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1 nsus was reached on the issue of subtypes of autoimmune pancreatitis.
2 s and development of new therapies for human autoimmune pancreatitis.
3 istics, course, and therapeutic responses in autoimmune pancreatitis.
4 n the rat, which we have termed experimental autoimmune pancreatitis.
5 t amylase may play a role in pathogenesis of autoimmune pancreatitis (6) 2-(18F)-Fluro-2-deoxy-D-gluc
6 h obstructive jaundice (77%) associated with autoimmune pancreatitis (92%), increased serum IgG4 leve
8 um immunoglobulin G4 (sIgG4) is a feature of autoimmune pancreatitis (AIP) and IgG4-associated cholan
10 ted pancreatic bicarbonate concentrations in autoimmune pancreatitis (AIP) by restoring mislocalized
11 k trial of corticosteroids may differentiate autoimmune pancreatitis (AIP) from pancreatic cancer in
18 tify current modalities for the diagnosis of autoimmune pancreatitis (AIP) with the objective of esta
20 is known about the pathogenic mechanisms of autoimmune pancreatitis (AIP), an increasingly recognize
21 m retrospective case series of patients with autoimmune pancreatitis (AIP), and follow-up periods hav
22 ith pancreatic cancer, chronic pancreatitis, autoimmune pancreatitis (AIP), and healthy controls (N).
24 re part of a spectrum of disorders including autoimmune pancreatitis and inflammatory pseudotumor.
25 patients, which may indicate an overlap with autoimmune pancreatitis and possible responsiveness to s
26 e the degree of inflammation and fibrosis in autoimmune pancreatitis and to monitor response to stero
27 ciations with inflammatory bowel disease and autoimmune pancreatitis, and medical therapy are discuss
28 ids rapidly reduce symptoms in patients with autoimmune pancreatitis, and micronutrient therapy to co
29 inical diagnostic and therapeutic aspects of autoimmune pancreatitis; and information on feeding stra
31 rts outlining the complexity in diagnosis of autoimmune pancreatitis; emerging roles of endoscopic ul
34 sed with primary sclerosing cholangitis have autoimmune pancreatitis in association with primary scle
35 st that gastric H. pylori infection triggers autoimmune pancreatitis in genetically predisposed indiv
37 tive descriptive evaluation of patients with autoimmune pancreatitis including dedicated radiology re
39 sh patients with IgG4-associated cholangitis/autoimmune pancreatitis (n = 34) from those with primary
40 res of IgG4-RSD in these 4 patients included autoimmune pancreatitis, sclerosing cholangitis, lymphop
41 nfirms that in non-expert practice settings, autoimmune pancreatitis scoring systems with a focus on
42 pancreatic lesions of rats with experimental autoimmune pancreatitis were characterized histologicall
44 ts with radiographic OR clinical features of autoimmune pancreatitis were initially identifiable (Mal
45 utility of published diagnostic criteria for autoimmune pancreatitis, when compared to the characteri
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