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1 nsus was reached on the issue of subtypes of autoimmune pancreatitis.
2 s and development of new therapies for human autoimmune pancreatitis.
3 istics, course, and therapeutic responses in autoimmune pancreatitis.
4 n the rat, which we have termed experimental autoimmune pancreatitis.
5 t amylase may play a role in pathogenesis of autoimmune pancreatitis (6) 2-(18F)-Fluro-2-deoxy-D-gluc
6 h obstructive jaundice (77%) associated with autoimmune pancreatitis (92%), increased serum IgG4 leve
7                                   Reviews of autoimmune pancreatitis, a newly described condition, pa
8 um immunoglobulin G4 (sIgG4) is a feature of autoimmune pancreatitis (AIP) and IgG4-associated cholan
9       To summarize the existing knowledge of autoimmune pancreatitis (AIP) and to review the progress
10 ted pancreatic bicarbonate concentrations in autoimmune pancreatitis (AIP) by restoring mislocalized
11 k trial of corticosteroids may differentiate autoimmune pancreatitis (AIP) from pancreatic cancer in
12                                              Autoimmune pancreatitis (AIP) has been divided into subt
13                                              Autoimmune pancreatitis (AIP) is a heterogeneous autoimm
14                                              Autoimmune pancreatitis (AIP) is a rare and underdiagnos
15                                              Autoimmune pancreatitis (AIP) is an increasingly recogni
16                                              Autoimmune pancreatitis (AIP) is the pancreatic manifest
17                                              Autoimmune pancreatitis (AIP) underlies 5%-11% of cases
18 tify current modalities for the diagnosis of autoimmune pancreatitis (AIP) with the objective of esta
19                                              Autoimmune pancreatitis (AIP), a recently defined diseas
20  is known about the pathogenic mechanisms of autoimmune pancreatitis (AIP), an increasingly recognize
21 m retrospective case series of patients with autoimmune pancreatitis (AIP), and follow-up periods hav
22 ith pancreatic cancer, chronic pancreatitis, autoimmune pancreatitis (AIP), and healthy controls (N).
23 treatment, and at follow-up in patients with autoimmune pancreatitis (AIP).
24 re part of a spectrum of disorders including autoimmune pancreatitis and inflammatory pseudotumor.
25 patients, which may indicate an overlap with autoimmune pancreatitis and possible responsiveness to s
26 e the degree of inflammation and fibrosis in autoimmune pancreatitis and to monitor response to stero
27 ciations with inflammatory bowel disease and autoimmune pancreatitis, and medical therapy are discuss
28 ids rapidly reduce symptoms in patients with autoimmune pancreatitis, and micronutrient therapy to co
29 inical diagnostic and therapeutic aspects of autoimmune pancreatitis; and information on feeding stra
30  Furthermore, the complexities of diagnosing autoimmune pancreatitis are being recognized.
31 rts outlining the complexity in diagnosis of autoimmune pancreatitis; emerging roles of endoscopic ul
32                  The clinical description of autoimmune pancreatitis has led to the realization that
33          Clinical description and studies of autoimmune pancreatitis have led to the realization that
34 sed with primary sclerosing cholangitis have autoimmune pancreatitis in association with primary scle
35 st that gastric H. pylori infection triggers autoimmune pancreatitis in genetically predisposed indiv
36                        Features that suggest autoimmune pancreatitis include focal or diffuse pancrea
37 tive descriptive evaluation of patients with autoimmune pancreatitis including dedicated radiology re
38                                              Autoimmune pancreatitis may belong to a multiorgan immun
39 sh patients with IgG4-associated cholangitis/autoimmune pancreatitis (n = 34) from those with primary
40 res of IgG4-RSD in these 4 patients included autoimmune pancreatitis, sclerosing cholangitis, lymphop
41 nfirms that in non-expert practice settings, autoimmune pancreatitis scoring systems with a focus on
42 pancreatic lesions of rats with experimental autoimmune pancreatitis were characterized histologicall
43 -82 years) with histopathologic diagnosis of autoimmune pancreatitis were examined.
44 ts with radiographic OR clinical features of autoimmune pancreatitis were initially identifiable (Mal
45 utility of published diagnostic criteria for autoimmune pancreatitis, when compared to the characteri

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