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1 sion signature consistent with an underlying autoimmune mechanism.
2 eta-cell death and dysfunction induced by an autoimmune mechanism.
3 the faulty BBB and neuronal death through an autoimmune mechanism.
4 al fullness, and believed to be caused by an autoimmune mechanism.
5 lity to detect BB: in the joint implicate an autoimmune mechanism.
6 The most favored hypothesis is based on autoimmune mechanisms.
7 ) in the course of HSE, suggesting secondary autoimmune mechanisms.
8 , suggesting a possible role for traditional autoimmune mechanisms.
9 onal damage, excitotoxicity and inflammatory/autoimmune mechanisms.
10 vide a unique opportunity to study potential autoimmune mechanisms.
11 ory synovitis whose pathogenesis may involve autoimmune mechanisms.
12 be a genetic susceptibility and a link with autoimmune mechanisms.
13 represent a useful tool in dissecting basic autoimmune mechanisms.
14 onimmune bronchial injury and alloimmune and autoimmune mechanisms.
15 The most favored hypothesis is based on autoimmune mechanisms.
16 men is unknown but may involve microbes and autoimmune mechanisms.
19 biliary cirrhosis (PBC), but the postulated autoimmune mechanisms behind these conditions are poorly
23 ted with autoimmune disease, but the role of autoimmune mechanisms in disease development is not well
25 of genetic predisposition, inflammation, and autoimmune mechanisms in the development of pulmonary ar
26 etection of allelic inclusion (an implicated autoimmune mechanism) in healthy individuals and (iii) t
28 In some patients, there is evidence that an autoimmune mechanism may be responsible for eliciting da
29 These findings support the hypothesis that autoimmune mechanisms may contribute to the pathogenesis
30 cell-mediated or humoral (antibody-mediated) autoimmune mechanisms may predominate in effecting a giv
31 at CASPR2 and LGI1 modulation via genetic or autoimmune mechanisms may share common intermediate mole
33 pathogen was identified, we examined whether autoimmune mechanisms played a role in initiating or mai
35 d autoantibodies will aid in elucidating the autoimmune mechanism(s) leading to the development of HG
36 e Syndrome (GBS) is believed to be caused by autoimmune mechanisms that are predominantly T-cell medi
37 s model will be a useful tool for addressing autoimmune mechanisms underlying gender-related differen
38 Programmed cell death is an important anti-autoimmune mechanism used to delete autoreactive lymphoc
40 l and clinical results, we concluded that an autoimmune mechanism was responsible for neutropenia.
41 reatment of non-neoplastic diseases in which autoimmune mechanisms were considered important to patho
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