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1 erts cysteine to cystine by copper-dependent autooxidation.
2 c conditions, and it was rapidly restored by autooxidation.
3 ins also caused decreased rates of alphaO(2) autooxidation as compared with wild-type AHSP.
4 Mito-HE for superoxide in vivo is limited by autooxidation as well as by nonsuperoxide-dependent cell
5                       MsrA catalyzes its own autooxidation as well as oxidation of free methionine an
6  by Pro-30 in wild-type AHSP promote alphaHb autooxidation by introducing strain into the proximal he
7 haHb O2 affinity roughly 4-fold and promotes autooxidation due primarily to a 3-4-fold increase in th
8 eviously was reported to undergo accelerated autooxidation during incubation in vitro.
9 e reaction limits methemoglobin formation by autooxidation; (iii) there is no gas-liquid interface, e
10 xidants from olive-oil pomace or a synthetic autooxidation inhibitor as dimethylsiloxane.
11 ntaining natural antioxidants or a synthetic autooxidation inhibitor on the metabolism of essential f
12 rocesses; however, the structure of many key autooxidation intermediates and the reactions leading to
13 mO was corroborated in vitro in an adrenalin autooxidation model.
14 n of highly oxygenated intermediates for the autooxidation of alkanes at 500-600 K builds upon prior
15                However, AHSP also stimulates autooxidation of alphaO(2) subunit and its rapid convers
16 ctive oxygen species (ROS) by the endogenous autooxidation of components of the electron transport ch
17 ighly oxygenated intermediates via extensive autooxidation of hydroperoxyalkylperoxy radicals.
18 complex I), Mac(8345) (complex II) underwent autooxidation of its cysteine residues, resulting in the
19 y the condensation of nitrous acid or by the autooxidation of nitric oxide, both of which are metabol
20            Peroxynitrite and products of the autooxidation of NO in the presence of oxygen, but not h
21                   Decades of research on the autooxidation of organic compounds have provided fundame
22 nce of a cellular pathway for countering the autooxidation of SoxR and confirm the hypothesis that in
23 tions made in atmospheric conditions for the autooxidation of terpenes and other unsaturated hydrocar
24 nation by a glutathione molecule triggers an autooxidation of the Cr(IV)-peroxo complex to Cr(VI) via
25 more hydroperoxy groups are prevalent in the autooxidation of various oxygenated (e.g., alcohol, alde
26 metabolite, analysis of gamma-linolenic acid autooxidation products and the compound present in freez
27                  The absorbance of quercetin autooxidation products at 320nm was correlated with the
28 l remains a significant increase in inherent autooxidation rate for HbS.
29 quilibrium constants, and reduced O2-alphaHb autooxidation rates.
30  These findings improve our understanding of autooxidation reaction mechanisms that are routinely use

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