ライフサイエンスコーパス: autofluorescence

1 oteins and lipids, which are also sources of autofluorescence.

2 ed tissue scattering, and diminishing tissue autofluorescence.

3 of poorly demarcated questionably decreased autofluorescence.

4 al separation, less phototoxicity, and lower autofluorescence.

5 e-gated detection is not needed to avoid the autofluorescence.

6 e in assessing areas of definitely decreased autofluorescence.

7 ated combined NADH and NADPH (i.e., NAD(P)H) autofluorescence.

8 ally overlapped immunofluorescence and Abeta autofluorescence.

9 of atrophic lesions as determined by fundus autofluorescence.

10 for poorly demarcated questionably decreased autofluorescence.

11 onic generation (SHG) and two photon excited autofluorescence.

12 bly because of macular pigmentation blocking autofluorescence.

13 ion completely eliminates the Stokes-shifted autofluorescence.

14 ain optical coherence tomography, and fundus autofluorescence.

15 al sensitivity, corresponded with absence of autofluorescence.

16 bility on funduscopy, ultrasound, and fundus autofluorescence.

17 istinct appearance and without intense hyper-autofluorescence.

18 t overcomes issues of background signals and autofluorescence.

19 rescence surrounded by a round area of hyper-autofluorescence.

20 ent mice were examined by immunostaining and autofluorescence.

21 lots), SD-OCT, and sometimes mfERG or fundus autofluorescence.

22 ue to deep tissue penetration and low tissue autofluorescence.

23 dative metabolism as was followed by NAD(P)H autofluorescence.

24 near-infrared FAF pattern, with central hypo-autofluorescence.

25 acuity was baseline area of confluent absent autofluorescence.

26 n vivo imaging because they eliminate tissue autofluorescence.

27 poor tissue penetration and high background autofluorescence.

28 /or the interference generated by biological autofluorescence.

29 ference associated with light scattering and autofluorescence.

30 er of a paracentral ring of increased fundus autofluorescence.

31 rowth of atrophic lesions measured by fundus autofluorescence.

32 y useful property of e-liquids, namely their autofluorescence.

33 and other undesirable effects such as tissue autofluorescence.

34 ividuals (24%) showed only reduced or absent autofluorescence, 3 patients (18%) displayed only increa

35 ent autofluorescence surrounded by increased autofluorescence, 4 individuals (24%) showed only reduce

36 All patients demonstrated autofluorescence abnormalities in the fovea and/or paraf

37 fundus of hCAPN5(R243L) mice showed enhanced autofluorescence (AF) and pigment changes indicative of

38 Autofluorescence (AF) images (30 degrees , 488-nm excita

39 ry image was compared with the corresponding autofluorescence (AF) images at 488 nm (SW-AF) and at 78

40 acuity (BCVA), widefield angiography, fundus autofluorescence (AF), and wnt signaling pathway mutatio

41 computed tomography detecting near-infrared autofluorescence allows in vivo monitoring of intraplaqu

42 cence imaging demonstrated foci of increased autofluorescence along the arcades.

43 A transient increase in autofluorescence also occurred.

44 also observed noninvasively by quantitative autofluorescence, an imaging technique used clinically,

45 .964 (0.929, 0.999) for definitely decreased autofluorescence and 0.268 (0.000, 0.730) for poorly dem

46 s overlap with endogenous activity-dependent autofluorescence and are affected by changes in blood vo

47 ere studied by evaluating areas of decreased autofluorescence and areas of increased autofluorescence

48 Fundus autofluorescence and electrophysiological testing (ERG a

49 ging and analysis (0.1-1 Hz frequency band), autofluorescence and hemodynamic effects contributed 3%

50 ajor challenges that still remain are tissue autofluorescence and hemoglobin absorption, which act to

51 better photon penetration, diminished tissue autofluorescence and high contrasts, its molecular mass

52 arising from non-specific bindings, material autofluorescence and leakage of excitation light, which

53 light (anti-Stokes emission) which prevents autofluorescence and light scattering of biological samp

54 robes for in vivo imaging conditions of high autofluorescence and low signal levels.

