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1 llary thymic epithelial cells expressing the autoimmune regulator.
2 ng the T cell repertoire and the role of the autoimmune regulator Aire in thymic MPO expression, we c
4 heckpoint occurs through the activity of the autoimmune regulator AIRE, which promotes central T cell
6 such as insulin, we are seeing the result of autoimmune regulator (AIRE) activity and the workings of
7 eted of T cells, showed a notable absence of autoimmune regulator (AIRE) and Foxp3(+) regulatory T ce
10 Both humans and mice with a mutation in the autoimmune regulator (aire) gene develop multiorgan auto
15 dullary thymic epithelial cells (mTECs), the Autoimmune regulator (Aire) gene plays an essential role
17 of mature lymphocytes from mice lacking the autoimmune regulator (aire) gene was performed to examin
18 e type I (APS-I), caused by mutations in the autoimmune regulator (AIRE) gene, and myasthenia gravis
19 syndrome, which is caused by mutation of the autoimmune regulator (AIRE) gene, is a highly variable d
20 yndrome type 1 is caused by mutations in the autoimmune regulator (AIRE) gene, located on chromosome
40 -antigens, regulated at least in part by the autoimmune regulator (AIRE) protein, is crucial for comp
41 PECED patients are defined by defects in the autoimmune regulator (AIRE) that mediates central T cell
42 also interacts with the transcription factor autoimmune regulator (AIRE) to promote central T-cell to
43 significant reduction in self-Ag-presenting, autoimmune regulator (AIRE)(+) medullary thymic epitheli
44 specific antigens that are controlled by the autoimmune regulator (AIRE), a key factor in central tol
47 ed antigens in the thymus, driven in part by autoimmune regulator (Aire), is critical for the protect
49 Here we show that in the presence of the autoimmune regulator (AIRE), the class III VNTR haplotyp
50 ctopic expression of TSAs largely depends on autoimmune regulator (Aire), which is expressed in matur
52 m our laboratory and others have established autoimmune regulator (Aire)-deficient mice as a useful m
54 e selection of T cells, with the presence of autoimmune regulator (Aire)-expressing medullary thymic
59 ecessive disorder caused by mutations in the autoimmune regulator (AIRE, which regulates immune toler
61 epithelial cells (mTEC(high)) expressing the autoimmune regulator are targets of donor T-cell alloimm
62 ssue antigens in a manner similar to that of autoimmune regulator-deficient (Aire-deficient) mice.
63 ctive form of the transcription factor AIRE (autoimmune regulator) develop multiorgan autoimmune dise
64 als, in spite of the inability to induce the autoimmune regulator, displayed a significant ameliorati
65 e mouse thymus could be assigned to strongly autoimmune regulator-expressing, mature medullary thymic
66 tioned with anti-CD3epsilon mAb, we detected autoimmune regulator expression together with the absenc
71 variable number of tandem repeats) and AIRE (autoimmune regulator) have been associated with the modu
73 interactions between developing T cells and autoimmune regulator-positive medullary thymic epithelia
75 s) in the thymus is under the control of the autoimmune regulator protein (AIRE), and polymorphisms i
78 cent lessons learned from the study of AIRE (autoimmune regulator), the gene responsible for the rare
79 hymi from NOD mice lacking expression of the autoimmune regulator transcription factor, which develop
80 affinity for the transcription factor AIRE (autoimmune regulator), which is highly expressed in thym
81 LNSC subset expressing the highest levels of autoimmune regulator, which responds potently to bystand
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