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1 a critical mechanism contributing to corneal avascularity.
2 antiangiogenic factor that maintains corneal avascularity.
3 ular regrowth, consequently reducing retinal avascularity.
4 as graded on a scale of 1 to 5 (1 indicating avascularity; 2, normal; 3, mild; 4, moderate; and 5, se
5 ietin (Tie2) and therefore contribute to the avascularity and transparency of the cornea in the devel
6 hought to be involved in maintaining corneal avascularity (angiogenic privilege).
7                                      Retinal avascularity, distance from the optic nerve head to the
8                                      Corneal avascularity during development is redundantly regulated
9 pellent, on angioblast migration and corneal avascularity during development.
10 he role of matrilysin in maintaining corneal avascularity during wound healing.
11 lay an important role in maintaining corneal avascularity during wound healing.
12 tant cue during the establishment of corneal avascularity in both chick and mouse embryos.
13 inant sflt-1 administration restores corneal avascularity in corn1 and Pax6+/- mice.
14 x-mediated gene disruption abolishes corneal avascularity in mice.
15 e of the few tissues in the human body where avascularity is actively maintained.
16                                      Corneal avascularity is critical for achieving transparency nece
17                                      Corneal avascularity is important for optical clarity and normal
18 uced hypertension called preeclampsia and to avascularity of normal cornea.
19       In this article, we exploit the unique avascularity of the cornea to examine a role for local o
20 neal allografts was based on the conspicuous avascularity of the cornea, which was believed to seques
21                      Retinal detachment with avascularity of the peripheral retina, typically associa
22       Here, an inhibitor responsible for the avascularity of these ocular compartments is identified
23 neo-vascularization of malignancies from the avascularity of thrombi and the sparse vascularity of st
24 treated rabbits did not show the thinning or avascularity that was seen in the MMC treatment group.
25                                      Corneal avascularity-the absence of blood vessels in the cornea-
26 ne diphosphatase and analyzed for peripheral avascularity, vascular density, and NV incidence and sev
27  This was associated with subsequent reduced avascularity, vascular leakage, and pathologic NV during
28                      A minimal difference in avascularity was observed at postnatal day 12 in the ret

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