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1 ance of opioid withdrawal (conditioned place aversion).
2 r 2; VGluT2), which play roles in reward and aversion.
3 tivation caused neither place preference nor aversion.
4 as icv TTR did not cause a conditioned taste aversion.
5 behavior when it was guided by innate taste aversion.
6 tificial activation is sufficient to mediate aversion.
7 al for imprinted, but not for adult-learned, aversion.
8 ed rewards, is associated with loss and risk aversion.
9 d by their efferent targets, code reward and aversion.
10 essary and sufficient to mediate behavioural aversion.
11 reward, whereas stimulating D2 MSNs produces aversion.
12 ral synchrony and spatial representations of aversion.
13 uppresses activity of a glomerulus wired for aversion.
14 ular control of opioid systems in reward and aversion.
15 nd sufficient for stress-induced anxiety and aversion.
16 dum hedonic cold spot led to real-time place aversion.
17 f a saliency spectrum ranging from reward to aversion.
18 nocifensive responses and conditional place aversion.
19 area (VTA) neurons play roles in reward and aversion.
20 sion and probability weighting to model risk aversion.
21 olerance, or conditioned-place preference or aversion.
22 g effects, which is contingent on their loss aversion.
23 the balance between drug-induced reward and aversion.
24 lutionary explanation for the origin of risk aversion.
25 er volume in this region exhibited less risk aversion.
26 eptors in mesohabenular stimulation-elicited aversion.
27 sponse speed, or promote a conditioned place aversion.
28 al area (VTA) plays roles in both reward and aversion.
29 at did not depend on sweetness perception or aversion.
30 and stress-related behaviors, such as social aversion.
31 betting, and impulsivity, measured as delay aversion.
32 h loss-sensitivity, and thus diminishes loss aversion.
33 whereas TRPM8 neurons are required for cold aversion.
34 nsitivity to losses than gains known as loss aversion.
35 ved in motor control, resulted in defects in aversion.
36 gustatory system are responsible for glucose aversion.
37 glutamate/Homer2 expression in MA preference/aversion.
38 (gain only)-were exclusively driven by risk aversion.
39 eption while activation of LC(:PFC) produced aversion.
40 five sessions that tested conditioned flavor aversion.
41 relin signaling can both inhibit and enhance aversion.
42 risk aversion but equivalent levels of loss aversion.
43 health, and individual prosociality and risk aversion.
44 reward as a measure of an individual's risk aversion.
45 secretion, body weight, or conditioned taste aversion.
46 d not induce conditioned place preference or aversion.
47 osite limb, as assessed by conditioned place aversion.
48 ecision-making tasks assessing risk and loss aversion.
49 the naloxone-precipitated MWD induced place aversion.
50 mesoaccumbens glutamatergic fibers promotes aversion.
51 ion and maintenance of taste preferences and aversions.
52 there thus be an evolutionary origin to risk aversion?
55 ateral habenula (LHb) is involved in reward, aversion, addiction and depression through descending in
57 estation of hyper-cooperation and inequality aversion, allowing exploration of high-quality, hard-to-
58 ndrome, a disorder characterized by eye-gaze aversion, among other social and cognitive deficits.
59 s, produced dose-dependent conditioned place aversion and a reduction in the above optical ICSS in Vg
61 ation of neural circuitry that mediates odor aversion and attraction will provide key insights into h
62 hila melanogaster, displays robust olfactory aversion and attraction, but how these behaviors are exe
67 ral habenula (LHb) is involved in reward and aversion and is reciprocally connected with dopamine (DA
68 d allergy, as they showed a reversal of food aversion and lower IgE production after 5 weeks of induc
69 and circuit logic of predator-driven innate aversion and may serve as a valuable model for studying
71 t-inducible stimulation of acute pain, place aversion and optogenetically mediated reductions in with
73 mists also make use of concepts such as loss aversion and probability weighting to model risk aversio
78 in regulating expression of ethanol-induced aversion and suggest a mechanism for its role in modulat
79 ic mice also demonstrated a reversal of food aversion and sustained visceral fat after 5 weeks of all
80 s, giving rise to phenomena such as betrayal aversion and taboo-tradeoffs, and specific patterns of f
81 were able to reliably estimate risk and loss aversion and their respective contribution to gambling d
82 ha3, beta4, and alpha5 receptors in nicotine aversion and withdrawal have been established, the cellu
83 prevented naloxone-induced conditioned place aversions and decreases in sensitivity to brain stimulat
84 gests behavioral factors (eg, increased risk aversion) and/or allocation inefficiencies may have play
85 variational, maxmin, constant absolute risk aversion, and constant relative risk aversion utilities.