55 ical coherence tomography (along with fundus autofluorescence and multifocal electroretinography as i

56 mography features and staging system, fundus autofluorescence and near-infrared reflectance features

57 Fundus autofluorescence and optical coherence tomography detect

58 rm needs no external light source and avoids autofluorescence and photobleaching, and target molecule

59 e near-infrared region, where reduced tissue autofluorescence and photon attenuation enable subsurfac

60 uorescent lesion types (definitely decreased autofluorescence and poorly demarcated questionably decr

61 Tissues were imaged using multiphoton autofluorescence and second harmonic generation microsco

62 best-corrected visual acuity (BCVA), fundus autofluorescence and spectral domain-optical coherence t

63 ated the multimodal imaging including fundus autofluorescence and spectral-domain optical coherence t

64 ule can be successfully used to image tissue autofluorescence and targeted fluorescence via fluoropho

65 e, 3 patients (18%) displayed only increased autofluorescence, and 1 individual (6%) exhibited decrea

66 foveal attenuation normally seen with fundus autofluorescence, and a concentric macular rings reflex

67 ng funduscopy, optical coherence tomography, autofluorescence, and angiography.

68 d reflectance, hypoautofluorescent on fundus autofluorescence, and as subretinal deposits on spectral

69 rve head (ONH), infrared reflectance, fundus autofluorescence, and color fundus photographs (CFP).

70 3 in lysosomes, decreases lipofuscin-related autofluorescence, and eliminates giant lipid-containing

71 , optical coherence tomography (OCT), fundus autofluorescence, and fundus photography.

72 photography, fluorescein angiography, fundus autofluorescence, and high-resolution optical coherence

73 g optical coherence tomography (OCT), fundus autofluorescence, and indocyanine green and fluorescein

74 n is challenging due to the plant cell wall, autofluorescence, and low effector abundance.

75 inical funduscopy as well as by pseudocolor, autofluorescence, and OCT imaging.

76 , and full-field electroretinography, fundus autofluorescence, and optical coherence tomography findi

77 photography, fluorescein angiography, fundus autofluorescence, and optical coherence tomography, at a

78 Color fundus photographs, fundus autofluorescence, and spectral-domain OCT were obtained,

79 infrared reflectance, red-free reflectance, autofluorescence, and spectral-domain optical coherence

80 n each image volume and the average collagen autofluorescence are significantly correlated with colla

81 photobleaching, photoblinking and background autofluorescence are unique and an added benefit when us

82 In the live animal, tissue autofluorescence arises from a number of biologically im

83 he nano-MOF emission signal and the cellular autofluorescence arising from biological material.

84 could produce a cross-shaped increase fundus autofluorescence artifact on subsequent imaging.

85 ased autofluorescence and areas of increased autofluorescence as a measure of retinal pigment epithel

86 time-delay imaging, thus eliminating tissue autofluorescence associated with fluorescence imaging.

87 luorescence (DDAF and questionably decreased autofluorescence) at first visit was 2.6 (2.8) mm2.

88 Autofluorescence, attributed to nicotinamide adenine din

89 or fundus and optic disc photography, fundus autofluorescence, automated perimetry, and optical coher

90 of increased autofluorescence on blue fundus autofluorescence (B-FAF).

91 g for studying cell biology under a cellular autofluorescence background and with a long observation

92 However, because of the high autofluorescence background of many tissue samples, it i

93 visualized, is made less sensitive by tissue autofluorescence background, and is usually incompatible

94 photography, near-infrared reflectance, blue autofluorescence, blue reflectance, and spectral-domain

95 enerally correlated with the area of reduced autofluorescence, but hyperautofluorescence extended int

96 after cell fixation can effectively suppress autofluorescence, but this approach is not conducive to

97 ia in COS-7 cells and showed that background autofluorescence can be identified through its distinct

98 ther achieved if the detection background of autofluorescence can be removed.

99 onclusion, our data suggest that e-cigarette autofluorescence can be used as a marker of e-cigarette

100 f-target binding of FISH probes and cellular autofluorescence-can become limiting in a number of impo

101 Because autofluorescence changes with metabolic state, it can be

102 Conjunctival UV autofluorescence (CUVAF) photography was developed to de

103 The total mean (SD) area of decreased autofluorescence (DDAF and questionably decreased autofl

104 Areas of definitely decreased autofluorescence (DDAF) and questionably decreased autof

105 Areas of definitely decreased autofluorescence (DDAF) and questionably decreased autof

106 study eyes had areas of definitely decreased autofluorescence (DDAF) with an average lesion area of 2

107 The autofluorescence decayed rapidly from the baseline immed

108 -wavelength light resulted in reduced fundus autofluorescence, decreased HPLC-quantified A2E, outer n

109 to fluorescent microscopy and found (i) that autofluorescence differs widely between e-liquids, (ii)

110 Fundus autofluorescence disclosed hypoautofluorescence (n = 18)

111 spite apparent differences in morphology and autofluorescence emission with traditional two-photon mi

112 both navigate to nodules and also to perform autofluorescence endomicroscopy.

