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1 oners, PMCTA could be used to avoid invasive autopsy.
2 rsity (ELA) were determined by psychological autopsy.
3 in 5 of 18 control aortas (28%) obtained at autopsy.
4 nd inversely correlated with brain weight at autopsy.
5 is associated with AD pathologic findings at autopsy.
6 nor had Balamuthia encephalitis confirmed on autopsy.
7 and a subset of ACT participants consent to autopsy.
8 or corticobasal degeneration (CBD) proved by autopsy.
9 lly examined 215 dogs, 28 of which underwent autopsy.
10 al Examiners for a cardiac channel molecular autopsy.
11 Of these, 1589 underwent autopsy.
12 h law, all subjects who die suddenly undergo autopsy.
13 et, and the final diagnosis was confirmed at autopsy.
14 of death after postmortem investigation and autopsy.
15 CIED removal, interrogation, and analysis at autopsy.
16 reproductive age were ascertained by verbal autopsy.
17 ents, with all deaths investigated by verbal autopsy.
18 ved xenografts at diagnosis, recurrence, and autopsy.
19 nd cognition, definitively diagnosed only at autopsy.
20 igious Orders Study) and had agreed to brain autopsy.
21 and underwent neuropathologic evaluation on autopsy.
22 but until recently could only be assessed at autopsy.
23 vestigated in ocular and cerebral vessels at autopsy.
24 codes and causes of death were confirmed by autopsy.
25 eart failure diagnosis; n=104) who underwent autopsy.
26 , community-dwelling individuals who came to autopsy.
27 ity Status Scale at attack leading to biopsy/autopsy.
28 elated with measures of amyloid pathology at autopsy.
29 pamine in human brain specimens collected at autopsy.
30 tern II among all active lesions examined at autopsy.
31 Each case was assessed by PMCTA, followed by autopsy.
32 aneurysm that was found on her grandmother's autopsy.
33 d normal brain pathologic characteristics at autopsy.
34 ations should include both PMCT and invasive autopsy.
35 , whole blood) are not retained routinely at autopsy.
36 one-quarter of MCI cases showed "pure" AD at autopsy.
37 -49 years) since January, 2008, using verbal autopsy.
38 Ocular tissue was obtained from 1 patient at autopsy.
39 rn consistent with tau pathology observed at autopsy.
40 eria and contributed 42 data sets and 12,834 autopsies.
41 replicated in an independent data set of 369 autopsies.
42 rs560380, P=3.8 x 10(-8)) in 909 prospective autopsies.
43 gments from adult human cortex obtained from autopsies.
45 atients with Parkinson's disease who came to autopsy 18 months and 16 years post-transplantation.
46 logically defined CAA were included: 52 with autopsies, 22 with brain biopsy specimens, and 31 with p
50 o Huntington's disease patients, who came to autopsy 9 and 12 years post-transplantation, and two pat
56 lysis as an important diagnostic tool in all autopsies and to assist manufacturers in identifying pot
59 rom 213 patients who had been followed up to autopsy and met inclusion criteria of Lewy body disorder
60 bsamples of BLSA, 191 participants underwent autopsy and neuropathological assessment, and 75 non-dem
62 ollect brain samples without performing full autopsy and show that a test currently marketed for vete
64 , biopsy specimen at hematoma evacuation, or autopsy) and available brain MRI sequences of adequate q
66 ivity and specificity in 272 cases that were autopsied, and evaluated the impact of mutations and pol
67 8 patients (38%) died during the study, were autopsied, and had neuritic plaque levels determined; 25
68 skeletal survey, (c) CMLs were confirmed at autopsy, and (d) non-CML fractures were also present.
