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1 tood disorder affecting 15% of patients with autoimmune hemolytic anemia.
2 dying within 3 to 5 wk from complications of autoimmune hemolytic anemia.
3 the BALB/c background, they rapidly die from autoimmune hemolytic anemia.
4 ion of autoantibodies and the development of autoimmune hemolytic anemia.
5 hemolytic anemia is the most common form of autoimmune hemolytic anemia.
6 lement may be exploited to prevent and treat autoimmune hemolytic anemia.
7 ocytes by Kupffer cells in a murine model of autoimmune hemolytic anemia.
8 emissions in patients with severe refractory autoimmune hemolytic anemia.
9 elops anti-erythrocyte Abs and is a model of autoimmune hemolytic anemia.
12 tive and autoimmune disease characterized by autoimmune hemolytic anemia (AHA) and inflammatory bowel
14 clones recovered ex vivo from a patient with autoimmune hemolytic anemia (AIHA) and characterize thei
15 retrospectively investigated in 308 primary autoimmune hemolytic anemia (AIHA) cases and correlated
16 he role of complement in the pathogenesis of autoimmune hemolytic anemia (AIHA) has been controversia
17 tly spontaneous autoimmune diseases, such as autoimmune hemolytic anemia (AIHA) in New Zealand Black
19 resulting from the deletion of IL-2 in which autoimmune hemolytic anemia (AIHA) is a prominent featur
22 ops a genetically imposed severe spontaneous autoimmune hemolytic anemia (AIHA) that is very similar
27 cases of immune thrombocytopenia followed by autoimmune hemolytic anemia and 1 Evans syndrome with rh
28 clonal B-cell lymphoproliferation results in autoimmune hemolytic anemia and cold-induced circulatory
29 en loss has been reported in the settings of autoimmune hemolytic anemia and transfusion of crossmatc
30 e treated similarly to those with idiopathic autoimmune hemolytic anemia, and additional therapy for
32 oimmune disease, usually characterized by an autoimmune hemolytic anemia, and NZB genes are essential
33 plegia, injection-site erythema and pyrexia, autoimmune hemolytic anemia, and suspected lack of vacci
34 epitopes that emerge on erythrocytes lead to autoimmune hemolytic anemia, and the causative auto-IgM
35 is, serositis, neuropsychiatric involvement, autoimmune hemolytic anemia, anti-double-stranded DNA, a
36 Reversible posterior leukoencephalopathy and autoimmune hemolytic anemia are newly reported clinical
38 eases like hereditary spherocytosis (HS) and autoimmune hemolytic anemia, but also with RBC senescenc
40 enotypes among descriptive diagnoses such as autoimmune hemolytic anemia, chronic immune thrombocytop
42 th BALB/c mice, but which are predisposed to autoimmune hemolytic anemia, develop auto-antibodies to
44 l as autoimmune complications manifesting as autoimmune hemolytic anemia, immune-mediated thrombocyto
45 children (none with serious infection), and autoimmune hemolytic anemia in two (resolved with a ster
46 isorder is cold agglutinin disease (CAD), an autoimmune hemolytic anemia in which autoantibodies (col
49 essary, the therapies used for warm antibody autoimmune hemolytic anemia may be tried with less likel
50 as immune thrombocytopenic purpura (ITP) or autoimmune hemolytic anemia, mouse models were used to e
51 severe refractory thrombocytopenia (n = 13), autoimmune hemolytic anemia (n = 15), and vasculitis (n
53 varies depending on whether the patient has autoimmune hemolytic anemia of warm antibody type, cold
54 yositis/dermatomyositis, systemic sclerosis, autoimmune hemolytic anemia, pernicious anemia, and anky
56 syndrome, paroxysmal cold hemoglobinuria, or autoimmune hemolytic anemia secondary to an underlying d
58 the transfusion management of patients with autoimmune hemolytic anemia, specifically focusing on pu
59 arthritis, immune thrombocytopenic purpura, autoimmune hemolytic anemia, systemic lupus erythematosu
60 o discovered a potential early biomarker for autoimmune hemolytic anemia that is based on different l
61 le mechanistic link between the induction of autoimmune hemolytic anemia, the reduction in naive T ce
62 and its major complications: hypersplenism, autoimmune hemolytic anemia, thrombocytopenia, and neutr
63 nic purpura (ITP), hereditary spherocytosis, autoimmune hemolytic anemia, thrombotic thrombocytopenic
64 crit level increased in 60% of patients with autoimmune hemolytic anemia undergoing successful laparo
66 oimmune hemolytic anemia (also known as warm autoimmune hemolytic anemia [WAHA]) in patients with bab
67 +6 and +12; the response was better in warm autoimmune hemolytic anemia (WAIHA; overall response, 10
69 hamide among patients with severe refractory autoimmune hemolytic anemia, we treated 9 patients with
71 tinues to be reported sporadically in severe autoimmune hemolytic anemia, with inconsistent results.
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