113 starch hydrolysis was followed by tryptophan autofluorescence (excitation at 280 nm, emission filter

114 han 6 months) on biomicroscopic examination, autofluorescence, FA, ICGA, and OCT.

115 rgement of the atrophic lesions using fundus autofluorescence (FAF) and color fundus photography (CFP

116 ce tomography (OCT), OCT-Angiography, fundus autofluorescence (FAF) and fluorescein-angiography (FA).

117 py, and fundus photography, including fundus autofluorescence (FAF) and near-infrared reflectance ima

118 corroboration for visual fields, and fundus autofluorescence (FAF) can show damage topographically.

119 Fundus autofluorescence (FAF) decays were detected in short (49

120 orithm and correlated with SD OCT and fundus autofluorescence (FAF) findings.

121 Eyes were evaluated on fundus autofluorescence (FAF) for GA.

122 The 200 degrees pseudocolor and fundus autofluorescence (FAF) images were captured on the Optos

123 olume scans centered at the fovea and fundus autofluorescence (FAF) images were obtained.

124 Fundus autofluorescence (FAF) imaging and optical coherence tom

125 graphy, optical coherence tomography, fundus autofluorescence (FAF) imaging, and audiologic and vesti

126 n optical coherence tomography (OCT), fundus autofluorescence (FAF) imaging, Humphrey visual field (H

127 graphy, fluorescein angiography (FA), fundus autofluorescence (FAF) imaging, optical coherence tomogr

128 ography, near-infrared (NIR) imaging, fundus autofluorescence (FAF) imaging, spectral domain optical

129 xamination, color fundus photography, fundus autofluorescence (FAF) imaging, spectral-domain (SD) opt

130 gement from baseline as assessed with fundus autofluorescence (FAF) imaging.

131 acteristics of GA were examined using fundus autofluorescence (FAF) imaging.

132 ical coherence tomography (SDOCT) and fundus autofluorescence (FAF) imaging.

133 cal coherence tomography (SD OCT) and fundus autofluorescence (FAF) imaging.

134 marily by color fundus photography or fundus autofluorescence (FAF) imaging.

135 ptical coherence tomography (SD OCT), fundus autofluorescence (FAF), and fluorescein angiography/indo

136 ptical coherence tomography (SD-OCT), fundus autofluorescence (FAF), and infrared reflectance (IR) to

137 nventional multimodal imaging (color, fundus autofluorescence (FAF), and infrared reflectance [IR] im

138 rared (NIR) and short-wavelength (SW) fundus autofluorescence (FAF), and NIR reflectance (REF).

139 with automated visual fields (AVFs), fundus autofluorescence (FAF), and optical coherence tomography

140 gether with color fundus photography, fundus autofluorescence (FAF), and spectral-domain optical cohe

141 or fundus photography (CFP), confocal fundus autofluorescence (FAF), confocal near-infrared reflectan

142 phthalmological examination including fundus autofluorescence (FAF), dynamic simultaneous fluorescein

143 , ophthalmoscopy, fundus photography, fundus autofluorescence (FAF), fluorescein angiography (FA), sp

144 photography, fluorescein angiography, fundus autofluorescence (FAF), near-infrared reflectance, and s

145 olor and red-free fundus photography, fundus autofluorescence (FAF), near-infrared reflectance, Multi

146 ptical coherence tomography (SD OCT), fundus autofluorescence (FAF), or multifocal electroretinograph

147 for at least 1 examination modality: fundus autofluorescence (FAF), spectral-domain (SD) optical coh

148 isual acuity (BCVA) determination and fundus autofluorescence (FAF).

149 yes were examined longitudinally with fundus autofluorescence (FAF; excitation wavelength, 488 nm; em

150 tical coherence tomography [OCT], and fundus autofluorescence [FAF]) then were reviewed.

151 In the area of the albipunctate spots, autofluorescence FAOSLO images (excitation, 561 nm; emis

152 em of SC lesions based on SS-OCTA and fundus autofluorescence findings.