69 d household demographics, completed a verbal autopsy, and randomly selected household members for ver
70 ed mortality from vital registration, verbal autopsy, and surveillance data using the Cause of Death
71 polymerase chain reaction, brain biopsy, or autopsy, and who had MR images available were included i
72 riple negative breast cancer underwent rapid autopsy as part of an institutional prospective communit
74 clinical assessments and had agreed to brain autopsy at the time of death, as part of one of two coho
75 pathology, and in 59 deceased subjects with autopsy-based amyloid beta neuritic plaque measure who u
76 quate vital registration, and updated verbal autopsy-based multicause models for high mortality count
79 In addition, we included 29 patients with autopsy/biomarker-defined Alzheimer's disease with a dys
80 plying voxel-based morphometry) with matched autopsy/biomarker-defined typical (amnestic-predominant)
82 brains from HIV-positive individuals and 78 autopsied brains from HIV-negative individuals were stai
86 of functional synaptic proteins as found in autopsied brains of patients with Alzheimer's disease (A
88 ticles from three sources: consecutive rapid autopsy brains from the Adult Changes in Thought Study,
89 ntation and decreased fusion-in neurons from autopsy brains of AD patients and from transgenic AD mic
91 tissue (FFPET) is ubiquitously collected at autopsy, but DNA quality hampers its use with traditiona
94 After sudden cardiac death with negative autopsy, clinical screening of relatives identifies a hi
95 al amyloid phase in a prospectively-followed autopsied cohort who underwent ante-mortem (11)C-Pittsbu
96 the National Alzheimer's Coordinating Center autopsy cohort across the entire clinicopathological con
98 ttsburgh compound B imaging; using the large autopsy cohort to broaden our perspective of (11)C-Pitts
104 rphisms in the hFPPS gene and mRNA levels in autopsy-confirmed AD subjects was undertaken, and a gene
105 rt of patients with Lewy body disorders with autopsy-confirmed alpha synucleinopathy (as of Oct 1, 20
106 nclusion criteria of Lewy body disorder with autopsy-confirmed alpha synucleinopathy, we identified 4
111 the primary clinical diagnosis of PPA and an autopsy-confirmed diagnosis of either AD (PPA-AD) or a t
112 l data from 100 consecutive patients with an autopsy-confirmed diagnosis of PD from the archives of t
116 sease (typical Alzheimer's disease, n = 58), autopsy-confirmed/Alzheimer's disease biomarker-negative
119 (95%) in SIDS infants (n = 61) compared with autopsied controls (n = 15) [SIDS, 177.2 +/- 15.1 (mean
121 abies transmission model fit to human rabies autopsy data and human rabies surveillance data from Tam
124 inment in most previous studies was based on autopsy data or clinical convenience samples, often with
125 in August 2015 using clinical and postmortem autopsy data that had been collected between August 1983
126 r metastases providing evidence in line with autopsy data that the metastatic potential, as shown by
127 d the National Alzheimer Coordinating Center autopsy data to evaluate the effect of different neurofi
128 disease severity variation in the absence of autopsy data, a rhesus macaque and common marmoset model
129 alysis because of procedural unmasking or no autopsy data, as were 24 cases with a clear diagnosis of
130 ssible clinical diagnosis, 117 had available autopsy data, including 98 with a primary pathological d
134 013 National Alzheimer's Coordinating Center autopsy database and found that ~14% of autopsied subjec
137 ed myocardial sections from 30 subjects with autopsy-documented AL (n=10), ATTR (n=10), and nonamyloi
139 the predictions from the EPICUP assay using autopsy examination, follow-up for subsequent clinical d
141 an evaluation of the vascular pattern in an autopsied eye were conducted at a community retina pract
144 escribe the clinical, imaging, molecular and autopsy features in 22 of 24 patients who have developed
145 lyzed the characteristics of SCD victims and autopsy findings in 1998 to 2002, 2003 to 2007, and 2008
147 gnosis from PMCTA against a gold standard of autopsy findings, modified by PMCTA findings only if add
153 ge tissues and chondrocytes were obtained at autopsy from normal knee joints and from OA-affected joi
154 19 is downregulated in brain tissue taken at autopsy from patients with AD and from those with severe
155 n profiles of purified microglia isolated at autopsy from the parietal cortex of 39 human subjects wi
158 eight regions-of-interest were obtained from autopsied human hearts, with plaque composition and clas
160 The method also effectively discriminated autopsy-identified amyloid-positive and -negative cases
163 monstrates a strategy to implement molecular autopsies in medicolegal investigations of young SD dece
164 l samples of the myocardium, obtained during autopsies in the period 2009 to 2013, from all patients
165 use of death data were drawn from the verbal autopsies in the Registrar General of India's ongoing Mi
166 hological samples were ascertained following autopsy in each individual brain bank, whereas clinical
167 pathologic changes are commonly observed at autopsy in individuals with DLB and PD dementia, but the
169 requently coexists with Lewy body disease at autopsy in patients with probable dementia with Lewy bod
171 istent with PHF-tau distribution observed at autopsy in subjects with a history of mild TBI and autop
172 removal and interrogation are recommended at autopsy in suspected cases of sudden cardiac death, but
174 ld of post-mortem genetic testing (molecular autopsy) in cases of SADS and comprehensive clinical eva
175 y serve as a potential forensic biomarker in autopsied infants with SIDS with serotonergic defects.
