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1 ides) are PtdIns3P and PtdIns(3,5)P2 binding autophagy related proteins.
2 s and enhances autophagic flux by modulating autophagy-related proteins.
3 phore assembly site and interacted with Atg (autophagy related) proteins.
5 t is a locus on chromosome 10 containing the autophagy-related protein 18 (ATG18) associated with dec
6 phagy receptors, such as the Pichia pastoris autophagy-related protein 30 (Atg30), which controls the
9 eracting serine-threonine kinase-2 (RIPK-2), autophagy-related protein-5 (ATG5), ATG7 and ATG16L1 but
11 autophagy-related protein beclin 1 (BCN1) or autophagy-related protein 7 (ATG7) in immortalized human
12 Atg7(Deltapan) mice that lack the essential autophagy-related protein 7 (ATG7) in pancreatic epithel
13 patocytes, autophagy was inhibited by 3MA or autophagy-related protein 7 (Atg7) small interfering RNA
15 es, vacuolar protein sorting 34 (VPS34), and autophagy-related protein 7 (ATG7), could rescue the PA-
17 ethyladenine (3-MA) or were transfected with autophagy-related protein 7 or TLR7 small interfering RN
19 may utilize autophagy for replication as the autophagy-related protein-7 (ATG-7), microtubule-associa
20 Here we describe a novel interaction between autophagy-related protein 8 (Atg8) and fatty acid syntha
21 evisiae, the integral membrane protein Atg9 (autophagy-related protein 9) cycles between mitochondria
22 lyses of the functions and dynamics of known autophagy-related proteins and for screening new genes.
25 found that ER content is expanded in mature autophagy-related protein (Atg) 7-deficient T lymphocyte
26 ential components of the autophagic pathway, autophagy-related protein (Atg)5, Beclin1, and Ulk1, wer
29 ed the previously described functions of the autophagy-related proteins Atg5-Atg12, but NLRX1 did not
30 gested that high expression of the essential autophagy-related protein, Atg7, was associated with sho
32 In this study, we found that knockdown of autophagy-related protein beclin 1 (BCN1) or autophagy-r
33 smodium vivax depends only on the downstream autophagy-related proteins Beclin 1, PI3K, and ATG5, but
35 ing compartment by a mechanism that involved autophagy-related proteins, but not the conventional aut
36 tion or knockdown of presenilins alters many autophagy-related proteins demonstrating a buildup of au
38 Western blot analysis indicated that several autophagy-related proteins fluctuated during the procedu
40 ubiquitin-binding protein SQSTM1/p62 and the autophagy-related protein LC3, silencing of p62 and LC3
41 he proximal tubules and reduced ratio of the autophagy-related protein LC3-II/I in the kidney post-UU
43 ar calcium overload that is dependent on the autophagy-related proteins LC3 and p62 upon hyperactivat
44 ganese superoxide dismutase-2 (SOD2) and the autophagy-related proteins LC3II and Gabarapl1 also are
45 during Plasmodium infection and the roles of autophagy-related proteins may provide an urgently neede
46 of autophagic vacuoles and conversion of an autophagy-related protein, microtubule-associated protei
49 embryonic fibroblasts deficient in Atg5, an autophagy-related protein required for autophagosome for
50 es autophagy by acting directly on essential autophagy-related proteins strategically located in the
51 genous and induced clusters co-localize with autophagy-related proteins such as Atg16L1, LC3B and Rab
53 rotein synthesis, including that of specific autophagy-related proteins that are up-regulated in resp
55 or with small interfering RNAs specific for autophagy-related proteins, which are essential for auto
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