153 B-scan ultrasonography, fundus photography, autofluorescence, fluorescein angiography (FA), optical

154 imaging, including color photographs, fundus autofluorescence, fluorescein angiography, and indocyani

155 hments based on clinical examination, fundus autofluorescence, fluorescein angiography, and optical c

156 opathy based on clinical examination, fundus autofluorescence, fluorescein angiography, and spectral-

157 usly unattainable levels of sensitivity, and autofluorescence-free imaging.

158 er it expressed, even in the specimens where autofluorescence from environment severely interferes fl

159 ology for the effective separation of tissue autofluorescence from extrinsic fluorophores, based on t

160 ng intrinsically expressed GFP fluorescence, autofluorescence from Flavin proteins, and exogenous ant

161 agellate fresh water green alga, but intense autofluorescence from photosynthesis pigments has hinder

162 pid contents in living algae, despite strong autofluorescence from the chloroplasts, at approximately

163 orescence and various types of adventitious "autofluorescence" from other molecules in the system bei

164 and retinal imaging by OCT, pseudocolor, and autofluorescence fundus photography.

165 nce including beta-galactosidase expression, autofluorescence, growth inhibition, and ATM pathway act

166 hyperfluorescent GA border zones, histologic autofluorescence (HAF) was measured at defined stages of

167 In recent years, autofluorescence has become an important diagnostic tool

168 giography, near-infrared reflectance, fundus autofluorescence, high-resolution OCT, and ultrawide-fie

169 eye at the most recent visit, and (2) fundus autofluorescence images for at least 2 visits with a min

170 A series of 3 fundus autofluorescence images using 3 different acquisition pa

171 Fundus autofluorescence images were not available.

172 tra-widefield (up to 200 degrees ) color and autofluorescence images were obtained using the Optos P2

173 patients, 215 had at least 2 gradable fundus autofluorescence images with atrophic lesion(s) present

174 escent AZOOR line in short-wavelength fundus autofluorescence images, delineating the peripapillary l

175 examination, Goldmann visual fields, fundus autofluorescence imaging (FAF), optical coherence tomogr

176 Fundus autofluorescence imaging and optical coherence tomograph

177 ed to hyperautofluorescent lesions on fundus autofluorescence imaging and subretinal hyperreflective

178 sease progression was followed by performing autofluorescence imaging at semi-regular intervals.

179 al retinal appearance, although their fundus autofluorescence imaging demonstrated foci of increased

180 coherence tomography and to describe fundus autofluorescence imaging in this condition.

181 We suggest that near-infrared autofluorescence imaging is a novel technology that allo

182 Fundus autofluorescence imaging is abnormal in children with a

183 The electrophysiological and fundus autofluorescence imaging presented here should facilitat

184 Fundus autofluorescence imaging remained normal.

185 reduced-illuminance and conventional fundus autofluorescence imaging showed good concordance in asse

186 Fundus autofluorescence imaging was used to evaluate GA progres

187 (CFP), near-infrared reflectance, and fundus autofluorescence imaging were performed in all participa

188 l responses were measured using flavoprotein autofluorescence imaging, and ageing-related changes in

189 in and indocyanine green angiography, fundus autofluorescence imaging, and corresponding eye-tracked

190 n optical coherence tomography (OCT), fundus autofluorescence imaging, and electroretinogram (ERG) re

191 ctroretinography, fundus photography, fundus autofluorescence imaging, and optical coherence tomograp

192 ally by wide-field color photography, fundus autofluorescence imaging, and spectral-domain optical co

193 examination, fundus photography, and fundus autofluorescence imaging, and visual function was assess

194 ed color photography, red-free imaging, blue autofluorescence imaging, fluorescein angiography, indoc

195 red-free images and blue reflectance, fundus autofluorescence imaging, indocyanine green angiography

196 ed chromoendoscopy, optical chromoendoscopy, autofluorescence imaging, or confocal laser endomicrosco

197 examination, wide-field photography, fundus autofluorescence imaging, sedated electroretinography, o

198 d eye examination, color fundus photography, autofluorescence imaging, spectral-domain optical cohere

199 th marked loss of autofluorescence on fundus autofluorescence imaging.

200 IR reflectance and hypofluorescent on fundus autofluorescence imaging.

201 The dots were hyperautofluorescent on fundus autofluorescence imaging.

202 ell 100 hue test, visual acuity testing, and autofluorescence imaging.

