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1 naling events that are required for TGFbeta1 autoinduction.
2 ii) reveal the origin of stereochemistry and autoinduction.
3 in a wild-type strain due to the absence of autoinduction.
4 as to examine the mechanism of PTC TGF-beta1 autoinduction.
5 significant induction period consistent with autoinduction.
6 oduce the optimum level of OmpR-P needed for autoinduction.
7 eta release, an amplification process termed autoinduction.
8 vels, possibly caused by enzyme induction or autoinduction.
9 o that present in 6TG1.1 cells after 18 h of autoinduction.
10 ng lacZ fused to a gene that is regulated by autoinduction.
11 ncreased only threefold in vivo during phoPR autoinduction, a role for unphosphorylated PhoP in Pho r
13 to the receptor is potentially amplified by autoinduction and cross-signaling through epidermal grow
14 arthritis by using IL-1R antagonist to block autoinduction and IL-1alpha stimulation to simulate auto
15 from LPS-stimulated cells is attributable to autoinduction and that GLA reduces autoinduction of IL-1
16 e examined group-specific differences in agr autoinduction and virulence gene regulation by utilizing
17 up-regulation of T(3) receptor beta (TRbeta; autoinduction) and BTEB1 (basic transcription element-bi
18 uxIR QS system produces an 'outward wave' of autoinduction, and the Lsr QS system yields dispersed au
19 in adenosine receptors, and adenosine-IL-13 autoinduction are critical events in IL-13-induced patho
21 th the TrbetaA promoter in vivo and enhances autoinduction, but this action does not depend on its DN
22 anscription of nisABTCIP and nisFEG requires autoinduction by external nisin via signal transducing b
25 ological noise present in the quorum-sensing autoinduction circuit and ensures that ICEMlSym(R7A) tra
26 factor (VEGF) interact in a newly described autoinduction circuit, in which each of these cytokines
27 tion, and the Lsr QS system yields dispersed autoinduction from spatially-localized secretion and upt
29 ucidation of cAMP-CRP involvement in E. coli autoinduction impacts many areas, including the growth o
30 dspI was shown to abolish biofilm dispersion autoinduction in continuous cultures of P. aeruginosa an
32 e we examined the role of km23-1 in TGFbeta1 autoinduction in TGFbeta-sensitive epithelial cells.
37 degradation of p27 is in the formation of an autoinduction loop that selectively controls the transit
45 receptor blockade reduces the IL-1-mediated autoinduction of IL-1, reduces the expression of ICAM-1
46 utable to autoinduction and that GLA reduces autoinduction of IL-1beta while leaving the initial IL-1
52 servations form the basis of a model for the autoinduction of the cytolysin by a quorum-sensing mecha
56 dulates the acyl-homoserine lactone-mediated autoinduction of Ti plasmid conjugative transfer by inte
57 or the clinically important oxazaphosphorine autoinduction pharmacokinetics seen with these drugs in
61 d in the extracellular matrix and acts as an autoinduction signal to stimulate neighboring cells to p
63 alter the activity of the protein on TrbetaA autoinduction, suggesting that BTEB1 can function in thi
64 uction system is controlled both by a second autoinduction system and by the RpoS(Rso) sigma factor.
66 e, in R. solanacearum the acyl-HSL-dependent autoinduction system is controlled both by a second auto
70 uction and IL-1alpha stimulation to simulate autoinduction that approximately 40% of IL-1beta release
72 Thus, while quorum sensing is dependent upon autoinduction, the two phenomena are not synonymous.
73 ndicate that km23-1 is required for TGFbeta1 autoinduction through Smad2-independent Ras/ERK/JNK path
74 ulated IL-33 receptors and facilitated IL-33 autoinduction, thus establishing a feed-forward circuit.
75 egulated in Lactococcus lactis ATCC 11454 by autoinduction via a two-component NisRK-mediated system.
76 m Agrobacterium tumefaciens is controlled by autoinduction via the transcriptional activator TraR and
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