176 ctively collected clinical, demographic, and autopsy information on all cases of sudden cardiac death
178 IDRalpha1.7 domain from a pediatric CM brain autopsy inhibited the barrier-protective properties of E
179 he objective of this study was to use verbal autopsy interviews to examine factors associated with st
183 h is based on evidence such as psychological autopsies, is that suicide attacks are caused by suicida
184 ly, Cx43 mislocalization was also evident in autopsied left ventricle tissue from HGPS patients, sugg
188 on emission tomography during life and brain autopsy (mean time positron emission tomography to autop
192 assist in the diagnosis of at least 6.3% of autopsy-negative child SUD cases and reduce risk of futu
195 are responsible for a significant portion of autopsy-negative sudden unexpected death (SUD) cases, bu
196 The most common findings at autopsy were autopsy-negative sudden unexplained death in 16 (25%), a
197 d putative pathogenic mutations in </=30% of autopsy-negative sudden unexplained death in the young (
200 life had other causes of death identified at autopsy, no MR, and scanty intracerebral sequestration.
202 (Cplx) 1/2, and synaptotagmin in brains from autopsies of individuals with and without schizophrenia.
205 5, 2012, and May 22, 2013, we did whole-body autopsies on inpatients aged at least 16 years who died
212 multiple-matched metastatic tumours from ten autopsied patients to infer the evolutionary history of
213 and compared in spinal cord samples from 18 autopsied patients with ALS and 12 neurologically health
214 obtained during a prospective series of 103 autopsies performed between 1996 and 2010 in Blantyre, M
216 renal, and nonrenal tissues were obtained at autopsy performed in the Department of Pathology at the
217 and ultrastructural findings from the first autopsy performed on a fatal case of MERS-CoV in the wor
220 in a large sample of multiple sclerosis (MS) autopsies provides insights into the dynamic nature of M
221 Metrics Research Consortium shortened verbal autopsy questionnaire was used for each interview, and c
222 facturer's technical analysis, and the final autopsy report were all cataloged in the Johns Hopkins P
223 ld reports with available death certificates/autopsy reports included sudden infant death syndrome (n
227 e sources, including ambulance call reports, autopsy reports, in-hospital data, and records of direct
232 ive impairment during life (N = 15) from the autopsy sample of the Baltimore Longitudinal Study of Ag
236 d exocrine sympathetic nerve fiber area from autopsy samples of patients with type 1 or 2 diabetes an
241 he prevalence of incidental DTC in published autopsy series and determine whether this prevalence has
247 on of mTORC was also found in the vessels of autopsy specimens from patients with catastrophic antiph
249 d during life, histopathological analysis of autopsy specimens is critical to understanding the cellu
252 cidence is not mirrored by prevalence within autopsy studies and, therefore, is unlikely to reflect a
256 de, growing recognition of this pathology in autopsy studies of nonboxers who were exposed to repetit
257 Although most AAOCA subtypes are benign, autopsy studies report an associated risk of sudden deat
259 s and the prevalence of adenomas reported in autopsy studies to determine the appropriate age at whic
260 me sequencing in cases and controls from the autopsy study "Pathobiological Determinants of Atheroscl
264 tudy using (89)Zr-labeled bevacizumab and an autopsy study, a 1-on-1 analysis of multiregional in viv
265 nter autopsy database and found that ~14% of autopsied subjects clinically diagnosed with mild-to-mod
266 ses of dementia, probable AD, and MCI in 636 autopsied subjects from the Religious Order Study and th
269 l of 1358 individuals had died and had brain autopsies that were approved by board-certified neuropat
270 ad no cause identified after a comprehensive autopsy that included toxicologic and histologic studies
275 stage-specific and type-specific markers in autopsy tissue from a representative cohort of 28 partic
280 nvestigated the HIV DNA levels in 229 varied autopsy tissues from 20 HIV-positive (HIV(+)) cART-treat
281 rticle by Ng et al, emphasizing the need for autopsies to better model infectious diseases such as Mi
282 ate cancer (CRPC) patients obtained at rapid autopsy to evaluate diverse genomic, transcriptomic, and
284 5 and 12 metastatic sites were collected at autopsy together with available primary tumours and long
285 unexpected death (SUD) cases, but molecular autopsy used to identify potentially causal variants is
286 pathologic measures of amyloid deposition at autopsy using all 4 reference regions, with the whole-ce
287 ng trauma and haemorrhage (p=0.008), whereas autopsy was better at identifying pulmonary thromboembol
300 ography (PMCTA), in adults to avoid invasive autopsy would have cultural, religious, and potential ec
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