1 by baroreceptor stimulation and abolished by autonomic ganglion blockade.

2 responses were specifically inhibited by the autonomic ganglion blocker hexamethonium bromide.

3 or 24 hrs, in the absence or presence of the autonomic ganglion blocker, pentolinium.

4 determine neuronal involvement, trimetaphan (autonomic ganglion blocker; 1 to 6 mg/min intravenous) w

5 tion was not attenuated by hexamethonium, an autonomic ganglion blocking agent, but was abolished by

6 spectral (Brainbow) labeling of the input to autonomic ganglion cells showed that the locations of ga

7 o exclude a potential developmental role for autonomic ganglion cells that are located in the posteri

8  to the CYP2J subfamily that is localized to autonomic ganglion cells, epithelial cells, smooth muscl

9 entially occur anywhere along the peripheral autonomic ganglion sites.

10                   Increased ROS also depress autonomic ganglion synaptic transmission by oxidizing th