87 capone exerted selective effects on inequity aversion, and not on other computational components such
88 b) nuclei play important roles in processing aversion, and recent work has focused on the critical in
89 isrupted anxiety and mPFC representations of aversion, and reduced theta synchrony in a pathway-, fre
91 from a single receptor can block innate odor aversion, and that instinctive behavioral responses to o
93 references changed substantially toward risk aversion as reward accumulated within a context, and blo
96 icted by interindividual differences in risk-aversion attitudes expressed during the gambling task.
97 s to DA neurons that might impact the reward/aversion balance of alcohol attributes, which may contri
99 d gene expression and eliminated the feeding-aversion behavior resulting from contact with the analog
103 ing uncertainty, eating: between craving and aversion, being different and professional help as a fur
104 dition, participants exhibited a strong risk aversion bias; strikingly, this bias reversed in the rea
106 l preference, are dispensable for egg-laying aversion but essential for movement aversion of UV.
108 ot produce a conditioned place preference or aversion, but elicited CB1 receptor-mediated antinocicep
111 2-mesohabenular neurons that plays a role in aversion by activating LHb glutamatergic receptors.
113 ehavioral economics, including inertia, loss aversion, choice overload, and relative social ranking.
114 ve losses, whereas individuals with low loss aversion choked for large prospective losses but not for
115 sregulated activity of the mesocorticolimbic aversion circuit may be etiologically related to errors
116 ne predominantly shapes the so-called reward/aversion circuitry, with major influence on negative aff
119 tion of sexual behavior in a conditioned sex aversion (CSA) paradigm where mating is paired with illn
123 ly regarded as integral to conditioned taste aversion (CTA) retention, a link that has been primarily
129 quisition and retention of conditioned taste aversions (CTAs) in rodents, but large lesions in this a
130 hould switch between risk proneness and risk aversion depending on state and circumstances, especiall
132 by the trade name Antabuse), an old alcohol-aversion drug that has been shown to be effective agains
135 imple binary-choice model, we show that risk aversion emerges by natural selection if reproductive ri
138 contrast to their role in inflammatory pain aversion, EP3 receptors on serotonergic cells were dispe
140 ccumbens glutamatergic fibers lack GABA, the aversion evoked by their photoactivation depended on glu
143 havior showed that citalopram increased harm aversion for both self and others, while levodopa reduce
144 mental oxygen also induces conditioned place aversion, for which Gucy1b2 and Trpc2 are required.
145 ploration of the action space for successful aversion formation and show how it predicts foraging beh
147 trust between dissimilar users and that risk aversion has an inverse relationship with trust given hi
149 urophysiological evidence suggests that loss aversion has its origins in relatively ancient neural ci
151 R-mediated dysphoria, assessed in rodents as aversion, has recently been attributed to the activation
152 ation analysis revealed that behavioral loss aversion hindered performance via the influence of ventr
157 on, constipation, and displayed no reward or aversion in CPP/CPA assays, suggesting that analogs migh
162 ative to either drug by itself, reduced loss aversion in the absence of an effect on risk attitude or
163 trategy, was positively correlated with loss aversion in the depressed patients, also implicating imp
166 endent; as the animals became "poorer," risk aversion increased, a finding incompatible with some mod
167 s cognate ligand from blocking predator odor aversion, indicating that the blocking requires sensory
169 able for acute nociceptive behaviors and for aversion induced by thermal pain or a kappa opioid recep
170 We tested the role of CRF in modulating aversion-induced changes in dopamine concentration and c
171 glutamatergic input to nAcc shown to mediate aversion instead of reward, and the first pathway shown
172 e that outcome anticipation and ensuing loss aversion involve multiple neural systems, showing functi
176 ur framework implies that the degree of risk aversion is determined by the stochastic nature of repro
181 first to reject it (i.e., conditioned taste aversion learning) and then to enjoy it again (i.e., ext
182 es ingestive behavior without inducing taste aversion may open the possibility of a therapeutic appli
184 teral habenula (LHb), which is implicated in aversion-mediated learning, show accelerated escalation
187 tenuated the extinction of established taste aversion memory without altering the initial associative
190 ther further modifications could reduce risk aversion, more efficiently allocate resources, and incre
192 t atrophy in an interoceptive, pain-related (aversion) network exhibited the most severe lack of soci
193 ocedure but did not affect conditioned place aversion, nor did JQ1 alone induce conditioned aversion
194 eep arousal (locus coeruleus) and preference/aversion (nucleus accumbens) demonstrate the unique capa
196 conditioned on the levels of risk acceptance/aversion of decision makers, and show how to extend the
198 retina suggest that egg-laying and movement aversion of UV are both mediated by the inner (R7) and n
207 athway, or a kappa-opiate receptor-dependent aversion pathway, directly within the ventral tegmental