203 a continuous hyperfluorescent ring on fundus autofluorescence imaging.

204 ain optical coherence tomography, and fundus autofluorescence imaging.

205 tical coherence tomography (OCT), and fundus autofluorescence imaging.

206 th Stargardt disease as determined by fundus autofluorescence imaging.

207 ed a striking leopard-spot pattern on fundus autofluorescence imaging.

208 We observed less progression of decreased autofluorescence in 4 out of 5 light-protected eyes rela

209 brother presented with irregular patterns of autofluorescence in both eyes characterized by concentri

210 opters) and area of conjunctival ultraviolet autofluorescence in mm(2).

211 The reduced progression of decreased autofluorescence in the light-protected eyes suggests th

212 fuscin, or aging pigment, is accreted as red autofluorescence in the lysosomes of motor neuron cell b

213 gical hydrogel, exhibiting new green and red autofluorescence in vitro and in vivo without the use of

214 fundus photography, ultrasonography, fundus autofluorescence, infrared reflectance (IR) imaging, and

215 imodal imaging including fundus photography, autofluorescence, infrared reflectance, ultrasonography,

216 d pattern), visual evoked potentials, fundus autofluorescence IRR, and optical coherence tomography (

217 Here we show that near-infrared autofluorescence is associated with the presence of intr

218 was larger than both the ONL-slab and fundus autofluorescence lesion areas.

219 er, thermoplastics are also known to exhibit autofluorescence levels that may hinder their utility fo

220 Stargardt disease with DDAF lesions, fundus autofluorescence may serve as a monitoring tool for inte

221 Areas of decreased autofluorescence may serve as a useful biomarker for mea

222 splasia using in vivo volumetric multiphoton autofluorescence microscopy and second harmonic generati

223 ophy were examined with light, electron, and autofluorescence microscopy.

224 -domain optical coherence tomography, fundus autofluorescence, multifocal electroretinography, visual

225 and followed wound closure up to 6 hours by autofluorescence multiphoton microscopy.

226 for poorly demarcated questionably decreased autofluorescence (n = 12).

227 2.04 +/- 1.87 mm(2) for definitely decreased autofluorescence (n = 15) and 1.86 +/- 2.14 mm(2) for po

228 Other methods, including fundus autofluorescence, near-infrared reflectance, and color i

229 enses did not explain the lower conjunctival autofluorescence observed in myopic subjects.

230 one density and spacing, and reflectance and autofluorescence of albipunctate spots.

231 from background signals, such as the typical autofluorescence of biological samples.

232 20 The latter likely accounts for the strong autofluorescence of Ca Entotheonella filaments.

233 Our system relies on monitoring the autofluorescence of cellulose and measuring the attenuat

234 Furthermore, we used the autofluorescence of e-liquids as a marker for tracking e

235 maging of a rat model of hepatic IR with the autofluorescence of mitochondrial flavins.

236 ations were associated with increases in the autofluorescence of NAD(P)H, whose amplitude was strongl

237 In tissue engineering, autofluorescence of polymer scaffolds often lowers the i

238 More interestingly, the strong red autofluorescence of the as-prepared hydrogel allows for

239 blished, taking advantage of the distinctive autofluorescence of these cells.

240 tomography (SDOCT) and an area of increased autofluorescence on blue fundus autofluorescence (B-FAF)

241 ient that was associated with marked loss of autofluorescence on fundus autofluorescence imaging.

242 The progression of increased autofluorescence, on the other hand, was highly variable

243 (OCT), en face near-infrared imaging, fundus autofluorescence, optical coherence tomography angiograp

244 hromophore maturation times and the cellular autofluorescence or phototoxicity that arises from light

245 glucose stimulation of respiration, NAD(P)H autofluorescence, or Ca(2+) responses between left- and

246 opy (P = 0.001), ultrasound (P = 0.013), and autofluorescence (P = 0.002).

247 pendently delineated boundaries of preserved autofluorescence (PAF) and preserved ellipsoid zone (EZ)

248 Interestingly, no changes in macular fundus autofluorescence pattern were evident after optical cohe

249 gic autorefraction, conjunctival ultraviolet autofluorescence photography, participant questionnaire.

