autism spectrum disorder- and schizophrenia-related dime
68 4), anencephaly (AOR 2.9, 95% CI: 1.0, 8.2), autism spectrum disorder [AOR 1.3, 95% credible interval
73 genes and pathways that are dysregulated in autism spectrum disorder as well as fragile X syndrome.
74 er risk for intellectual disability (ID) and autism spectrum disorder (ASD) and affects an estimated
75 t is common among individuals diagnosed with autism spectrum disorder (ASD) and attention-deficit hyp
77 LTD dysregulation in several mouse models of autism spectrum disorder (ASD) and discuss whether LTD d
79 mmon monogenic and highly penetrant cause of autism spectrum disorder (ASD) and intellectual disabili
80 communication in children with non-syndromic autism spectrum disorder (ASD) and language impairment i
83 s neurodevelopmental disorders, particularly autism spectrum disorder (ASD) and schizophrenia (SCZ).
84 orts have demonstrated a higher incidence of autism spectrum disorder (ASD) and substantially elevate
85 psychiatric disorders with high comorbidity: autism spectrum disorder (ASD) and Tourette syndrome (TS
86 the largest collection of trios (5,947) with autism spectrum disorder (ASD) available, including 282
88 oximately one quarter of individuals with an autism spectrum disorder (ASD) display self-injurious be
90 tion of emerging epigenetic information with autism spectrum disorder (ASD) genetic results may eluci
91 eviously reported that infants who developed autism spectrum disorder (ASD) had increased cerebrospin
95 the association between maternal smoking and autism spectrum disorder (ASD) in offspring have produce
122 ividuals with a clinical diagnosis of either autism spectrum disorder (ASD) or intellectual disabilit
123 d flame retardants (BFRs) is associated with autism spectrum disorder (ASD) or intellectual disabilit
124 nctional impact of novel mutations linked to autism spectrum disorder (ASD) provides a deeper mechani
125 dysfunction and the high male prevalence in autism spectrum disorder (ASD) remain poorly understood.
132 n patients with developmental delay (DD) and autism spectrum disorder (ASD) suggests a different path
133 ing whole-genome sequencing of families with autism spectrum disorder (ASD) to build a resource (MSSN
134 rrently, there is no effective treatment for autism spectrum disorder (ASD), a developmental disabili
135 a (SCZ), major depressive disorder (MDD) and autism spectrum disorder (ASD), and CACNA1D for BD and A
136 tor MEF2C regulates multiple genes linked to autism spectrum disorder (ASD), and human MEF2C haploins
137 human MET gene imparts a replicated risk for autism spectrum disorder (ASD), and is implicated in the
138 bility (ID) and are strongly associated with autism spectrum disorder (ASD), attention deficit hypera
139 ntal factors contribute to the etiologies of autism spectrum disorder (ASD), but evidence of specific
140 change are among the diagnostic criteria for autism spectrum disorder (ASD), but little research has
141 ationship between peripheral BDNF levels and autism spectrum disorder (ASD), but the findings are inc
142 largement has been observed in children with autism spectrum disorder (ASD), but the timing of this p
143 isorders such as intellectual disability and autism spectrum disorder (ASD), but their dysfunction is
144 hannel NaV1.2 are important risk factors for autism spectrum disorder (ASD), developmental delay, and
145 FMRP loss causing the most common heritable autism spectrum disorder (ASD), fragile X syndrome (FXS)
146 rks in neuropsychiatric disorders, including Autism Spectrum Disorder (ASD), has been well establishe
147 n in at least a subgroup of individuals with autism spectrum disorder (ASD), including in those with
149 and environmental factors in the etiology of autism spectrum disorder (ASD), our limited understandin
150 syndrome (FXS), a heritable intellectual and autism spectrum disorder (ASD), results from the loss of
152 pmental process are seen in individuals with autism spectrum disorder (ASD), schizophrenia and intell
153 mber variant most frequently associated with autism spectrum disorder (ASD), schizophrenia, and comor
154 Fine motor skill impairments are common in autism spectrum disorder (ASD), significantly affecting
157 rce Exchange (AGRE) cohorts of children with autism spectrum disorder (ASD), we find strong statistic
158 nal disturbances are common in children with autism spectrum disorder (ASD), we sought to define the
159 nnectivity have been extensively reported in autism spectrum disorder (ASD), while their effects on t
160 ioral phenotypes.SIGNIFICANCE STATEMENT Many autism spectrum disorder (ASD)-linked mutations disrupt
181 PTEN is mutated in a subset of children with autism spectrum disorder (ASD); however, the mechanism b
182 h improved outcomes for children with severe autism spectrum disorder (ASD); however, there are long
192 ess in understanding the genetic etiology of autism spectrum disorders (ASD) has fueled remarkable ad
195 etween labor induction and offspring risk of autism spectrum disorders (ASD) reflected causal influen
197 nly implicated factors in neurodevelopmental autism spectrum disorders (ASD), characterized, in part,
198 f heritable intellectual disability (ID) and autism spectrum disorders (ASD), Fragile X syndrome (FXS
199 tor 5 (mGluR5) have therapeutic potential in autism spectrum disorders (ASD), including tuberous scle
207 .2 (one of the most common genetic causes of autism spectrum disorder [ASD]) and assess how these fea
208 populations - specifically, individuals with autism spectrum disorder, ASD - may further illuminate s
209 pain sensitivity is commonly associated with autism spectrum disorders (ASDs) and affects the life qu
210 stnatal onset neurological disorders such as autism spectrum disorders (ASDs) and intellectual disabi
212 dent that there is a genetic contribution to autism spectrum disorders (ASDs) and other neural disord
213 e de novo synonymous mutations identified in autism spectrum disorders (ASDs) and schizophrenia (SCZ)
214 With an onset under the age of 3 years, autism spectrum disorders (ASDs) are now understood as d
215 have been described in some individuals with autism spectrum disorders (ASDs) as well as their family
216 (LGD) mutations contribute significantly to autism spectrum disorders (ASDs) but have been primarily
218 een prenatal exposure to antidepressants and autism spectrum disorders (ASDs) in children, with incon
221 hanisms driving brain development at risk in autism spectrum disorders (ASDs) remain mostly unknown.
