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1 onse has previously not been demonstrated in autonomic ganglia.
2 enteric neurons but not in glial cells or in autonomic ganglia.
3 n was not consistently altered in any of the autonomic ganglia.
4 3 and beta 4, which are the most abundant in autonomic ganglia.
5 iary neurons does not likely derive from the autonomic ganglia.
6 of intact sensory or major, posterior tongue autonomic ganglia.
7 pair was adjacent to the fat pad containing autonomic ganglia (AG) at the veno-left atrial (LA) junc
8 cific inhibition of synaptic transmission in autonomic ganglia and antibody levels correlate in a pre
9 ous agent primarily uses synaptically linked autonomic ganglia and efferent fibers of the vagus and s
11 ssion across parasympathetic and sympathetic autonomic ganglia and in the adrenal medulla and, theref
12 ining alpha3 and beta2 subunits are found in autonomic ganglia and mediate ganglionic transmission.
14 to establish latent infection in sensory or autonomic ganglia and to reactivate on physical, hormona
15 to establish latent infection in sensory or autonomic ganglia and to reactivate on physical, hormona
16 aricella), becomes latent in dorsal root and autonomic ganglia, and reactivates decades later to caus
17 for nicotinic acetylcholine receptors in the autonomic ganglia are potentially pathogenic and may ser
18 ariety of derivatives, including sensory and autonomic ganglia, cartilage and bone of the face and pi
19 cranial ganglia, and peripheral sensory and autonomic ganglia during the embryonic and neonatal peri
20 ere present at high levels in nerve cells of autonomic ganglia, epithelial cells, intestinal smooth m
21 eral targets, modulates neurotransmission in autonomic ganglia, has an important role in local enteri
24 n of NO bioavailability from nNOS in cardiac autonomic ganglia in response to training is dependent o
27 ain disease states, synaptic transmission in autonomic ganglia is depressed and the periphery becomes
28 Neonatal neurodegeneration in sensory and autonomic ganglia is followed by loss of neurons from la
29 ory synaptic transmission through vertebrate autonomic ganglia is mediated by postsynaptic nicotinic
32 ations (brain, raphe nuclei, spinal cord and autonomic ganglia) may modulate rat sexual behavior in o
33 the incubation period in enteric ganglia and autonomic ganglia of splanchnic or vagus circuitry prior
34 -1 spread via the dorsal root ganglia to the autonomic ganglia of the enteric nervous system (ENS) in
35 rgic synaptic transmission in adrenal gland, autonomic ganglia, pineal gland, and several nuclei in t
38 present study measured nAChRs in several rat autonomic ganglia: the superior cervical ganglia (SCG),
40 terrupting neurotransmission at the level of autonomic ganglia to determine its effect on the QT inte
41 ls with prior supranodose de-efferentations, autonomic ganglia were stained with Fluoro-gold, and tis
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