250 ivery of excitation light, which can trigger autofluorescence, photoxicity, and photobleaching, impai

251 Free label imaging, using UV autofluorescence, provides a great tool to follow one si

252 Quantitative fundus autofluorescence (qAF) and spectral-domain optical coher

253 fundus autofluorescence (quantitative fundus autofluorescence [qAF]) and spectral-domain optical cohe

254 uorescence (DDAF) and questionably decreased autofluorescence (QDAF) were outlined and quantified.

255 sions by quantifying short-wavelength fundus autofluorescence (quantitative fundus autofluorescence [

256 s not associated with corresponding abnormal autofluorescence resolved without clinical scarring afte

257 We observe that the autofluorescence response induced by two NADH-oxidation

258 on of treatment, the age-related increase in autofluorescence resumed.

259 h eyes.Retinal anatomy was investigated with autofluorescence, retinal angiography and optical cohere

260 Non-invasive skin autofluorescence (SAF) measurement serves as a proxy for

261 aging, cognitive tests, and noninvasive skin autofluorescence (SAF; a measure of tissue AGE levels) o

262 Fundus autofluorescence showed little or no change except in se

263 RNFL thinning and autofluorescence showed relative sparing of the temporal

264 Autofluorescence showed sharply demarcated areas of RPE

265 ound NADH conformations) separately from the autofluorescence signal as a whole.

266 The results show that the autofluorescence signals emitted from polycaprolactone (

267 iddle ear tissue based on the characteristic autofluorescence signals.

268 Next, the autofluorescence, solvent compatibility, and biocompatib

269 2 visual field pattern density plots, fundus autofluorescence, spectral-density optical coherence tom

270 he basis of the quantification of UV-excited autofluorescence spectrum shape.

271 The autofluorescence suppressive effect does not substantial

272 ients tested had relatively preserved foveal autofluorescence surrounded by a ring of high density, 4

273 ected a pattern consisting of a central hypo-autofluorescence surrounded by a round area of hyper-aut

274 : 9 participants (53%) had reduced or absent autofluorescence surrounded by increased autofluorescenc

275 ography (SD-OCT) and short wavelength fundus autofluorescence (SW-AF).

276 yopia in the lowest quartile of conjunctival autofluorescence than the highest quartile (33.0% vs 15.

277 herosclerotic plaques generate near-infrared autofluorescence that can be detected via emission compu

278 erformed without influence from the inherent autofluorescence that commonly affects fluorescence-base

279 by HPLC analysis and quantitation of fundus autofluorescence; this effect is consistent with photoox

280 tep uses a thiolene-based resin with minimal autofluorescence to create an array of microwells to cap

281 tokes Raman scattering (CARS) and two-photon autofluorescence (TPAF).

282 This scheme simply avoids both autofluorescence under infrared excitation and many tedi

283 , to monitor the long excited-state lifetime autofluorescence (usually associated with protein-bound

284 the disease extent on ultra-widefield fundus autofluorescence (UWF-FAF) in patients with ABCA4 Starga

285 2-0.61 mm2/y), and of total decreased fundus autofluorescence was 0.35 mm2/y (95% CI, 0.28-0.43 mm2/y

286 Autofluorescence was due to riboflavin accumulation in m

287 nts exhibiting annular RPE lesions on fundus autofluorescence was included for chart review and exami

288 We show further that the autofluorescence was produced predominantly from mitocho

289 Median area of autofluorescence was significantly lower in myopic than

290 and poorly demarcated questionably decreased autofluorescence) was measured.

291 ptical coherence tomography (OCT) and fundus autofluorescence were evaluated at the baseline and duri

292 uorescence (DDAF) and questionably decreased autofluorescence were quantified by a reading center.

293 Longitudinal changes in autofluorescence were studied by evaluating areas of dec

294 roduce pre-culture SBB treatment to suppress autofluorescence, where SBB is applied to polymeric scaf

295 urements or images with decreased background autofluorescence while eliminating the need for expensiv

296 g near-infrared (NIR) reflectance and fundus autofluorescence with a confocal scanning laser ophthalm

297 ission of the bound UCNPs was imaged free of autofluorescence with anti-Stokes photoluminescence micr

298 trate that autoluminescence is solely due to autofluorescence with lifetimes of about 5 ns in the vis

299 all eyes, a cross-shaped increase in macular autofluorescence with variable intensity occurred after

300 off-target probe binding and in the cellular autofluorescence without detectable loss in RNA.