222 coding polymorphism has been associated with autism spectrum disorders (ASDs) within a subgroup of pa
223 sory hypersensitivity is a common symptom in autism spectrum disorders (ASDs), including fragile X sy
230 mood affective disorders, anxiety disorders, autism spectrum disorders, attention deficit/hyperactivi
232 ore, our finding of enrichment was unique to autism spectrum disorders; brain gene expression signatu
233 (2-AG) have been reported in mouse models of autism spectrum disorder, but a causal role for striatal
234 ed vs 2.14% of unexposed were diagnosed with autism spectrum disorder by age 15 years, and 12.63% of
236 ntribute to many clinical disorders, such as autism spectrum disorders, corpus callosum agenesis, Jou
237 tric and neurodegenerative diseases, such as autism spectrum disorder, depression and Alzheimer's dis
239 ed clusters for enrichment of schizophrenia, autism spectrum disorders, developmental delay and intel
240 d a weak but significant association between autism spectrum disorder diagnosis and increased cerebel
241 ought to determine a combined effect size of autism spectrum disorder diagnosis on different measures
242 higher rates than UK controls of symptoms of autism spectrum disorder, disinhibited social engagement
243 patients display intellectual disability and autism spectrum disorder due to silencing of the X-linke
244 able research into the genetic background of autism spectrum disorders, environmental factors are lik
245 rios across four neuropsychiatric disorders: autism spectrum disorder, epileptic encephalopathy, inte
246 ysiology of macrocephaly/autism syndrome and autism spectrum disorders featuring dysregulated mTOR si
247 ificantly enriched for genes associated with autism spectrum disorder, giving support to the idea tha
249 ed together in typical and atypical (notably autism spectrum disorder) groups: imitation, biological
251 re our findings showed that male adults with autism spectrum disorder have regional differences in br
252 estational age OR, 1.15 [95% CI, 1.06-1.25]; autism spectrum disorder hazard ratio [HR], 2.02 [95% CI
253 many neurodevelopmental disorders including autism spectrum disorder; however, little is known about
255 tational age (OR, 1.01 [95% CI, 0.81-1.25]), autism spectrum disorder (HR, 0.83 [95% CI, 0.62-1.13]),
258 lity of the parent-mediated intervention for autism spectrum disorder in south Asia (PASS) in India a
261 or speech and language and are implicated in autism spectrum disorder, in which language function can
262 direct measures of IQ to measure symptoms of autism spectrum disorder, inattention and overactivity,
264 tive immune system function is implicated in autism spectrum disorders, including Fragile X syndrome.
265 omatin-remodeling subunit, in short stature, autism spectrum disorder, intellectual disability, and c
266 al disorders including primary microcephaly, autism spectrum disorder, intellectual disability, and s
267 ing aggregated data from 9,246 families with autism spectrum disorder, intellectual disability, or de
268 y of neurodevelopmental disorders, including autism spectrum disorders, intellectual disability and s
272 though human RIM-BPs have been implicated in autism spectrum disorders, little is known about the rol
273 ne of the most prevalent monogenic causes of autism spectrum disorder, making it imperative to unders
275 logy of neurodevelopmental disorders such as autism spectrum disorders; neurodegenerative disorders s
276 ltural bridges shaped public discourse about autism spectrum disorders on Facebook over the course of
278 een described in individuals presenting with autism spectrum disorder or mild intellectual disability
280 icated in neurodevelopmental disorders, like autism spectrum disorders or attention-deficit hyper-act
281 was somewhat elevated for ADHD with comorbid autism spectrum disorder (OR, 1.76; 95% CI, 1.37-2.26).
282 increased risk of small for gestational age, autism spectrum disorder, or attention-deficit/hyperacti
283 ity in the etiology across schizophrenia and autism spectrum disorder, PVI circuits are altered in th
284 oter, enriched in subgroups of children with autism spectrum disorder, reduces transcription and disr
286 translation of dyrk1a, a Down syndrome- and autism spectrum disorders-related gene, is dependent on
288 es of many neurological disorders, including autism spectrum disorder, schizophrenia, and Alzheimer's
289 t (attention-deficit/hyperactivity disorder, autism spectrum disorder, schizophrenia, bipolar disorde
290 nsight into the neural substrates underlying autism spectrum disorder social symptom severity, and fu
291 it/hyperactivity disorder, eating disorders, autism spectrum disorder, substance use disorders, anxie
292 typically developing individuals but not in autism spectrum disorder, the magnitude of social predic
294 eneration of epilepsy, neuropathic pain, and autism spectrum disorders; thus, it is important to char
295 understanding of the complex neurobiology of autism spectrum disorders, valid disease models are pivo
296 mpared with unexposed siblings (incidence of autism spectrum disorder was 3.40 per 1000 person-years
298 Tract-based spatial statistics revealed that autism spectrum disorder was associated with significant
299 mutations have recently been associated with autism spectrum disorder, which parallels previous evide
300 vious observations of a higher risk of child autism spectrum disorder with serotonergic antidepressan
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