ライフサイエンスコーパス: autoantibody-positive

1 gative first-degree relatives who were never autoantibody positive.

2 wly diagnosed IDDM patients tested were IA-2 autoantibody positive.

3 ually diminished in alpha-cells of NOD mice, autoantibody-positive (AA(+)) and overtly type 1 diabeti

4 In contrast, autoantibody positive (Ab+) type 2 diabetic patients (n

5 time in situ, that in pancreas sections from autoantibody-positive (Ab+) donors, insulin area and bet

6 e predictive power of autoantibodies because autoantibody-positive (AbP) children have heterogeneous

7 b in the treatment of acetylcholine receptor autoantibody-positive (AChR+) generalized MG.

8 (HLA) alleles for anti-citrullinated-protein-autoantibody-positive (ACPA(+)) rheumatoid arthritis (RA

9 e polymorphisms (SNPs) were genotyped in 556 autoantibody-positive African Americans with RA and 791

10 No differences between anti-islet cell autoantibody-positive and -negative children were found

11 ent of juvenile DM in both myositis-specific autoantibody-positive and -negative patients.

12 The magnitude of association was assessed in autoantibody-positive and -negative subgroups.

13 be useful in predicting disease severity in autoantibody-positive and autoantibody-negative patients

14 ucleatum) were significantly associated with autoantibody-positive and high-risk status (P < 0.05).

15 ears, associations that were evident both in autoantibody-positive and in autoantibody-negative disea

16 We compared anti-U11/U12 RNP autoantibody-positive and negative SSc patients on demog

17 n of CD8 T cells in pancreata from T1D, T2D, autoantibody-positive, and healthy control subjects.

18 in children with newly diagnosed T1D and in autoantibody-positive at-risk children with impaired glu

19 atives followed to diabetes were ICA512/IA-2 autoantibody-positive but anti-phogrin autoantibody-nega

20 In preclinical autoantibody-positive cases, the number of elevated cyto

21 ntribute to type 1 diabetes (T1D) risk among autoantibody-positive children in The Environmental Dete

22 tetramer staining in 6 patients and 10 islet autoantibody-positive children showed large diversity wi

23 We studied 28 autoantibody-positive children, out of which 22 progress

24 gnificantly higher in pretransplantation GAD autoantibody-positive daclizumab-treated recipients comp

25 Autoantibody-positive diabetic subjects (n = 1,384) and

26 hat T1D is more similar genetically to other autoantibody-positive diseases, significantly most simil

27 levels of interleukin-4 (IL-4) in anti-islet autoantibody-positive first-degree relatives of patients

28 complement factor H-related (CFHR) genes and autoantibody-positive form of hemolytic uremic syndrome

29 ons were higher in high-risk (P = 0.011) and autoantibody positive group (P = 0.010) than in the auto

30 alysis suggested that the gut microbiomes of autoantibody-positive individuals and seronegative FDRs

31 beta-cells in pancreatic sections from GAD65 autoantibody-positive individuals who have not yet progr

32 ide association study meta-analysis of 5,539 autoantibody-positive individuals with rheumatoid arthri

33 Individuals were considered autoantibody positive (n = 113) if they had >/=1 RA-rela

34 ed whether immunoglobulin G from aquaporin-4-autoantibody-positive neuromyelitis optica patients has

35 antibodies on 2 consecutive visits and still autoantibody positive or having diabetes at last follow-

36 h focal accumulation in the muscle fibers of autoantibody-positive patients compared with a homogeneo

37 Immunoglobulin G from autoantibody-positive patients induced hyperpolarization

38 estive evidence of a stronger association in autoantibody-positive patients with RA (OR 0.55, 95% con

39 king is associated not only with the risk of autoantibody-positive RA but also with the risk of autoa

40 enome-wide association study (GWAS) of 5,539 autoantibody-positive RA cases and 20,169 controls.

41 of 33 [42%]; 3.8+/-4.5 at 10 microg/ml) and autoantibody-positive relatives at increased risk for ID

42 l insulin delays onset of type 1 diabetes in autoantibody-positive relatives of patients with type 1

43 Among autoantibody-positive relatives of patients with type 1

44 We examined a cohort of 3,358 autoantibody-positive relatives of T1D patients in the P

45 ly regulated (interleukin-10-positive, pauci-autoantibody-positive) responses.

46 ritis in an independent replication of 3,929 autoantibody-positive rheumatoid arthritis cases and 5,8

47 toantibodies in 88 dual (ICA512 and phogrin) autoantibody-positive sera could be completely blocked b

48 No autoantibody-positive subject had inflammatory arthritis

49 HRCT revealed that 76% of autoantibody-positive subjects had airways abnormalities

50 The median age of autoantibody-positive subjects was 54 years, 52% were fe

51 valence and type of lung abnormalities among autoantibody-positive subjects were similar to those amo

52 In 2 autoantibody-positive subjects with airways disease, inf

53 e consistent with inflammation are common in autoantibody-positive subjects without inflammatory arth

54 lls in early stages of T1D (i.e., in healthy autoantibody-positive subjects) and in more advanced pha

55 edicts a low first-phase insulin response in autoantibody-positive subjects.

56 iagnosed patients ages 1-18 years with islet autoantibody-positive type 1 diabetes (n = 970) and cont