210 fairness decisions: disadvantageous inequity aversion (peer receives more than self) and advantageous
213 ases of choice-outcome anticipation and loss aversion provided inconsistent results, showing either b
214 ynorphinerigic cells regulate preference and aversion provides insight into motivated behaviors that
217 s on the human brain, particularly on reward/aversion regions implicated in addiction, such as the nu
219 ns occurred in connectional patterns of pain/aversion-related nodes, including the mu receptor-enrich
220 se results suggest that kappa opioid-induced aversion requires regulation of VTA dopaminergic neuron
221 indicate that HA-NMDAR modulators can reduce aversion-resistant alcohol drinking, and support testing
222 ion of Syt2 expression in the mPFC increased aversion-resistant alcohol drinking, supporting a mechan
225 impact of D-serine and D-cycloserine on this aversion-resistant alcohol intake (that persists despite
226 eased alcohol self-administration, increased aversion-resistant alcohol intake and enhanced stress-in
232 (DA) neurons have been implicated in reward, aversion, salience, cognition, and several neuropsychiat
233 ve behavior in wild-type CD1 mice similar to aversion seen previously after central (intracerebrovent
235 slaf = ball, slaflaf = balls), the doubling aversion shifts into a reliable (morphological) preferen
236 of choice such as motor impulsivity or loss aversion, suggesting a direct and specific influence of
237 enorhabditis elegans leads to a long-lasting aversion that requires distinct sets of neurons for the
239 ons and that this signaling sequence induced aversion through GABA-mediated inhibition of dopaminergi
240 e found that systemic inflammation triggered aversion through MyD88-dependent activation of the brain
241 rea (VTA), a region implicated in reward and aversion, thus providing a candidate neural substrate fo
242 central sensitization and conditioned place aversion, thus providing a novel paradigm to investigate
243 oth a TAAR ligand and a common odorant block aversion to a predator odor, indicating that this abilit
244 ared with the other groups exhibited greater aversion to both risk and loss during gambles involving
247 raft survival, accepting risk, and desperate aversion to dialysis), guilt and responsibility (jeopard
251 rodent pain assay based on conditioned place aversion to formalin injection, an inflammatory noxious
252 f the dorsal raphe nucleus failed to form an aversion to formalin-induced pain, as did mice lacking t
253 have rapidly evolved an adaptive behavioral aversion to glucose (a phagostimulant component of baits
255 ong many mammals, giving birth leads from an aversion to infant stimuli to irresistible attraction.
256 sly observed that most people show a greater aversion to inflicting pain on others than themselves.
258 p cooperating even if conditions change, and aversion to information gathering helps to interact pref
259 ed conditioned flavor (i.e., taste and odor) aversion to intravenously self-administered (IVSA) nicot
261 as alpha6 KO showed both a conditioned place aversion to lithium chloride as well as CPP to palatable
263 nes focus on risk, often assuming negligible aversion to medication, yet most patients discontinue pr
266 ychopathic traits correlated negatively with aversion to pain for both self and others, in line with
267 ceptor activation elicited conditioned place aversion to pain via inhibition of serotonergic neurons.
268 cidal behavior is associated with heightened aversion to risk and loss, which might produce negative
269 tion that despite the effects of warnings on aversion to smoking, intention to quit smoking is an inv
271 stimulation, mice showed a conditioned place aversion to the chamber that was previously associated w
272 just in particular research areas but in an aversion to the explanatory depth available from politic
274 with delta opioid-induced seizure activity, aversion to the kappa drug U50, 488H, or alpha2-mediated
276 g with pathogenic bacteria induces a learned aversion to the smell of the pathogen, a behavioral plas
277 to pathogenic bacteria results in persistent aversion to those bacterial odors, whereas adult exposur
279 wever, this bias could be driven by enhanced aversion to uncertainty about the decision outcome (e.g.
280 ever, was mediated by winter severity, where aversion to well pads decreased as winter severity incre
281 the paradox of how flies maintain reflexive aversion to your approaching swatter, whilst tolerating
282 experiences that shape odor preferences and aversions to explore developmental plasticity in circuit
286 the degree of participants' behavioral loss aversion was correlated with their susceptibility to cho
289 y, we demonstrated that inflammation-induced aversion was not an indirect consequence of fever or ano
290 induce memory given that a conditioned taste aversion was obtained for a novel taste, presented just
293 g in the LHb contributing to ethanol-induced aversion, we recorded neural firing in the LHb of freely
294 nd loss frames: individuals with higher loss aversion were susceptible to choking for large prospecti
295 mixed)-could be driven by both loss and risk aversion, whereas choices on the other type-featuring on
296 ation of the RMTg produced conditioned place aversion, whereas cocaine-induced avoidance behaviors in
297 red with the expression of conditioned place aversions while leaving intact cocaine-induced place pre
300 and associated hormonal changes affect loss aversion, yet the underlying neuroendocrine mechanisms a
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