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1      Seventeen of these 19 deaths were fully autopsied.
2 ffspring were raised to terminal illness and autopsied.
3 descending coronary arteries (LADs) from 760 autopsied 15- to 34-year-old victims of accidents, homic
4                      Our similar findings in autopsied AD brains validate the discovery in AD mouse m
5  were replicated in an independent sample of autopsied AD cases and controls recruited from a geograp
6 ivity and specificity in 272 cases that were autopsied, and evaluated the impact of mutations and pol
7 8 patients (38%) died during the study, were autopsied, and had neuritic plaque levels determined; 25
8 omas, and four adenomas developed in 35 rats autopsied at 24 weeks after the first injection of DMH.
9                             All animals were autopsied at the end of the experiments, and organ probe
10 y included 20 consecutive patients with HSCT autopsied at the University of Oklahoma, between 1994 an
11                                     Among 24 autopsied behavioural Alzheimer's disease/dysexecutive A
12 , we performed real-time quantitative PCR in autopsied brain from women without clinical or pathologi
13                 Neurochemical analysis of an autopsied brain showed a pronounced decrease in choline
14     Isolation of the ameba into culture from autopsied brain tissue confirmed the presence of Balamut
15 one) were cloned and sequenced directly from autopsied brain, spleen, and lymph node specimens from f
16 ost of the Abeta species detected in rapidly autopsied brains (<3 h) with sporadic AD were Abeta(1-x)
17  brains from HIV-positive individuals and 78 autopsied brains from HIV-negative individuals were stai
18                       Large arteries from 84 autopsied brains from HIV-positive individuals and 78 au
19                                Among the 286 autopsied brains of 544 participants, brain mercury leve
20                       The study included 286 autopsied brains of 554 deceased participants (51.6%).
21  of functional synaptic proteins as found in autopsied brains of patients with Alzheimer's disease (A
22 claudin-5 were observed in microvessels from autopsied brains of patients with HIV-1-associated demen
23 DA storage vesicles from the striatum of six autopsied brains of PD patients and four controls and me
24  a collection of 708 prospectively collected autopsied brains to assess the methylation state of the
25 DISC1 protein in a well characterized set of autopsied brains, including brains of patients with SZ,
26                             Subjects were 62 autopsied cases from a prospective study of vascular con
27           Neuropathological examination in 4 autopsied cases showed neuronal loss in the substantia n
28                     Pathological analyses of autopsied cases strongly suggest that unlike MS, astrocy
29    Although the pathological findings in the autopsied cases were well characterized, only limited an
30  64 patients with FTD, retrospectively in 26 autopsied cases with FTD or Alzheimer's disease (AD), an
31 on cause of death and was found in 158 (36%) autopsied cases, followed by 135 (31%) cases of sudden u
32 esponding pathology were investigated in 430 autopsied coal miners from West Virginia.
33 al amyloid phase in a prospectively-followed autopsied cohort who underwent ante-mortem (11)C-Pittsbu
34 (95%) in SIDS infants (n = 61) compared with autopsied controls (n = 15) [SIDS, 177.2 +/- 15.1 (mean
35                                 All patients autopsied, excluding preterm fetuses, at the Medical Cen
36  an evaluation of the vascular pattern in an autopsied eye were conducted at a community retina pract
37                                          The autopsied eyes were examined with light microscopy, immu
38                                       Of the autopsied group with acute MI, 97% (31 of 32 patients) w
39                                    Among the autopsied group with severe coronary atherosclerosis, HD
40                 Three AIDP subjects who were autopsied had had symptoms for 3 to 9 days at the time o
41 validated by comparing microscopic images of autopsied hearts with corresponding CT images.
42 udy, O-GlcNAc glycosylation of proteins from autopsied human brains with confirmed AD and non-AD age-
43 nique to isolate living neurons from rapidly autopsied human elderly brains, and have succeeded in ke
44 oatrial segments (32 veins) harvested from 8 autopsied human hearts using antibodies to tyrosine hydr
45 eight regions-of-interest were obtained from autopsied human hearts, with plaque composition and clas
46 In the present work, analysis of biopsied or autopsied human skeletal muscle revealed the absence or
47  15 to 34 years dying of external causes and autopsied in forensic laboratories.
48 anying pathology is occasionally reported in autopsied infant lungs.
49 y serve as a potential forensic biomarker in autopsied infants with SIDS with serotonergic defects.
50 ly, Cx43 mislocalization was also evident in autopsied left ventricle tissue from HGPS patients, sugg
51                                              Autopsied lungs of infants with bronchopulmonary dysplas
52                                    Among 333 autopsied men, 120 had dementia, 115 had marginal result
53        Quantitative histological analysis of autopsied multiple sclerosis spinal cord specimens suppo
54 eight active demyelinating lesions from four autopsied multiple sclerosis subjects.
55  in longitudinally followed, well documented autopsied normal control subjects and patients with amne
56 zheimer disease in the first 35 participants autopsied (out of 152 individuals enrolled in the PET pa
57 c nigral neurons from MPTP-treated mice, and autopsied Parkinsonian tissue from late-onset sporadic c
58 the Rush Memory and Aging Project) among 740 autopsied participants 66.0 to 108.3 years old.
59                       The study included 483 autopsied participants from the Religious Orders Study a
60                                          One autopsied patient had predominantly CD8(+) T lymphocytes
61 multiple-matched metastatic tumours from ten autopsied patients to infer the evolutionary history of
62  and compared in spinal cord samples from 18 autopsied patients with ALS and 12 neurologically health
63  also found in spinal cord tissue samples of autopsied patients with ALS.
64   Genetic analysis was performed to identify autopsied patients with the most common Lrrk2 mutation (
65 sis affecting the frontal lobes in all three autopsied patients, and severe caudate atrophy was noted
66  the decrease observed in the hippocampus of autopsied patients.
67 mi-quantitative pathological phenotype in 10 autopsied patients.
68  arteries and tissue from approximately 3000 autopsied persons aged 15-34 y who died of accidental in
69  atherosclerosis in a geographically defined autopsied population, while capitalizing on the autopsy
70  atherosclerosis was analyzed in a cohort of autopsied residents of Rochester, Minnesota, age 30 year
71 hologically, five of the six patients so far autopsied shared frontotemporal atrophy with involvement
72 s, cognitive decline and fluctuations in 872 autopsied subjects (mean age = 87.9 years) from the Rush
73 nter autopsy database and found that ~14% of autopsied subjects clinically diagnosed with mild-to-mod
74 ses of dementia, probable AD, and MCI in 636 autopsied subjects from the Religious Order Study and th
75                    Participants included 148 autopsied subjects of the Rush Memory and Aging Project
76                                              Autopsied subjects were classified into 5 categories: de
77                                    Fifty-six autopsied subjects were identified with a clinical diagn
78                             We scanned human autopsied temporal lobe blocks in a 7T human scanner usi
79 o study their function in cells derived from autopsied tissue.
80 red clinicopathological data from 11 sets of autopsied twin-twin transfusion syndrome fetuses, includ

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